ライフサイエンスコーパス: cerebrocortical

1 nd-B positron emission tomography imaging of cerebrocortical abeta-amyloid deposition.

2 the effect of IGF analogues on cell death of cerebrocortical and cerebellar granule cell cultures.

3 arge number of neurons distributed over many cerebrocortical and limbic brain regions, but the multif

4 atment with 6% PN401 decreased mortality and cerebrocortical apoptosis caused by azide.

5 s (both Na(+)-dependent and -independent) in cerebrocortical astrocytes and hippocampal neurons.

6 Initial data in rat cerebrocortical astrocytes demonstrated that similar eff

7 posure (gestational days 4-21) decreased the cerebrocortical binding of paroxetine (PXT), a marker fo

8 We tested the hypothesis that cerebrocortical blood flow and O2 consumption would be p

9 suggest a proportional relationship between cerebrocortical blood flow and oxygen consumption in the

10 Such upregulation did not alter basal cerebrocortical blood flow or O(2) consumption.

11 A receptor upregulation does not alter basal cerebrocortical blood flow or O2 consumption but in the

12 otein (A beta P) forms senile plaques in the cerebrocortical blood vessels and brain parenchyma of pa

13 ately 100 mug of tryptic peptides from mouse cerebrocortical brain tissue.

14 he protein solubilized from postmortem human cerebrocortical brain tissue.

15 We previously showed in cerebrocortical cell cultures from rodents containing mi

16 In cerebrocortical cell cultures, HIV-1 gp120 stimulated a

17 In cerebrocortical cell cultures, neuroprotection by IFNbet

18 Using cerebrocortical cell cultures, we find that LCN2 is neur

19 1 cells were prevented in microglia-depleted cerebrocortical cells pretreated with a pharmacological

20 GSH via GCL activation was observed in mixed cerebrocortical cultures and N27 dopaminergic cells.

21 ial release of cytochrome c were observed in cerebrocortical cultures exposed to the HIV coat protein

22 whether p53 participates in HAD, we exposed cerebrocortical cultures from wild-type and p53 deficien

23 Here we show in rat cerebrocortical cultures that recapitulate the type and

24 Furthermore, primary rat fetal cerebrocortical cultures were transfected with APP and B

25 These results indicate that, in cerebrocortical cultures, excessive NMDA receptor activa

26 Here, using rat primary cerebrocortical cultures, we demonstrate that Abeta1-42

27 iuretic furosemide reduces alcohol-dependent cerebrocortical damage by 75-85% while preventing brain

28 triatal vesicular monoamine transporters and cerebrocortical deposition of abeta-amyloid in mild deme

29 cluding reelin, a gene that is essential for cerebrocortical development.

30 te gyrus, and olfactory bulbs accompanied by cerebrocortical edema and electrolyte (Na+, K+) accumula

31 o been implicated in the modulation of mood, cerebrocortical excitability, hypothalamic-pituitary sig

32 itates with receptor subunits NR1 and NR2 in cerebrocortical extracts.

33 Cerebrocortical injuries such as stroke are a major sour

34 cific and are expressed during the period of cerebrocortical lamination.

35 Cerebrocortical manifestations (including cognitive impa

36 esults in the formation of a focal region of cerebrocortical microdysgenesis resembling, in many ways

37 the cortex and thalamus of rats with induced cerebrocortical microgyria.

38 t the end of normothermic bypass diameter of cerebrocortical microvessels increased to 116+/-9% (alph

39 ty, regional distribution, associations with cerebrocortical morphology and effect sizes of cerebella

40 ndependent component of glutamate release in cerebrocortical nerve terminals after blocking Na(+) cha

41 dent pathway to enhance glutamate release at cerebrocortical nerve terminals.

42 l axons (TCAs) are essential elements of the cerebrocortical network.

43 In primary cerebrocortical neuron cultures under basal conditions,

44 Parallel to this response, PbTx-2 increased cerebrocortical neuron synaptic density.

45 xic (N-methyl-D-aspartate) insults to mature cerebrocortical neurons activate caspase-3, -7, in turn

46 in I at MAP kinase-dependent sites in intact cerebrocortical neurons and PC12 cells, respectively, wh

47 In cerebrocortical neurons containing NR3 family members, g

48 Nitric oxide (NO) synthase (NOS)-containing cerebrocortical neurons degenerate in patients with amyo

49 to attenuate hypoxic/hypoglycemic injury to cerebrocortical neurons in culture and excitotoxic injur

50 aspartate (NMDA) toxicity in cultured murine cerebrocortical neurons in vitro and mouse cerebral cort

51 wever, during 2 hr of anoxia, [Ca(2+)](i) in cerebrocortical neurons increased by 35%, and suppressio

52 ACEA 1021 to produce morphologic changes in cerebrocortical neurons of the rat was assessed since it

53 -dependent neuronal structural plasticity in cerebrocortical neurons through an increase in [Na(+)](i

54 rents gated by 100 microM NMDA from cultured cerebrocortical neurons were examined in the presence of

55 Transfection of cerebrocortical neurons with a dominant interfering form

56 tected in rat brain homogenates, in cultured cerebrocortical neurons, and in isolated presynaptic ter

57 neuron/muscle transcription factor found in cerebrocortical neurons, and study its regulation of the

58 In cultured cerebrocortical neurons, mild excitotoxic insults or sta

59 gating modifier, to manipulate [Na(+)](i) in cerebrocortical neurons.

60 veratridine-induced sodium influx in murine cerebrocortical neurons.

61 of NMDAR function were evaluated in immature cerebrocortical neurons.

62 on-derived clonal cell line) and rat primary cerebrocortical neurons.

63 nt Zn2+ release from intracellular stores in cerebrocortical neurons.

64 ate (NMDA)-induced apoptosis in cultured rat cerebrocortical neurons.

65 oses they cause irreversible degeneration of cerebrocortical neurons.

66 in amounts sufficient to injure cultured rat cerebrocortical neurons.

67 revented NMDA receptor-mediated apoptosis of cerebrocortical neurons.

68 beta peptides from N2a cells and rat primary cerebrocortical neurons.

69 01 treatment, nor show neurotoxic changes in cerebrocortical neurons.

70 ncreased dendritic spines in early postnatal cerebrocortical neurons.

71 ns for beta- and gamma-actin within cultured cerebrocortical neurons.

72 y cultures of rat, mouse and human embryonic cerebrocortical neurons.

73 d not affect Ca(2+) dynamics in 2 d in vitro cerebrocortical neurons; however, this treatment robustl

74 t a dose (0.05 mg/kg s.c.) that prevents the cerebrocortical neurotoxic effects of MK-801, decreased

75 ion-evoked L-glutamate efflux from adult rat cerebrocortical prisms.

76 in vitro enhanced glutamate efflux from rat cerebrocortical prisms.

77 unoreactivities were assessed in 4 different cerebrocortical regions derived from 16 elderly controls

78 between level of SLI and CRF-IR in different cerebrocortical regions to the earliest signs of cogniti

79 ever, anatomic hemispheric asymmetry of this cerebrocortical site is clearly not unique to humans, as

80 Superfused P7 rat cerebrocortical slices (350 mum) were treated with 2 mM

81 Respiring neonatal rat cerebrocortical slices were exposed for 30 min to toxic

82 ach experiment used eighty 350 microns thick cerebrocortical slices, obtained from twenty 7-day old r

83 r glutamate accumulation in monkey and mouse cerebrocortical slices.

84 In Percoll-purified rat cerebrocortical synaptosomes, depolarization with 60 mM

85 With cerebrocortical synaptosomes, inhibition of glutamate up

86 -induced release of [3H]D-aspartate from rat cerebrocortical synaptosomes, with an IC50 value of 0.39

87 ed by reductions in cerebral myelination and cerebrocortical volumes and is associated with secondary