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1 shes and from the ectocervix and vagina with cervicovaginal lavage.
2   HSV-2 DNA and HIV-1 RNA were quantified in cervicovaginal lavage.
3                                              Cervicovaginal lavage and plasma from 122 HIV-uninfected
4   Schistosoma PCR was done on urine, biopsy, cervicovaginal lavage, and genital mucosal surface speci
5                                    Gingival, cervicovaginal lavage, and plasma specimens were collect
6 ADCC mediated by antibodies present in sera, cervicovaginal lavages, and breast milk from HIV-1-infec
7 ysis of 16S ribosomal RNA gene sequencing of cervicovaginal lavage clustered each participant visit i
8                                              Cervicovaginal lavages collected from 19,512 women atten
9           Twenty-four couples were enrolled; cervicovaginal lavage (CVL) and tissue were collected 2
10 ducted to analyze the presence of HCV RNA in cervicovaginal lavage (CVL) fluid from 71 women (58 HCV/
11 ty (CMI) was evaluated for the first time in cervicovaginal lavage (CVL) fluid from RVVC patients.
12 sence of a heat-stable soluble factor in the cervicovaginal lavage (CVL) fluid of both HIV-infected a
13                                              Cervicovaginal lavage (CVL) fluid was evaluated for tota
14 ory cytokine concentrations were measured in cervicovaginal lavage (CVL) from 49 women 6, 17, 30, and
15 igned to determine the antiviral activity in cervicovaginal lavage (CVL) samples collected after intr
16 tic cells secreted TNF- alpha in response to cervicovaginal lavage (CVL) samples from women with BV.
17 ng into the genital tract, paired plasma and cervicovaginal lavage (CVL) samples were obtained from 1
18                                              Cervicovaginal lavage (CVL) samples were obtained from 2
19 nerella vaginalis, and Mycoplasma hominis in cervicovaginal lavage (CVL) samples were quantified by p
20 he Women's Interagency HIV Study contributed cervicovaginal lavage (CVL) samples.
21 pothesis-generating study, 17 women provided cervicovaginal lavage (CVL) specimens at baseline (all h
22 irus (CMV) was studied in blood, saliva, and cervicovaginal lavage (CVL) specimens from 33 HIV-1-infe
23 Abbott RealTime HIV-1 assay using plasma and cervicovaginal lavage (CVL) specimens.
24 formalin-fixed-tissue specimens collected by cervicovaginal lavage (CVL) within 90 days of each other
25 y, and inflammatory cells were quantified in cervicovaginal lavages (CVLs) of 24 women enrolled in th
26 in plasma, female reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabo
27  and innate resistance) was also detected in cervicovaginal lavage fluid from both species.
28 rrent quantified HIV-1 RNA concentrations in cervicovaginal lavage fluid in 301 women infected with t
29                                      Testing cervicovaginal lavage fluids for >40 HPV genotypes using
30 p160-specific IgA responses were detected in cervicovaginal lavage fluids in 6 of 13 HEPS CSWs but 0
31 nd prevalence of human papillomavirus DNA in cervicovaginal lavage fluids were all >50% and were 2-30
32  endocervical swabs were more sensitive than cervicovaginal lavage for HIV-1 RNA detection by PCR but
33 py and tests for human papillomavirus DNA in cervicovaginal lavage-for a median follow-up of 3.2 year
34 L-10 were measured prior to HIV infection in cervicovaginal lavages from 58 HIV seroconverters and 58
35  tryptophan, indole, and IFN-gamma levels in cervicovaginal lavages from women with either naturally
36  [RLU/PC]) using Hybrid Capture 2 testing of cervicovaginal lavages obtained at enrolment.
37 d in the plasma of 2 women and in at least 1 cervicovaginal lavage sample from all 6 women.
38                                              Cervicovaginal lavage samples (n = 19) were collected, c
39 s and proviral DNA in cervical, vaginal, and cervicovaginal lavage samples by polymerase chain reacti
40 + (GP+-PCR), for the detection of HPV DNA in cervicovaginal lavage samples from 208 women.
41 nerella vaginalis, and Mycoplasma hominis in cervicovaginal lavage samples were quantified by PCR.
42                         In an analysis of 95 cervicovaginal lavage samples, we found that 12 (12.6%)
43 5) ng/mL)] and ex vivo antiviral activity of cervicovaginal lavage samples.
44 ens from endocervical canal wick and most in cervicovaginal lavage samples.
45 t lifestyle and sexual behavior and obtained cervicovaginal-lavage samples for the detection of HPV D
46                                              Cervicovaginal lavage sequencing (n = 109) resulted in a
47 ty-two cytokines were measured by Luminex in cervicovaginal lavage specimens at enrollment.
48 al neoplasia grade 3 and cancer (CIN3+) with cervicovaginal lavage specimens collected at enrollment
49                                              Cervicovaginal lavage specimens from enrollment were tes
50 NA testing were conducted annually in serial cervicovaginal lavage specimens obtained over 8-10 years
51 land (of whom 184 were HIV+), provided 1,426 cervicovaginal lavage specimens tested for HPV DNA by a
52                                    Antenatal cervicovaginal lavage specimens were assessed for HIV-1
53                                              Cervicovaginal lavage specimens were collected from each
54                                         When cervicovaginal lavage specimens, the reverse line-blot a
55 09/11 system was evaluated with a set of 262 cervicovaginal lavage specimens.
56  were used to characterize HPV-16-containing cervicovaginal lavage specimens.
57  wicks should be considered as an adjunct to cervicovaginal lavage, to improve the sensitivity and pr
58 munoglobulins G and A and some antibodies in cervicovaginal lavages varied with the stages of the men
59 w ProTalpha variants from CD8(+) T cells and cervicovaginal lavage with potent anti-HIV-1 activity.
60 blot analysis, secreted HD-5 was detected in cervicovaginal lavages, with the highest concentrations

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