ライフサイエンスコーパス: chemotaxin

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1 -related factors, inducers of cytokines, and chemotaxins).

2 induce the production of IL-6 and neutrophil chemotaxins.

3 irways epithelial cells to induce neutrophil chemotaxins.

4 ich specifically cleaves mouse and human C5a chemotaxins.

5 hepatic amyloidosis, leukocyte cell-derived chemotaxin 2 (LECT2) amyloidosis (ALect2) accounted for

6 ke chemotactic factor leukocyte cell-derived chemotaxin 2 (LECT2) and invariant NKT (iNKT) cells were

7 Leukocyte cell-derived chemotaxin 2 (LECT2) is a cytokine and preferentially ex

8 that we must now add leukocyte cell-derived chemotaxin 2 (LECT2) to the list of proteins that can ca

9 ologous to vertebrate leukocyte cell-derived chemotaxin 2 (LECT2)/Chondromodulin II.

10 mine synthetase (GS), leukocyte cell-derived chemotaxin 2, Regucalcin, and Cyclin-D1 and of K-Ras eff

11 CT-2, the ortholog of leukocyte cell-derived chemotaxin-2 (LECT2), is secreted from the muscles and r

12 leukin-8 (IL-8) is the major lung neutrophil chemotaxin, and we have shown that Ad7 induces IL-8 rele

13 al of those occurring in cells stimulated by chemotaxins, and that the nucleotides ADP, ATP, and UTP

14 e conclude that some of the effects of these chemotaxins are mediated by translocation of an intracel

15 (SCPA) specifically cleaves the human serum chemotaxin C5a at the polymorphonuclear leukocyte (PMNL)

16 s, including IL-1, IL-36, and the neutrophil chemotaxins CXCL1 and CXCL8.

17 Anaphylatoxin C5a was a potent chemotaxin (EC50 approximately 0.5 nM) for J774 monocyte

18 n from lamina propria to lumen caused by the chemotaxin, FMLP, and that LTB4-induced eosinophil migra

19 ponsiveness both to C5a and to the bacterial chemotaxin, fMLP.

20 ADP was also a chemotaxin for murine J774 monocytes.

21 ally cleaves the complement component C5a, a chemotaxin for polymorphonuclear leukocytes.

22 Intralumenal instillation of the chemotaxin, formyl-met-leu-phe (FMLP) caused migration o

23 d virus-induced expression of the neutrophil chemotaxin IL-8, previously implicated in adenoviral pat

24 sely related chemokines are major neutrophil chemotaxins in experimental gout.

25 stals induce different classes of neutrophil chemotaxins, including certain chemokines (e.g., interle

26 5a promoted the production of the neutrophil chemotaxin interleukin-8, and recruitment of neutrophils

27 kotriene A4 (LTA4) and subsequently into the chemotaxin LTB4, which has no direct bronchoconstrictor

28 le derivatives of ADP and ATP were effective chemotaxins, obviating a role for adenosine receptors.

29 ng the SDF-1-CXCR4-axis by cleaving SDF-1, a chemotaxin recruiting CXCR4(+) SC.

30 These findings highlight the complexity of chemotaxin signaling pathways and offer one mechanism by

31 by which neutrophils may spatially organize chemotaxin signaling within the plasma membrane.

32 their role in recognition systems for other chemotaxins such as leukotriene B4 (LTB4) and fMLP is un

33 peptide-78 (ENA-78), is a potent neutrophil chemotaxin whose expression is increased in inflamed syn

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