ライフサイエンスコーパス: cerebrospinal fluid

1 giotensin II into angiotensin-(1-7) in human cerebrospinal fluid.

2 lepsy, and elevated lactate in the blood and cerebrospinal fluid.

3 r discovery and validates its feasibility on cerebrospinal fluid.

4 persistence were observed in bone marrow and cerebrospinal fluid.

5 of recent ZIKV infection in the serum and/or cerebrospinal fluid.

6 in the inflammatory milieu of the serum and cerebrospinal fluid.

7 the mammalian brain as well as in the human cerebrospinal fluid.

8 n MRI; positive spinal cord MRI; or positive cerebrospinal fluid.

9 is present at notably high concentrations in cerebrospinal fluid.

10 16 bacterial, viral, and fungal pathogens in cerebrospinal fluid.

11 lated tau, and neurogranin concentrations in cerebrospinal fluid.

12 erneuron activity in human versus artificial cerebrospinal fluid.

13 ts applied intranasally appears to reach the cerebrospinal fluid.

14 cell repertoires in the primary melanoma and cerebrospinal fluid.

15 d IL-1beta levels were elevated in serum and cerebrospinal fluid.

16 levels of cytokines and immune cells in the cerebrospinal fluid.

17 termination and the transport of gametes and cerebrospinal fluid.

18 tects multiple pathogens simultaneously from cerebrospinal fluid.

19 throat swabs (33 of 39 [85%]) rather than in cerebrospinal fluid (10 of 41 [24%]).

20 leacetic acid (5-HIAA) concentrations in the cerebrospinal fluid 12 months after gene therapy.

21 bic wounds (30%), blood (5.5%), urine (22%), cerebrospinal fluid (24%), ophthalmologic cultures (8%),

22 In cerebrospinal fluid 27-hydroxycholesterol was reduced by

23 Pittsburgh compound B and centrally analysed cerebrospinal fluid, a minority of subjects showed disco

24 osinor measures of each participant's serial cerebrospinal fluid Abeta concentrations and Abeta turno

25 the diurnal fluctuations in interstitial and cerebrospinal fluid Abeta levels observed in both the ro

26 ds, from salty biofluids such as aritificial cerebrospinal fluid (aCSF) and serum.

27 nal fluid (hCSF), using a matched artificial cerebrospinal fluid (aCSF) as control.

28 types of microorganisms spiked in artificial cerebrospinal fluid (ACSF) were directly detected simult

29 imal were pre-incubated in normal artificial cerebrospinal fluid (aCSF) while half were pre-incubated

30 ocial cognition and behavior, accumulates in cerebrospinal fluid after intranasal administration in m

31 lfonylurea receptor-1 is measurable in human cerebrospinal fluid after severe traumatic brain injury

32 the exchange of brain interstitial fluid and cerebrospinal fluid along a brain-wide network of periva

33 ll, players with PCS had significantly lower cerebrospinal fluid amyloid-beta levels compared with co

34 vement of the medial temporal lobe, abnormal cerebrospinal fluid amyloid-beta1-42 values, and proport

35 ls based on biomarkers of amyloid pathology (cerebrospinal fluid amyloid-beta42/amyloid-beta40 assay

36 = 5) or vitreous humor (n = 2) analysis and cerebrospinal fluid analysis (n = 2).

37 mination, 4.1 [0.7] months), 7 who underwent cerebrospinal fluid analysis for ZIKV had positive findi

38 Cerebrospinal fluid analysis was abnormal in 9 of 12 pat

39 sonance imaging, electroencephalography, and cerebrospinal fluid analysis) and molecular data (prion

40 etagenomic next generation sequencing of his cerebrospinal fluid and brain biopsy tissue was performe

41 test further these small molecules in human cerebrospinal fluid and in a Caenorhabditis elegans mode

42 , cis P-tau is induced in cortical axons and cerebrospinal fluid and positively correlates with axona

43 Total cerebrospinal fluid and ventricular volume change may be

44 ll subsets and immunoglobulin indices in the cerebrospinal fluid and whether retinal layer volumes al

45 physiological fluids (blood, saliva, urine, cerebrospinal fluid) and brain tissue, it is unclear whe

46 cognitive, biomarker (structural imaging and cerebrospinal fluid) and other PD psychosis risk factor

47 nt mice, and IL-1beta is found in the blood, cerebrospinal fluid, and CNS lesions of MS patients.

48 cipation, decreased DA and DA metabolites in cerebrospinal fluid, and decreased availability, and rel

49 after acute SCI, and were detected in serum, cerebrospinal fluid, and in the injured tissue.

50 apeutic drug concentrations in the brain and cerebrospinal fluid, and that non-cross-resistant drugs

51 reatly to exosomal microRNAs (miRNAs) in the cerebrospinal fluid, and these exosomal miRNAs declined

52 be the effects of complex biofluids, such as cerebrospinal fluid, and thus has wide applicability.

53 Clinical specimens (blood, cerebrospinal fluid, and urine) and environmental specim

54 -reaction (RT-PCR) assays for ZIKV in blood, cerebrospinal fluid, and urine, as well as antiflaviviru

55 Oligoclonal Ig bands (OCBs) of the cerebrospinal fluid are a hallmark of multiple sclerosis

56 ron emission tomography amyloid imaging or a cerebrospinal fluid assay of amyloid beta.

57 11-labeled Pittsburgh Compound B (PiB), and cerebrospinal fluid assays of tau and phosphorylated tau

58 ividuals, elevated brain amyloid (defined by cerebrospinal fluid assays or positron emission tomograp

59 nds, Propionibacterium species in joints and cerebrospinal fluid associated with central nervous syst

60 Moreover, microinjection of artificial cerebrospinal fluid at a pH of 6.5 into the VLM increase

61 ssed by endothelial cells of the mouse blood-cerebrospinal fluid barrier and of the blood-leptomening

62 ealthy CNS by endothelial cells of the blood-cerebrospinal fluid barrier and of the spinal cord menin

63 strate that leukemic cells transit the blood-cerebrospinal fluid barrier situated close to the dural

64 associated with brain injury as evaluated by cerebrospinal fluid biochemical markers for axonal damag

65 RI, amyloid positron emission tomography and cerebrospinal fluid biomarker analysis.

66 previously identified proSAAS as a potential cerebrospinal fluid biomarker in various neurodegenerati

67 out to have the least abnormal cognitive and cerebrospinal fluid biomarker profiles and the slowest c

68 heimer's disease neuroanatomical, cognitive, cerebrospinal fluid biomarkers and clinical profile, who

69 ere were significant relationships among all cerebrospinal fluid biomarkers and tracer uptake, notabl

70 When ND was defined using cerebrospinal fluid biomarkers, individuals with stage 2

71 We relate these neuroimaging patterns to cerebrospinal fluid biomarkers, white matter hyperintens

72 DS cohort, using multimodal neuroimaging and cerebrospinal fluid biomarkers.

73 the amount of tau tracer uptake to levels of cerebrospinal fluid biomarkers.

74 eference for a novel face) robustly predicts cerebrospinal fluid, but not blood, oxytocin concentrati

75 Apolipoprotein E isolated from human cerebrospinal fluid carries sulfatide that can be captur

76 evidence exists that differences in blood or cerebrospinal fluid chemokine concentration discriminate

77 ctivity disruption during polysomnogram, and cerebrospinal fluid collection the following morning for

78 Serial cerebrospinal fluid collection via indwelling lumbar cat

79 tment 1), extracellular (compartment 2), and cerebrospinal fluid (compartment 3).

80 Cerebrospinal fluid-contacting (CSF-c) cells containing

81 e B cells, and a neuronal-like population of cerebrospinal fluid-contacting cells.

82 96% (27 of 28) had cryptococcal meningitis (cerebrospinal fluid CRAG-positive) whereas 50% (7 of 14)

83 se in the released form of galectin-3 in the cerebrospinal fluid (CSF) 24 h after head injury.

84 plaques as evidenced by amyloid imaging and cerebrospinal fluid (CSF) Abeta1-42 (Abeta42) is an earl

85 mptoms (N=331) in the BioFINDER study, using cerebrospinal fluid (CSF) Abeta40 as a surrogate for amy

86 infected participants enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis unde

87 We studied whether continuous lower normal cerebrospinal fluid (CSF) amyloid beta1-42 (>/=640pg/ml)

88 tin C-Terminal Hydrolase L1 (UCHL1) in mouse cerebrospinal fluid (CSF) and Abeta in the mouse brains.

89 and neurofilament levels were determined in cerebrospinal fluid (CSF) and blood and analysed with re

90 iewing the literature to date on the role of cerebrospinal fluid (CSF) and blood biomarkers, we highl

91 peptide secondary structure distribution in cerebrospinal fluid (CSF) and blood plasma of 141 patien

92 p between SVD, multiple isoforms of Abeta in cerebrospinal fluid (CSF) and cortical Abeta in 831 subj

93 as a system that allows CNS perfusion by the cerebrospinal fluid (CSF) and interstitial fluid (ISF).

94 Here we show that ZIKV can persist in cerebrospinal fluid (CSF) and lymph nodes (LN) of infect

95 iral RNA was also detected in saliva, urine, cerebrospinal fluid (CSF) and semen, but transiently in

96 Neurofilaments are elevated in the cerebrospinal fluid (CSF) and serum of amyotrophic later

97 elin Basic Protein (MBP) and Tau proteins in cerebrospinal fluid (CSF) and serum, obtained from Multi

98 yloid precursor protein (APP) are present in cerebrospinal fluid (CSF) and their potential as biomark

99 We also examined cerebrospinal fluid (CSF) biomarker measures for patient

100 mpal volume, normalized whole-brain volume), cerebrospinal fluid (CSF) biomarkers (amyloid-beta1-42,

101 ical disease, we sought to determine whether cerebrospinal fluid (CSF) biomarkers are intraindividual

102 bar punctures were performed and assayed for cerebrospinal fluid (CSF) biomarkers of AD pathology, in

103 levels of differentially abundant genera and cerebrospinal fluid (CSF) biomarkers of AD.

104 The data show that LXR is a regulator of cerebrospinal fluid (CSF) both at the choroid plexus and

105 In hydrocephalus, cerebrospinal fluid (CSF) builds up in the cranial cavit

106 detection of human cytomegalovirus (hCMV) in cerebrospinal fluid (CSF) by polymerase chain reaction (

107 we showed that abnormal levels of Abeta42 in cerebrospinal fluid (CSF) can be detected before abnorma

108 min B6 (pyridoxal phosphate (PLP))) in human cerebrospinal fluid (CSF) can be used as indicators of t

109 The primary outcome was 2-week cerebrospinal fluid (CSF) clearance rate of cryptococcus

110 he absence of markers of inflammation in the cerebrospinal fluid (CSF) commonly predicts the absence

111 In the present study, cerebrospinal fluid (CSF) concentrations of GABA were an

112 Notably, cerebrospinal fluid (CSF) contains microRNAs that may se

113 Cerebrospinal fluid (CSF) continuously flows through the

114 of 10 LFA-positive cases agreeing to LP, the cerebrospinal fluid (CSF) CrAg LFA was positive.

115 quency and/or a delay in the positivity of a cerebrospinal fluid (CSF) culture or CSF antibody, parti

116 hile the concentration of Abeta(1-42) in the cerebrospinal fluid (CSF) decreases in APPswe, PS1M146V

117 central nervous system, resulting in either cerebrospinal fluid (CSF) discordance (viral load [VL] i

118 ts, perhaps owing to persistent HIV-1 RNA in cerebrospinal fluid (CSF) during antiretroviral therapy

119 nt diagnostic criteria involving analysis of cerebrospinal fluid (CSF) for parasites and/or pleocytos

120 study, we performed metabolomics analysis of cerebrospinal fluid (CSF) from cognitively intact elderl

121 undred twenty-two C57BL/6J mice infused with cerebrospinal fluid (CSF) from patients with anti-NMDAR

122 he apoE isoform amounts are not different in cerebrospinal fluid (CSF) from young normal controls, su

123 (>/= 55%), absent or low titers of serum and cerebrospinal fluid (CSF) immunoglobulin M, and occasion

124 We performed detailed neurological exams, cerebrospinal fluid (CSF) immunophenotyping and biomarke

125 her pyrimethamine lowered SOD1 levels in the cerebrospinal fluid (CSF) in patients carrying SOD1 muta

126 autism spectrum disorder (ASD) had increased cerebrospinal fluid (CSF) in the subarachnoid space (i.e

127 Plasma and cerebrospinal fluid (CSF) inflammatory markers were asse

128 5, and inflammatory phenotype was defined by cerebrospinal fluid (CSF) leukocyte and cytokine concent

129 LTA4H promoter polymorphism, which predicted cerebrospinal fluid (CSF) leukocyte count and survival o

130 eak positive correlations between plasma and cerebrospinal fluid (CSF) levels for both Abeta42 and Ab

131 We investigated whether plasma or cerebrospinal fluid (CSF) levels of cytokines and chemok

132 Elevated cerebrospinal fluid (CSF) levels of the glia-derived N-m

133 l protein that has demonstrated utility as a cerebrospinal fluid (CSF) marker of synaptic loss in AD.

134 Biological variables included cerebrospinal fluid (CSF) markers and dopamine transport

135 he innate immune system, was associated with cerebrospinal fluid (CSF) markers of neuronal injury in

136 Blood and cerebrospinal fluid (CSF) of children with TBM and hydro

137 High levels of G6 dendrimers were found in cerebrospinal fluid (CSF) of injured animals with a CSF/

138 nt heavy chain (pNfH) levels are elevated in cerebrospinal fluid (CSF) of patients with amyotrophic l

139 in levels were significantly elevated in the cerebrospinal fluid (CSF) of Plxna4(-/-) mice and, in hu

140 Existing cerebrospinal fluid (CSF) or imaging (tau positron emiss

141 e symptoms and measles antibody detection in cerebrospinal fluid (CSF) or medical record documentatio

142 disease-specific pathologic prion protein in cerebrospinal fluid (CSF) or olfactory mucosa (OM) sampl

143 Although studies have shown increased cerebrospinal fluid (CSF) OT levels following IN adminis

144 Cerebrospinal fluid (CSF) pleocytosis (>5 white blood ce

145 SCS patients had constitutional symptoms, cerebrospinal fluid (CSF) pleocytosis, and hilar adenopa

146 orylated neurofilament heavy chain (pNFH) in cerebrospinal fluid (CSF) predict disease status and sur

147 sually self-limiting condition caused by low cerebrospinal fluid (CSF) pressure, usually due to CSF l

148 fied XCAT phantom with additional and edited cerebrospinal fluid (CSF) regions to produce voxel-level

149 zed symptoms and signs, and neuroimaging and cerebrospinal fluid (CSF) results.

150 We examined longitudinal cerebrospinal fluid (CSF) samples (median, 5 samples/pat

151 ting Xpert MTB/RIF diagnostic performance on cerebrospinal fluid (CSF) samples are lacking in resourc

152 Here, using cerebrospinal fluid (CSF) samples from 239 patients with

153 During this period, serum and/or cerebrospinal fluid (CSF) samples from 3,969 patients we

154 High throughput metabolite screening of cerebrospinal fluid (CSF) samples from a canine model of

155 myloid1-42 (Abeta) levels were determined in cerebrospinal fluid (CSF) samples from all patients and

156 ing strategy to procure bone marrow (BM) and cerebrospinal fluid (CSF) samples from May 1, 2007, thro

157 al of 363 patients with HSV PCR results from cerebrospinal fluid (CSF) samples were included in this

158 Infant cerebrospinal fluid (CSF) samples were tested for ZIKV-s

159 tion (PCR) testing for Treponema pallidum in cerebrospinal fluid (CSF) samples.

160 We prospectively obtained diagnostic cerebrospinal fluid (CSF) specimens during screening for

161 and neonates with microcephaly and collected cerebrospinal fluid (CSF) specimens from 30 neonates.

162 Archival serum or cerebrospinal fluid (CSF) specimens were available from

163 strategy, defined as the absence of definite cerebrospinal fluid (CSF) sterilization or related morta

164 Cerebrospinal fluid (CSF) tau is an excellent surrogate

165 To test the hypotheses that (1) antemortem cerebrospinal fluid (CSF) tau levels correlate with post

166 ency virus (HIV) infection by evaluating the cerebrospinal fluid (CSF) to serum albumin quotient (QAl

167 tral distribution of macromolecules from the cerebrospinal fluid (CSF) to the brain and spinal cord r

168 asculature and the path of drainage from the cerebrospinal fluid (CSF) to the lymphatics remain poorl

169 We measured cerebrospinal fluid (CSF) tryptophan and kynurenine meta

170 lly was observed in animals with episodes of cerebrospinal fluid (CSF) viral rebound or sustained pla

171 pants with low or undetectable plasma VL and cerebrospinal fluid (CSF) VL prior to death who were enr

172 re unique to neuronal cells, are shed to the cerebrospinal fluid (CSF), and are detectable at low con

173 monocyte subpopulations in peripheral blood, cerebrospinal fluid (CSF), and brain biopsy material der

174 issues or prior probability maps (nlTPMs) of cerebrospinal fluid (CSF), gray matter (GM), and white m

175 ave been proposed for detection of prions in cerebrospinal fluid (CSF), most recently, the measuremen

176 which are diagnosed by detection of JCPyV in cerebrospinal fluid (CSF), often with the support of cer

177 The 102 included patients had: (1) serum, cerebrospinal fluid (CSF), or both that yielded a charac

178 ssion from MCI to AD were found for abnormal cerebrospinal fluid (CSF), phosphorylated tau (p-tau) (r

179 ngeal metastasis, or spread of cancer to the cerebrospinal fluid (CSF), which is a frequent and fatal

180 Current cerebrospinal fluid (CSF)- or imaging-based single bioma

181 tin in the blood and translocate it into the cerebrospinal fluid (CSF).

182 notherapeutics) access brain tissue from the cerebrospinal fluid (CSF).

183 eloped to measure beta-amyloid (Abeta) 42 in cerebrospinal fluid (CSF).

184 whether circulating NMDAR1 AB appear in the cerebrospinal fluid (CSF).

185 he ionic constituency of the bathing medium [cerebrospinal fluid (CSF)], or by electrical stimulation

186 ubsequent placement of an LD for drainage of cerebrospinal fluid (CSF; intervention group).

187 ls (PBMCs; n = 72), seminal plasma (n = 20), cerebrospinal fluid (CSF; n = 36), dried blood spots (DB

188 lt Malawian patients with ABM (n = 523 [65%] cerebrospinal fluid [CSF] culture positive).

189 Localization of a cerebrospinal fluid [CSF] fistula is a diagnostic challe

190 lium (CPE) secretes higher volumes of fluid (cerebrospinal fluid, CSF) than any other epithelium and

191 All infants had either cerebrospinal fluid cultures or 7-day follow-up.

192 erences (blood CCL5, CCL7, CXCL9, CXCL10 and cerebrospinal fluid CXCL8 and CXCL10).

193 Cerebrospinal fluid data from all 37 laboratories perfor

194 based on immediate and sustained response to cerebrospinal fluid diversion.

195 f meningeal LVs should allow manipulation of cerebrospinal fluid drainage and neuropathological proce

196 s or situations: cerebrospinal fluid shunts, cerebrospinal fluid drains, implantation of intrathecal

197 Subsequent edema can hinder cerebrospinal fluid efflux and can lead to locally incre

198 They are found in the cerebrospinal fluid even in the absence of inflammation;

199 ther supportive information is obtained from cerebrospinal fluid examination and neurophysiological t

200 Cerebrospinal fluid examination revealed mildly elevated

201 Serial cerebrospinal fluid examinations were consistent with ch

202 the formation of ependymal cells, disrupting cerebrospinal fluid flow and causing hydrocephalus.

203 lper T-cell subsets from peripheral blood or cerebrospinal fluid for cytokine production, gene expres

204 Testing of cerebrospinal fluid for ZIKV using IgM antibody-capture

205 We analyzed cerebrospinal fluid from 32 cases, 40 subjects with mult

206 The endopeptidome of cerebrospinal fluid from 40 Alzheimer's disease (AD) pat

207 ve proteomics to identify protein changes in cerebrospinal fluid from children with Streptococcus pne

208 to have an altered consciousness and higher cerebrospinal fluid fungal burden at presentation.

209 (+) T cells pervade lymphatic organs and the cerebrospinal fluid, have a strong ability to produce di

210 Human cerebrospinal fluid (hCSF) substantially increases the e

211 om pyramidal cells, in the presence of human cerebrospinal fluid (hCSF), using a matched artificial c

212 we characterize exosomes directly from human cerebrospinal fluid (hCSF).

213 Higher cerebrospinal fluid HTLV-1 proviral load (p = 0.01) was

214 ci) and possibly promoted by low flow of the cerebrospinal fluid in these areas.

215 s and the frequencies of immune cells in the cerebrospinal fluid (including B cells, T cells, and nat

216 iagnosis, exercise-induced miRNA patterns in cerebrospinal fluid indicated distinct mechanisms for po

217 hether meningitis is present and analysis of cerebrospinal fluid is essential for diagnosis.

218 des quantitative data on the dynamics of the cerebrospinal fluid, it can help test new therapies and

219 ons, vital signs, clinical laboratory tests, cerebrospinal fluid laboratory tests, and electrocardiog

220 We assessed for surgical complications (cerebrospinal fluid leakage and intracerebral haemorrhag

221 monomeric glycoprotein, is known to indicate cerebrospinal fluid leakage.

222 hetical preclinical AD groups using baseline cerebrospinal fluid levels of Abeta and tau or Abeta and

223 e lateral temporal lobe, and lower mean (SD) cerebrospinal fluid levels of tau (59.2 [32.8] vs 111.3

224 s and tracer uptake, notably for tau-related cerebrospinal fluid markers.

225 rmal expression of Keratin 9 in AD blood and cerebrospinal fluid may be a result of blood brain barri

226 tic end feet and defects in the synthesis of cerebrospinal fluid may be targeted by LXR agonists to f

227 adjusted for magnetic resonance imaging and cerebrospinal fluid measurements, sCD27 levels were asso

228 ntal cortices is correlated with tau-related cerebrospinal fluid measures.

229 Total cerebrospinal fluid (median, 47.6 mL [35.1-69.4 mL] to 6

230 lesterol was confirmed in serum (n = 19) and cerebrospinal fluid (n = 17) of SPG5 patients.

231 levels of 38 mediators in serum (n = 78) and cerebrospinal fluid (n = 37) specimens from patients wit

232 Conclusions and Relevance: Increased cerebrospinal fluid neurofilament light proteins and red

233 Cerebrospinal fluid NFL concentration (median [interquar

234 Cerebrospinal fluid NFL concentration is increased by th

235 biomarker in ALS and the prognostic value of cerebrospinal fluid NFL in patients with ALS.

236 ethyl-D-aspartate receptor antibodies in the cerebrospinal fluid of 1 patient.

237 t here altered levels of gangliosides in the cerebrospinal fluid of amyotrophic lateral sclerosis pat

238 a markedly disturbed immune signature in the cerebrospinal fluid of cases that is consistent with imm

239 at shed PrP(c) (sPrP(c)) is increased in the cerebrospinal fluid of HIV-infected people with cognitiv

240 observed an increase in ACE2 activity in the cerebrospinal fluid of hypertensive patients, which was

241 ence essential for JCV neurovirulence and in cerebrospinal fluid of immunosuppressed patients with mu

242 f DN and CD21(low) B cells were found in the cerebrospinal fluid of MS patients compared with paired

243 influenza-infected mice and was elevated in cerebrospinal fluid of MS patients during relapse compar

244 even in the absence of inflammation; in the cerebrospinal fluid of MS patients they occur at a frequ

245 tion marker soluble CD27 (sCD27) measured in cerebrospinal fluid of patients at time of a first attac

246 The cerebrospinal fluid of patients with active MS was enric

247 Soluble CD27 in cerebrospinal fluid of patients with CIS was associated

248 individual neurotropic JCPyV strains in the cerebrospinal fluid of patients with PML.

249 in VH4-containing genes in B cells from the cerebrospinal fluid of patients with relapsing remitting

250 s from laboratory studies were notable for a cerebrospinal fluid opening pressure of 28 mm H2O and 8

251 xperience with the safety, tolerability, and cerebrospinal fluid penetration of the Src family kinase

252 ippocampal volume), neurofibrillary tangles (cerebrospinal fluid phosphorylated tau181 assay levels),

253 cteristics, including non-motor symptoms and cerebrospinal fluid phosphorylated-tau/total-tau ratio.

254 rmacokinetic findings of approximately 0.001 cerebrospinal fluid:plasma ratio further supported limit

255 t with the homozygous PNPO variant had a low cerebrospinal fluid pyridoxal-5-phosphate level.

256 N/MCP, DN-to-pons, GP-to thalamus, and GP-to-cerebrospinal fluid ratios were measured and compared by

257 Dural cerebral veins (CV) are required for cerebrospinal fluid reabsorption and brain homeostasis,

258 underpinning mechanisms include the role of cerebrospinal fluid regulation as well as metabolic and

259 In the cerebrospinal fluid sample of a 73-year-old woman with r

260 ologic and molecular testing using blood and cerebrospinal fluid samples can serve as a less expensiv

261 evel of tumor necrosis factor-alpha in those cerebrospinal fluid samples confirmed that ADAM17 was up

262 Examination of cerebrospinal fluid samples detected lymphomatous mening

263 osorbent assay for ZIKV was performed on the cerebrospinal fluid samples of 7 of the 8 infants (88%),

264 Serum samples of cases and controls and cerebrospinal fluid samples of cases were tested for Zik

265 A total of 119 cerebrospinal fluid samples were collected from 28 sever

266 Cerebrospinal fluid samples were obtained from 176 patie

267 is presentation and found Ebola virus in all cerebrospinal fluid samples.

268 female (66.7%).Patients with CIS had higher cerebrospinal fluid sCD27 levels than control individual

269 .20%]) and intrathecal IgG synthesis (median cerebrospinal fluid/serum IgG index, 0.78 [IQR, 0.53-1.0

270 er CRD (HR, 1.07; 95% CI, 1.03 to 1.11), and cerebrospinal fluid shunting (HR, 2.02; 95% CI, 1.07 to

271 fter the following procedures or situations: cerebrospinal fluid shunts, cerebrospinal fluid drains,

272 High TSPyV loads in blood and cerebrospinal fluid, sometimes coinciding with cerebral

273 Among 4301 cerebrospinal fluid specimens tested, 1603 (37.3%) were

274 Most of the serum and cerebrospinal fluid studies were unremarkable.

275 fonylurea receptor-1 is available, assessing cerebrospinal fluid sulfonylurea receptor-1 in larger st

276 This is the first report quantifying human cerebrospinal fluid sulfonylurea receptor-1.

277 Cerebrospinal fluid testing was less sensitive than seru

278 f specific IgM antibodies in either blood or cerebrospinal fluid that appear in the second phase of t

279 post sacrifice and maintained in artificial cerebrospinal fluid to create near-physiological conditi

280 monkeys, where it achieved 10-fold enhanced cerebrospinal fluid to plasma ratio versus DON.

281 umes of whole brain, tissue compartments and cerebrospinal fluid, to test the hypothesis that HCA is

282 rived measures of neurodegeneration/atrophy (cerebrospinal fluid-to-brain volume ratio, and hippocamp

283 Transthyretin (TTR) is a blood and cerebrospinal fluid transporter of thyroxine and retinol

284 Limited data suggest that the cerebrospinal fluid Treponema pallidum particle agglutin

285 genic bacteria in positive culture of blood, cerebrospinal fluid, urine, or stool samples, including

286 Soluble CD27 levels were determined in cerebrospinal fluid using a commercially available enzym

287 crease was associated with increase in total cerebrospinal fluid volume (r = 0.83, p < 0.001), and ch

288 quantitative technique to measure brain and cerebrospinal fluid volume changes.

289 e (r = 0.83, p < 0.001), and change in total cerebrospinal fluid volume was associated with ventricul

290 Associations among total cerebrospinal fluid volume, ventricular volume, serum so

291 ing brain-predicted age with grey matter and cerebrospinal fluid volumes (themselves strong predictor

292 Fungal clearance in cerebrospinal fluid was slower in the dexamethasone grou

293 itive RT-PCR results), although 3 samples of cerebrospinal fluid were also positive.

294 MicroRNAs (miRNA) in cerebrospinal fluid were amplified by quantitative PCR.

295 ension and the soluble CD14 concentration in cerebrospinal fluid were associated with lower fractiona

296 MIF levels in the cerebrospinal fluid were associated with systemic compli

297 Samples of serum and cerebrospinal fluid were tested for ZIKV using real-time

298 t contains a solution that closely resembles cerebrospinal fluid with the addition of extracellular m

299 s typically confined to the brain, eyes, and cerebrospinal fluid without evidence of systemic spread.

300 ce showed substantial levels of scFvs in the cerebrospinal fluid without significant effects on total