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1 lear staining of beta-catenin in 18 cases of cervical dysplasia.
2 umerous HPV types, and frequently results in cervical dysplasia.
3 s also evidence for reduced gap junctions in cervical dysplasia.
4 may improve sensitivity in the detection of cervical dysplasia.
5 equired some form of destructive therapy for cervical dysplasia.
6 important early event in the development of cervical dysplasia.
7 ns targeting the vasculature in premalignant cervical dysplasia.
8 was also found in a subset of 10 high-grade cervical dysplasias.
9 human papillomavirus types and prevalence in cervical dysplasia and cancer in HIV-infected women sugg
11 on of E7 caused the regression of high-grade cervical dysplasia and established cervical tumors, indi
12 a potentially important marker of high-grade cervical dysplasia and squamous cell carcinoma (SCC).
13 F of most patients with untreated high-grade cervical dysplasia but disappeared if the dysplasia was
16 , chondrodysplasia punctata, coronal clefts, cervical dysplasia, congenital cataracts, profound postn
17 genic HPV infection predicted progression of cervical dysplasia from normal to abnormal SIL (HR, 2.8;
18 human papillomavirus (QHPV) vaccine against cervical dysplasia has not been estimated using populati
19 umcision protecting against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamyd
20 een and treat women for preinvasive disease, cervical dysplasia, is the key factor leading to the red
21 ubjects, at least 25% of HPV16(+) high-grade cervical dysplasia lesions undergo complete regression.
22 V) in the cervix, the proportion of cases of cervical dysplasia missed, or the false-negative rate, h
23 , and 7.4% of the methylprednisolone group), cervical dysplasia (seen in 11% of the cyclophosphamide
26 fectiveness (VE) of the QHPV vaccine against cervical dysplasia using data collected routinely in Man
27 shold of microvessel leakage associated with cervical dysplasia was <17 kDa and highlighted the poten
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