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1 or regression of histological or cytological cervical abnormalities, or incidence of invasive cervcal
2 r with aphthous stomatitis, pharyngitis, and cervical adenitis (PFAPA) is a relatively common autoinf
3 ehaviors were compared to incidence data for cervical adenocarcinoma, oropharyngeal cancer, and anal
4 ded a correct attribution for KB, as HeLa or cervical adenocarcinoma.
5 sits included questionnaires, chart reviews, cervical/anal cytologic and cervical/anal HPV testing fo
6 , chart reviews, cervical/anal cytologic and cervical/anal HPV testing for 2 years.
7              Thirty-day POM was higher after cervical anastomosis (8.8% vs 4.9%, P < 0.001).
8    After adjustment for confounding factors, cervical anastomosis was associated with 30-day POM [odd
9 provides a lower 30-day POM rate compared to cervical anastomosis, and performing a thoracotomy is no
10                            Intrathoracic (vs cervical) anastomosis and a thoracotomy (vs absence) hav
11 n patients having received intrathoracic (vs cervical) anastomosis and between those having received
12 -wide mRNA expression and DNA methylation in cervical and anterior thoracal spinal cord segments of f
13  MIA PaCa-2 pancreatic, DU145 prostate, HeLa cervical and CaCo-2 colon, as well as normal human MCF10
14  and PPV for detecting distant metastasis in cervical and endometrial cancer and should be included i
15 ic curve (AUC) values were 0.78 and 0.89 for cervical and endometrial cancer, respectively; these wer
16 et HPV integration is frequently reported in cervical and head and neck cancer specimens.
17 ditions, as are often found in subregions of cervical and head and neck cancers, enable HPV-positive
18 inked to major human malignancies, including cervical and head and neck cancers.
19 ronounced differences were higher anxiety in cervical and laryngeal, lower anxiety in upper cranial a
20 inings, as well as Western blot analyses, on cervical and lumbar sections of the spinal cord in patie
21 he anterior horn of the gray matter, in both cervical and lumbar sections.
22                         Interactions between cervical and lumbar spinal circuits are mediated by long
23 uron histological analyses were performed in cervical and lumbar spinal cords.
24                       Coordinated control of cervical and mandibular musculatures, which is necessary
25 justed incidence of HPV-related cancer (both cervical and non-cervical) in all women in England in vi
26                                              Cervical and thoracic lesions that persisted from pain o
27                                  We compared cervical and vulvar areas of the vagina in young nullipa
28 BS colonization (GBS isolation from vaginal, cervical, and/or rectal swabs; with separate subanalysis
29 se 5% of all cancer cases worldwide, notably cervical, anogenital, and oropharyngeal cancers.
30 s have suggested a link between migraine and cervical artery dissection (CEAD), any association betwe
31  coronary artery disease, migraine headache, cervical artery dissection, fibromuscular dysplasia, and
32 g to the temporal progression of thoracic to cervical atrophy described here.
33                                            A cervical basis for the parturition delay was confirmed b
34                                     In human cervical biopsies, IL-17A(+) gammadelta T cells could on
35 rmal cervical cytology, no previous abnormal cervical biopsy results, and no more than four lifetime
36 , 55.5; fatigue, 57.3; depression, 51.4) and cervical cancer (anxiety, 53.2; sleep disturbance, 53.4)
37  to prevent 1 anogential warts (AGW) case or cervical cancer (CC) was similar for routine + catch-up
38  discovery: human embryonic kidney (HEK293), cervical cancer (HeLa), and liver cancer (HepG2).
39 f human papillomavirus-associated metastatic cervical cancer after tumor-infiltrating adoptive T cell
40 distant metastasis was 13.7% (21 of 153) for cervical cancer and 11.8% (24 of 203) for endometrial ca
41         Results There were 153 patients with cervical cancer and 203 patients with endometrial cancer
42 est for gliomas and sarcomas and highest for cervical cancer and oropharyngeal head and neck cancer.
43 hat wider implementation will help to reduce cervical cancer and precursors incidence.
44                                   Breast and cervical cancer are major threats to the health of women
45  2019 could reduce age-standardised rates of cervical cancer at ages 25-64 years by 19%, from 15.1 in
46 wanted to examine whether IPSA could improve cervical cancer brachytherapy plans giving D90 < 6 Gy (w
47          We obtained relative risks (RRs) of cervical cancer by screening history (never screened, re
48  provide broader coverage and prevent 90% of cervical cancer cases worldwide.
49                              The efficacy of cervical cancer cell elimination was drastically increas
50 liferation of a conventional HPV-16 positive cervical cancer cell line.
51 n and, in contrast to all other HPV-positive cervical cancer cell lines, they harbored a gain-of-func
52 xenograft in vivo in nude mice, and suppress cervical cancer cell migration and invasion.
53  of human cervical cancer cells in vitro and cervical cancer cell xenograft in vivo in nude mice, and
54 nergistically to inhibit the growth of human cervical cancer cells in vitro and cervical cancer cell
55 RISPR/Cas9), the SIRT1 gene was removed from cervical cancer cells.
56 ts Twenty-four patients were enrolled in the cervical cancer cohort.
57  present study aimed to investigate the anti-cervical cancer effects of metformin, a first-line thera
58  cohort effects to provide absolute risks of cervical cancer for unscreened women in different birth
59 tify proteins interacting with TCTP in human cervical cancer HeLa cells.
60    In the next 25 years, the epidemiology of cervical cancer in England, UK, will change: human papil
61  We estimated age-specific absolute risks of cervical cancer in the absence of screening (derived fro
62  was associated with a reduction in invasive cervical cancer incidence (crude HR 0.40, 95% CI 0.18-0.
63 ce, progression, or regression; and invasive cervical cancer incidence among women living with HIV.
64 redict an initial widening of this gap, with cervical cancer incidence in Asian women up to 2.5 times
65 wever, the significance of LKB1 mutations in cervical cancer initiation and progress has not been exa
66  driver of transformation in this aggressive cervical cancer is not HPV oncogene expression but rathe
67 ir "causal" HPV infection that develops into cervical cancer is poorly understood and practically uno
68                                        Human cervical cancer is the fourth most common carcinoma in w
69 y maintain HPV 31 or 16 episomes, as well as cervical cancer lines that contain integrated genomes su
70 al tumor microenvironment is associated with cervical cancer maintenance and progression.
71  value of 47.6%, 93.9%, 55.6%, and 91.9% for cervical cancer metastasis and 66.7%, 93.9%, 59.3%, and
72  value of 54.8%, 97.7%, 79.3%, and 93.1% for cervical cancer metastasis versus 64.6%, 98.6%, 86.1%, a
73 sis drug bevacizumab for women with advanced cervical cancer on the basis of improved overall surviva
74 pplicators were developed for 30 consecutive cervical cancer patients on the basis of computed tomogr
75 assays based on nfGNPs for HPV detection and cervical cancer prevention.
76 oncogenic HPV types could make prevention of cervical cancer programmatically feasible.
77 s time period, and more modest reductions in cervical cancer screening and sexual risk behaviors.
78 idered when clinical practice guidelines for cervical cancer screening are reassessed.
79         Recent changes in the periodicity of cervical cancer screening have led to questions about th
80 lomavirus (HPV) tests are needed for primary cervical cancer screening in lower-resource regions.
81 I] 1.1-2.7) more likely to be diagnosed with cervical cancer than white women (22.8 vs 13.4 cases per
82  predicted to have a relatively high risk of cervical cancer throughout their lives.
83 ns, the direct reduction in lifetime risk of cervical cancer varied from 55% (53-56%) among women vac
84 se was observed in a patient with metastatic cervical cancer who received 2.7 x 10(9) cells (ongoing
85 eady recommends screening (ie, screening for cervical cancer with a Papanicolaou smear, screening for
86 el the dynamics of cell cycle in HeLa (human cervical cancer) and S. cerevisiae cells.
87  for their anticancer activity against HeLa (cervical cancer), MCF-7 (breast cancer), HL-60 (Human pr
88 ccination coverage, changes in screening for cervical cancer, and risk behaviors for acquiring HPV.
89 s, with male circumcision protecting against cervical cancer, cervical dysplasia, herpes simplex viru
90 pelvic examination for conditions other than cervical cancer, gonorrhea, and chlamydia, for which the
91 r any gynecologic cancer or condition except cervical cancer, gonorrhea, and chlamydia, which are cov
92  programmed death ligand 1-positive advanced cervical cancer, pembrolizumab demonstrated antitumor ac
93  model that simulates the natural history of cervical cancer, we estimated the cumulative number of c
94 ancers, we describe the burden of breast and cervical cancer, with an emphasis on global and regional
95 women worldwide are diagnosed with breast or cervical cancer, yet where a woman lives, her socioecono
96 (HPV) screening will be the primary test for cervical cancer.
97  including human papillomavirus (HPV)-caused cervical cancer.
98 extremely rare and aggressive neuroendocrine cervical cancer.
99 e agents of many human cancers, most notably cervical cancer.
100 ificant potential for the treatment of human cervical cancer.
101 has been established as the primary cause of cervical cancer.
102 ts from the cohort of patients with advanced cervical cancer.
103 lity of antiangiogenesis therapy in advanced cervical cancer.
104                                   Breast and cervical cancers are the commonest cancers diagnosed in
105 nce were performed on 30 women with advanced cervical cancers at three time points (within 2 weeks be
106 omavirus 58 (HPV58) is found in 10 to 18% of cervical cancers in East Asia but is rather uncommon els
107           Our model projected that among all cervical cancers, 50% and 75% of women acquired their ca
108 s including penile, vulva, oropharyngeal and cervical cancers.
109 ICs and high-income countries for breast and cervical cancers.
110 2 years, persistent HPV16 causes half of all cervical cancers.
111 papillomavirus (HPV) plays a central role in cervical carcinogenesis and its oncogene E7 is essential
112 ent Human Papillomavirus (HPV) infection and cervical carcinogenesis.
113 omavirus (HPV) infections cause nearly every cervical carcinoma and a subset of tumors in the orophar
114 bined chemotherapy and radiation therapy for cervical carcinoma and evaluate the risk of local recurr
115 for the anchorage-independent growth of HeLa cervical carcinoma cells.
116 ts with metastatic, persistent, or recurrent cervical carcinoma from 81 centres in the USA, Canada, a
117 encing of RNA prepared using RPAD from human cervical carcinoma HeLa cells and mouse C2C12 myoblasts
118 eliminated detectable clonogens in some SiHa cervical carcinoma xenografts, and in combination with g
119 ade 2 or 3, adenocarcinoma in situ, invasive cervical carcinoma), vulvar disease (vulvar intraepithel
120                                     However, cervical CD4+ T-cell number was associated with HSV-2 in
121 or C-to-T mutations in human papilloma virus cervical cell lines and genital warts.
122  suppressing migration and invasion of human cervical cell lines HeLa, SiHa and CaSki.
123   miR-143 and miR-145 transfection decreased cervical cell number by increasing apoptosis and decreas
124 ificant increases in miR-143 and miR-145, in cervical cells of women destined to have a PTB.
125     Histology revealed dramatically expanded cervical cementum in GACI teeth, including cementocyte-l
126 om 3 GACI subjects revealed 4-fold increased cervical cementum thickness ( P = 0.00007) and a 23% inc
127 fore, this study sought to determine whether cervical changes could explain the parturition differenc
128             In this observational study, all cervical cytology and HPV testing reports from January 1
129 ho were healthy, with no history of abnormal cervical cytology, no previous abnormal cervical biopsy
130 be associated with patient discomfort due to cervical dentin hypersensitivity (CDH) and esthetic diss
131 rimary outcomes were incidence of high-grade cervical disease (cervical intraepithelial neoplasia gra
132 wed statistically significant changes during cervical disease progression in clinical samples.
133 led to faster and more complete diagnosis of cervical disease, but 55.8% more biopsies and 20.0% more
134 England reported a disproportionate share of cervical disease.
135 1-26.4; P = .04) after adjusting for sex and cervical dissection.
136 caudal to rostral, as in other species, into cervical dorsal horn, subnucleus caudalis, subnucleus in
137 umcision protecting against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamyd
138 obustly associated with dystonic symptoms in cervical dystonia and may be a useful biomarker for adap
139 ine the effect of miR-143 and miR-145 on the cervical epithelial barrier and to elucidate the mechani
140 R-143 and miR-145 play a significant role in cervical epithelial barrier breakdown through diverse me
141  the mechanisms by which these miRNAs modify cervical epithelial cell function.
142 increased in miR-143 and miR-145 transfected cervical epithelial cells.
143  expression in laser-captured regions of the cervical epithelium and stroma of untreated or estrogen-
144               HPV oncogene expression in the cervical epithelium elicited significant gene-expression
145          High intrathoracic (Ivor Lewis) and cervical esophagogastrostomy (McKeown) were performed in
146  birth is reported to correlate with size of cervical excision and therefore balancing the risk of ad
147 amples, with levels highest in cocultures of cervical fibroblasts and cancer-derived epithelial cells
148 g, non-Hodgkin lymphoma, breast, uterine, or cervical) from 2010 to 2012 (N = 5,284) were recruited t
149 minal ganglion (TG) and sympathetic superior cervical ganglion (SCG) neurons expressed adrenergic rec
150 blastoma neuro-2A cells and primary superior cervical ganglion neurons, where APP is highly expressed
151        Finally, our data identified superior cervical ganglion-10 (SCG10) as an interacting partner o
152   Our aim is to investigate the evolution of cervical gene expression as related to putative mechanis
153 % had anal HPV, 51% had anal HR-HPV, 50% had cervical HPV, and 29% had cervical HR-HPV.
154 al HR-HPV, 50% had cervical HPV, and 29% had cervical HR-HPV.
155                                Although most cervical human papillomavirus type 16 (HPV16) infections
156 aphy images to assess whether there was true cervical ICA occlusion.
157  to demonstrate a lack of attenuation in the cervical ICA on the symptomatic side (24 men and 13 wome
158  patients (32.4%) with nonattenuation of the cervical ICA on the symptomatic side.
159                                      Results Cervical ICA pseudo-occlusion occurred in 12 of 37 patie
160 of HPV-related cancer (both cervical and non-cervical) in all women in England in view of differences
161 with an adverse obstetrical outcome included cervical incompetence (adjusted odds ratio, 24.29; 95% C
162 The major risk factors for such outcomes are cervical incompetence, preterm labor during current preg
163             Patients received a standardised cervical injection of indocyanine green and sentinel-lym
164 graphic (CT) angiography to distinguish true cervical internal carotid artery (ICA) occlusion from ps
165  Extracranial carotid disease was defined as cervical internal carotid artery stenosis (>50%) or occl
166 nown about the long-term yield of high-grade cervical intraepithelial neoplasia (CIN) and the influen
167 ce; squamous intraepithelial lesion (SIL) or cervical intraepithelial neoplasia (CIN) prevalence, inc
168            To determine the population-based cervical intraepithelial neoplasia (CIN) trends when adj
169 d with tests for hrHPV and HPV 16/18 to find cervical intraepithelial neoplasia (grade >/=2 [CIN2+] o
170 gnificantly increased with disease severity (cervical intraepithelial neoplasia [CIN] 3, 17.9% [+/-7.
171 re incidence of high-grade cervical disease (cervical intraepithelial neoplasia grade 2 or 3, adenoca
172 lasia grades 2/3 and adenocarcinoma in situ (cervical intraepithelial neoplasia grade 2 or higher [CI
173 lial neoplastic lesions and invasive cancer (cervical intraepithelial neoplasia grade 2+ [CIN2+]) com
174  and premalignancies in women diagnosed with cervical intraepithelial neoplasia grade 3 (CIN3).
175 icut (CT) implemented mandatory reporting of cervical intraepithelial neoplasia grades 2/3 and adenoc
176 currence of residual or recurrent high-grade cervical intraepithelial neoplasia of grade two or worse
177  screening increases detection of high-grade cervical intraepithelial neoplastic lesions and invasive
178 t sites were grouped into five body regions: cervical, laryngeal, limb, lower cranial and upper crani
179 s 0.59 (95% CI, 0.56-0.62), for transvaginal cervical length alone was 0.67 (95% CI, 0.64-0.70), and
180                                 Transvaginal cervical length and quantitative vaginal fetal fibronect
181 bronectin and serial transvaginal ultrasound cervical length had low predictive accuracy for spontane
182                                          For cervical length of 20 mm or shorter, women in both group
183  Among women with spontaneous preterm birth, cervical length of 25 mm or less occurred in 35 of 439 (
184 ior spontaneous preterm birth but with short cervical length on transvaginal ultrasound, use of a cer
185 singleton pregnancies and short transvaginal cervical length, use of a cervical pessary, compared wit
186 ents of vaginal fetal fibronectin levels and cervical length.
187  no previous spontaneous preterm births, and cervical lengths of 25 mm or less at 18 weeks 0 days to
188 ted the association of ART with longitudinal cervical lesion outcomes.
189 ity on DCE MR images that was greater in the cervical lesion than in the myometrium was defined as ti
190  (6537 women living with HIV) and high grade cervical lesions (HSIL-CIN2+; 9288 women living with HIV
191 nds in human papillomavirus (HPV)-associated cervical lesions can provide an indication of vaccine im
192 creased detection of high-grade precancerous cervical lesions compared to cytology, in a population w
193         Our purpose was to measure trends in cervical lesions during 2008-2015 and to consider possib
194 s (hrHPV) testing to triage women with minor cervical lesions generates many referrals.
195            Studies of large numbers of early cervical lesions revealed frequent viral integration int
196 s also observed in normal and HPV-associated cervical lesions.
197 step after hrHPV testing in women with minor cervical lesions.
198 us blood flow, that is, in epidural veins at cervical level 3, uniquely demonstrated CSF and venous f
199 a 53-year-old man with a spinal cord injury (cervical level 4, American Spinal Injury Association Imp
200 e profile was observed in ex vivo culture of cervical lymph node cells and splenocytes, indicating th
201 hat BMI1(+) CSCs mediate invasive growth and cervical lymph node metastasis in a mouse model of HNSCC
202 tribution of peripheral activation events in cervical lymph nodes (CLN) to driving humoral immune res
203 s in the intestinal lamina propria (LPL) and cervical lymph nodes (CLN).
204 rew from 68% of lungs and 36% of spleens and cervical lymph nodes but fewer than 20% of axillary lymp
205 ntigen-loaded bone marrow DC migrate to deep cervical lymph nodes where they prime antigen-specific T
206 d spinal cord while wild type DC migrated to cervical lymph nodes.
207 focal lesion in the right parotid gland, and cervical lymphadenopathy.
208                                              Cervical mediastinoscopy was positive for carcinoma in l
209 ent uniquely affected gene expression in the cervical microenvironment of HPV-transgenic mice compare
210 he cervix and must swim through viscoelastic cervical mucus and other mucoid secretions to reach the
211 dysfunction, we assessed the permeability of cervical mucus from non-pregnant ovulating (n = 20) and
212 ss may alter the overall permeability of the cervical mucus plug, our findings suggest that the latte
213 m bacterial ascension across a dysfunctional cervical mucus plug.
214                          Patients had either cervical (n = 548) or intrathoracic (n = 2738) anastomos
215 er appreciation of the potential role of the cervical nerves, and the recognition of the crucial role
216 ion of tumor thickness and predicting occult cervical nodal metastasis.
217 own for early hominins, to date, no complete cervical or thoracic series has been recovered.
218 tranded DNA viruses that are responsible for cervical, oropharyngeal, and various genitourinary cance
219                           It is unclear if a cervical pessary can reduce the risk of spontaneous pret
220  length on transvaginal ultrasound, use of a cervical pessary would reduce the rate of spontaneous pr
221 short transvaginal cervical length, use of a cervical pessary, compared with no pessary use, resulted
222 in the L1 gene, to the histological grade of cervical precancer and cancer.
223 r viral genetic variation influences risk of cervical precancer and cancer.
224                       Incomplete excision of cervical precancer is associated with therapeutic failur
225 f the margins of the tissue excised to treat cervical precancer.
226 gical abnormalities, high-grade lesions, and cervical procedures related to HPV 31, 33, 45, 52, and 5
227 bers were found in the lamina propria of the cervical region of the vagina, where a higher number of
228 mechanisms and could contribute to premature cervical remodeling associated with PTB.
229 ms regulating preterm birth (PTB)-associated cervical remodeling remain unclear.
230 n-invasively investigate normal and impaired cervical remodeling.
231 -resistant dimethyl-PGE2 resulted in preterm cervical ripening and delivery in mice.
232 tely 10% of pregnancies undergo induction of cervical ripening and labor with prostaglandin (PG) E2 o
233 s, and 15-PGDH activators to prevent preterm cervical ripening and preterm birth.
234 nt implications for the control of premature cervical ripening and prevention of preterm birth in hum
235 ine contractions and are clinically used for cervical ripening during pregnancy.
236                   On the other hand, preterm cervical ripening in the second trimester predicts prete
237 o initiate or amplify low-dose PGE2-mediated cervical ripening or (ii) EP2 receptor antagonists, HDAC
238  in the E/P4 signaling ratio is critical for cervical ripening.
239 adiographic evidence of unusually protruding cervical root morphology in primary and/or secondary den
240 igh-risk human papillomavirus infection from cervical samples and 78.6% (95% CI: 69.4-87.7%) from ana
241 cocultures of early-passage cells from human cervical samples, with levels highest in cocultures of c
242 uman papillomavirus (HPV)-associated uterine cervical SCC, the third-leading cause of death by cancer
243 ervations reveal that humans with incomplete cervical SCI have an altered ability to modulate cortico
244 viduals with anatomically incomplete chronic cervical SCI responded to a startle stimulus, a test tha
245  females with and without incomplete chronic cervical SCI were tested.
246 program, receives mandatory reporting of all cervical screening (cytologic and HPV testing) and any c
247              After adjustment for changes in cervical screening across the period, reductions in the
248 ation of new technologies to enhance current cervical screening among unvaccinated women.
249 se vaccinated women who attended for routine cervical screening at age 20-21 years.
250  a dynamic transmission model, calibrated to cervical screening data from Italy, to estimate vaccinat
251 These data provide essential information for cervical screening guidelines and public health policy.
252 g in women who turn 25 in 2018 or later; and cervical screening in all ages.
253 les to match that in whites for vaccination; cervical screening in women who turn 25 in 2018 or later
254 imary human papillomavirus (HPV) testing for cervical screening increases detection of high-grade cer
255 a (CIN) trends when adjusting for changes in cervical screening practices that overlapped with HPV va
256  captures population-based estimates of both cervical screening prevalence and CIN, was used to compu
257 ine choice and models to shape the future of cervical screening programmes.
258                                    Increased cervical screening uptake in vaccinated women from ethni
259  effect of ethnic disparities in vaccine and cervical screening uptake on inequality in disease incid
260 m-specific IgG, but not IgA, was detected in cervical secretions of serum antibody-positive animals,
261 ase in the density of small blood vessels in cervical sections of HD cases.
262 luding that small ganglia were restricted to cervical segments, epithelial fibers were mainly present
263 nd clinicians were blinded to results unless cervical shortening less than 15 mm was identified.
264 urpose To determine if the obliteration of a cervical space, the paraspinal fat pad (PFP), can be use
265 nvenience sample of 516 residual exfoliative cervical specimens from the Kaiser Permanente Northern C
266 s into the contralesional gray matter of the cervical spinal cord administered 28 d after stroke indu
267  electrodes over motor cortex and the dorsal cervical spinal cord in rats; motor evoked potentials (M
268  preserved in humans with incomplete chronic cervical spinal cord injury (SCI).
269 ls in 15 individuals with chronic incomplete cervical spinal cord injury and 17 uninjured participant
270 ped a low-cost portable BMI for survivors of cervical spinal cord injury and investigated it as a mea
271 indings of an individual with traumatic high-cervical spinal cord injury who coordinated reaching and
272 People with chronic tetraplegia, due to high-cervical spinal cord injury, can regain limb movements t
273 n atrophy (whole brain and gray matter), and cervical spinal cord lesions (T2LV) and atrophy.
274 tissue bridges at the epicenter of traumatic cervical spinal cord lesions in 24 subacute tetraplegic
275  of nongenomic estrogen signaling within the cervical spinal cord to recover respiratory neuroplastic
276 that pairing stimulation of motor cortex and cervical spinal cord would strengthen motor responses th
277 n-like c-Fos immunoreactivity pattern in the cervical spinal cord.
278                         The impact of AIH on cervical spinal interneuron (C-IN) discharge and connect
279 rve growth and selective trochlear and first cervical spinal nerve guidance abnormalities.
280 d as a well-circumscribed fatty area between cervical spine and posterior muscles.
281 a, readers 2 and 3 reviewed in consensus the cervical spine CT (reference for fracture and luxation)
282  at computed tomography (CT) of an injury of cervical spine posterior ligamentous complex (PLC).
283       Then, in 85 patients with suspicion of cervical spine trauma following high-velocity trauma, re
284 rations in the motor cortex of patients with cervical spondylotic myelopathy (CSM) by using proton ma
285                                   Monitoring cervical structure and composition during pregnancy has
286 esulting in well-documented life-threatening cervical swelling and cyst-like bone formation.
287 ntiretroviral regimen, and genital ulcers or cervical tenderness.
288                                              Cervical tissues from older mice were more distensible t
289  with injuries of PLC in patients with spine cervical trauma.
290  identified in the expression profile of the cervical tumor microenvironment.
291                              Our analysis of cervical tumors from four separate data sets found a sig
292 y expressed in advanced stages of breast and cervical tumors.
293 n melanoma; nonmelanoma skin cancer; breast; cervical; uterine; ovarian; prostate; testicular; kidney
294 ully or partially concordant for 9 of the 15 cervical-vaginal samples, with 95.83% overall type-speci
295                   Efferent activation of the cervical vagus nerve (cVN) dampens systemic inflammatory
296 ally implement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selec
297 INTS: The evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynami
298 tralopithecus skeleton to preserve all seven cervical vertebrae and provides evidence for 12 thoracic
299 ocol population, the incidence of high-grade cervical, vulvar and vaginal disease related to HPV 31,
300 creening (cytologic and HPV testing) and any cervical, vulvar, and vaginal histopathological findings

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