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1 imer disease, hydrocephalus, and pseudotumor cerebri.
2 incontinence, venous stasis, and pseudotumor cerebri.
3 athic intracranial hypertension (pseudotumor cerebri), (3) nonarteritic anterior ischemic optic neuro
6 orders, such as optic neuritis, pseudotumour cerebri and nonarteritic anterior ischaemic optic neurop
9 is one cause of the syndrome of pseudotumour cerebri but the cause and the link with a rise in body w
10 le leukoencephalopathy syndrome, pseudotumor cerebri, disturbances in eye movements, accommodation pr
11 ecially diffuse astrocytomas and gliomatosis cerebri, for diagnostic, therapeutic, and prognostic pur
12 glioma, glioblastoma multiforme, gliomatosis cerebri, gliosarcoma, ependymoma, and subependymoma, for
13 sclerosis, neuromyelitis optica, pseudotumor cerebri, migraine, optic nerve head drusen, compressive
15 tigated motor fiber organization in the crus cerebri of the cerebral peduncle (ccCP) in the rhesus mo
18 athies, the ocular motor system, pseudotumor cerebri, posterior reversible encephalopathy syndrome an
20 rd in diagnosis and treatment of pseudotumor cerebri (PTC), the syndrome of increased intracranial pr
21 idiopathic (n = 59) or secondary pseudotumor cerebri syndrome (n = 16), as rigorously defined by rece
22 served in the possible secondary pseudotumor cerebri syndrome group included Down syndrome, vitamin A
23 sures seen in definite secondary pseudotumor cerebri syndrome included tetracycline-class antibiotics
24 bri syndrome, definite secondary pseudotumor cerebri syndrome patients were on average older (15.0 vs
25 tertiary children's hospital for pseudotumor cerebri syndrome were classified as having either primar
26 , 79% of children with secondary pseudotumor cerebri syndrome were either overweight or obese (36% ov
28 sure is identified for pediatric pseudotumor cerebri syndrome, the possible contribution of overweigh
29 liferative diabetic retinopathy, pseudotumor cerebri, thyroid orbitopathy, and cystoid macular edema.
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