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1 lly patients with generalized, segmental, or cervical dystonia.
2 han sham stimulation at reducing symptoms of cervical dystonia.
3 pear not to be a common cause of adult-onset cervical dystonia.
4 ts, and finally discuss its putative role in cervical dystonia.
5 tation in a family with adult onset, primary cervical dystonia.
6 striatal cholinergic interneuron density in cervical dystonia.
7 brain regions in 13 subjects with idiopathic cervical dystonia.
8 ervation and/or change in the pattern of the cervical dystonia.
9 the DYT1 gene, as well as for patients with cervical dystonia.
10 halopathy (n = 4), ocular motor palsies (2), cervical dystonia (2), focal weakness (2), and facial pa
11 A case-control study of 67 patients with cervical dystonia and 67 of their age-matched unaffected
12 sease, one with myoclonic dystonia, two with cervical dystonia and five with primary generalized dyst
13 asians with familial or sporadic adult onset cervical dystonia and matching controls for sequence var
14 obustly associated with dystonic symptoms in cervical dystonia and may be a useful biomarker for adap
15 nths (myoclonic dystonia), 14 and 24 months (cervical dystonia) and 3-24 months (primary generalized
16 report three new phenotypes at presentation: cervical dystonia; autonomic dysfunction and peripheral
17 abismus, blepharospam, hemificial spasm, and cervical dystonia, because of the toxin's tropism for ne
18 patterns of head movements in patients with cervical dystonia can be predicted by deficits in a neur
19 thophysiology of arm tremor in patients with cervical dystonia (CD) and its relationship to other typ
20 of selective peripheral denervation (SPD) in cervical dystonia (CD) patients with primary or secondar
23 pathway impairment in patients with sporadic cervical dystonia, due to rare coding variation in the e
24 controlled trial, we recruited patients with cervical dystonia from centres in Germany, Norway, and A
27 a appears to increase risk of development of cervical dystonia in genetically predetermined individua
29 rge number of familial and sporadic cases of cervical dystonia led to the identification of a total o
30 ality in families with inherited adult-onset cervical dystonia; linkage to chromosome 18p has been de
31 15), benign essential blepharospasm (n = 9), cervical dystonia (n = 10) and in age-matched controls.
32 examining environmental exposure history in cervical dystonia patients and their similarly aged unaf
34 ctive antibodies against toxin from mice and cervical dystonia patients undergoing BoNT/A treatment r
36 tions in CIZ1 may cause adult onset, primary cervical dystonia, possibly by precipitating neurodevelo
37 ntrol, as well as a conceptual framework for cervical dystonia that departs considerably from current
38 Caucasian pedigree with adult onset, primary cervical dystonia to identify a cosegregating mutation.
39 es with selective peripheral denervation for cervical dystonia were retrospectively analysed concerni
41 to a novel pathophysiological explanation in cervical dystonia, which proposed that the abnormalities
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