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1 complementation assay to screen against this chimeric receptor.
2 e specificities not directly targeted by the chimeric receptor.
3  in the sustained signaling activity of this chimeric receptor.
4 sts demonstrated greater EC50 values for the chimeric receptor.
5  to compete for (33)P-relaxin binding to the chimeric receptor.
6 tations of signaling exclusively through the chimeric receptor.
7  the activation of PLCgamma mediated by this chimeric receptor.
8 tracytoplasmic portion of the IL-2R via this chimeric receptor.
9 yrosines contributed to the function of this chimeric receptor.
10 toreactive T cells by a MBP89-101-IA(s)-zeta chimeric receptor.
11 ell receptor and tumor targets through their chimeric receptors.
12  domain is critical for proliferation of the chimeric receptors.
13 based reporter assay employing LXRalpha-GAL4 chimeric receptors.
14 nic receptors was evaluated at wild-type and chimeric receptors.
15  stimulating hormone to bind or activate the chimeric receptors.
16 necessary to impart full BZD potentiation to chimeric receptors.
17 nd antitumor activity mediated through their chimeric receptors.
18 CCR5 inhibitor binding site using CCR5/CCR2b chimeric receptors.
19 R/syntrophin interaction was confirmed using chimeric receptors.
20 ce expression and abrogated signaling of the chimeric receptors.
21 t1 fibroblasts transfected with EGFR or HER2 chimeric receptors activated by synthetic ligands withou
22                             Testing of these chimeric receptors against a battery of AIP analogs loca
23 he melatonin-related receptor, the resultant chimeric receptors all displayed specific 2-[(125)I]iodo
24 isulfide-mediated oligomerization of another chimeric receptor, alpha zetazeta, enhances signaling.
25                                              Chimeric receptor analyses raise the possibility that th
26                       The combination of the chimeric receptor and GFP-Smad2 makes it possible to fol
27                                              Chimeric receptor and molecular modeling studies also we
28 D8(+) T cells were engineered to express the chimeric receptor and were costimulated ex vivo with bea
29 se differences was investigated by preparing chimeric receptors and by site-directed mutagenesis.
30 18 inhibition of cells expressing CXCR4/CCR5 chimeric receptors and CCR5 with a truncated N terminus
31 esponsible for this selectivity, a series of chimeric receptors and mutant receptors was constructed
32  its ligand-selective activation, we assayed chimeric receptors and receptor variants containing subs
33 d this dichotomy through the construction of chimeric receptors and site-directed mutagenesis in orde
34                                              Chimeric receptors and structurally guided amino acid su
35 eptor is tyrosine phosphorylated through the chimeric receptors and the endogenous IL-6 and OSM recep
36  gastrin-releasing peptide (GRPR), we used a chimeric receptor approach and a site-directed mutagenes
37 n and therefore validated the utility of the chimeric receptor approach for signaling pathway identif
38                          Here we have used a chimeric receptor approach in which the extracellular do
39        To address this issue, we developed a chimeric receptor approach that permits a structure/func
40 To investigate the basis for this, we used a chimeric receptor approach to make both GRPR loss of aff
41                                            A chimeric receptor approach was utilized to identify key
42                        Nevertheless, using a chimeric receptor approach, we previously found that RYK
43 procally, reporter cells with a CD3zeta-LLT1 chimeric receptor are stimulated by NKR-P1A.
44                                     Although chimeric receptors are not pathogenic per se, the freque
45 specifically validate humanized MBP-DR2-zeta chimeric receptors as a potential therapeutic in MS.
46 scence microscopy revealed that internalized chimeric receptors, as identified with fluorescent ligan
47  binding specificity of alpha(L)beta(2), the chimeric receptor became competent to support cell migra
48                                         This chimeric receptor became localized to detergent-insolubl
49                                  All but one chimeric receptor bound IL-8 and mediated signal transdu
50                                          The chimeric receptor bound radiolabeled TGF-beta and could
51                                        These chimeric receptors bound CPV capsids and mediated uptake
52 tion induces tyrosine phosphorylation of the chimeric receptor but does not enhance its binding to SH
53 ecific antibody OKT3 and expressing the same chimeric receptor but lacking virus specificity.
54            Furthermore, cells expressing the chimeric receptor, but not alpha(L)beta(2), were able to
55  demonstrate activation of a TREM-2A/CD3zeta chimeric receptor by both bacteria and dextran sulfate.
56 ty of a constitutively active RARgamma-VP-16 chimeric receptor by the inverse agonist AGN193109 requi
57                            Engagement of the chimeric receptor by the TCR of autoreactive T cells act
58 l domain of SV2A or SV2B alone, expressed in chimeric receptors by replacing the extracellular domain
59                  In THP-1 cells expressing a chimeric receptor CD4-TLR4, which triggers constitutive
60  B cell lines expressing the human CD40-LMP1 chimeric receptor, CD40- and LMP1-mediated NF-kappaB and
61                                              Chimeric receptor chains of the gammaC tail fused to the
62 rm of DEP-1 formed a stable complex with the chimeric receptor colony stimulating factor 1 (CSF)-Met
63 t effective of these interventions created a chimeric receptor combining the ligand-binding domain of
64 myoblast proliferation in situ, we created a chimeric receptor composed of a modified FK506-binding p
65      Second, we use a recently characterized chimeric receptor composed of rhodopsin and the beta2-ad
66 ith the AChBP, Torpedo californica nAChR and chimeric receptor composed of the alpha7 nAChR extracell
67                                            A chimeric receptor composed of the extracellular domain o
68 e first, or regulatory, cassette comprises a chimeric receptor composed of the hinge and ligand bindi
69                                            A chimeric receptor composed of the N-terminal extracellul
70                         Here, we generated a chimeric receptor composed of the TrkB extracellular dom
71 y of PC12 cell lines stably transfected with chimeric receptors composed of the extracellular domain
72 C12 cell lines expressing similar amounts of chimeric receptors composed of the extracellular domain
73  hypothesis, we created ligand-regulated TLR chimeric receptors composed of the extracellular region
74                            Cotransfection of chimeric receptors comprised of the extracellular region
75                     CD8 T cells expressing a chimeric receptor comprising Ly49H extracellular and CD3
76            We show that wild-type Rfz2 and a chimeric receptor consisting of the extracellular and tr
77 M1 failed to induce autophosphorylation of a chimeric receptor consisting of the extracellular domain
78 -15Ralpha cytoplasmic domain, we generated a chimeric receptor consisting of the extracellular domain
79  dimer peptide, but not IL-5, can activate a chimeric receptor consisting of the IL-5Ralpha extracell
80          We observed that cross-linking of a chimeric receptor consisting of the murine CD28 extracel
81                                            A chimeric receptor construct is utilized to show that cle
82                                      Using a chimeric receptor construct, we provide evidence that pl
83                                              Chimeric receptors constructed from receptors with oppos
84                                              Chimeric receptors, constructed by switching the cytopla
85 in and p75(NTR), we analyzed binding between chimeric receptor constructs and truncated p75(NTR) vari
86 ansmembrane domains to AGRP binding by using chimeric receptor constructs of the human melanocortin-1
87 both receptors, we engineered a series of 14 chimeric receptor constructs, allowing us to determine t
88 these receptors using LRP minireceptors, its chimeric receptor constructs, and full-length VLDLR and
89 nsulin receptor and a mutated version of the chimeric receptor containing a 12-amino acid deletion of
90 ary CD4+ T cells expressing a CD8 alpha/CD28 chimeric receptor containing a mutation at tyrosine 200
91 essed, in bone marrow-derived macrophages, a chimeric receptor containing a range of tyrosine to phen
92 ammaRII(b)(-/-) mice or upon clustering of a chimeric receptor containing CD8 and the immunoreceptor
93 ensitizes signaling mediated by a transduced chimeric receptor containing extracellular domains of pl
94 ionally significant, we transduced EC with a chimeric receptor containing extracellular domains of pl
95  GP Ibalpha was inhibited by co-expressing a chimeric receptor containing interleukin 2 receptor alph
96 in the receptors, individual exoloops of the chimeric receptor containing the ectodomain of the LH re
97 uate potential ligand activators of SmRK1, a chimeric receptor containing the extracellular domain of
98                    We recently constructed a chimeric receptor containing the extracellular domain of
99 cient to block EGF-mediated stimulation of a chimeric receptor containing the intracellular domain of
100 s proliferation in vitro and activation of a chimeric receptor containing the TACI intracellular doma
101          T cell lines expressing CD8alpha/28 chimeric receptors containing a mutation in tyrosine 173
102                    In this work we show that chimeric receptors containing amino acids 1-1,680 of RyR
103                                         Only chimeric receptors containing both JAK2 kinase activity
104  in the V3 crown had fusogenic activity with chimeric receptors containing either the N terminus or l
105         To test this hypothesis we expressed chimeric receptors containing fragments of Sstr3 and Htr
106 oped a functional assay utilizing a panel of chimeric receptors containing the extracellular and tran
107               The generation and analysis of chimeric receptors containing the extracellular recognit
108 stoma cells when compared with expression of chimeric receptors containing wild-type gp130 cytoplasmi
109                                            A chimeric receptor, containing the extracellular domain o
110              We produced mice that express a chimeric receptor, containing the juxtamembrane region o
111 loss of transactivation seen with the mutant chimeric receptor correlated with a decrease in Vav tyro
112                                              Chimeric receptors created from distant members of the h
113 characterize this survival pathway we used a chimeric receptor (CSF1R/IR) consisting of the ligand-bi
114                                          The chimeric receptor CXP, in which the NT of mCRFR1 was ann
115                                        Using chimeric receptor DBDs, the higher intrinsic DNA binding
116 s expressing the dileucine-mutated CD28-zeta chimeric receptor demonstrated enhanced proliferation, c
117  compounds for as little as 15 s resulted in chimeric receptor dimerization detectable as beta-gal en
118 tance by transducing CD56+CD3- NK cells with chimeric receptors directed against CD19, a molecule wid
119                                          The chimeric receptor displayed a pharmacological binding pr
120 eta4 signaling, however, dimerization of the chimeric receptor does not activate either Akt or Erk1/2
121 d neutralization by engineered antibodies or chimeric receptors, downregulation of its expression wit
122 two receptors intact, we, recently developed chimeric receptors (EGDR and EGLT) in which the extracel
123                                    Using the chimeric-receptor EGLT in which the extracellular domain
124               In this study we constructed a chimeric receptor, EGNP-1, by fusing the extracellular d
125  behind these divergent responses, we made a chimeric receptor (ErbB1/2) composed of the extracellula
126 nstitution experiments demonstrated that the chimeric receptor, even in the phosphorylated state, exi
127                            The generation of chimeric receptors exchanging different domains of noncl
128                                              Chimeric receptor experiments demonstrated that the extr
129                 Fluorescence, because of the chimeric receptors expressed, was over the whole oocyte
130                                The alpha(FR) chimeric receptor fails to rescue any aspect of the PDGF
131                       After endocytosis, the chimeric receptor first enters sorting endosomes, and it
132  differences, we constructed two Flag-tagged chimeric receptors, Flag-h/rkor and Flag-r/hkor, in whic
133       For this purpose we have constructed a chimeric receptor for VEGFR-1 (CTR) and VEGFR-2 (CKR) in
134 ion, a method for tailoring spacer length of chimeric receptors for optimal function, and a functiona
135 nd should prove useful in the development of chimeric receptors for therapeutic purposes.
136  we first studied four loss-of-affinity GRPR chimeric receptors formed by exchanging the four extrace
137 ignaling in a cellular context, a c-fms-Tie1 chimeric receptor (fTie1) was expressed in NIH 3T3 cells
138 ate that inactivation of this motif enhances chimeric receptor function, and illustrate a potential n
139                        Nitrite activated the chimeric receptor Gal-ER containing the DNA-binding doma
140 er ex vivo expansion and transduction with a chimeric receptor gene (CD4/CD3-zeta) between identical
141                               Examination of chimeric receptors, generated by domain exchange between
142 mary) T cells transduced with tumor-specific chimeric receptor genes can be expanded and maintained l
143                                            A chimeric receptor, GerU*, demonstrates interchangeabilit
144            However, 32D cells expressing the chimeric receptor had less IL-15-induced proliferation t
145                              Moreover, these chimeric receptors had a fast-desensitizing component, e
146                             Since studies of chimeric receptors have indicated that the extracellular
147                                            A chimeric receptor (hGM/beta(c)), comprising the extracel
148           Expression of active Notch1/Notch3 chimeric receptors implicated the RBPjk-association mole
149 ne receptor (nAChR) agonist activity using a chimeric receptor in a functional, cell-based, high-thro
150                              By expressing a chimeric receptor in an NK-like cell line, we found that
151                           Expression of this chimeric receptor in beta4-null cancer cells enabled us
152 ther than CD28 could be included in a single chimeric receptor in series with TCRzeta to mediate the
153 ptor (TfR/SR-A) resulted in retention of the chimeric receptor in the endoplasmic reticulum suggestin
154        The subsequent testing of a "reverse" chimeric receptor in which sequences encoding transmembr
155                                   However, a chimeric receptor in which the DD of CD120a was fused to
156 nities are similar to those for binding to a chimeric receptor in which the ECD1 of CRFR2beta replace
157  gain insight into this mechanism, we used a chimeric receptor in which the ligand binding domain of
158 emonstrated by its weakened antagonism for a chimeric receptor in which the membrane-spanning domains
159              An analogous completely foreign chimeric receptor in which the Y440 motif is replaced wi
160               Using full-length and Gal4-LBD chimeric receptors in functional assays, 20:4,n6 induced
161 e homology and equivalent expression by both chimeric receptors in the ventral prostate gland, only F
162 s pharmacological difference, we constructed chimeric receptors in which individual extracellular loo
163                                              Chimeric receptors in which residues 310-312 were replac
164 CH-8 cells were transfected with CXCR1/CXCR2 chimeric receptors in which the 40-amino acid C-terminal
165                                        Using chimeric receptors in which the C terminus of PAR(2) is
166         To test this premise, we constructed chimeric receptors in which the extracellular and transm
167                              We next studied chimeric receptors in which the pattern of beta-arrestin
168                         Experiments with the chimeric receptors indicate that Shc also signals to the
169 ; site-directed mutagenesis in wild-type and chimeric receptors indicated that the threonine residue
170                                       Use of chimeric receptors indicates that the CSF-1 receptor is
171  Upon arginine vasopressin stimulation, this chimeric receptor induced robust calcium mobilization an
172                                          The chimeric receptor initiates plant defense responses in r
173                  Introduction of EphB1-EphB2 chimeric receptors into RGCs reveals that both extracell
174                                          The chimeric receptor is unable to amplify growth factor-med
175                                  We analyzed chimeric receptor kinases by fusing CLV1 and BRASSINOSTE
176                                     However, chimeric receptors lacking an intact box2 domain of Mpl
177                                              Chimeric receptor LGR7/8 with the ectodomain from LGR7 b
178                                We designed a chimeric receptor linking NKG2D, a natural killer (NK) c
179                                        These chimeric receptors manifest both nuclear translocation a
180                                 This type of chimeric receptor may now be used to discover the most p
181                                      Using a chimeric receptor model where the granulocyte/monocyte c
182                                 Finally, the chimeric receptor-modified CTL ameliorated or blocked ex
183 l, its cytoplasmic zeta-chain stimulates the chimeric receptor-modified T cell (RMTC).
184 pecific ligand binding regions in mutant and chimeric receptor molecules.
185                                        Using chimeric receptors, mutagenesis, and molecular modeling,
186        Using the antibody and point/deletion/chimeric receptor mutants, we demonstrate that changes i
187  for the IFN-gamma-like response through the chimeric receptors, nor does it mediate an IFN-gamma-lik
188     Using a series of deletion mutations and chimeric receptors of p75(NTR) and the related Fas recep
189 en coupled to the green fluorescent protein, chimeric receptors offer a powerful new tool to 1) study
190  required the presence of the tumor-specific chimeric receptor on T cells.
191 rated cell lines that express either EGFR-H2 chimeric receptor or HER2 and HER3 receptors in an EGFR-
192            Using ErbB-2 mutants expressed as chimeric receptors or green fluorescent protein fusion p
193 imulating factor receptor (GCSFR)-gp130(133) chimeric receptor, overexpression of Stat3 induced gamma
194                               The reciprocal chimeric receptor (P1-NP2) was not activated by TIP39 an
195  with their PTH1R counterparts resulted in a chimeric receptor, PEC, which had normal CRFR1 functiona
196    An epidermal growth factor receptor/gp130 chimeric receptor previously shown by us to transactivat
197 elium is rescued by an exogenously expressed chimeric receptor (prl-EpoR) containing the PrlR extrace
198 rly, TCR transgenic CD8 cells expressing the chimeric receptor produced higher effector numbers durin
199                   CD8 T cells expressing the chimeric receptor protect mice in vivo from lethality in
200 st a functionally unimportant component of a chimeric receptor protein.
201 s using the EIAV SU gp90 protein and various chimeric receptor proteins derived from exchanges betwee
202  transduction by the gamma-like chains using chimeric receptor proteins.
203 ng T cells to attack cancer using engineered chimeric receptors provides powerful new therapeutic cap
204                                              Chimeric receptors providing B7 and TNFR family costimul
205 constitutively activated RARalpha or RARbeta chimeric receptors (RARVP16) in branching airways of tra
206 lar loop (E3) in the rat BRS-3 resulted in a chimeric receptor (RB3-E3) that behaved almost identical
207  reporter cells expressing a CD3zeta-NKR-P1A chimeric receptor; reciprocally, reporter cells with a C
208  modification of alloreactive T cells with a chimeric receptor recognizing folate-binding protein, an
209                           Expression of this chimeric receptor reduced hippocampal lesion size after
210                            By using the Gal4 chimeric receptor/reporter cotransfection system, the li
211                           Expression of this chimeric receptor restores presynaptic and postsynaptic
212                          Mice expressing the chimeric receptor retain a severe bleeding phenotype, co
213 tory function in a single receptor maximizes chimeric receptor sensitivity and potency.
214         Analysis of FGF8 and FGF9 binding to chimeric receptors showed that a broad region spanning I
215 eceptor cocapping studies indicate that this chimeric receptor signals T cells independently of the r
216                   Primary T cells expressing chimeric receptors specific for tumor or viral antigens
217                                Next, using a chimeric receptor strategy, we found that two independen
218                            This was shown by chimeric receptor studies in recombinant cell line assay
219                                              Chimeric receptor studies support the importance of this
220                         In addition, through chimeric receptor studies, we determined that the second
221 g a set of alpha7-5-hydroxytryptamine type 3 chimeric receptor subunit cDNAs, we expressed these cons
222                                              Chimeric receptor subunits and site-directed mutants wer
223  replaced with the I domain of alpha(M), the chimeric receptor supported cell migration to Fg; howeve
224         Inactivation of this motif increased chimeric receptor surface expression 2- to 5-fold.
225 ly, we used an erythropoietin (EPO) receptor chimeric receptor system in which IL-2-dependent HT-2 T
226 h these two receptors intact, we developed a chimeric receptor system in which the N terminus of the
227    T-cell precursors transduced to express a chimeric receptor targeting hCD19 resulted in significan
228 ns to activate the TCR or a CD8alpha/CD3zeta chimeric receptor (termed CD8zeta).
229                 Importantly, expression of a chimeric receptor that consisted of the extracytoplasmic
230 xpression of transgenic wild-type IL-7R or a chimeric receptor that consisted of the extracytoplasmic
231 s of IL-2 from PBT than a similar transduced chimeric receptor that contains a wild-type CD28 CYT.
232          First, Argos is unable to inhibit a chimeric receptor that contains an extracellular domain
233 nically expressed on T cells a heterodimeric chimeric receptor that genetically links an autoantigeni
234 m and TMB-8 had much higher affinity for the chimeric receptor that included the M(2) second outer lo
235 The cells are engineered with a cell-surface chimeric receptor that presents the nonmammalian enzyme
236  with that of the rP2Y(4) receptor yielded a chimeric receptor that was activated fully by UTP and ne
237 quirement for Wnt ligand can be abrogated by chimeric receptors that allow formation of Frizzled-LRP
238 embrane domains 4 or 6, however, resulted in chimeric receptors that displayed no detectable 2-[(125)
239 ocket was obtained in signaling studies with chimeric receptors that exhibited improved responses to
240                                              Chimeric receptors that include CD28 signaling in series
241 ors, we constructed a series of single-chain chimeric receptors that incorporate extracellular human
242 atory cells are transgenically modified with chimeric receptors that link antigen-major histocompatib
243               These modified T cells express chimeric receptors that link ligand recognition and sign
244 When coexpressed with dIRS in COS-7 cells, a chimeric receptor (the extracellular domain of human IR
245 ons of TM4 and TM5 than for any of the other chimeric receptors (the affinities of which remained sim
246                                        These chimeric receptors then were used in binding and entry a
247 one of the alphav subunit did not enable the chimeric receptor to bind alphaIIbbeta3-specific ligands
248                                  Here we use chimeric receptors to address the mechanisms of PIR-A si
249                               We constructed chimeric receptors to dissect the role of the transmembr
250 developed a cellular immunotherapy that uses chimeric receptors to selectively redirect therapeutic T
251                 We also demonstrate that two chimeric receptors, Torextracellular-Egfrcytoplasmic and
252            In response to IL-2 binding, this chimeric receptor transduces IL-4-specific signals and d
253                                      Several chimeric receptor transgene constructs where domains of
254 IA was exacerbated in IL-2R beta/IL-4R alpha chimeric receptor transgenic mice, with increased diseas
255 us, CD8 and CD4 T cells transduced with this chimeric receptor underwent an enhanced proliferative re
256                    Upon IL-8 activation, the chimeric receptors underwent receptor phosphorylation an
257                            Activation of the chimeric receptor upon the addition of M-CSF readily med
258 rosine was efficiently phosphorylated in the chimeric receptor upon treating the cells with pervanada
259       The pattern of phenotype rescue by the chimeric receptors used in this study supports a model w
260 or-related receptor 1 (VRR1) by generating a chimeric receptor, V1a/VRR1.
261                             We constructed a chimeric receptor (VEGFR2Fas) combining the extracellula
262               Our results indicate that each chimeric receptor was expressed at the cell surface and
263                             Furthermore, the chimeric receptor was internalized and recycled at rates
264                                 However, the chimeric receptor was still capable of binding to a RGD
265                                         This chimeric receptor was used to conduct an innovative frag
266 for the ALV-J receptor activity, a series of chimeric receptors was created by exchanging the extrace
267                         The dose response of chimeric receptors was ligand specific.
268                            Using a series of chimeric receptors, we demonstrated that this ligand act
269                                        Using chimeric receptors, we mapped the region for this differ
270              Transgenic animals expressing a chimeric receptor were generated and bred into the murin
271 interleukin-3-dependent cell line Ba/F3, the chimeric receptors were appropriately expressed on the c
272                                              Chimeric receptors were constructed in which the cytopla
273 eptor (and to further study the mGluRs), two chimeric receptors were constructed in which the large E
274 address this, alpha(2A)/alpha(2C) third loop chimeric receptors were constructed.
275                                     Finally, chimeric receptors were engineered containing the extrac
276 ical residues, site-directed mutagenesis and chimeric receptors were evaluated in functional calcium
277                                        These chimeric receptors were expressed in hamster cells.
278 hese three ligands for the two related LGRs, chimeric receptors were generated to elucidate the mecha
279                          Human CD33-specific chimeric receptors were generated with CD28, inducible c
280                                          The chimeric receptors were mostly M(5) sequence; the amount
281 virus receptor interaction on infection, two chimeric receptors were prepared which contained antibod
282                     To test this hypothesis, chimeric receptors were required in which the cytoplasmi
283                        From this library, 80 chimeric receptors were tested against 26 odorants after
284                                              Chimeric receptors were used to reconstitute development
285 ally, proline recognition was conferred to a chimeric receptor when TM regions associated with the pu
286 so conferred potent alphaBgtx sensitivity to chimeric receptors when co-expressed with the beta4 subu
287                                     However, chimeric receptors, which are composed of combinations o
288  Modulating the signaling characteristics of chimeric receptors will be important for their applicati
289               Third, a retroviral transduced chimeric receptor with a CD28 CYT that is specifically u
290  Long-term treatment of cells expressing the chimeric receptor with agonists, antagonists, and invers
291 ation of neurons transfected with a Met-Robo chimeric receptor with Hepatocyte growth factor leads to
292  created by engineering T cells to express a chimeric receptor with high affinity for human mesotheli
293  in parallel sets of mice as IgM, IgG1, or a chimeric receptor with IgM extracellular domains and tra
294                             We constructed a chimeric receptor with the ligand-binding and transmembr
295                                              Chimeric receptors with the ectodomain and exoloop 2, bu
296 ptor subunit to Stat recruitment we employed chimeric receptors with the extracellular domain of eith
297                                              Chimeric receptors with the hCaR VFT and mGluR1 seven-tr
298                   We constructed functional, chimeric receptors with the ligand-binding and transmemb
299                                        Using chimeric receptors with wild-type and mutant Ig alpha cy
300 FcR vs NKG2D-DAP10) or ectopically expressed chimeric receptors (with ITAM-containing cytoplasmic tai

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