ライフサイエンスコーパス: childhood-onset schizophrenia

1 on-deficit/hyperactivity disorder (ADHD) and childhood-onset schizophrenia.

2 ttern of abnormalities seen in patients with childhood-onset schizophrenia.

3 r volume during adolescence in patients with childhood-onset schizophrenia.

4 is seen through adolescence in patients with childhood-onset schizophrenia.

5 e decreases were significantly correlated in childhood-onset schizophrenia.

6 ere assessed in a cohort of 49 patients with childhood-onset schizophrenia.

7 rbid developmental impairments are common in childhood-onset schizophrenia.

8 ral lobe abnormalities have been observed in childhood-onset schizophrenia.

9 on may be a new finding in the literature on childhood-onset schizophrenia.

10 d raw scores on subtests of 31 patients with childhood-onset schizophrenia.

11 ted with a diagnosis of treatment-resistant, childhood-onset schizophrenia.

12 be structures occurs with ongoing illness in childhood-onset schizophrenia.

13 een clinical and neurobiological measures of childhood-onset schizophrenia.

14 asymmetry as a basis for psychopathology in childhood-onset schizophrenia.

15 n severely ill children and adolescents with childhood-onset schizophrenia.

16 nd negative symptoms in treatment-refractory childhood-onset schizophrenia.

17 morphology was examined in adolescents with childhood-onset schizophrenia.

18 6 +/- 2.1 years; range, 10 to 18 years) with childhood-onset schizophrenia (13 males, eight females)

19 with schizophrenia, parents of patients with childhood-onset schizophrenia (95 parents), patients wit

20 so reported for a cohort of 47 patients with Childhood Onset Schizophrenia, a rare and severe form of

21 examined large-scale network interactions in childhood-onset schizophrenia, a severe form of the dise

22 Study of patients with childhood-onset schizophrenia allows the opportunity to

23 ch subject, in a sample of 106 patients with childhood-onset schizophrenia and 102 age-matched health

24 ly compared in 55 young adolescents (29 with childhood-onset schizophrenia and 26 with psychotic diso

25 The birth records of 36 patients with childhood-onset schizophrenia and 35 sibling comparison

26 mately 2-year intervals for 39 subjects with childhood-onset schizophrenia and 43 healthy subjects.

27 to test the hypothesis of continuity between childhood-onset schizophrenia and adult-onset schizophre

28 ly healthy full siblings of 15 patients with childhood-onset schizophrenia and from 32 matched commun

29 elopment during adolescence in patients with childhood-onset schizophrenia and healthy comparison sub

30 otherwise specified to that of children with childhood-onset schizophrenia and healthy comparison sub

31 In the present study, subjects with childhood-onset schizophrenia and healthy subjects were

32 ship of this group of children to those with childhood-onset schizophrenia and other childhood psychi

33 hood, by reviewing sibling studies from both childhood-onset schizophrenia, and the more common adult

34 Brain anatomic abnormalities in childhood-onset schizophrenia are similar to those repor

35 as independently replicated in patients with childhood-onset schizophrenia as compared with their par

36 enlargement for a group of adolescents with childhood-onset schizophrenia at 2-year follow-up, with

37 normalities in children and adolescents with childhood-onset schizophrenia by using whole-brain voxel

38 Childhood onset schizophrenia (COS), defined as onset be

39 rogressive cortical gray matter (GM) loss in childhood-onset schizophrenia (COS) across both lateral

40 atter (GM) loss is marked and progressive in childhood-onset schizophrenia (COS) during adolescence b

41 Childhood-onset schizophrenia (COS) is a rare and severe

42 genetic liability to schizophrenia and that childhood-onset schizophrenia (COS) is a variant of adul

43 This study tested the hypothesis that childhood-onset schizophrenia (COS) is a variant of adul

44 NTEXT Nonpsychotic siblings of patients with childhood-onset schizophrenia (COS) share cortical gray

45 f cortical gray matter (GM) in patients with childhood-onset schizophrenia (COS), which appears great

46 resonance imaging scans for 15 patients with childhood-onset schizophrenia (defined as onset of psych

47 scence in the full-scale IQ of patients with childhood-onset schizophrenia does not reflect dementia

48 Children with childhood-onset schizophrenia exhibit a pattern of eye-t

49 Thus, the childhood-onset schizophrenia group showed a distinctive

50 For the childhood-onset schizophrenia group, there was a decreas

51 Parents of patients with childhood-onset schizophrenia had a significantly higher

52 Subjects with childhood-onset schizophrenia had qualitatively poorer e

53 Patients with childhood-onset schizophrenia had significantly smaller

54 Siblings of patients with childhood-onset schizophrenia had smaller total cerebral

55 Parents of patients with childhood-onset schizophrenia have a higher rate of schi

56 volumetric studies of brain abnormalities in childhood-onset schizophrenia have revealed anomalies si

57 The present study shows that patients with childhood-onset schizophrenia have smaller than normal r

58 cross-sectional study of brain morphology in childhood-onset schizophrenia indicated sparing of the t

59 Childhood-onset schizophrenia is a rare but severe form

60 Childhood-onset schizophrenia is a rare but severe form

61 Childhood-onset schizophrenia is a severe and unremittin

62 Childhood-onset schizophrenia is associated with signifi

63 cale IQ during adolescence for patients with childhood-onset schizophrenia is due to a dementing proc

64 Although childhood-onset schizophrenia is relatively rare, a siza

65 ospinal fluid (CSF) monoamine metabolites in childhood-onset schizophrenia may help to elucidate both

66 ested that deficits in cortical thickness in childhood-onset schizophrenia may normalize over time, s

67 Developmental brain abnormalities in childhood-onset schizophrenia may thus be genetic trait

68 ing scans were acquired for 60 subjects with childhood-onset schizophrenia (mean age=14.5 years, SD=2

69 The patient groups include childhood-onset schizophrenia (n = 22), obsessive-compul

70 N=95, mean age=11.7 years) and patients with childhood-onset schizophrenia (N=24, mean age=14.6 years

71 of some neurodevelopmental disorders such as childhood-onset schizophrenia or autism.

72 in magnetic resonance scans were obtained in childhood-onset schizophrenia probands (N=89, 198 scans)

73 Childhood-onset schizophrenia probands had a fixed reduc

74 Healthy siblings of patients with childhood-onset schizophrenia share brain MRI abnormalit

75 ne whether healthy siblings of patients with childhood-onset schizophrenia show structural brain abno

76 Childhood-onset schizophrenia shows progressive brain ma

77 etic abnormalities appear to be increased in childhood-onset schizophrenia, suggesting an association

78 were significantly higher for patients with childhood-onset schizophrenia than for healthy compariso

79 tected a dynamic wave of gray matter loss in childhood-onset schizophrenia that started in parietal a

80 In childhood-onset schizophrenia, the rate of gray matter r

81 ared with olanzapine in treatment-refractory childhood-onset schizophrenia, the study suggests that c

82 volumes in relation to age for patients with childhood-onset schizophrenia, their nonpsychotic health

83 l records were examined for individuals with childhood-onset schizophrenia to determine if birth comp

84 e authors performed 1H-MRSI of patients with childhood-onset schizophrenia to specifically test wheth

85 ties in autonomic nervous system activity in childhood-onset schizophrenia to those found in adult ch

86 te of gray matter reduction in patients with childhood-onset schizophrenia was examined in relation t

87 Brain abnormalities in childhood-onset schizophrenia were examined in relation

88 ts participating in a comprehensive study of childhood-onset schizophrenia were screened for chromoso

89 Fixed hippocampal volume loss seen in childhood-onset schizophrenia, which is not shared by he

90 Caudate enlargement in patients with childhood-onset schizophrenia who are taking typical neu

91 ned the planum temporale of adolescents with childhood-onset schizophrenia who had a high rate of pre

92 treatment-refractory sample of patients with childhood-onset schizophrenia who had had more prepsycho

93 in a group of children and adolescents with childhood-onset schizophrenia who had severe, chronic sy