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1 amycin delays tumor recurrence following the cessation of treatment.
2 ncerning for progression of disease prompted cessation of treatment.
3 n effects tend to wane 6-12 months after the cessation of treatment.
4 sed during SIT but started to decrease after cessation of treatment.
5 nist mifepristone, evident 2 weeks after the cessation of treatment.
6 ormance, which was evident 7 weeks after the cessation of treatment.
7 ned virologic response at 12 weeks after the cessation of treatment.
8 Neutrophil counts returned to normal after cessation of treatment.
9 n was observed, with effects sustained after cessation of treatment.
10 ot understood, its effects persist after the cessation of treatment.
11 siently during treatment but recovered after cessation of treatment.
12 growth, however, rapidly advanced following cessation of treatment.
13 ertebral BMD during treatment and 6 mo after cessation of treatment.
14 raft during the rejection observed following cessation of treatment.
15 ist or become enhanced by 48 h following the cessation of treatment.
16 normal and rebound hyperphagia occurring on cessation of treatment.
17 nograft tumours without recurrence after the cessation of treatment.
18 e changes persisted for several months after cessation of treatment.
19 have a sustained clinical response after the cessation of treatment.
20 well tolerated and only occasionally require cessation of treatment.
21 of elevated ALT levels within 3 years after cessation of treatment.
22 sion per RECIST v1.1 might prevent premature cessation of treatment.
23 uated every 2-4 weeks through 2 months after cessation of treatment.
24 n chronic treatment but reverses slowly upon cessation of treatment.
25 nsgenic mice as evident within 21 days after cessation of treatment.
26 ghtly subnormal almost immediately after the cessation of treatment (50 days of age) with no further
29 ever, the cTEC compartment regenerated after cessation of treatment, accompanied by the restoration o
30 functional adipocytes can be recovered upon cessation of treatment, allowing the study of adipogenes
31 prognosis, but relapses occur, usually after cessation of treatment and occasionally many years later
33 rapeutic effects were not reversed following cessation of treatment, and IL-13 anti-serum treatment d
36 the emergence of new local adipocytes, upon cessation of treatment, enables the ductal epithelium to
40 a condition that can persist long after the cessation of treatment in as many as 75% of survivors.
41 stored almost to control levels 4 days after cessation of treatment in vivo and fully normalised by 4
44 ndomized trials rarely are utilized to study cessation of treatment, most of what we know about the e
45 V617F) cells persisted and MPN recurred upon cessation of treatment, suggesting that JAK2 inhibitors
48 GP returned to control level at 3 days after cessation of treatment with either drug but did not retu
50 ll as the time course for its recovery after cessation of treatment with the SSRI sertraline were inv
51 rved between the time to disease flare after cessation of treatment with TNFalpha antagonists and the
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