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1 be M. abscessus and 29 were identified as M. chelonae.
2 ily identify isolates of M. abscessus and M. chelonae.
3 submitted as M. abscessus was found to be M. chelonae.
4 an incorrect identification as Mycobacterium chelonae.
5 ycobacterium abscessus but not Mycobacterium chelonae.
6 r distinguishing between M. abscessus and M. chelonae.
7 en Mycobacterium abscessus and Mycobacterium chelonae.
8 mycobacteria including M. llatzerense and M. chelonae.
9 Mycobacterium sp. most closely resembling M. chelonae.
11 against clarithromycin; M. abscessus and M. chelonae against the aminoglycosides; and all three spec
16 acin, clarithromycin, tobramycin (only in M. chelonae), and cefoxitin (only in M. abscessus) was show
17 complex (MCC), including M. immunogenum, M. chelonae, and M. abscessus, have been associated with no
18 test species, Mycobacterium immunogenum, M. chelonae, and M. abscessus, showed various susceptibilit
19 ended incubation may not be necessary for M. chelonae, and the erm(41) genotype is a useful adjunct f
20 Mycobacterium abscessus and Mycobacterium chelonae are two closely related species that are often
23 ly, uncapped LAM purified from Mycobacterium chelonae did not induce IL-12 secretion or apoptosis.
24 not harbor functional erm genes and, like M. chelonae, do not require extended clarithromycin suscept
25 . smegmatis, primarily secrete Ndk, while M. chelonae does not appear to secrete either of these two
26 istance and that isolates of M. abscessus/M. chelonae from CF patients are more likely than those fro
31 M. avium, M. intracellulare, M. kansasii, M. chelonae group, M. gordonae, M. xenopi, and M. scrofulac
34 case of disseminated cutaneous Mycobacterium chelonae infection in a patient with head and neck cance
37 nts, showed abnormalities in all 17, with M. chelonae isolated from 14 and confirmed by means of DNA
38 41) sequencing of 285 M. abscessus and 45 M. chelonae isolates was compared to 14-day susceptibility;
39 bacterium abscessus, and three Mycobacterium chelonae isolates) were tested against amikacin, cefoxit
40 al of 82 isolates (58 M. abscessus and 24 M. chelonae isolates) were tested blindly against 15 antimi
41 e of a detectable erm gene in isolates of M. chelonae, M. senegalense, and M. peregrinum and a nonfun
42 rRNA gene sequencing (26 isolates of the M. chelonae-M. abscessus complex and 64 remaining isolates,
43 ex (MTC), the M. avium complex (MAC), the M. chelonae-M. abscessus group (MCAG), the M. fortuitum gro
44 m isolates previously identified as being M. chelonae/M. abscessus and identified M. massiliense from
45 that is indistinguishable from Mycobacterium chelonae/M. abscessus by partial 16S rRNA gene sequencin
46 matis, Mycobacterium gordonae, Mycobacterium chelonae, Mycobacterium fortuitum, and Mycobacterium scr
49 outbreak of tattoo-associated Mycobacterium chelonae skin and soft-tissue infections in Rochester, N
50 ify 75 isolates as either M. abscessus or M. chelonae that were originally submitted for drug suscept
51 Eight isolates originally submitted as M. chelonae were identified as M. abscessus, and one isolat
52 or =16 microg/ml), while all isolates of M. chelonae were susceptible to this drug (MIC of < or = 4
53 terium abscessus, and three of Mycobacterium chelonae) were tested against amikacin, cefoxitin, cipro
54 Another 2 skin specimens grew Mycobacterium chelonae, which also grew from a bottle of graywash ink
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