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1 rval change, in milliseconds, divided by the change in blood pressure).
2 There was no significant change in blood pressure.
3 tic output occurred despite an insignificant change in blood pressure.
4 1.60 in the second group (six dogs) with no change in blood pressure.
5 e A/B status was not associated with 10-year change in blood pressure.
6 in goats with persistent AF, independent of changes in blood pressure.
7 cal fluctuations, such as those arising from changes in blood pressure.
8 ides (NPs) control natriuresis and normalize changes in blood pressure.
9 s maintenance of sinus rhythm and to monitor changes in blood pressure.
10 endothelial function, even in the absence of changes in blood pressure.
11 .30; 95% CI, 0.12 to 0.73), independently of changes in blood pressure.
12 notype had no effect on pioglitazone-induced changes in blood pressure.
13 -induced cardiac hypertrophy, independent of changes in blood pressure.
14 eft ventricular ejection fraction but not to changes in blood pressure.
15 The benefit exceeded that attributable to changes in blood pressure.
16 hether these are accompanied by simultaneous changes in blood pressure.
17 best for smoothing respiratory (mechanical) changes in blood pressure.
18 e decline in MI incidence (23%), followed by changes in blood pressure (13%), HDL cholesterol (12%),
19 nt blood pressure level, the previous 2-year change in blood pressure adds important predictive infor
22 changes in drinking status predicted 5-year changes in blood pressure among the 652 women and 318 me
23 AT4R-mediated effects were independent from changes in blood pressure, amyloidosis, and oxidative st
24 ects of the potassium source and dose on the change in blood pressure and augmentation index (AIx) we
25 cm visual analog sedation scale, the percent change in blood pressure and heart rate from the previou
26 majority of studies reported no significant change in blood pressure and mixed results were found re
30 he association between levels of 25(OH)D and changes in blood pressure and incident hypertension in 4
31 diabetes mellitus and metabolic syndrome and changes in blood pressure and lung function were similar
32 impact upon microvascular reactivity without changes in blood pressure and suggest that a vasoconstri
33 serum levels of 25(OH)D were not related to changes in blood pressure, and evidence for an associati
35 ertensive individuals to regulate short-term changes in blood pressure arise as emergent properties o
36 w studies have examined how the longitudinal change in blood pressure associated with aging differs a
39 ts were followed for acute rejection and for changes in blood pressure, body weight, and serum creati
42 on the transfer function between spontaneous changes in blood pressure (BP) and cerebral blood flow v
43 studies, but their relations to longitudinal changes in blood pressure (BP) and hypertension incidenc
45 cidence of hypertension and in aging-related changes in blood pressure by neighborhood and individual
46 The arterial baroreflex acts to buffer acute changes in blood pressure by reciprocal modulation of sy
49 vivo have cardiac hypertrophy independent of changes in blood pressure, corroborating earlier studies
51 nary creatinine-adjusted arsenic with annual change in blood pressure during follow-up (median, 6.7 y
53 resistance were assessed in the forearm from changes in blood pressure (Finapres) divided by brachial
54 stance responses in the brachial artery from changes in blood pressure (Finapres) divided by brachial
57 heart rate were investigated by studying the changes in blood pressure, heart rate, and neurotransmis
59 n mild, some patients have experienced brief changes in blood pressure, heart rate, or respiratory ra
60 n with hemoperfusion through a pig liver for changes in blood pressure, hematocrit, platelets, and NA
61 as not observably toxic, with no significant changes in blood pressure, hepatic artery blood flow, se
63 arsenic exposure in relation to longitudinal change in blood pressure in 10,853 participants in the H
64 e did a randomised parallel trial to compare change in blood pressure in 118 men with obstructive sle
65 We attempted to characterize age-related changes in blood pressure in both normotensive and untre
67 measured by the response in heart rate to a change in blood pressure induced by phenylephrine i.v. w
68 intained a normal electroretinogram, with no changes in blood pressure, intraocular pressure, or hear
69 value during a specific time period) and the changes in blood pressures levels that occur, with sleep
73 There were no significant differences in the change in blood pressure or AIx with the treatment sourc
75 ty indices may not relate in the same way to changes in blood pressure or blood lipid concentrations.
76 mption was not significantly associated with changes in blood pressure or hypertension incidence (sys
79 or 12 wk was not associated with significant changes in blood pressure or platelet function compared
84 (+/-)) plays no persistent role: the in vivo changes in blood pressure reflect the in vitro changes i
85 fusion) or sustained (steady-state infusion) changes in blood pressure regardless of the thermal cond
88 to be responsible for the treatment effect, changes in blood pressure should depend on the dose of i
89 th the 10-year (1988) follow-up there was no change in blood pressure standard deviation scores (SDS)
90 ealth-based intervention did not result in a change in blood pressure that differed from usual care,
92 roducing a bradycardia of >50 bpm, without a change in blood pressure, using D,L homocysteic acid (DL
93 ociation of the Type A behavior pattern with change in blood pressure was examined in a multiethnic s
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