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5 y cell leukemia (HCL) after treatment with 2-chlorodeoxyadenosine (2-CdA) using immunohistochemical (
7 eroids, radiation, vinblastine/prednisone, 2-Chlorodeoxyadenosine (2-CdA), or cytosine arabinoside (A
12 with interferon alfa (IFN-alpha; P = .27), 2-chlorodeoxyadenosine (2CDA; P = .37), or deoxycoformycin
13 a clinical protocol was designed combining 2-chlorodeoxyadenosine (CdA), an inhibitor of ribonucleoti
14 fludarabine, 2'-deoxycoformycin (DCF), and 2-chlorodeoxyadenosine (CdA), commonly used in the treatme
15 the absolute bioavailability (F value) of 2-chlorodeoxyadenosine (cladribine; 2-CdA) after multiple
17 ox-binding protein (TBP) were disrupted on 2-chlorodeoxyadenosine monophosphate (CldAMP)-substituted
19 efined cases have been shown to respond to 2-chlorodeoxyadenosine therapy after failing treatment wit
20 l processes would therefore be affected by 2-chlorodeoxyadenosine triphosphate (CldATP) incorporation
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