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1 aqueduct to the canalostomy due to influx of cerebral spinal fluid.
2 9 areas of normal appearing white matter and cerebral spinal fluid.
3 al canal where they block circulation of the cerebral spinal fluid.
4 infected hamsters using 2-microl aliquots of cerebral spinal fluid.
5 mission tomography and glucose monitoring in cerebral spinal fluid.
6 mixture of peptides dissolved in artificial cerebral spinal fluid.
7 ular space where they are transported to the cerebral spinal fluid.
9 jority of interneurons (21/25) in artificial cerebral spinal fluid (aCSF) exhibited spontaneous tonic
11 ted context, BLA was infused with artificial cerebral spinal fluid (ACSF) or ACSF containing an N-met
14 is followed by microdialysis with artificial cerebral spinal fluid allowed isolation of 14C-glutamine
15 nclusion were absence of correlation between cerebral spinal fluid amyloid-beta1-42 and Boston naming
16 nal fluid t-tau, brain amyloid-beta load via cerebral spinal fluid amyloid-beta1-42 and vascular dise
17 rebral spinal fluid t-tau) and amyloid load (cerebral spinal fluid amyloid-beta1-42) all independentl
22 ulin forms in human neocortex, white matter, cerebral spinal fluid, and serum by immunostaining and m
25 S induces nuclear ASPP2 in vivo at the blood-cerebral spinal fluid barrier (the brain's barrier to in
27 isolated primary viruses from autopsy brain, cerebral spinal fluid, blood, spleen, and lymph node sam
28 observed to correlate with viral RNA in the cerebral spinal fluid but not with plasma viral load, co
34 ng were reviewed to identify the presence of cerebral spinal fluid (CSF) clefts around the tumors.
41 v compartmentalization between the blood and cerebral spinal fluid (CSF) was apparent with all neurol
42 orectal and lymph node tissues, bone marrow, cerebral spinal fluid (CSF), plasma, and very large numb
43 nvolved in the production and circulation of cerebral spinal fluid (CSF), suggesting that loss of mFu
46 age fluid (n = 152), pleural fluid (n = 76), cerebral spinal fluid (CSF; n = 152), pericardial fluid
49 tify a proteolytic fragment of cystatin C in cerebral spinal fluids from patients diagnosed with mult
51 -67 exhibited robust in vivo activity in the cerebral spinal fluid glycine biomarker model in both ro
53 Notice of retraction: the article "Role of Cerebral Spinal Fluid in Space Flight Induced Ocular Cha
54 4 (ST4) was the predominant sequence type of cerebral spinal fluid isolates from cases of meningitis.
58 ell tolerated in humans and elevates cGMP in cerebral spinal fluid of healthy volunteers, confirming
62 ent and lumbar puncture for determination of cerebral spinal fluid phosphorylated tau (P-tau) and bet
65 R analysis was used to genotype parasites in cerebral spinal fluid samples from 8 of 10 human immunod
68 y (structural magnetic resonance imaging and cerebral spinal fluid t-tau) and amyloid load (cerebral
69 gnetic resonance images, neuronal injury via cerebral spinal fluid t-tau, brain amyloid-beta load via
70 plate brain as well as GM, white matter, and cerebral spinal fluid tissue probability maps to facilit
71 n increase in CXCL1 and CXCL10 levels in the cerebral spinal fluid, TNF-alpha expression in the epend
75 e it exists in steady-state equilibrium with cerebral spinal fluid where it has the potential to serv
76 regulin activity is also detectable in human cerebral spinal fluid where its expression appears to be
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