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1 omosome 11, which encodes a Ca(2+)-dependent chloride ion channel.
2 GABAA receptors are brain inhibitory chloride ion channels.
3 mission between neurons through formation of chloride ion channels.
4 ve identified two salt bridges in human CFTR chloride ion channels, Arg(352)-Asp(993) and Arg(347)-As
5 ct enterotoxin enhancement of the intestinal chloride ion channel as a basis for diarrhoeal disease.
6 abscisic acid (ABA) regulates potassium and chloride ion channels at the plasma membrane of guard ce
7 nsmembrane conductance regulator (CFTR) is a chloride ion channel, but its relationship to the primar
10 nsmembrane conductance regulator (CFTR) is a chloride ion channel constructed from two membrane-spann
12 he benzodiazepine binding site on the GABA-A chloride ion channel enhance cognitive performance in an
13 ntly been shown to express a glioma-specific chloride ion channel (GCC) that is sensitive to chloroto
16 appreciation of the central roles played by chloride ion channels in cellular functions, such as ele
17 he benzodiazepine-gamma-aminobutyric acid(A)-chloride ion channel macromolecular complex in the patho
18 identified that a proapoptotic mitochondrial chloride ion channel, mtCLIC/CLIC4, is induced by Myc.
19 n-3 (5-HT(3)) receptors, and glutamate-gated chloride ion channels of proteasome invertebrate phyla.
22 nsmembrane conductance regulator (CFTR) is a chloride ion channel regulated by protein kinase A and a
23 e fragment of Nt-Syr1 prevents potassium and chloride ion channel response to ABA in guard cells and
24 ng the insulin receptor, the muscle-specific chloride ion channel, sarco(endo)plasmic reticulum Ca(2+
25 nsmembrane conductance regulator (CFTR) is a chloride ion channel that also serves as a receptor for
26 ransmembrane conductance regulator (CFTR), a chloride ion channel that controls fluid and electrolyte
27 ulator (CFTR) protein is a small conductance chloride ion channel that may interact directly with oth
28 Ligand-gated heteropentameric GlyRs form chloride ion channels that contain the alpha(1) and beta
29 expressed in L929 cells produced functional chloride ion channels that were both spontaneously activ
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