ライフサイエンスコーパス: chemokinesis

1 ced osteoclast conditioned media-induced hMS chemokinesis.

2 is a common feature both for chemotaxis and chemokinesis.

3 calpain, induced neutrophil polarization and chemokinesis.

4 chemotaxis, but dimeric TFF1 stimulated some chemokinesis.

5 active CREB decreased both proliferation and chemokinesis.

6 ated that SPP stimulates both chemotaxis and chemokinesis.

7 d monocytes induced by CC chemokines, termed chemokinesis.

8 gher for faster cells, and was compounded by chemokinesis, an increase in speed with resource concent

9 tively active CREB abolished the increase in chemokinesis and cell cycle progression induced by eithe

10 oxidative concentrations of H2O2 also impede chemokinesis and chemotaxis of previously activated huma

11 nsient overexpression of PLD1 increased both chemokinesis and chemotaxis toward IL-8 and FMLP but not

12 (PLD1) and PLD2 reduced cell migration (both chemokinesis and chemotaxis) by approximately 60% and >8

13 says do not definitively distinguish between chemokinesis and chemotaxis, single-cell chemotaxis assa

14 sufficient to promote SMC migration by both chemokinesis and chemotaxis, which was inhibited by prot

15 ming of stem cells to this site by mediating chemokinesis and chemotaxis.

16 ve stress decrease SMC CREB content increase chemokinesis and entry into the cell cycle, which is blo

17 se decreased CREB function but increased SMC chemokinesis and entry into the cell cycle.

18 by endothelial activation with IL-4 improved chemokinesis and lateral migration toward a monocyte che

19 /neogenin interactions augment CD4(+) T cell chemokinesis and promote cellular infiltration in associ

20 ed, AMPK increased microtubule synthesis and chemokinesis and provided adaptation to energy demand du

21 dependent PIP3 accumulation, PKB activation, chemokinesis and reactive oxygen species (ROS) formation

22 show defective chemokine-induced chemotaxis, chemokinesis, and adhesion to integrin ligands.

23 -mediated actin rearrangement and eosinophil chemokinesis, and impairs eosinophil viability.

24 predominantly stimulated chemotaxis and some chemokinesis, and it was chemotactic for a variety of hu

25 -/-) mice exhibited an increased chemotaxis, chemokinesis, and transendothelial migration in vitro.

26 te cell migration in chemotaxis, haptotaxis, chemokinesis, and wound healing assays.

27 Increases in DC accumulation and chemokinesis are partially dependent on ccr6, a crucial

28 d of similar magnitude during chemotaxis and chemokinesis, at 18 +/- 1.4 and 16 +/- 1.3 nN/cell, resp

29 In addition, thrombin induced chemokinesis, but not chemotaxis, of CD8(+) T cells, whi

30 thionylleucylphenylalanine (FMLP)-stimulated chemokinesis by >60%, markedly reduced polar morphology,

31 une reactions to the joint through leukocyte chemokinesis, cell-cell adhesion, and matrix specializat

32 of PA and EF) significantly impair human PMN chemokinesis, chemotaxis, and ability to polarize.

33 eased stem cell migration when compared to a chemokinesis control.

34 taxis induced by IL-8 and fMLP but decreased chemokinesis for neutrophils.

35 Chemokinesis has been most thoroughly studied in the per

36 transport, epilepsy, behavioral abnormality, chemokinesis, histone deacetylation, and immunity.

37 ld involve chemotaxis toward infected cells, chemokinesis (i.e., increased motility) combined with CT

38 d cells, indicating that F. alocis increases chemokinesis in human neutrophils.

39 Lysophosphatidic acid induced chemokinesis in T cells.

40 neutrophil adhesion, polarization, and rapid chemokinesis in the absence of exogenous activators.

41 ne CCL21, a ligand of CCR7, strongly induces chemokinesis in vitro, and T cell motility in LNs from C

42 reas micromolar GABA predominantly initiated chemokinesis (increased random movement).

43 n contrast to IL-8-induced chemokinesis, the chemokinesis induced by calpain inhibition was not reduc

44 PE increased cell velocity, suggesting that chemokinesis may be at least partly responsible for dire

45 ndicated that LPA causes both chemotaxis and chemokinesis of beta1-replete cells.

46 ll m.w. cytokines that induce chemotaxis and chemokinesis of leukocytes.

47 LPA stimulates both chemotaxis and chemokinesis of ovarian cancer cells and LPA-stimulated

48 PGE2-elicited chemokinesis of RNK-16 cells across a Matrigel model bas

49 Furthermore, this dependence of chemokinesis on PI(3)Kgamma activity is context dependen

50 Chemokinesis on VCAM-1 appeared to be associated with tr

51 e constitutively active Ras has no effect on chemokinesis or chemotaxis.

52 sures true macrophage chemotaxis rather than chemokinesis or fugetaxis.

53 (2)O(2)) demonstrated a 3.5-fold increase in chemokinesis (p < 0.05) and accelerated entry into cell

54 with ET causes an additive inhibition of PMN chemokinesis, polarization, chemotaxis, and FMLP (N-form

55 PCNs utilized FGF-2 found in situ to induce chemokinesis, potentiate SDF-1alpha chemotactic recruitm

56 r-1alpha (SDF-1alpha)-induced chemotaxis and chemokinesis (random migration).

57 ne airways primarily by increasing velocity (chemokinesis) rather than directional migration (chemota

58 vidual eggs reveals that both chemotaxis and chemokinesis significantly promote contacts.

59 Chemokinesis stimulated by IL-8 was inhibited in the pre

60 PLD1 isoform, led to an abolishment of basal chemokinesis that could not be rescued with chemoattract

61 In contrast to IL-8-induced chemokinesis, the chemokinesis induced by calpain inhibi

62 analysis indicated that both chemotaxis and chemokinesis took place.

63 we describe reduced chemotaxis but preserved chemokinesis toward a range of inflammatory stimuli in m

64 Chemokinesis was not mediated by the enzymatic activity

65 Chemotaxis and chemokinesis were markedly impaired, but staphylococcal

66 sulted in elevation of both S6K activity and chemokinesis, whereas PLD silencing was inhibitory.

67 s only, is a potent stimulator of macrophage chemokinesis, whose activity is enhanced by yeast cell w