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1 n the plasmid were selected according to the chloramphenicol resistance.
2 and > 50 compounds were tested for promoting chloramphenicol resistance.
3 n resistance developed both erythromycin and chloramphenicol resistance, and plasmid DNA isolated fro
5 ml), 4/242 isolates tested were resistant to chloramphenicol (resistance breakpoint >/= 32 mug/ml), 1
6 Chlamydomonas reinhardtii, was replaced by a chloramphenicol resistance cartridge in the cyanobacteri
7 ylori rpoB-rpoC fusion gene with a non-polar chloramphenicol resistance cassette containing a new tra
8 various H. pylori strains by insertion of a chloramphenicol resistance cassette into lpxEHP and exam
11 oter, pOX38-tra719-traD411, which contains a chloramphenicol-resistance cassette in place of the kana
12 ng promoterless, terminatorless kanamycin or chloramphenicol resistance cassettes into the HindIII si
13 previously employed, using tetracycline and chloramphenicol resistance cassettes, and non-polar stra
14 individually engineered into a plasmid-borne chloramphenicol-resistance (cat) gene driven by the lac
15 orientations were disrupted by promoterless chloramphenicol resistance (Cm(r)) and kanamycin resista
18 ying the ColE1 origin of replication and the chloramphenicol-resistance (CmR) gene to facilitate the
19 ene therapy by the addition of mitochondrial chloramphenicol resistance, conferred by a point mutatio
20 ed to clone and express the promoterless Tn9 chloramphenicol resistance gene (cat, CmR) in P. haemoly
22 or by introduction of both a kanamycin and a chloramphenicol resistance gene to produce a double muta
25 the fla operon promoter and a staphylococcal chloramphenicol resistance gene, was constructed to help
31 ac promoter or the cat start codon abolished chloramphenicol resistance, indicating that E. coli init
32 codon in the intergenic region, between the chloramphenicol resistance marker and ccsB, in frame wit
33 resulted in the introduction of a selectable chloramphenicol resistance marker into the chromosome.
34 containing transposon-based tetracycline and chloramphenicol resistance markers were combined to allo
38 r protein RepD encoded by the Staphylococcus chloramphenicol resistance plasmid pC221 stimulates the
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