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   1  myocytes and fibroblasts induced by physico-chemical stimulation.                                   
     2 ies of these nanoassemblies in response to a chemical stimulation.                                   
     3 s elegans animals under precise microfluidic chemical stimulation.                                   
     4 cell death in response to virus infection or chemical stimulation.                                   
     5 ternally applied mechanical, electrical, and chemical stimulation.                                   
     6 cal stimulation and all responded to noxious chemical stimulation.                                   
     7 ked in experimental animals by electrical or chemical stimulation.                                   
     8 docrine cells respond to both mechanical and chemical stimulation.                                   
     9 cted from simple occlusion between light and chemical stimulation.                                   
    10 mical receptors responsive to mechanical and chemical stimulation.                                   
    11 d for sensitivity to mechanical, thermal and chemical stimulation.                                   
    12 somatic afferents to mechanical, thermal and chemical stimulation.                                   
    13  adjacent to individual mast cells following chemical stimulation.                                   
  
  
    16 ic multifunctional device that is capable of chemical stimulation and electrical sensing at the same 
    17 ry effects on dorsal horn neurones evoked by chemical stimulation at identified pressor and depressor
  
    19 m were necessary for tears evoked by noxious chemical stimulation (CO2 pulses) or drying of the ocula
    20 brain region, such as lesion, electrical and chemical stimulation, electrophysiology and in vivo micr
    21 plate, tail flick) and to persistent noxious chemical stimulation (formalin) and (2) the antinocicept
    22  matrix and when presented with a controlled chemical stimulation from the artificial vasculature, th
    23 tivation of LC neurons through electrical or chemical stimulation has also led to important insights,
  
    25 s throughout the spinal dorsal horn, noxious chemical stimulation in the normal rat only induces inte
    26 ation is consistent with a potential role of chemical stimulation in triggering dyspeptic symptoms.  
    27 ding facilitation produced by electrical- or chemical-stimulation in the NGC and NGCalpha also signif
  
  
    30 esponding to mechanical, noxious thermal and chemical stimulation; mechanothermal nociceptors respond
  
  
    33 icrom) and deeper thoracic spinal neurons to chemical stimulation of cardiac afferents and effects of
  
    35 , both spontaneous activity and responses to chemical stimulation of cardiac afferents significantly 
    36    This study examined whether site-directed chemical stimulation of DMH/PeF neurons evoked changes i
  
  
  
    40 ve examined the influences of electrical and chemical stimulation of Gi and DPGi inhibitory sites on 
    41 bition of glycolysis decreased SSC activity, chemical stimulation of glycolysis or transfection of ac
    42  hypoxia-inducible factor (HIF-1alpha) after chemical stimulation of human pancreatic tumor cells enc
  
  
    45 asured cardiovascular responses to localized chemical stimulation of neurons in area infraradiata of 
  
    47 been considered to result from mechanical or chemical stimulation of pain-sensitive structures of the
    48 characterize cardiovascular responses to the chemical stimulation of sites located in the medullary l
    49 t to caudal regions of the PBN responsive to chemical stimulation of the anterior tongue as well as m
  
  
    52 displayed increased nociceptive responses to chemical stimulation of the face and hyperalgesic respon
    53 The mechanism of cardiovascular responses to chemical stimulation of the hypothalamic arcuate nucleus
  
  
  
  
    58 acterized the effects of both electrical and chemical stimulation of the LH on 36 DMNV and 14 NST neu
  
  
  
    62 r the increase in sympathetic tone caused by chemical stimulation of the nasal passages with ammonia 
  
    64 ervated rats: 1) tachycardia elicited by the chemical stimulation of the PVN was mediated via both in
    65 halamic activity on Up states, electrical or chemical stimulation of the thalamus triggered cortical 
  
  
    68 tivation of DA neuron firing, the effects of chemical stimulation of the vSub on ventral tegmental ar
  
    70 , cAMP, or MAPK pathway activation by either chemical stimulation or cotransfection of active cascade
    71 threat and is also elicited by electrical or chemical stimulation over the rostro-caudal extent of th
    72 , for the first time, that nerve impulses or chemical stimulation promote Ca2+ entry into PSNTs, incl
  
  
    75  afferent sensitivity to both mechanical and chemical stimulations, suggesting that somatostatin play
  
    77 mplants capable of delivering electrical and chemical stimulation to targeted regions of the central 
  
    79 t, chorda tympani nerve responses to lingual chemical stimulation were maintained at 10 d but were el
    80 e responses of thermally-sensitive fibers to chemical stimulation were modest at best with an absence
    81 The CBF effects evoked from DPAG and VPAG by chemical stimulation were preserved in spinalized rats s
    82 nteract with cocultured cells by physical or chemical stimulation, which significantly mediate their 
  
    84 l stimulus (up to 265 +/- 42%, P = 0.022) or chemical stimulation with formyl-methylleucylphenylalani
    85 menon have been limited to digital (or step) chemical stimulation with little control over the tempor
  
    87  PAG comparably elevated AP and CBF, whereas chemical stimulation with the D,L-homocysteine produced 
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