ライフサイエンスコーパス: childhood leukemia

1 to an increased risk of cancer, particularly childhood leukemia.

2 ration of breastfeeding and the incidence of childhood leukemia.

3 s associated with the most common subtype of childhood leukemia.

4 dicted RF-EMF exposure from broadcasting and childhood leukemia.

5 evaluate the association between allergy and childhood leukemia.

6 ctase 1 (NQO1), had no significant effect on childhood leukemia.

7 y of residential magnetic field exposure and childhood leukemia.

8 examined the association between NQO1*2 and childhood leukemia.

9 ears to be associated with a reduced risk of childhood leukemia.

10 ociation between magnetic field exposure and childhood leukemia.

11 sive smoking may be important in the risk of childhood leukemia.

12 ron supplement use may be protective against childhood leukemia.

13 thogenesis, natural history, and etiology of childhood leukemia.

14 cations in utero leading to infant and early childhood leukemia.

15 ated in human leukemias, but rarely in early childhood leukemias.

16 en identified in Noonan syndrome and various childhood leukemias.

17 Noonan syndrome and a significant portion of childhood leukemias.

18 ighlight parallels between DS AMKL and other childhood leukemias.

19 ich had long been recognized as disrupted in childhood leukemia, also plays a role in the CNS.

20 Relative to those survivors with childhood leukemia, an increased risk was observed for a

21 amples from a study of electrical wiring and childhood leukemia and a study of diet and glioma.

22 ty of combination chemotherapy to cure acute childhood leukemia and advanced Hodgkin's disease in the

23 Survivors of childhood leukemia and brain tumors, particularly those

24 kes that occur in > or = 5-year survivors of childhood leukemia and brain tumors.

25 widely used drugs for the treatment of acute childhood leukemia and chronic myelocytic leukemia.

26 rts describing the effect of NQO1 in de novo childhood leukemia and conducted a meta-analysis of 7 ca

27 the role of P-gp-mediated drug resistance in childhood leukemia and confirms that P-gp expression and

28 new development in the induction therapy of childhood leukemia and lymphoma in the United States is

29 a significant late toxicity of treatment for childhood leukemia and lymphoma.

30 nce known as "wire coding"), but not between childhood leukemia and measurements of 60-Hz residential

31 e suggested that meta-analyses of studies on childhood leukemia and proximity to gasoline stations sh

32 identified evidence of associations between childhood leukemia and several different potential metri

33 de novel insight into the natural history of childhood leukemia and suggest that consequent to a pren

34 Previous studies found associations between childhood leukemia and surrogate indicators of exposure

35 Studies of childhood leukemia and the potential etiologic role of g

36 f-function Shp2 mutations have been found in childhood leukemias and Noonan syndrome.

37 cell signaling pathways, are associated with childhood leukemias and solid tumors.

38 r all cancers, 0.55 (95% CI: 0.26, 1.19) for childhood leukemia, and 1.68 (95% CI: 0.98, 2.91) for ch

39 ment of further evidence for fetal origin of childhood leukemias, and additional evidence to support

40 associated with a markedly increased risk of childhood leukemias, and identification of chromosome 21

41 NX1) is the most common translocation in the childhood leukemias, and is a prenatal mutation in most

42 hatase) are associated with Noonan syndrome, childhood leukemias, and sporadic solid tumors.

43 Thiopurines are a standard treatment for childhood leukemia, but like all chemotherapeutics, thei

44 The present analysis included 327 acute childhood leukemia cases (281 acute lymphoblastic leukem

45 The study compared the 2,760 childhood leukemia cases diagnosed in France between 200

46 ent meta-analyses results, 14% to 19% of all childhood leukemia cases may be prevented by breastfeedi

47 e was associated with a reduction in risk of childhood leukemia diagnosed between the ages of 2 and 1

48 nuous doxorubicin infusion over 48 hours for childhood leukemia did not offer a cardioprotective adva

49 The molecular characterization of childhood leukemias directly affects our treatment strat

50 in methionine synthase could mediate risk of childhood leukemia, either via effects on DNA methylatio

51 ole of NQO1 polymorphisms in the etiology of childhood leukemia, especially among MLL-positive leukem

52 in C and/or potassium may reduce the risk of childhood leukemia, especially if they are consumed on a

53 a matched case-control study of the risk of childhood leukemia from exposure to residential electric

54 Recent identification of SHP-2 mutations in childhood leukemia further emphasizes the importance of

55 of birth, in contrast to previously studied childhood leukemia fusions, t(12;21), t(8;21), and t(4;1

56 ation between child's early diet and risk of childhood leukemia has remained largely unexplored.

57 Significant elevations in the risk of childhood leukemia have been associated with environment

58 s in therapy, primarily intensification, for childhood leukemias have significantly improved cure rat

59 ys the progress of epidemiologic research on childhood leukemia in 1996.

60 (first 2 years) are associated with risk of childhood leukemia in a 1995-2002 case-control study of

61 t has been associated with increased risk of childhood leukemia in Los Angeles and elsewhere in North

62 inone carcinogens, have been associated with childhood leukemia in some studies, although the observe

63 The odds ratios for developing childhood leukemia in the highest exposure category were

64 phoblastic leukemia, the predominant type of childhood leukemia in the United States and many Europea

65 An expert panel reviewed a cluster of childhood leukemias in Fallon, Nevada, and suggested the

66 tfeeding for 6 months or more may help lower childhood leukemia incidence, in addition to its other h

67 Remaining challenges in the treatment of childhood leukemia include 1) the development of specifi

68 tained from 12 case-control studies from the Childhood Leukemia International Consortium (CLIC, 1974-

69 11 case-control studies participating in the Childhood Leukemia International Consortium (enrollment

70 d data from 13 case-control studies from the Childhood Leukemia International Consortium done in nine

71 hed cause of adult leukemia, but its role in childhood leukemia is less clear.

72 motherapy, but its role in the management of childhood leukemia is unclear.

73 Childhood leukemia may be associated with traffic-relate

74 In contrast to childhood leukemia, no pooled analyses of childhood brai

75 22) is the most common gene rearrangement in childhood leukemia, occurring in approximately 25% of pe

76 ger was associated with a 19% lower risk for childhood leukemia (odds ratio, 0.81; 95% CI, 0.73-0.89)

77 ed was associated with an 11% lower risk for childhood leukemia (odds ratio, 0.89; 95% CI, 0.84-0.94)

78 V6-RUNX1), the most common fusion protein in childhood leukemia, on miRNA expression and the leukemic

79 No increase in childhood leukemia or other cancers has been documented.

80 d to be associated with an increased risk of childhood leukemia, particularly among young children.

81 40%) and 52 of 190 (27.4%) bone marrows from childhood leukemia patients demonstrated hypermethylatio

82 ng associations between parental smoking and childhood leukemia, possibly because previous studies us

83 ons and applied to a candidate gene study of childhood leukemia (Quebec Childhood Leukemia Study, 198

84 ortality; whether a similar effect exists in childhood leukemia remains controversial.

85 leukemia, but whether it is associated with childhood leukemia remains unclear.

86 the association between parental smoking and childhood leukemia remains unclear.

87 ic leukemia (ALL) is the most common form of childhood leukemia, representing 75% to 80% of cases of

88 llicit) during pregnancy might be related to childhood leukemia risk.

89 469 households from the Northern California Childhood Leukemia Study (1999-2007).

90 (n=567) enrolled in the Northern California Childhood Leukemia Study (NCCLS) compared with populatio

91 ollected from participants of the California Childhood Leukemia Study at various intervals from 1999

92 useholds enrolled in the Northern California Childhood Leukemia Study during 2001-2006, trained inter

93 om 293 households in the Northern California Childhood Leukemia Study during two sampling rounds (fro

94 e collected from 289 homes in the California Childhood Leukemia Study during two sampling rounds from

95 ate gene study of childhood leukemia (Quebec Childhood Leukemia Study, 1980-2000).

96 mia were examined in the Northern California Childhood Leukemia Study, a case-control study, between

97 etreatment tumor samples from the California Childhood Leukemia Study.

98 race/ethnicity from the Northern California Childhood Leukemia Study.

99 ata (1995-2002) from the Northern California Childhood Leukemia Study.

100 lasts of adult patients and early relapse in childhood leukemia, suggest that GRP78 is a novel therap

101 nation in the first year of life and risk of childhood leukemia (summary odds ratio (OR) 0.58 [95% co

102 Epidemiological studies of childhood leukemia survivors reveal an alarmingly high i

103 The Therapeutic Advances in Childhood Leukemia (TACL) Consortium was assembled to as

104 erinatal factors have been linked to risk of childhood leukemia, testicular cancer, and breast cancer

105 phoblastic leukemia (T-ALL) is an aggressive childhood leukemia that is caused by the accumulation of

106 tween paternal military service and risk for childhood leukemia, the authors analyzed data from three

107 e success of L-asparaginase as a therapy for childhood leukemia, the data suggest that intracellular

108 s study did not understand randomization for childhood leukemia trials.

109 1987 to 2014, the summary relative risk for childhood leukemia was 1.96 (95% confidence interval (CI

110 ng the association between breastfeeding and childhood leukemia was conducted on PubMed, the Cochrane

111 Parental smoking and the risk of childhood leukemia were examined in the Northern Califor

112 tion strategies were evaluated in a study of childhood leukemia, which commenced in California in 199

113 additional new evidence for associations of childhood leukemia with both residential proximity to ga

114 stigated recently include the association of childhood leukemia with infection and with birth weight.

115 meta-analysis, we identified associations of childhood leukemia with occupational and household produ