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1 rons with this anatomical specialization are chemosensitive.
2  also respond to local glutamate and thus be chemosensitive.
3 er of thermosensitive mitral cells were also chemosensitive.
4 ndependent growth, and rendered glioma cells chemosensitive.
5 nsory integration centers and are themselves chemosensitive.
6 ned 83.3% of the cases as chemorefractory or chemosensitive.
7 the brainstem chemoreceptor areas are highly chemosensitive.
8  inclusion of bexarotene, the cells remained chemosensitive.
9  found that 34% (61 of 178) of GC units were chemosensitive.
10             Ninety-four patients with either chemosensitive (50 patients) or chemoresistant (44 patie
11 me treatment on progression in patients with chemosensitive advanced colorectal cancer.
12 the muscle interstitium where they stimulate chemosensitive afferent nerves that reflexly increase ef
13  differentially affects mechanosensitive and chemosensitive afferent pathways.
14 ly evoked plantar flexion, where only muscle chemosensitive afferents were likely to be stimulated, B
15 solidation therapy for PTCL patients who are chemosensitive after induction chemotherapy.
16 nting number of competing nuclei proposed as chemosensitive along with an ever increasing list of pot
17  was significantly higher than that noted in chemosensitive AML (P=0.018).
18 atient-derived xenografts to generate paired chemosensitive and chemoresistant cancers.
19 e-dependently inhibited the proliferation of chemosensitive and chemoresistant glioma cells but did n
20  augmented chemotherapeutic efficacy in both chemosensitive and chemoresistant models of small cell l
21 itously released in high abundance from both chemosensitive and chemoresistant ovarian cancer cells v
22 nomycin-induced PARC down-regulation in both chemosensitive and chemoresistant ovarian cancer cells.
23 n results from ischemic episodes that excite chemosensitive and mechanoreceptive receptors in the hea
24 rs with oligodendroglial differentiation are chemosensitive and this correlates with allelic loss of
25  both RTN and pfRG neurons are intrinsically chemosensitive and tonically firing neurons whose respir
26 x vivo in tissue culture are also relatively chemosensitive and undergo apoptotic death in response t
27 ical astrocytes, astrocytes of the brainstem chemosensitive area(s) possess signaling properties that
28 urface of the brainstem (central respiratory chemosensitive area) respond to physiological decreases
29  the retrotrapezoid nucleus (RTN), a central chemosensitive area, and the rostral ventrolateral medul
30  cells were not found in any of the putative chemosensitive areas.
31 ellular PCO2/[H(+)] with Ca(2+) responses in chemosensitive astrocytes remain unknown.
32 lthough Burkitt lymphoma cells are extremely chemosensitive, biologically targeted therapies should b
33 between neurones from the medullary raphe (a chemosensitive brain region) and hippocampus (a non-chem
34 ct the drive to breathe, we hypothesize that chemosensitive brain regions have adapted a means of pre
35 tzinger rhythm-generating complex and in the chemosensitive brainstem respiratory regions, thereby in
36 d nuclear p53 accumulation, and apoptosis in chemosensitive but not resistant OVCA cells.
37 ggressive disease-the condition is initially chemosensitive but then relapses with acquired chemoresi
38 Small-cell lung cancer (SCLC) is exquisitely chemosensitive, but few patients are cured by convention
39   Small cell lung cancer (SCLC) is initially chemosensitive, but rapidly relapses in a chemoresistant
40 en patients designated as chemorefractory or chemosensitive by using the baseline CNA classifier.
41                               Interestingly, chemosensitive cancer cell lines (A2780 and PEO1) displa
42                                          The chemosensitive cancer cells could not survive glucose de
43                                              Chemosensitive carotid body glomus cells exhibited toll-
44  are associated with poor prognosis, even in chemosensitive cases.
45 nd higher P-glycoprotein expression than the chemosensitive cell lines MCF7-wt and MES-SA.
46 ye-filled neurons showed that ASIC-dependent chemosensitive cells (cells responding to pH 7.0) cluste
47 omponents of transduction to the identity of chemosensitive cells and chemosensitive nuclei.
48           Conversely, upregulation of MVP in chemosensitive cells does not confer increased drug resi
49 ar composition of the native KO2 channels in chemosensitive cells is important to understand the mech
50 e therapy to maintain a stable population of chemosensitive cells that can, in turn, suppress the gro
51 en by permitting a significant population of chemosensitive cells to survive so that they, in turn, s
52 arwinian environment of a cancer, the fitter chemosensitive cells will ordinarily proliferate at the
53                             Rat carotid body chemosensitive cells, and human neutrophils, were treate
54 cellular components and trigger apoptosis in chemosensitive cells.
55 K+ channel similar to that recorded in other chemosensitive cells.
56 s offer the basis of methods for reversible, chemosensitive control of the interfacial organization o
57 fter cPGI but is more excitable and markedly chemosensitive (CS) with a pronounced enhancement of max
58        One hundred fifty-three patients with chemosensitive disease after ICE (ifosfamide, carboplati
59 ion may be considered in young patients with chemosensitive disease and in newly diagnosed patients w
60                  All patients presented with chemosensitive disease at transplantation.
61            Five of six patients (83.3%) with chemosensitive disease continue to be alive compared wit
62 at relapse from five patients with initially chemosensitive disease did not switch to a chemorefracto
63                       Fourteen patients with chemosensitive disease evident on neuroimaging then rece
64                               For those with chemosensitive disease the PFS and OS were 42% and 55%,
65  1 year for patients with chemoresistant and chemosensitive disease were 75% and 25%, respectively (P
66  rates for patients with chemorefractory and chemosensitive disease were 80% and 87%, respectively (P
67 dministering HDT/ASCT to those patients with chemosensitive disease were reviewed.
68         A subgroup of patients with residual chemosensitive disease who attained complete remission a
69    We retrospectively analyzed patients with chemosensitive disease who underwent high-dose chemoradi
70 rs); median prior regimens, 6 (range, 3-12); chemosensitive disease, 6 (15%); bulk > 5 cm, 17 (range,
71                      In select patients with chemosensitive disease, autologous stem cell transplanta
72  and was significantly better for those with chemosensitive disease, Hodgkin disease (HD), and low-gr
73 iled in five (28%) patients and 16 (89%) had chemosensitive disease.
74 tent of administering HDT/ASCT to those with chemosensitive disease.
75 llowed by autologous stem cell transplant in chemosensitive disease.
76 gous transplant consolidation for those with chemosensitive disease.
77 esistant disease versus 33% of patients with chemosensitive disease; P = 0.005) and decreased overall
78         Forty-three patients (50.6%) had ICE-chemosensitive disease; there was no difference in the r
79  mismatch repair (MMR) and the repair of the chemosensitive DNA lesion, O6-methylguanine (O6-MeG), by
80 xed (anaplastic oligoastrocytoma [AOA]), are chemosensitive, especially if codeleted for 1p/19q, but
81                                          CO2-chemosensitive firing responses using single-cell patch
82 ituximab (FCR) conditioning for relapsed and chemosensitive follicular lymphoma.
83  The molecular mechanisms that differentiate chemosensitive from chemorefractory disease are currentl
84 o identify genetic features that distinguish chemosensitive from chemorefractory disease, we examined
85                             Although carotid chemosensitive glomus cells have been the most extensive
86                              In carotid body chemosensitive glomus cells, activation of toll-like rec
87 dicate that there is severe loss of putative chemosensitive glutamatergic and serotonergic neurons as
88                                              Chemosensitive glutamatergic and serotonergic neurons lo
89 ity as well as an increase in the numbers of chemosensitive group III/IV muscle afferents as assessed
90 wever, the proliferation rate is low in many chemosensitive human cancers, and it is not clear how a
91 n medullary serotonergic neurones, which are chemosensitive in vitro.
92 nd cell lines derived from these tumours are chemosensitive in vitro.
93 atal day 12 (P12), when 5-HT neurones become chemosensitive in vitro.
94 e many neurons that make up this network are chemosensitive in vitro.
95 ow that hTRPA1 is an intrinsically cold- and chemosensitive ion channel.
96                  Small cell lung cancer is a chemosensitive malignancy, yet long-term survival remain
97 tive strategy for patients with relapsed and chemosensitive mantle-cell lymphoma.
98 f the estrogen receptor alpha (ER)-positive, chemosensitive MCF7 breast cancer cell line to tumor nec
99 nding and suggesting mechanosensitive and/or chemosensitive mechanisms for upregulation.
100 n TRPA1 (hTRPA1-HEK), as well as a subset of chemosensitive mouse trigeminal neurons.
101 uli and the BAO-induced phenotypic switch in chemosensitive muscle afferents, potentially through reg
102  suggest that an important characteristic of chemosensitive neurones is an unusually large change of
103 its presence is not uniquely associated with chemosensitive neurones.
104 n involve direct H(+)-mediated activation of chemosensitive neurons and indirect modulation of chemos
105 sensitive neurons and indirect modulation of chemosensitive neurons by purinergic signalling.
106 /H(+)-dependent drive to breathe produced by chemosensitive neurons in the retrotrapezoid nucleus (RT
107                                              Chemosensitive neurons in the retrotrapezoid nucleus (RT
108 Isoflurane also increased firing rate of RTN chemosensitive neurons in urethane-anesthetized rats, ag
109 o and the excitability of isolated pulmonary chemosensitive neurons in vitro, and this effect of PAR2
110 als are a probable marker of RTN; and 4) the chemosensitive neurons of RTN may provide a chemical dri
111 he main stimulus for breathing by activating chemosensitive neurons that control respiratory output.
112 onal brain slices (postnatal days 7-12), RTN chemosensitive neurons were silent at pH 7.55.
113  stimulation of Phox2b-lineage, putative CO2-chemosensitive neurons.
114 ing reduced CO2 -induced firing responses of chemosensitive neurons.
115  sensitivity of isolated rat vagal pulmonary chemosensitive neurons.
116 europil likely regulates the activity of RTN chemosensitive neurons; 2) the catecholaminergic, cholin
117  create 5-FdUMP resistant enzymes to protect chemosensitive normal cells and further understand mecha
118 also augmented action potential discharge in chemosensitive nTS neurons.
119  to the identity of chemosensitive cells and chemosensitive nuclei.
120  we show that CTCs from patients with either chemosensitive or chemorefractory SCLC are tumorigenic i
121 n effective option in lymphoma patients with chemosensitive or stable disease who experience disease
122               Patients were required to have chemosensitive or stable disease.
123 s of whether neurones were cultured from the chemosensitive or the non-chemosensitive region, pH(i) d
124 r processes in the RTN/Ppy region are either chemosensitive or they modulate chemosensitivity.
125 serotonergic neurones (most of which are not chemosensitive), or from the DeltapH(i)/DeltapH(o) of hi
126 ess visceral organs and are only temporarily chemosensitive, ovarian carcinoma is a deadly disease, w
127 lenge, increases calcium oscillations in the chemosensitive parafacial respiratory group (pFRG/RTN).
128 re affected by a selection bias because only chemosensitive patients actually proceeded to AHCT, wher
129 r delaying development of drug resistance in chemosensitive patients represents another therapeutic s
130  cancer stem cells differentiated into their chemosensitive progeny, they lost tumor-initiating capac
131 to metabolic acidosis, the neurones from the chemosensitive region do not have a uniquely high Deltap
132 he perinatal pFRG and appears lateral to the chemosensitive region of adult RTN.
133 These glutamatergic neurons are located in a chemosensitive region of the ML and their projections ar
134 nsitive brain region) and hippocampus (a non-chemosensitive region), we used BCECF to monitor pH(i) i
135  cultured from the chemosensitive or the non-chemosensitive region, pH(i) did not recover during any
136 gers the immediate release of ATP from three chemosensitive regions located on the ventral surface of
137 s reinforce past findings that patients with chemosensitive relapse are better candidates for high-do
138 of lenalidomide maintenance in patients with chemosensitive relapse of DLBCL not eligible for ASCT or
139  first CR, compared with 37% for patients in chemosensitive relapse, and 7% for chemoresistant patien
140 patients with relapsed disease, particularly chemosensitive relapse, are superior to those reported f
141 lapse rates within the first 3 years for the chemosensitive relapse, chemoresistant, and induction fa
142 lantation (ASCT) is the standard of care for chemosensitive relapsed and refractory diffuse large B-c
143 y, similar to the outcomes for patients with chemosensitive relapsed disease.
144                                          The chemosensitive response of locus coeruleus (LC) neurones
145 gnals play a greater role in determining the chemosensitive response to changes in CO2/H+ than previo
146                                   Therefore, chemosensitive RTN neurons appear to be critically impor
147 urochemical phenotype previously defined for chemosensitive RTN neurons in vivo.
148 stic BM samples from primary refractory AML (chemosensitive) showed methylation of WIT-1.
149  to an increase in P(CO2)/[H+] (hypercapnia) chemosensitive structures located on the ventral surface
150 n even in seminoma and that seminoma is more chemosensitive than nonseminoma, a renewed clinical need
151 r, the widely accepted view that BAC is less chemosensitive than other NSCLCs is not clearly supporte
152 s cancer cells and why some cancers are more chemosensitive than others.
153 te when it was focally applied to the apical chemosensitive tips of receptor cells.
154  direct evidence that pre-LSCs are much less chemosensitive to existing chemotherapy drugs than leuke
155 er cells expressing MT1-MMP were selectively chemosensitive to ICT2588, whereas MCF7 cells that did n
156 rons in the midbrain of rats are also highly chemosensitive to small changes in CO2/pH and are closel
157                                Patients with chemosensitive transformed lymphoma should be seriously
158 n tumor in children, is a radiosensitive and chemosensitive tumor.
159 OF REVIEW: Synovial sarcomas are regarded as chemosensitive tumors compared to other types of soft ti
160              Further trials in children with chemosensitive tumors, with minimal residual disease, ar
161 l tumors (TGCT) are considered a paradigm of chemosensitive tumors.
162                Pharmacological activation of chemosensitive vagal afferents with phenyl biguanide pro
163                          The two subtypes of chemosensitive VMM neurones could be distinguished by ch
164 atients that entered CR after therapy (i.e., chemosensitive) were studied and only 10 (37%) of the di
165 ed carboplatin-induced apoptosis in the more chemosensitive WERI, but not the chemoresistant Y79 line
166 that epithelial ovarian cancer is moderately chemosensitive, what distinguishes it most from other me
167            Small cell lung cancer was termed chemosensitive with the introduction of combination chem
168  relatively uncommon carcinomas is initially chemosensitive, with a high overall response rate to com

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