ライフサイエンスコーパス: cerebrovascular accident

1 tcomes were death, myocardial infarction, or cerebrovascular accident.

2 nt was withdrawn from the study because of a cerebrovascular accident.

3 eath of any cause, myocardial infarction, or cerebrovascular accident.

4 due to any cause, myocardial infarction, or cerebrovascular accident.

5 entricular arrhythmias, current smoking, and cerebrovascular accident.

6 mg/dL, history of hypertension, or death by cerebrovascular accident.

7 the follow-up period, one mother died from a cerebrovascular accident.

8 increased risk for death and potentially for cerebrovascular accidents.

9 ogenesis of anatomic derangements underlying cerebrovascular accidents.

10 ve Q-wave myocardial infarctions, and 6 (5%) cerebrovascular accidents.

11 significant reductions in rates of confirmed cerebrovascular accidents (0%, P = .015) and mortality (

12 jor hemorrhage (1.7% vs. 2.3%; p = 0.61), or cerebrovascular accident (1.2% vs. 1.5%; p = 0.77).

13 trial fibrillation (4.2% vs 18.0%, p=0.001), cerebrovascular accident (1.7% vs 7.0%, p=0.04), and pos

14 ence interval, 1.06-1.21) and a trend toward cerebrovascular accident (10-year hazard ratio, 1.08; 95

15 had the highest percentage of damage due to cerebrovascular accident (12.8%) and venous thrombosis (

16 793 subjects died, including 279 who died of cerebrovascular accident, 217 who died of cancer, and 20

17 ardiovascular death (6.0 vs 6.0%, p=0.86) or cerebrovascular accident (3.6 vs 3.5%, p=0.31).

18 schaemic heart disease (6.3 million deaths), cerebrovascular accidents (4.4 million deaths), lower re

19 Cerebrovascular accident (40%) was the most prevalent ca

20 e neurologic deficit, and 5% developed a new cerebrovascular accident after dissection repair.

21 ent coronary artery bypass graft surgery, or cerebrovascular accident after PCI.

22 Cerebrovascular accidents after PCI, although rare, are

23 Cerebrovascular accidents after PCI, although rare, can

24 ferences in death, myocardial infarction, or cerebrovascular accident among patients enrolled in the

25 ho do not undergo surgical removal, rates of cerebrovascular accident and mortality are increased.

26 ) died because of an adverse event (one [7%] cerebrovascular accident and one [7%] respiratory failur

27 (CHF), abdominal aortic aneurysm (AAA), and cerebrovascular accident and tracked triglyceride, high-

28 inically evident, radiographically confirmed cerebrovascular accidents and 60-day mortality and evalu

29 Primary end points included cerebrovascular accidents and all-cause mortality in the

30 astrocytes in tissue surrounding new and old cerebrovascular accidents and brain tumours.

31 ns in cerebral blood flow (CBF), hemorrhage, cerebrovascular accidents and death.

32 acute care hospitals, aged </= 70 years from cerebrovascular accidents and trauma.

33 tomatic fibrillation and reduces the risk of cerebrovascular accidents and ventricular tachycardia.

34 symptoms included transient ischemic attack, cerebrovascular accident, and amaurosis fugax.

35 rt (1.0% vs. 1.2%) and no difference in AAA, cerebrovascular accident, and CHF.

36 associated with increased risk of death, HF, cerebrovascular accident, and myocardial infarction (p <

37 Subjects were followed for death, HF, cerebrovascular accident, and myocardial infarction with

38 coma lasting >24 hours, acute renal failure, cerebrovascular accident, and pulmonary embolism.

39 e = 2.5 mg/dL), peripheral vascular disease, cerebrovascular accident, and severe obesity (body mass

40 n, tachydysrythmias, myocardial infarctions, cerebrovascular accidents, and deaths.

41 bidity (such as the acute coronary syndrome, cerebrovascular accidents, and heart failure) was collec

42 omposite of death, myocardial infarction, or cerebrovascular accidents, and occurrence of the key saf

43 ore the invasion were myocardial infarction, cerebrovascular accidents, and other chronic disorders w

44 rial fibrillation and related cardio-embolic cerebrovascular accidents are two well-defined major hea

45 hree patients (2%) in the pravastatin group (cerebrovascular accident, arteriosclerosis coronary arte

46 organs, especially from ECD and donors with cerebrovascular accident as cause of death, and to impro

47 These factors included donor age, cerebrovascular accident as the cause of death, renal in

48 ath, nonfatal myocardial infarction (MI), or cerebrovascular accident at 24 months.

49 I, 3.9-6.1; OR, 2.3; 95% CI, 1.7-3.2), acute cerebrovascular accident (beta coefficient, 6.6; 95% CI,

50 neurologic disorders and injuries (including cerebrovascular accidents, brain trauma, brain tumors, a

51 dial infarction, coronary revascularization, cerebrovascular accident, carotid endarterectomy, periph

52 -year-old female patient with a history of a cerebrovascular accident caused by a right pontine arter

53 Subject Headings alcohol drinking, ethanol, cerebrovascular accident, cerebrovascular disorders, and

54 nsion, a silent myocardial infarction, and a cerebrovascular accident complicated by seizures.

55 ient death from acute myocardial infarction, cerebrovascular accident, congestive heart failure, or p

56 on (AMI), congestive heart failure (CHF), or cerebrovascular accident (CVA) from 1991 to 1994.

57 Twenty patients had at least one cerebrovascular accident (CVA) in follow-up; 35 patients

58 tion (AF), use of warfarin and prevalence of cerebrovascular accident (CVA) in paced versus unpaced p

59 There was a higher prevalence of cerebrovascular accident (CVA) in patients with SEC when

60 Cerebrovascular accident (CVA) is a major complication o

61 rimary end point was the first occurrence of cerebrovascular accident (CVA) or death.

62 e rate of resolution of iBCVIs, freedom from cerebrovascular accident (CVA) or transient ischemic att

63 rative predictors of complication were prior cerebrovascular accident (CVA), chronic obstructive pulm

64 rated contralateral carotid occlusion, prior cerebrovascular accident (CVA), or SPs less than 35 mmHg

65 ding mortality, intraoperative/postoperative cerebrovascular accident (CVA), postoperative bleeding,

66 ite of death, myocardial infarction (MI), or cerebrovascular accident (CVA).

67 hospitalized myocardial infarctions (MI) and cerebrovascular accidents (CVA) in patients with diabeti

68 42 339 in-hospital deaths </= 70 years from cerebrovascular accidents (CVA) or trauma that were corr

69 ce, predictors, and clinical implications of cerebrovascular accidents (CVAs) after percutaneous coro

70 dial Infarction, ventricular arrhythmia, and cerebrovascular accident during follow-up, albeit with d

71 dial Infarction, ventricular arrhythmia, and cerebrovascular accident during follow-up.

72 ardial infarction, congestive heart failure, cerebrovascular accident, gastrointestinal tract hemorrh

73 eline variables (death/myocardial infarction/cerebrovascular accident: hazard ratio: 1.11, 99% confid

74 >50 years, female sex, death attributable to cerebrovascular accident, hypertension, diabetes mellitu

75 ), and ischemic stroke (CV death/MI/ischemic cerebrovascular accident [iCVA]).

76 boli despite prophylaxis and an unrecognized cerebrovascular accident in one patient each.

77 phropathy in one patient, and death due to a cerebrovascular accident in one patient.

78 l cell carcinoma in the ozanezumab group and cerebrovascular accident in the placebo group).

79 Major bleeding events occurred in 33% and cerebrovascular accidents in 14%.

80 igate the role of ANP in the pathogenesis of cerebrovascular accidents in humans.

81 may represent an independent risk factor for cerebrovascular accidents in humans.

82 could underlie the unfavorable prognosis of cerebrovascular accidents in sleep apnea patients.

83 matics in a female patient recovering from a cerebrovascular accident involving anterior regions of t

84 2 years and reported the outcome measures of cerebrovascular accidents, myocardial infarctions, arter

85 dical cause: cardiovascular disease (n = 2), cerebrovascular accident (n = 1), metabolic collapse and

86 Reported AEs were thromboembolic cerebrovascular accident (n = 39), acute myocardial infa

87 Indications for placement were cerebrovascular accident (n = 80), failure to thrive (n

88 Cerebrovascular accident occurred in 0.3%, and transfusi

89 ped hemodynamic compromise and no documented cerebrovascular accident occurred within one month after

90 Vision loss and cerebrovascular accidents often complicate giant cell ar

91 n-hospital mortality, myocardial infarction, cerebrovascular accident or transient ischemic attack, r

92 1) or having a residual deficit from a prior cerebrovascular accident (OR, 1.17; 95% CI, 1.11-1.22; P

93 mg ranibizumab yielded an increased risk for cerebrovascular accidents (OR, 2.33; 95% CI, 1.04-5.22;

94 ns for myocardial infarction, heart failure, cerebrovascular accident, or angina after the index angi

95 omposite end point of myocardial infarction, cerebrovascular accident, or cardiovascular death during

96 story of myocardial infarction, a history of cerebrovascular accident, or history of alcohol abuse.

97 f cardiac death, myocardial infarction (MI), cerebrovascular accident, or re-vascularization occurred

98 idual risks of death, myocardial infarction, cerebrovascular accident, or stent thrombosis did not di

99 ent (myocardial infarction, angina pectoris, cerebrovascular accidents, or major coronary surgery).

100 riates were defined: cardiovascular disease, cerebrovascular accident, peripheral vascular disease, d

101 irculatory collapse, wound infection, ileus, cerebrovascular accident [possibly treatment related], a

102 mia, coronary stenosis, coronary restenosis, cerebrovascular accident, randomized controlled trial, c

103 ing 30-day mortality, myocardial infarction, cerebrovascular accident, rebleeding, pneumonia, or thro

104 come was a composite of all-cause mortality, cerebrovascular accident, reinfarction, or unplanned tar

105 .610; 95% confidence interval, 0.440-0.847), cerebrovascular accidents (relative risk, 0.840; 95% con

106 Rates of cerebrovascular accident, return to cardiopulmonary bypa

107 Cerebrovascular accident risk in group 2 at 1 and 5 year

108 > or =6 months) transient ischemic attack or cerebrovascular accident, smoking history, and creatinin

109 r composite of death, myocardial infarction, cerebrovascular accident, target vessel revascularizatio

110 oped adverse neurological events, defined as cerebrovascular accident, transient ischemic attack (TIA

111 idences of death or readmission for embolic (cerebrovascular accident, transient ischemic attack, and

112 The composite primary end point was cerebrovascular accident, transient ischemic attack, and

113 Cerebrovascular accidents/transient ischemic attacks occ

114 ed arterial events (myocardial infarction or cerebrovascular accident), venous events (deep vein thro

115 Although cerebrovascular accident was associated with an increase

116 The incidence of cerebrovascular accident was not significantly reduced b

117 monary vein isolation or after cardioembolic cerebrovascular accident was performed using standard ap

118 a come almost exclusively from patients with cerebrovascular accidents where brain damage extends int

119 onfatal myocardial infarctions, and nonfatal cerebrovascular accidents, which are possible effects fr

120 ty analyses revealed that the annual risk of cerebrovascular accident would have to be at least 1.5%