ライフサイエンスコーパス: cervical cord

1 nia or optogenetic photoactivation of the C4 cervical cord.

2 l and lateral columns and grey matter of the cervical cord.

3 rsal column extending from the lumbar to the cervical cord.

4 injury to the craniocervical junction or the cervical cord.

5 sion load and the presence of atrophy of the cervical cord.

6 ns in the upper cervical cord, who underwent cervical cord (1) H-magnetic resonance spectroscopy.

7 of all labeled cells were located within the cervical cord, 18% in thoracic cord, and 19% in the lumb

8 tion of the corticospinal tract (CST) in the cervical cord above a traumatic lesion and explored its

9 s showed progressive degenerative changes in cervical cord and brain morphometry across the sensory s

10 DTI of the cervical cord and brain provided measurements of fractio

11 nificantly lower in both CSTs throughout the cervical cord and brain when compared with controls (p</

12 rest (ROI) included the bilateral CST in the cervical cord and brain.

13 om sporadic PAPT in having marked atrophy of cervical cord and brainstem with corticospinal signs but

14 ance spectroscopy and q-space imaging of the cervical cord and conventional brain and spinal magnetic

15 ing single-voxel (1)H-MR spectroscopy of the cervical cord and diffusion-based tractography of the ma

16 -like immunoreactivity in all laminae of the cervical cord and in laminae I and II and the ventral ho

17 expression in laminae I-II of the lumbar and cervical cord and in the rostral ventromedial medulla in

18 data provided information on cross-sectional cervical cord area and volumetric brain changes in 30 in

19 A smaller cervical cord area was associated with impaired upper li

20 inal cord gray and white matter areas, upper cervical cord area, and the ratio of gray matter to the

21 a, and the ratio of gray matter to the upper cervical cord area.

22 Cervical cord atrophy was also assessed by using an acti

23 In patients with MS, cervical cord atrophy was correlated with clinical disab

24 PAPT is associated with marked brainstem and cervical cord atrophy with corticospinal tract findings,

25 g syndrome that involves the optic nerve and cervical cord but differs pathologically from multiple s

26 Seven of the 366 patients had cervical cord contusions, four patients had ligamentous

27 ated with Ambulation Index, whereas only the cervical cord correlated with disease duration (p < 0.05

28 Upper cervical cord cross-sectional area (UCCA), brain and spi

29 global and regional brain volumes and upper cervical cord cross-sectional area that are highly repro

30 tures of patients with HSP who had brain and cervical cord damage were also investigated.

31 sion loads, age, sex, and disease duration), cervical cord GM areas had the strongest correlation wit

32 In multiple sclerosis (MS), upper cervical cord gray matter (GM) atrophy correlates more s

33 These unfolded cervical cord images were coregistered into a common sta

34 s was thin compared to HVs, whereas only the cervical cord in MS patients was thinner than in HVs (p

35 Reduced FA of the cervical cord in patients with SCI was associated with s

36 lanning, but its routine use for imaging the cervical cord in shaken, abused infants without clinical

37 of low signal intensity that compressed the cervical cord in six patients.

38 f the regional distribution of damage in the cervical cord is feasible and might improve our understa

39 l nucleus synapse in the brainstem and upper cervical cord is the most likely site of action for brai

40 Patients showed lower tNAA of the cervical cord, lower connectivity and lower fractional a

41 A significant difference in cervical cord mean fractional anisotropy (FA) was found

42 lesions in an MS cord are seen in the upper cervical cord, most of the pathology in HAM/TSP is seen

43 A greater number of cervical cord neurons responded to hypocretin than anoth

44 alities in the glutamatergic pathways in the cervical cord of early primary progressive multiple scle

45 rror corrected for multiple comparisons) and cervical cord (P < .001) in patients with HSP relative t

46 copy and diffusion-based tractography of the cervical cord provide measures that are sensitive to the

47 onal degeneration of the CST in the atrophic cervical cord, proximal to the site of injury, parallels

48 Fourteen patients at the onset of a cervical cord relapse with at least one lesion between C

49 jor spinal cord pathways, in patients with a cervical cord relapse, differed from controls and correl

50 ge occurring in this area in patients with a cervical cord relapse.

51 ord, but were 10 times more prevalent in the cervical cord than the lumbar cord.

52 sition or the trigeminal subnucleus caudalis-cervical cord (Vc/C1) junction region in the lower brain

53 The upper cervical cord volumes (r = -0.39, P < 0.01), but not the

54 red cortex that innervated the contralateral cervical cord was five times that of controls, and in th

55 lution magnetic resonance (MR) images of the cervical cord were acquired from 45 patients with RR MS,

56 istribution patterns on the two sides of the cervical cord were compared.

57 rexin 1 receptor protein in the ventral C3-5 cervical cord were statistically diminished in WNV-infec

58 ongitudinally extensive lesions in the upper cervical cord, who underwent cervical cord (1) H-magneti