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1 on using sequence polymorphisms induced by a chemical mutagen.
2 increased to within an order of magnitude of chemical mutagens.
3 xposed to ionizing radiation and a number of chemical mutagens.
4 g the utility of non-null alleles created by chemical mutagens.
5 including via the handling and metabolism of chemical mutagens.
6 erfering particles, (ii) the presence of the chemical mutagen 5-fluorouracil (5-FU), or (iii) an incr
8 op skin lesions following treatment with the chemical mutagen 9,10-dimethyl-1,2-benzanthracene, or th
9 or investigating the effects of physical and chemical mutagens and for exploring the potential of mut
10 hotriester (PTE) lesions that are induced by chemical mutagens and refractory to DNA repair have not
11 f mutational changes can be used to identify chemical mutagens and to explore the molecular mechanism
15 ethal alleles created by either radiation or chemical mutagens (Ednrb27Pub, Ednrb17FrS, Ednrb1Chlc, a
21 unodeficiency virus type 1 (HIV-1) by use of chemical mutagens [i.e., 5-azacytidine (5-AZC)] as well
22 oses of alpha radiation, gamma radiation, or chemical mutagens in the presence and absence of caffein
24 , the killing of a microbial pathogen with a chemical mutagen, is a potential broad-spectrum antivira
25 reen of recessive mutations generated by the chemical mutagen n-ethyl-n-nitrosourea (ENU) mapped a ne
27 ations was isolated after treatment with the chemical mutagen N-methyl-N'-nitro-N-nitrosoguanidine.
30 in wild-type E. coli in <24 h, outperforming chemical mutagens, ultraviolet light and the mutator str
31 iently by densely ionizing radiation than by chemical mutagens, x-rays, or endogenous aging processes
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