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1 tertiary alcohols or C-tertiary amines using chloramine.
2 ably, OSCN(-) reacts faster than SCN(-) with chloramines.
3 moke might promote nucleobase oxidation by N-chloramines.
4  to yield long-lived, selectively reactive N-chloramines.
5 ve and quantitative determination of organic chloramines.
6 st hydrogen peroxide, hypochlorous acid, and chloramines.
7  is of significance because they are organic chloramines, a family of compounds that is perceived to
8                The reaction pathway involves chloramine and aldehyde intermediates, and converts the
9  peaks correlated with the formation of an N-chloramine and the product of its subsequent reaction wi
10 nts of HOBr produced by the decomposition of chloramines and bromamines.
11 s, we propose a reaction mechanism involving chloramines and chloramides as reaction intermediates, w
12           Net formation and decomposition of chloramines and cyanogen chloride (CNCl) were measured f
13 pathways in both cases (i.e., reactions with chloramines and free chlorine).
14       These results indicate that long-lived chloramines and HOCl generated by myeloperoxidase can ge
15 fter the AOP from reactions between residual chloramines and NDMA precursors in the AOP product water
16 labeled dT(40) and SSB was also generated by chloramines and the complete myeloperoxidase-hydrogen pe
17 er (NOM) concentrates treated with chlorine, chloramine, and medium pressure ultraviolet (UV) irradia
18 tion of apoB lysine residues, formation of N-chloramines, and increases in the relative electrophoret
19 nts were the dominant source of NDMA and its chloramine- and ozone-reactive precursors.
20 (RO) permeate often includes the de facto UV/chloramine AOP because chloramines applied upstream perm
21 hlorine AOP and approximately 35% for the UV/chloramine AOP such that (*)OH was generally more import
22                    The performance of the UV/chloramine AOP was comparable to the UV/H2O2 AOP for deg
23 ludes the de facto UV/chloramine AOP because chloramines applied upstream permeate RO membranes.
24 e the principal initial targets of HOCl, and chloramines are a significant product.
25 -offs between Legionella and mycobacteria if chloramines are applied as secondary disinfectant residu
26                                              Chloramines are the second most used secondary disinfect
27 an occur in water distribution systems where chloramines are used as the disinfectant.
28 ons were incorporated into a widely accepted chloramine autodecomposition model, providing a comprehe
29 inogenic N-nitrosamines as byproducts during chloramine-based drinking water disinfection.
30 ard free energies of formation for inorganic chloramines, bromamines, and bromochloramines, based on
31 acity of the hemodialysis equipment to clear chloramine can vary as a function of external factors.
32                                              Chloramine chemistry is complex, with a variety of react
33                     Effects of disinfectant (chloramines, chlorine), water age (2.3 days, 5.7 days),
34     Model system studies demonstrated that N-chloramines convert uracil to 5-chlorouracil.
35 otolysis) accounted for approximately 50% of chloramine decay and approximately 80% of free chlorine
36                     After formation, organic chloramines degrade into aldehydes, nitriles and N-chlor
37 artmannella vermiformis and the depletion of chloramine disinfectant residuals by nitrifying bacteria
38 h chlorine, ozone, chlorine dioxide, UV, and chloramine disinfection are critically reviewed.
39 , conventional practices, including chlorine-chloramine disinfection, flushing of DWDS, nutrient remo
40 ammonia are rapidly converted to bromine and chloramines during chlorination.
41              Typically, electron-deficient N-chloramines effectively react with aryne intermediates g
42                                              Chloramines eliminated culturable Legionella and prevent
43                                              Chloramines eventually decompose to irreversibly damage
44 using chlorine as a primary disinfectant and chloramines for maintenance of a distribution system res
45 s initially chlorinated, which suggests that chloramine formation is the first step in tyrosine chlor
46 e oocyte recording medium identified organic chloramines formed by the interaction of HOCl with HEPES
47                                 In addition, chloramines formed by the interaction of HOCl with tauri
48 sodes were caused by inadequate clearance of chloramine from the tap water used for portable dialysis
49      In this paper, the formation of organic chloramines from chlorination of lysine, tyrosine and va
50 is proposed to be a Lys-epsilonNH-Cl (lysine chloramine) from reaction of enzyme-generated HOCl with
51 s with taurine chloramine or with long lived chloramines generated from modified Lys or Arg.
52 l-induced apoptosis is mediated by aminoacyl chloramines generated in the culture media and can be mi
53                                              Chloramines in drinking water may form N-nitrosodimethyl
54 onstrate that SCN(-) reacts efficiently with chloramines in small molecules, in proteins, and in Esch
55     Using a reliable test method to identify chloramines in the water prior to entering the hemodialy
56  its reactive intermediates (such as organic chloramines) inhibit ENaC by affecting channel gating, w
57                         This covalent enzyme chloramine likely plays a key role in directing regiospe
58   These reactions of SCN(-) and OSCN(-) with chloramines may repair some of the damage that is inflic
59  HE was exposed to hypochlorous acid (HOCl), chloramines, MPO/H2O2/chloride, and activated human neut
60 has been developed by aryne insertion into N-chloramine, N-bromoamine, and N-iodoamine bonds via two
61  relative rates of chlorination at amine and chloramine nitrogens are a function of pH and depend on
62      During formation potential testing with chloramines, nitrosamine yields from IOM were measured f
63 (precursor test conducted in the presence of chloramines) of TSNAs in treated water from two wastewat
64 of UV-C and UV-C/H(2)O(2) in the presence of chloramines on the N-nitrosodimethylamine formation pote
65 o commonly applied disinfectants, copper and chloramines, on water chemistry and the growth of Legion
66  mimicked by treatment of cells with taurine chloramine or with long lived chloramines generated from
67 (-) and I(-) after disinfection by chlorine, chloramines, or ozone.
68 rometry (LC-MS) to demonstrate that HOCl and chloramines oxidize GSSG to two irreversible products in
69 one may serve as a key branching point, with chloramines promoting the formation of dichloroacetamide
70 mbient specific and total N-nitrosamines and chloramine-reactive and ozone-reactive N-nitrosamine pre
71 specific and total N-nitrosamines as well as chloramine-reactive and ozone-reactive precursors in 47
72 itidine substantially increased NDMA and its chloramine-reactive precursors in urine.
73  involved synthesis of polymers bearing less chloramine-reactive quaternary ammonium groups with dipr
74  polymer-bound tertiary amine groups to less chloramine-reactive quaternary ammonium groups.
75 se DBPs measured after re-establishment of a chloramine residual within the facilities than prior to
76 vailable sulfonyl nitrenoid sources known as chloramine salts (R3SO2NClNa), essentially by simply sti
77 hat incorporated subsequent free chlorine or chloramine scavenging by the (*)Cl and (*)OH daughter ra
78 chloramine transport profiling measured by a chloramine-sensitive microelectrode revealed a broader d
79                          Dichloramine is the chloramine species known to react with NDMA precursors t
80 ctivation of sqKv1A channels in oocytes, and chloramine T (Chl-T) accelerated inactivation.
81    125I-labeled-IGF-I was prepared using the chloramine T method.
82  These products could then be iodinated with chloramine T to give the 125I-derivatives.
83 se of labeling human FGF-1 with Na(125)I and chloramine T, it was observed that the protein lost its
84 te was mimicked by the thiol-oxidizing agent chloramine T.
85 o methionine oxidation caused by the oxidant chloramine-T (Ch-T) without altering other functional ch
86 tion targets in catalase were detected using chloramine-T as a milder oxidant.
87 es, we labeled protomeric fibronectin by the chloramine-T method or with 125I-Bolton-Hunter reagent,
88 was compared with (131)I-RS7 (labeled by the chloramine-T method) in paired-label biodistribution stu
89 furo[2,3-b]pyridine 4 was synthesized by the chloramine-T method.
90 on to 131I linked to MAb by the conventional chloramine-T methodology.
91 the oxidant, which is a fluorous analogue of chloramine-T, was obtained.
92 block of persistent late hNav1.4 currents in chloramine-T-pretreated cells by mexiletine was 7.5 +/-
93 postulated to involve the actions of taurine chloramine (Tau-Cl) derived via halide-dependent myelope
94                                      Taurine chloramine (TN-Cl) is one of the most abundant compounds
95 regeneration upon Cl[+1] transfer from their chloramines to tyrosines.
96  more cytotoxic than those treated with only chloramine using either dose metric.
97                      Samples treated with UV+chloramine were more cytotoxic than those treated with o
98                                              Chloramines were applied to organic precursors serving a
99 let (UV) irradiation followed by chlorine or chloramine, with and without nitrate or iodide spiking.
100 h continuous flow, and it did not occur with chloramine, with intermittent flow, or if orthophosphate

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