ライフサイエンスコーパス: cerebral ventricle

1 orpus callosum, and enlargement of the third cerebral ventricle.

2 e, cannulas were placed in the right lateral cerebral ventricle.

3 ntagonist, BMS-200261 into the right lateral cerebral ventricle.

4 s to the infusion of oleic acid in the third cerebral ventricle.

5 al inhibitors of its activity into the third cerebral ventricle.

6 nd administered this directly into the third cerebral ventricle.

7 e brain through an infusion into the lateral cerebral ventricle.

8 a 5-HT1A receptor agonist, into the lateral cerebral ventricle.

9 subthreshold for effect if injected into the cerebral ventricles.

10 spatial order from germinal cells lining the cerebral ventricles.

11 al cells were observed in the linings of all cerebral ventricles.

12 ology resulting in abnormal expansion of the cerebral ventricles.

13 e olfactory bulb, and in cells bordering the cerebral ventricles.

14 ary neuronal damage in regions bordering the cerebral ventricles.

15 ood intake in the rat when injected into the cerebral ventricles.

16 e developed criteria for leakage of PRV into cerebral ventricles.

17 duces obesity in rats when injected into the cerebral ventricles.

18 istration of ATA by injection into the right cerebral ventricle 1 h before (but not 6 h after) bilate

19 0.3, 3.0, or 10 nmol) into the left lateral cerebral ventricle 5 min before a smaller non-aggressive

20 l fluid (CSF) continuously flows through the cerebral ventricles, a process essential for brain homeo

21 Finally, third-cerebral-ventricle administration of inhibitors of ATP-s

22 afish larva as an in vivo model for studying cerebral ventricle and blood-brain barrier function.

23 ted in the germinal zones near the embryonic cerebral ventricle and migrate radially to the overlying

24 id plexus epithelial cells extends into each cerebral ventricle and secretes signaling factors into t

25 velopment leads to marked enlargement of the cerebral ventricles and destruction of brain tissue, due

26 These epithelial cells line the cerebral ventricles and generate intermediate progenitor

27 ical disorder that leads to expansion of the cerebral ventricles and is associated with a high rate o

28 a critical role in the development of normal cerebral ventricles and neuroependymal integrity as well

29 nd venous catheters, a catheter in the third cerebral ventricle, and ultrasonic flow probes on the le

30 al longitudinal anastomotic vessel, enlarged cerebral ventricles, and pericardial edema, in addition

31 giotensin II, we created anteroventral third cerebral ventricle (AV3V) lesions in mice.

32 nding the anteroventral portion of the third cerebral ventricle (AV3V) of the preoptic recess is crit

33 When administered acutely into the 3rd cerebral ventricle, both DET and NPH insulin reduced foo

34 ) mice, and leptin infusion into the lateral cerebral ventricles decreases feeding with short latency

35 gic hydrocephalus (PHH), an expansion of the cerebral ventricles due to CSF accumulation following in

36 ore, the infusion of oleic acid in the third cerebral ventricle failed to decrease the expression of

37 t continuously released NTX into the lateral cerebral ventricle for 7 days.

38 in the subependymal (SZ) zone of the lateral cerebral ventricle generate neuroblasts that migrate in

39 Proliferative cells lining the cerebral ventricles generate all of the phenotypically d

40 (15 nmol) was also injected into the lateral cerebral ventricle (i.c.v.).

41 in or an active cysteine mutant in the third cerebral ventricle (icv) or in the mediobasal hypothalam

42 , we directly infused the ASO into a lateral cerebral ventricle in adult mice expressing a human SMN2

43 lacZ), or saline was injected into the right cerebral ventricle in mice.

44 rain arise from progenitors located near the cerebral ventricles in a temporally segregated manner su

45 The walls of the cerebral ventricles in the developing embryo harbor the

46 ormal mice resistin delivered in the lateral cerebral ventricle increased endogenous glucose producti

47 Acute insulin infusion in the third cerebral ventricle inhibits endogenous glucose productio

48 on and is more potent when injected into the cerebral ventricle (intracerebroventricular [ICV]).

49 thesis, injection of alpha2-antiplasmin into cerebral ventricles markedly ameliorated HI-induced dama

50 w dose of leptin (3.5 microg) into the third cerebral ventricle (n = 15) during a 40-h period of fast

51 e (2 microl) was administered into the third cerebral ventricle of age- and weight-matched male and f

52 and administration of insulin into the third cerebral ventricle of fasted rats increased expression o

53 ion of 60 fmol of gamma-MSH into the lateral cerebral ventricle of hypertensive mice also lowered MAP

54 intracerebroventricular infusion to the left cerebral ventricle of male C57Black6 mice.

55 to c-fos, rncfosr115, infused into the left cerebral ventricle of male Long-Evans rats and the effec

56 ed continuously for 2 weeks into the lateral cerebral ventricle of male Wistar rats rendered obese by

57 hat chronic resistin infusion in the lateral cerebral ventricle of normal rats markedly affects both

58 tracerebroventricularly (ICV) into the third cerebral ventricle of ovariectomized, estradiol benzoate

59 d that administration of hUCB cells into the cerebral ventricle of presymptomatic Naglu mice had a be

60 -6 to brain organotypic cultures or into the cerebral ventricles of adult rats did not activate the J

61 ving rise to both neurons and glia, line the cerebral ventricles of all adult animals, including huma

62 cycle exit in mammalian neocortex--into the cerebral ventricles of chicks at embryonic day (ED) 4.

63 ique system for maintaining catheters in the cerebral ventricles of conscious mice to test whether th

64 Fetal human brain cells implanted into the cerebral ventricles of embryonic rats incorporate indivi

65 al populations, and infusion of CRF into the cerebral ventricles of infant rats produces severe age-d

66 ar injection of Abeta1-42 oligomers into the cerebral ventricles of mice, a validated Alzheimer's dis

67 A single microinjection of FGF2 into cerebral ventricles of rat embryos at E15.5 increased th

68 ctor, called satietin, was injected into the cerebral ventricles of rats that were either fed ad libi

69 lantation of glial cell progenitors into the cerebral ventricles of the embryonic myelin-deficient ra

70 and had a cannula implanted into the lateral cerebral ventricle on day 24.

71 ive inhibitor of CPT1A activity in the third cerebral ventricle or in the mediobasal hypothalamus (MB

72 mplanted with cannulas to either the lateral cerebral ventricle or the ventromedial hypothalamic nucl

73 administration of PNU120596 either into the cerebral ventricles or directly into the mediodorsal tha

74 small-molecule insulin mimetic in the third cerebral ventricle suppressed glucose production indepen

75 onist dizocilpine (100 nmol infused into the cerebral ventricles) suppressed drinking following eithe

76 to the contralateral hemisphere, or into the cerebral ventricles), the donor cells migrate through no

77 ve EP receptor agonists into the rat lateral cerebral ventricle under unrestrained condition.

78 subpopulation of ependymal cells lining the cerebral ventricle wall, and other nonneuronal cells.

79 plets were asymmetrically distributed in the cerebral ventricle walls, mainly located in the lateral

80 a-MSH), or antagonist, SHU9119, in the third cerebral ventricle while food intake was maintained cons