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1  myocytes and fibroblasts induced by physico-chemical stimulation.
2 ies of these nanoassemblies in response to a chemical stimulation.
3 s elegans animals under precise microfluidic chemical stimulation.
4 cell death in response to virus infection or chemical stimulation.
5 ternally applied mechanical, electrical, and chemical stimulation.
6 cal stimulation and all responded to noxious chemical stimulation.
7 ked in experimental animals by electrical or chemical stimulation.
8 docrine cells respond to both mechanical and chemical stimulation.
9 cted from simple occlusion between light and chemical stimulation.
10 mical receptors responsive to mechanical and chemical stimulation.
11 d for sensitivity to mechanical, thermal and chemical stimulation.
12 somatic afferents to mechanical, thermal and chemical stimulation.
13  adjacent to individual mast cells following chemical stimulation.
14           Also striking was the finding that chemical stimulation (50 mm Glu) of the vBNST yielded lo
15                  Conversely, the response to chemical stimulation alone (based on physiological CO2/H
16 ic multifunctional device that is capable of chemical stimulation and electrical sensing at the same
17 ry effects on dorsal horn neurones evoked by chemical stimulation at identified pressor and depressor
18                                         This chemical stimulation caused consistent excitatory cardio
19 m were necessary for tears evoked by noxious chemical stimulation (CO2 pulses) or drying of the ocula
20 brain region, such as lesion, electrical and chemical stimulation, electrophysiology and in vivo micr
21 plate, tail flick) and to persistent noxious chemical stimulation (formalin) and (2) the antinocicept
22  matrix and when presented with a controlled chemical stimulation from the artificial vasculature, th
23 tivation of LC neurons through electrical or chemical stimulation has also led to important insights,
24  Class 2 neurones were strongly inhibited by chemical stimulation in either sector of the PAG.
25 s throughout the spinal dorsal horn, noxious chemical stimulation in the normal rat only induces inte
26 ation is consistent with a potential role of chemical stimulation in triggering dyspeptic symptoms.
27 ding facilitation produced by electrical- or chemical-stimulation in the NGC and NGCalpha also signif
28                                Electrical or chemical stimulation increased AbetaO synaptic localizat
29                    We observed that repeated chemical stimulation led to a reversible reduction in th
30 esponding to mechanical, noxious thermal and chemical stimulation; mechanothermal nociceptors respond
31                  In addition, electrical and chemical stimulation of A7 neurons produces antinocicept
32                                              Chemical stimulation of cAMP production by Mtb within ma
33 icrom) and deeper thoracic spinal neurons to chemical stimulation of cardiac afferents and effects of
34                          Results showed that chemical stimulation of cardiac afferents excited superf
35 , both spontaneous activity and responses to chemical stimulation of cardiac afferents significantly
36    This study examined whether site-directed chemical stimulation of DMH/PeF neurons evoked changes i
37                                              Chemical stimulation of DMH/PeF neurons evoked significa
38                                              Chemical stimulation of DMH/PeF neurons evokes substanti
39               The demonstration of localized chemical stimulation of excitable cells illustrates the
40 ve examined the influences of electrical and chemical stimulation of Gi and DPGi inhibitory sites on
41 bition of glycolysis decreased SSC activity, chemical stimulation of glycolysis or transfection of ac
42  hypoxia-inducible factor (HIF-1alpha) after chemical stimulation of human pancreatic tumor cells enc
43 iation of central sensitization triggered by chemical stimulation of meningeal nociceptors.
44                          Focal electrical or chemical stimulation of MPO in anesthetized rats induced
45 asured cardiovascular responses to localized chemical stimulation of neurons in area infraradiata of
46                                              Chemical stimulation of nonischemic ipsilateral cortex b
47 been considered to result from mechanical or chemical stimulation of pain-sensitive structures of the
48 characterize cardiovascular responses to the chemical stimulation of sites located in the medullary l
49 t to caudal regions of the PBN responsive to chemical stimulation of the anterior tongue as well as m
50                           In the middle LPBN chemical stimulation of the dorsal PAG selectively incre
51                        Tachycardia evoked by chemical stimulation of the dorsomedial hypothalamus can
52 displayed increased nociceptive responses to chemical stimulation of the face and hyperalgesic respon
53 The mechanism of cardiovascular responses to chemical stimulation of the hypothalamic arcuate nucleus
54                                              Chemical stimulation of the insula using L-glutamate was
55 ransiently abolishes behavioral responses to chemical stimulation of the intestine.
56                                              Chemical stimulation of the lateral or ventrolateral col
57                                              Chemical stimulation of the lateral PAG produces hyperte
58 acterized the effects of both electrical and chemical stimulation of the LH on 36 DMNV and 14 NST neu
59                           We now report that chemical stimulation of the MeA enhanced levels of extra
60                                              Chemical stimulation of the MPO excited 17/97 cells and
61  responses to electrical, mechanical, and to chemical stimulation of the muscle.
62 r the increase in sympathetic tone caused by chemical stimulation of the nasal passages with ammonia
63                                              Chemical stimulation of the PVN by unilateral microinjec
64 ervated rats: 1) tachycardia elicited by the chemical stimulation of the PVN was mediated via both in
65 halamic activity on Up states, electrical or chemical stimulation of the thalamus triggered cortical
66 -, or Q-best on the basis of its response to chemical stimulation of the tongue.
67                            However, stronger chemical stimulation of the vBNST (100 mm Glu) completel
68 tivation of DA neuron firing, the effects of chemical stimulation of the vSub on ventral tegmental ar
69                                              Chemical stimulation of their dural receptive fields wit
70 , cAMP, or MAPK pathway activation by either chemical stimulation or cotransfection of active cascade
71 threat and is also elicited by electrical or chemical stimulation over the rostro-caudal extent of th
72 , for the first time, that nerve impulses or chemical stimulation promote Ca2+ entry into PSNTs, incl
73         Neuronal responses to electrical and chemical stimulation showed specific firing patterns of
74             Yet, no subretinal electrical or chemical stimulation study has stimulated the OFF and ON
75  afferent sensitivity to both mechanical and chemical stimulations, suggesting that somatostatin play
76                        Direct mechanical and chemical stimulation to peripheral nociceptors, peripher
77 mplants capable of delivering electrical and chemical stimulation to targeted regions of the central
78 energy levels for mechanical stimulation and chemical stimulation were comparable.
79 t, chorda tympani nerve responses to lingual chemical stimulation were maintained at 10 d but were el
80 e responses of thermally-sensitive fibers to chemical stimulation were modest at best with an absence
81 The CBF effects evoked from DPAG and VPAG by chemical stimulation were preserved in spinalized rats s
82 nteract with cocultured cells by physical or chemical stimulation, which significantly mediate their
83 itization in dorsal horn neurons produced by chemical stimulation with capsaicin.
84 l stimulus (up to 265 +/- 42%, P = 0.022) or chemical stimulation with formyl-methylleucylphenylalani
85 menon have been limited to digital (or step) chemical stimulation with little control over the tempor
86             Furthermore, RSR was enhanced by chemical stimulation with potassium chloride.
87  PAG comparably elevated AP and CBF, whereas chemical stimulation with the D,L-homocysteine produced

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