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1 tes the neurotoxic and nephrotoxic byproduct chloroacetaldehyde.
2 f an etheno ring after reaction of ADPR with chloroacetaldehyde.
3 caused by exogenous chemical agents such as chloroacetaldehyde.
4 r cells under the influence of the cytotoxin chloroacetaldehyde.
7 1, treated with 10 and 35 mumol.liter(-1) of chloroacetaldehyde, a metabolite of the anti-cancer drug
8 rated with the isophosphoramide mustard; and chloroacetaldehyde, a neurotoxic and nephrotoxic compoun
9 ly, treatment of cultured human cells with 2-chloroacetaldehyde, a reactive metabolite of vinyl chlor
12 esized that inhibition of complex I (C-I) by chloroacetaldehyde (CAA), a metabolite of IFO, is the ch
15 ective condensation of aminotriazine 15 with chloroacetaldehyde provided the desired imidazotriazine
16 adducts produced by chloroethylene oxide and chloroacetaldehyde, reactive metabolites of vinyl chlori
19 nt acyl etheno CoA esters in the presence of chloroacetaldehyde, separated by ion-paired reversed-pha
20 substrate; furthermore, each mutant oxidized chloroacetaldehyde slower than propionaldehyde, and a pr
21 lboronate agarose columns and incubated with chloroacetaldehyde to convert the adenosine present in t
22 dissociation to deacylation was finding that chloroacetaldehyde was oxidized more rapidly than acetal
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