ライフサイエンスコーパス: chemical agent

1 25(OH)D in the therapeutic use against these chemical agents.

2 or under the action of various physical and chemical agents.

3 ks, it is reversible and can be modulated by chemical agents.

4 o identify the macromolecular targets of new chemical agents.

5 ave so far been identified are determined by chemical agents.

6 It is stable against a variety of chemical agents.

7 nmentally benign production of key synthetic chemical agents.

8 resistant to drought and other physical and chemical agents.

9 ting the physiologically relevant targets of chemical agents.

10 robust cell wall that is impermeable to many chemical agents.

11 e (+1.25 V) to release precise quantities of chemical agents.

12 for the metabolism of nutrients, drugs, and chemical agents.

13 y disrupting the partnering with peptide and chemical agents.

14 in their biochemical optima and responses to chemical agents.

15 s frequently damaged by various physical and chemical agents.

16 a wide spectrum of biological, physical and chemical agents.

17 teins are extremely stable to heat, acid and chemical agents.

18 attle and chickens, and also to carcinogenic chemical agents.

19 y than wild-type controls in response to the chemical agents.

20 e the potential health impacts of individual chemical agents?

21 of IFI16 reduced lytic gene induction by the chemical agent 12-O-tetradecoylphorbol-13-acetate (TPA).

22 Here we identify the first chemical agent able to act upstream of the proteasome to

23 mechanisms have been proposed: movement of a chemical agent and a pressure wave through the vasculatu

24 ain sequences important for gene response to chemical agents and fungal attack.

25 it a target for the rational design of novel chemical agents and genetic modifications that improve p

26 macronucleus in response to DSBs induced by chemical agents and in the micronucleus during prophase

27 rium appears to be relatively insensitive to chemical agents and inflammatory mediators, in contrast

28 major DNA repair pathway for SSB induced by chemical agents and ionizing radiation, we initially ass

29 ion was measured after cells were exposed to chemical agents and radiation and after HO endonuclease

30 in sampling hundreds of chemicals, including chemical agents and their signatures, pharmaceutics, met

31 rrent methods require proteolytic enzymes or chemical agents and typically a second reagent to discon

32 nduced by endonucleases, ionizing radiation, chemical agents, and mechanical forces or by replication

33 P expression sensitizes cells to a number of chemical agents, and mutations at predicted channel-faci

34 ces obviate bacterial resistance common with chemical agents, and therefore a robust and stable means

35 lucidate the roles of shear stress, specific chemical agents, and thermal fluctuations in cytoskeleto

36 aptation under the effect of physical and/or chemical agents, and thus function as adaptive polymers

37 umors by inducing the viral lytic cycle with chemical agents are hindered by inefficient responses to

38 city of 97%) in assessing whether particular chemical agents are irritating or not for human skin.

39 These chemical agents are known to confer protection on heart

40 ne that does not require the presence of the chemical agent before or during UVR exposure.

41 dged to destroy approximately 25,000 tons of chemical agents by the end of the decade.

42 esult if, using this accumulating knowledge, chemical agents can be developed that can enhance repair

43 Certain chemical agents can force some tumor cells to resume the

44 However, DNA structures bound to chemical agents cannot be PCR-amplified, and therefore a

45 ied to CNVs monitoring in cells exposed to a chemical agent capable of deletion induction, such as ci

46 tudy, we have demonstrated that a variety of chemical agents capable of denaturing or dissociating pr

47 y neurons grown in culture to the excitatory chemical agent capsaicin was examined.

48 control elements, and a unique modulation by chemical agents, cytokines, and serum-factors.

49 he wasabi receptor) is a detector of noxious chemical agents encountered in our environment or produc

50 ould facilitate development of site-directed chemical agents for bioimaging or therapeutic applicatio

51 our newly discovered p18SMIs represent novel chemical agents for murine and human HSCs ex vivo expans

52 Use of chemical agents for scar homogenization represents an al

53 -renewal and may help develop more effective chemical agents for therapeutic expansion of HSC.

54 Metabolites and certain chemical agents (for example methyl methanesulfonate) ca

55 uses extreme sensitivity to the [PSI]-curing chemical agent guanidine hydrochloride.

56 l residues of proteins with a broad range of chemical agents has been proposed to be dependent on the

57 on of these metal-peptide interactions using chemical agents holds considerable promise as a therapeu

58 sis of mass flow in the xylem transporting a chemical agent, however, is able to reproduce experiment

59 threshold VC exposure and the role of other chemical agents in TASH are as yet unknown.

60 These questions, as well as how chemical agents, including therapeutic substances, might

61 As the public pressure to limit the use of chemical agents increases, the control of thinning becom

62 al route for a "quick fix." Thus, the use of chemical agents is on the rise.

63 Conditions and chemical agents known to activate ER stress response (ER

64 ims from severe sunburn or exposure to other chemical agents known to trigger the p38 pathway.

65 wn to impart cellular resistance to multiple chemical agents, many of which are commonly used in canc

66 d previous research has suggested that these chemical agents may disrupt circulating levels of total

67 estriction enzyme Pvu II or the DNA-damaging chemical agents methyl methanesulfonate (MMS) or 4-nitro

68 he combined actions of ricin holotoxin and a chemical agent, N,N'-dimethylethylenediamine.

69 Sulfur mustard is a chemical agent of high military and terroristic signific

70 data, could be used to predict the effect of chemical agents on mobility, adhesion, and proliferation

71 ly, the reduction of GO has relied on either chemical agents or high temperature treatment.

72 The blockade of NF-kappaB activation via chemical agents or the overexpression of the mutant form

73 noflagellates is evoked by a factor (i.e., a chemical agent) present in a homogenate of host tissue.

74 ently alert the cell to an array of reactive chemical agents, regardless of their structure.

75 The increase in excitability is caused by chemical agents released at the site of injury.

76 High resolution mapping with chemical agents selective for non-B DNA provides evidenc

77 elp repair damage to DNA caused by exogenous chemical agents such as chloroacetaldehyde.

78 tiate in vivo by treatment with a variety of chemical agents such as N, N-hexamethylene bisacetamide

79 concept of enhancing antibody sensitivity by chemical agents that affect the structural stability of

80 A number of detergents and chemical agents that are capable of breaking ionic and h

81 s susceptible to damage by a wide variety of chemical agents that are generated either as byproducts

82 duced by ultraviolet irradiation and various chemical agents that cause bulky adducts.

83 he development of aneuploidy when exposed to chemical agents that disrupt the mitotic spindle and pre

84 High-throughput screening for chemical agents that exert greater inhibitory effects ag

85 Consequently, there is a need for chemical agents that increase DNA damage by ionizing rad

86 Several environmental conditions and chemical agents that influence the expression of VEGF ca

87 Chemical agents that inhibit infected cells from enterin

88 t can therefore be used for the screening of chemical agents that may have gamma-globin gene inducibi

89 nting challenge because of the wide array of chemical agents that must be screened.

90 modulus, and resistance to heat, flame, and chemical agents that normally degrade conventional macro

91 e in vitro pharmacological evaluation of new chemical agents that target bacterial transcription.

92 The focus of this Perspective is on chemical agents that target the most common mechanisms o

93 ed for targeted delivery of radioisotopes or chemical agents to diseased tissues.

94 Moreover, our results suggest that designing chemical agents to disrupt Rhl-Pqs crosstalk could be an

95 lar ion concentrations, and the screening of chemical agents to identify molecules targeting ion tran

96 is the use of specific natural or synthetic chemical agents to reverse, suppress, or prevent progres

97 pyelonephritis by delivering bacteriostatic chemical agents to the lower urinary system.

98 with the modes of application of anti-plaque chemical agents to their interdental and subgingival sit

99 throughput approaches evaluating toxicity of chemical agents toward bacteria typically rely on optica

100 We therefore suggest that chemical agents transported by mass flow within the xyle

101 ated release were dependent upon the type of chemical agent used to evoke the release.

102 The primary chemical agents used for controlling mosquitoes are inse

103 bon monoxide-releasing molecules (CORMs) are chemical agents used to administer CO as an endogenous,

104 As representative chemical agents, we selected a chemotherapeutic drug (ci

105 roblem associated with CF and to identifying chemical agents, which correct this problem.

106 Unlike chemical agents, which typically lead to violent disease

107 biological targets or signaling pathways and chemical agents, with a focus on small molecules, to ach