ライフサイエンスコーパス: chemotherapeutic drug

1 dly decreased at 24 h after treatment with a chemotherapeutic drug.

2 zyme activity in single cells treated with a chemotherapeutic drug.

3 e more sensitive to doxorubicin, a classical chemotherapeutic drug.

4 mitogenic activity in mice pretreated with a chemotherapeutic drug.

5 t brusatol can be developed into an adjuvant chemotherapeutic drug.

6 to paclitaxel, a commonly used breast cancer chemotherapeutic drug.

7 not expressing the oncogene in response to a chemotherapeutic drug.

8 own activity for this widely utilized cancer chemotherapeutic drug.

9 om dose-limiting toxicity of streptozocin, a chemotherapeutic drug.

10 in cancer cells, and thus sensitise them to chemotherapeutic drugs.

11 fection that is resistant to the majority of chemotherapeutic drugs.

12 f HER2 confers resistance of cancer cells to chemotherapeutic drugs.

13 is a major dose-limiting side effect of many chemotherapeutic drugs.

14 ed G1 arrest and induced hypersensitivity to chemotherapeutic drugs.

15 and are still the most widely used forms of chemotherapeutic drugs.

16 ntly sensitize breast cancer cell to various chemotherapeutic drugs.

17 which the HIV-1 reservoir may be limited by chemotherapeutic drugs.

18 sensitize cells cross-resistant to multiple chemotherapeutic drugs.

19 ty to cisplatin, one of the most widely used chemotherapeutic drugs.

20 and calcium signaling associated with these chemotherapeutic drugs.

21 Rac activation and increased sensitivity to chemotherapeutic drugs.

22 ity of cells to cell death stimuli including chemotherapeutic drugs.

23 insults to cells, including cytotoxic cancer chemotherapeutic drugs.

24 ance (MDR) restricts the efficacy of current chemotherapeutic drugs.

25 istance to a variety of chemically unrelated chemotherapeutic drugs.

26 d increased cytotoxicity in combination with chemotherapeutic drugs.

27 ivity of certain types of cancer to standard chemotherapeutic drugs.

28 s and A549 xenografts to cisplatin and other chemotherapeutic drugs.

29 rtmental and intracellular concentrations of chemotherapeutic drugs.

30 s by which brusatol enhances the efficacy of chemotherapeutic drugs.

31 es, and they are often resistant to standard chemotherapeutic drugs.

32 otential adjuvant to improve the efficacy of chemotherapeutic drugs.

33 ith blood transfusions or myeloablation with chemotherapeutic drugs.

34 of the efficacy and toxicity profile of the chemotherapeutic drugs.

35 rapy in combination with several widely used chemotherapeutic drugs.

36 f Nrf2 expression sensitizes cancer cells to chemotherapeutic drugs.

37 sis and a potential differential response to chemotherapeutic drugs.

38 -kappaB), which becomes further activated by chemotherapeutic drugs.

39 cells lacking certain MSHs are resistant to chemotherapeutic drugs.

40 rs in part because it is insensitive to many chemotherapeutic drugs.

41 tumor cells to growth suppression by various chemotherapeutic drugs.

42 nd is potentially important in resistance to chemotherapeutic drugs.

43 Ca cell survival, viability, and response to chemotherapeutic drugs.

44 ecific inhibitors combined with conventional chemotherapeutic drugs.

45 rovides a major resistance mechanism to some chemotherapeutic drugs.

46 t cancer patients treated with commonly used chemotherapeutic drugs.

47 state cancer cells from apoptosis induced by chemotherapeutic drugs.

48 ghts into the mechanism of this new class of chemotherapeutic drugs.

49 dered them sensitive to apoptosis induced by chemotherapeutic drugs.

50 ereas its deregulation reduces resistance to chemotherapeutic drugs.

51 (I.P./I.V.) injection alone or combined with chemotherapeutic drugs.

52 siRNA knockdown and following treatment with chemotherapeutic drugs.

53 like receptors, reactive oxygen species, and chemotherapeutic drugs.

54 hich is upregulated following treatment with chemotherapeutic drugs.

55 Akt activation and increased sensitivity to chemotherapeutic drugs.

56 igature or embolization, and radiotherapy or chemotherapeutic drugs.

57 ture, improving the delivery and efficacy of chemotherapeutic drugs.

58 f tumor hypoxia and reduction of delivery of chemotherapeutic drugs.

59 on the response of cervical cancer cells to chemotherapeutic drugs.

60 nment which nurtures and protects cells from chemotherapeutic drugs.

61 molar IC50 values, superior to many clinical chemotherapeutic drugs.

62 tase (DHFR), a key target of antibiotics and chemotherapeutic drugs.

63 ncer-cell motility, and higher resistance to chemotherapeutic drugs.

64 lications in tumor growth and treatment with chemotherapeutic drugs.

65 mediates the cytotoxicity of the anti-cancer chemotherapeutic drug 5-fluorouracil (5-FU) by prolongin

66 kinase activity of Mirk was increased by the chemotherapeutic drug 5-fluorouracil (5-FU).

67 The chemotherapeutic drug 5-fluorouracil (5FU) disrupts DNA

68 ity of cells to the cytotoxic effects of the chemotherapeutic drug 5-fluorouracil, as shown by increa

69 screens in two bacterial species using three chemotherapeutic drugs: 5-fluorouracil (5-FU), 5-fluoro-

70 used to describe the patient response to the chemotherapeutic drug 6-mercaptopurine, with some model

71 P-gp expression and function coincided with chemotherapeutic drug accumulation in brains of WT mice

72 treatment of p53-null cancer cells with the chemotherapeutic drug adriamycin (doxorubicin) induces A

73 nce since this enzyme is currently used as a chemotherapeutic drug against several types of cancer an

74 by paclitaxel, one of the most commonly used chemotherapeutic drugs against various human cancers.

75 Thus, application of chemotherapeutic drugs along with HO-1 inhibitor may ele

76 Chemotherapeutic drugs also activated an alternative cas

77 of both a low-molecular-weight conventional chemotherapeutic drug and a nanotherapeutic agent, leadi

78 Stratified by chemotherapeutic drug and comorbid pain risk, patients w

79 synergic combinational dosage ratios of the chemotherapeutic drug and the anticancer flavonoid were

80 ffects on cancer cells, several conventional chemotherapeutic drugs and agents used in targeted thera

81 lls within a tumor, cancer stem cells resist chemotherapeutic drugs and can regenerate the various ce

82 esistant tumor cells to apoptosis by various chemotherapeutic drugs and chemosensitization was, in la

83 plasma membrane ABC transporters can export chemotherapeutic drugs and confer drug resistance, it is

84 ment with radiation and different classes of chemotherapeutic drugs and describe mechanisms determini

85 endogenous and exogenous chemicals, such as chemotherapeutic drugs and formaldehyde.

86 The finding that cancer chemotherapeutic drugs and ionizing radiation often prom

87 Moreover, the combined use of chemotherapeutic drugs and metalloproteinase inhibitors

88 cking cholesterol uptake sensitizes cells to chemotherapeutic drugs and potentiates the effect of che

89 inamide enhanced the ROS produced by several chemotherapeutic drugs and produced synergistic cell kil

90 cancer cells as a survival strategy against chemotherapeutic drugs and promotes growth of tumor cell

91 applied to reverse cross-resistance to other chemotherapeutic drugs and restore treatment efficacy.

92 high incidence of resistance to DNA-damaging chemotherapeutic drugs and severe side effects of chemot

93 discuss the most common interactions between chemotherapeutic drugs and supportive-care drugs-such as

94 ity, the multi-drug resistance (MDR) to free chemotherapeutic drugs and the deadly metastases of canc

95 The combination of conventional chemotherapeutic drugs and those that block NF-kappaB ac

96 eveloped laboratory biosensors for screening chemotherapeutic drugs and to aid in the assessment of D

97 Nrf2 inhibitors to increase the efficacy of chemotherapeutic drugs and to combat chemoresistance, th

98 ensitize tumor cells to apoptosis by various chemotherapeutic drugs and tumor necrosis factor-related

99 ated with tumour formation and resistance to chemotherapeutic drugs, and epigenetic modifications are

100 vated by a range of xenochemicals, including chemotherapeutic drugs, and has been suggested to play a

101 AEG-1 directly contributes to resistance to chemotherapeutic drugs, another important hallmark of ag

102 rtion and extension of a ribonucleotide, the chemotherapeutic drug arabinofuranosylcytosine triphosph

103 Current chemotherapeutic drugs are effective only transiently be

104 Platinum-based chemotherapeutic drugs are front-line therapies for the

105 ons, a surgical approach is limited and most chemotherapeutic drugs are ineffective owing to the bloo

106 Since several chemotherapeutic drugs are known to increase rAAV transd

107 Systemically administered chemotherapeutic drugs are often ineffective in the trea

108 lementation strategy for assessing potential chemotherapeutic drugs as well as a sensitive and dynami

109 (MTAs), widely used as biological probes and chemotherapeutic drugs, bind directly to tubulin subunit

110 as maintained after DNA damage caused by the chemotherapeutic drug bleomycin.

111 showed markedly increased sensitivity to two chemotherapeutic drugs, bleomycin and etoposide (P < 0.0

112 cancer cells were resistant to cell death by chemotherapeutic drugs but E-cadherin null cells or thos

113 Gemcitabine is a widely used chemotherapeutic drug, but limited therapeutic efficacy

114 ies acquire resistance to apoptosis-inducing chemotherapeutic drugs by downregulating the key effecto

115 (ATM-RAD3-related) signaling pathway by the chemotherapeutic drug camptothecin (CPT).

116 r knowledge, this is the first report that a chemotherapeutic drug can induce HIF-1alpha accumulation

117 Convection enhanced delivery (CED) of chemotherapeutic drugs can successfully bypass the blood

118 RAPA), which is used as the antiangiogenesis chemotherapeutic drug, can cutdown the tumor vessels and

119 KK2 knockdown potentiated the effects of the chemotherapeutic drugs carboplatin and PX-866 to reduce

120 low levels of Ag with local irradiation or a chemotherapeutic drug causes sufficient release of Ag to

121 The chemotherapeutic drug cisplatin (cis-diamminedichloropla

122 The chemotherapeutic drug cisplatin is actively transported

123 As Ctr1 also transports the chemotherapeutic drug cisplatin via direct binding to th

124 as those caused by UV light exposure and the chemotherapeutic drug cisplatin.

125 h in high-grade tumor cells resistant to the chemotherapeutic drug cisplatin.

126 se-dependent resistance to the cross-linking chemotherapeutic drug cisplatin.

127 tosis both alone and in combination with the chemotherapeutic drug cisplatin.

128 epresentative chemical agents, we selected a chemotherapeutic drug (cisplatin) which forms covalent a

129 rt of our hypothesis that DIM could abrogate chemotherapeutic drug (cisplatin, gemcitabine, and/or ox

130 RNAi-chemotherapeutic drug combinations have also been found

131 By achieving higher chemotherapeutic drug concentrations in target lesions,

132 In mice, DNA damage initiated by chemotherapeutic drug cyclophosphamide activated caspase

133 lowing the induction of neutropenia with the chemotherapeutic drug cyclophosphamide, ExPEC translocat

134 TMF, sensitivity to six clinically relevant chemotherapeutic drugs (dacarbazine, doxorubicin, paclit

135 pre-treatment with N6L efficiently improved chemotherapeutic drug delivery and increased the antitum

136 ultidrug resistance protein 1 (MRP1), before chemotherapeutic drug delivery in vivo with a single loc

137 blood brain barrier (BBB) is used to enhance chemotherapeutic drug delivery.

138 ow) and potentially enhance co-administrated chemotherapeutic drug delivery.

139 tate-specific target for CaP chemopreventive/chemotherapeutic drug development.

140 tent oncogene that is a prominent target for chemotherapeutic drug development.

141 ed drug sensitivity to the two commonly used chemotherapeutic drugs, docetaxel and etoposide, judging

142 e the anticancer activity of the traditional chemotherapeutic drug, DOX, by multiple mechanisms inclu

143 y mild hyperthermia-triggered release of the chemotherapeutic drug doxorubicin (DOX) after hCTMO1 mon

144 PTEN showed increased susceptibility to the chemotherapeutic drug doxorubicin but not paclitaxel.

145 cycle increased MDA cell apoptosis, and the chemotherapeutic drug doxorubicin had a synergistic effe

146 e xenografts, injection of metformin and the chemotherapeutic drug doxorubicin near the tumor is more

147 Here, we report the ability to enhance chemotherapeutic drug doxorubicin penetration using ultr

148 hemosensitizer curcumin and the encapsulated chemotherapeutic drug doxorubicin to maximize a synergis

149 The chemotherapeutic drug doxorubicin, a potent inducer of a

150 Doxil, a liposomal formulation of the chemotherapeutic drug doxorubicin, is FDA-approved for m

151 In contrast to the conventional chemotherapeutic drug doxorubicin, which induces p53 act

152 tizes MCF-7 human breast cancer cells to the chemotherapeutic drug doxorubicin.

153 st cancer cells to DNA damage induced by the chemotherapeutic drug doxorubicin.

154 allows their subsequent depletion using the chemotherapeutic drug doxorubicin.

155 lls were anchorage-independent, resistant to chemotherapeutic drugs doxorubicin and paclitaxel, and r

156 f a membrane-impermeable dye (calcein) and a chemotherapeutic drug (doxorubicin) are demonstrated.

157 were then subjected to the action of a model chemotherapeutic drug, doxorubicin (DOX), to assess the

158 h s.c. and i.p. models and the presence of a chemotherapeutic drug, doxorubicin, further inhibited MM

159 ) binding to MDR1 resulting in the efflux of chemotherapeutic drugs (e.g. doxorubicin and paclitaxel

160 We show that E2F1 causes chemotherapeutic drug efflux both in vitro and in vivo v

161 matic enhancement of the cytotoxic effect of chemotherapeutic drugs, either active or nonactive as si

162 sive, but it has become evident that certain chemotherapeutic drugs elicit immunogenic danger signals

163 ian cells and that certain antibacterial and chemotherapeutic drugs elicit thymine deficiency, a mech

164 eficient cells; treatment of both lines with chemotherapeutic drugs elicited similar intrinsic apopto

165 enrichment of CD44(hi) cells exposed to the chemotherapeutic drug epirubicin, which suggests a feed-

166 imately 10-fold) to certain TopoII-targeting chemotherapeutic drugs (etoposide, mitoxantrone, amsacri

167 y itself nor did it increase the toxicity of chemotherapeutic drug exposure in mice.

168 ensitizer, and gemcitabine - an FDA approved chemotherapeutic drug for lung cancer chemo-radiotherapy

169 5-fluorouracil (5-FU) is a widely used chemotherapeutic drug for the treatment of a variety of

170 have created a large arsenal of targeted and chemotherapeutic drugs for precision medicine.

171 dioisotopes into tumors for internal RIT, or chemotherapeutic drugs for synergistically combined chem

172 her therapeutic agents (such as gene, DNA or chemotherapeutic drug) for targeting permeability glycop

173 rattles combined with triggered release of a chemotherapeutic drug from the nanorattles.

174 ployed to investigate the suitability of six chemotherapeutic drugs from the perspective of intratumo

175 s, we found that bacteria can metabolize the chemotherapeutic drug gemcitabine (2',2'-difluorodeoxycy

176 Treatment with the chemotherapeutic drug gemcitabine, which reduces MDSC, p

177 mproved when the virus was combined with the chemotherapeutic drug gemcitabine.

178 enhanced when BMTP-11 was combined with the chemotherapeutic drug gemcitabine.

179 Conditioning of tumor cells with chemotherapeutic drug has been shown to enhance the anti

180 Chemotherapeutic drugs have made significant contributio

181 Traditional chemotherapeutic drugs have modest but limited efficacy

182 by which NF-kappaB inhibits PCD triggered by chemotherapeutic drugs, however, remains poorly understo

183 mergence of resistance to multiple unrelated chemotherapeutic drugs impedes the treatment of several

184 , an anti-microtubule agent, is an effective chemotherapeutic drug in breast cancer.

185 erministic analysis of the success rate of a chemotherapeutic drug in less than 12h.

186 ted resistance to cisplatin, a commonly used chemotherapeutic drug in the treatment of ovarian cancer

187 tance by testing the cytotoxicity of several chemotherapeutic drugs in a panel of human glioma cell l

188 ted brain and the ability to locally deliver chemotherapeutic drugs in disease.

189 s been shown to be involved in resistance to chemotherapeutic drugs in many forms of cancers.

190 m that can profile the mechanisms of various chemotherapeutic drugs in minutes.

191 mechanisms of Pim-1L-mediated resistance to chemotherapeutic drugs in prostate cancer cells, we empl

192 vated Akt activation to confer resistance to chemotherapeutic drugs in Rb-proficient cells, which can

193 ld nanoparticles (AuNPs) functionalized with chemotherapeutic drugs in so-called "complexes" (supramo

194 nografts, and (c) increase biliary uptake of chemotherapeutic drugs in swine.

195 ak is activated normally in response to many chemotherapeutic drugs in the presence of Bax, but remai

196 elodendrimers), for the targeted delivery of chemotherapeutic drugs in the treatment of cancers, are

197 We discovered that resistance to chemotherapeutic drugs in these lines broadly correlates

198 n TACE effectiveness and supports the use of chemotherapeutic drugs in transarterial therapy.

199 The effectiveness of chemotherapeutic drugs in tumors is reduced by multiple

200 STRAP sensitized colorectal cancer cells to chemotherapeutic drugs in vitro and in vivo STRAP deplet

201 reduced the IC50 inhibitory concentration of chemotherapeutic drugs in vitro This MCAM-mediated sensi

202 sensitized GBM cells to several FDA-approved chemotherapeutic drugs including cisplatin, lomustine an

203 ide effects caused by treatment from several chemotherapeutic drugs, including paclitaxel (Taxol(R))

204 been demonstrated to increase sensitivity to chemotherapeutic drugs, including the DNA-crosslinking a

205 We recently showed that several chemotherapeutic drugs induce intratumoral expression of

206 Because many classical chemotherapeutic drugs induce reactive oxygen species (R

207 Both irradiation and chemotherapeutic drugs induced upregulation of HMGB1 and

208 were resistant to both serum starvation- and chemotherapeutic drug-induced apoptosis, whereas cells t

209 hannels in the release of nucleotides during chemotherapeutic drug-induced apoptosis.

210 wth, and results in increased sensitivity to chemotherapeutic drug-induced cell death in vitro and in

211 ation of Rad17 disrupts cellular response to chemotherapeutic drug-induced DNA damage and enhances ce

212 Here, we show that chemotherapeutic drug-induced NF-kappaB activation promo

213 mprehensive review of all published cases of chemotherapeutic drug-induced SCLE.

214 bine and discuss 16 other published cases of chemotherapeutic drug-induced SCLE.

215 tal cancer treated with a combination of the chemotherapeutic drug irinotecan and the monoclonal anti

216 seeded in a 3D extra cellular matrix when a chemotherapeutic drug is flown next to the matrix.

217 Uptake of radiopharmaceuticals and chemotherapeutic drugs is nonuniform at the microscopic

218 fficacy of cancer treatment by radiation and chemotherapeutic drugs is often limited by severe side e

219 The use of anthracycline chemotherapeutic drugs is restricted owing to potentiall

220 Many chemotherapeutic drugs kill only a fraction of cancer ce

221 We used doxorubicin (DOX), a chemotherapeutic drug known to induce oxidative stress a

222 ancer strategy alone and in combination with chemotherapeutic drugs known to induce ROS.

223 ed with temozolomide (TMZ; a DNA-methylating chemotherapeutic drug), LB-1.2 causes complete regressio

224 sotypes, the primary targets for antimitotic chemotherapeutic drugs like taxanes, has implications fo

225 ee delivery of a predetermined amount of the chemotherapeutic drug (liposomal doxorubicin) into the b

226 ugs; in the case of neoplasia, the export of chemotherapeutic drugs may facilitate cellular chemoresi

227 ration and resensitization of tumor cells to chemotherapeutic drugs may hold promise for cancer treat

228 The change in impedance magnitude on flowing chemotherapeutics drugs measured at 12h for drug-suscept

229 by replacing polystyrene particles with pure chemotherapeutic drug nanoparticles of comparable dimens

230 chanisms underlying the impact of individual chemotherapeutic drugs on cognition.

231 The effect of many contemporary chemotherapeutic drugs on pregnancy and livebirth is not

232 cyclophosphamide, a bone marrow-suppressive chemotherapeutic drug, on the development and growth of

233 rug resistance (MDR) and sensitize HDECCs to chemotherapeutic drugs, or directly reduce the tumorigen

234 cancer resistance protein (ABCG2) transports chemotherapeutic drugs out of cells, which makes it a ma

235 flammatory mediator prostaglandin E2 and the chemotherapeutic drug oxaliplatin, modeling the inherent

236 In TNBC cell lines and mouse xenografts, the chemotherapeutic drug paclitaxel increased autocrine TGF

237 el EphA2-targeting agent conjugated with the chemotherapeutic drug paclitaxel.

238 er cells treated with ionizing radiation and chemotherapeutic drug, paclitaxel.

239 The chemotherapeutic drug pemetrexed, an inhibitor of thymid

240 ts of Pt(II) complexes, we have modified the chemotherapeutic drug picoplatin with an azide moiety fo

241 ful for cancer treatment in association with chemotherapeutic drugs, possibly allowing a reduction of

242 differently to immunotherapies compared with chemotherapeutic drugs, raising questions about the asse

243 n of two candidate compounds to the existing chemotherapeutic drug regime: lithium and ibudilast.

244 contribution of bacteria to the response to chemotherapeutic drugs remains poorly understood.

245 Recurrence and chemotherapeutic drug resistance are two of the most pro

246 , which may contribute to the acquisition of chemotherapeutic drug resistance.

247 regulation of cell division and inducing the chemotherapeutic drug resistance.

248 nifying an important role of CAF exosomes in chemotherapeutic drug resistance.

249 ism by which this p73 isoform contributes to chemotherapeutic drug response remains to be explored.

250 Widely used anti-cancer treatments involving chemotherapeutic drugs result in cancer cell damage due

251 percent inhibitory concentrations (IC50) of chemotherapeutic drugs routinely used to treat breast ca

252 ological processes, disease diagnostics, and chemotherapeutic drug screening.

253 s also affect treatment outcome, and certain chemotherapeutic drugs stimulate cancer-specific immune

254 colon cancer cell death after treatment with chemotherapeutic drugs such as etoposide, cisplatin, and

255 f aggressive cancers, that is, resistance to chemotherapeutic drugs, such as 5-fluorouracil (5-FU).

256 r microtubule-targeted agents, because other chemotherapeutic drugs, such as doxorubicin, have no eff

257 BCRP and resensitized the resistant cells to chemotherapeutic drugs suggesting that BCRP phosphorylat

258 We found that the chemotherapeutic drug susceptibility-associated SNPs are

259 OA, cycloheximide, or the chemotherapeutic drug Taxol suppressed HBC cell growth.

260 This is especially important for the chemotherapeutic drug Taxol, an important anticancer age

261 umors from 6 of 10 patients treated with the chemotherapeutic drug temozolomide (TMZ) followed an alt

262 Etoposide (ETO) is a commonly used chemotherapeutic drug that inhibits topoisomerase II act

263 Etoposide, a widely used chemotherapeutic drug that inhibits topoisomerase II, is

264 Bortezomib, a proteasome inhibitor, is a chemotherapeutic drug that is commonly used to treat a v

265 Consequently, chemotherapeutic drugs that activate miR106b could initi

266 ulations has been demonstrated for classical chemotherapeutic drugs that are mostly hydrophobic, smal

267 anthracyclines, a class of clinically useful chemotherapeutic drugs that includes doxorubicin and dau

268 inimal side effects in cancer treatment with chemotherapeutic drugs that produce bulky adducts in DNA

269 there is considerable interest in developing chemotherapeutic drugs that specifically disrupt the fun

270 development of novel, less cytotoxic cancer chemotherapeutic drugs that specifically target the cell

271 reduction in P-gp when used with an existing chemotherapeutic drug (that is, doxorubicin).

272 ncer cells significantly more susceptible to chemotherapeutic drugs, that is, cisplatin or etoposide.

273 bits apoptosis and counteracts the effect of chemotherapeutic drugs, the underlying mechanism by whic

274 d a strategy to enhance tumor penetration of chemotherapeutic drugs through use of iRGD peptide (CRGD

275 mits delivery of effective concentrations of chemotherapeutic drugs to brain tumors.

276 lgae-derived nanoporous biosilica to deliver chemotherapeutic drugs to cancer cells.

277 minating against devastating cytotoxicity of chemotherapeutic drugs to healthy cells.

278 s for using the combination of metformin and chemotherapeutic drugs to improve treatment of patients

279 rnary complex of topoisomerase, DNA, and the chemotherapeutic drug topotecan show important differenc

280 A well-known chemotherapeutic drug, topotecan hydrochloride, was used

281 cumulation of endogenous adenosine following chemotherapeutic drug treatment and exposure to hypoxia.

282 ample, ionizing and ultraviolet radiation or chemotherapeutic drug treatment) can activate p53, but a

283 ed mediator of breast cancer senescence upon chemotherapeutic drug treatment.

284 c cytokine secretion and reduced the IC50 of chemotherapeutic drug treatments in AML cells.

285 924 sensitized ovarian cancer cells to other chemotherapeutic drug treatments.

286 Our recent unbiased functional screen of 54 chemotherapeutic drugs unveiled striking heterogeneity i

287 be killed by arsenic trioxide (As(2)O(3)), a chemotherapeutic drug used in the treatment of acute pro

288 -32 before treatment with arabinocytosine, a chemotherapeutic drug used to treat human AML.

289 he risk of relapse, treatment phase, and the chemotherapeutic drugs used concomitantly.

290 ed to the pH, the pulsatile delivery, or the chemotherapeutic drugs used.

291 iamminedichloroplatinum(II), CDDP), a cancer chemotherapeutic drug utilized clinically to treat a var

292 mediators of resistance to puromycin and the chemotherapeutic drug vincristine by performing genetrap

293 te a pathway for pain that is induced by the chemotherapeutic drug vincristine sulfate (VCR).

294 AF1 expression is induced in cancer cells by chemotherapeutic drug vincristine that regulates cytopla

295 nce of Star-PAP, treatment of cells with the chemotherapeutic drug VP-16 dramatically reduced E6 and

296 f2 rendered SPEC-2 cells more susceptible to chemotherapeutic drugs, whereas it had a limited effect

297 , carcinogenesis, and altered sensitivity to chemotherapeutic drugs, whereas PXR contributes to chemo

298 resistance (MDR) after prolonged exposure to chemotherapeutic drugs, which is a severe impediment to

299 nnecessary and irreversible damage caused by chemotherapeutic drugs while still maintaining therapeut

300 hese results reflect a direct interaction of chemotherapeutic drugs with both the vascular endotheliu