ライフサイエンスコーパス: cerebral vasospasm

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1 opment of an effective therapy to ameliorate cerebral vasospasm.

2 approach for the prevention and treatment of cerebral vasospasm.

3 currence and severity of sequelae, including cerebral vasospasm.

4 much has been written about the treatment of cerebral vasospasm.

5 plicated by pathological vasoconstriction or cerebral vasospasm.

6 ic oxide synthase, have been used to prevent cerebral vasospasm after aneurysmal subarachnoid hemorrh

7 hether recombinant OPN (r-OPN) could prevent cerebral vasospasm after subarachnoid hemorrhage (SAH) i

8 iovascular variables, detecting and treating cerebral vasospasm and managing systemic complications.

9 acid peptide that has been closely linked to cerebral vasospasm and more recently to oxidative stress

10 , emphasizing the detection and treatment of cerebral vasospasm and the management of systemic compli

11 orts regarding hyperthermia, cerebral edema, cerebral vasospasm, and lethal interactions with commonl

12 95 fasudil was approved for the treatment of cerebral vasospasm, and more recently, ripasudil was app

13 n (oxyhb) has been implicated in SAH-induced cerebral vasospasm as it causes cerebral artery constric

14 ng sequential brain imaging and investigated cerebral vasospasm by angiography or time-of-flight magn

15 ns in normal cerebral blood flow, such as in cerebral vasospasm, can induce neurological deficits.

16 Besides classical contributors like proximal cerebral vasospasm, CSD clusters may reduce O(2) supply

17 Cerebral vasospasm (CV) and the resulting delayed cerebr

18 rotective in animal models and may attenuate cerebral vasospasm (cVSP) in human aneurysmal subarachno

19 Cerebral vasospasm following aneurysmal subarachnoid hem

20 Cerebral vasospasm has traditionally been regarded as an

21 oppler has an established role in diagnosing cerebral vasospasm in patients with aneurysmal subarachn

22 mortality, and the occurrence of symptomatic cerebral vasospasm in patients with aneurysmal subarachn

23 ch that potentiates vascular remodeling, and cerebral vasospasm, in bTBI patients.

24 Cerebral vasospasm is a frequent complication after suba

25 Cerebral vasospasm is a recognised but poorly understood

26 endothelin-1 (ET-1) is induced resulting in cerebral vasospasm, ischemia, reperfusion and the activa

27 Cerebral vasospasm occurs more frequently, and with earl

28 ation of cerebral blood flow, SAH grade, and cerebral vasospasm of SAH mice.

29 ructures in the cerebrospinal fluid (CSF) of cerebral vasospasm patients has been made.

30 ia ink-gelatin casting for the assessment of cerebral vasospasm, permits outstanding immunohistochemi

31 l, a ROCK inhibitor used clinically to treat cerebral vasospasm, restored platelet counts in adult mi

32 we developed a novel technique for assessing cerebral vasospasm using cerebrovascular perfusion with

33 In the overall population, cerebral vasospasm was significantly less common in the

34 thromboxane A2 implicated in the etiology of cerebral vasospasm, we observed significant increases in

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