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1 cts were induced in a virulent strain of the chestnut blight fungus Cryphonectria parasitica (Murr.)
3 patibility (vic)] loci were disrupted in the chestnut blight fungus Cryphonectria parasitica using an
4 ion of one of two dicer genes, dcl-2, of the chestnut blight fungus Cryphonectria parasitica was rece
5 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica were use
8 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica, could s
9 V-1/Euro7, and CHV-1/EP721, which infect the chestnut blight fungus Cryphonectria parasitica, differ
10 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica, encodes
11 silencing antiviral defense response in the chestnut blight fungus Cryphonectria parasitica, is indu
12 on and sporulation by the infected host, the chestnut blight fungus Cryphonectria parasitica, while b
17 nce attenuation) observed for strains of the chestnut blight fungus, Cryphonectria parasitica, harbor
19 We now report that DI RNA production in the chestnut blight fungus, Cryphonectria parasitica, persis
20 st characterized of a number of genes in the chestnut blight fungus, Cryphonectria parasitica, that a
21 itor global transcriptional responses of the chestnut blight fungus, Cryphonectria parasitica, to inf
22 ed phenotype, such as the hypoviruses of the chestnut-blight fungus, have been studied for their pote
23 he oah gene in Cryphonectria parasitica, the chestnut blight fungus, reduces the ability of the fungu
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