ライフサイエンスコーパス: chlorosis

1 r fresh weight, reduced root elongation, and chlorosis.

2 'h1 mutant plants from Fe deficiency-induced chlorosis.

3 uch as accumulation of carbohydrates or leaf chlorosis.

4 s in tobacco (Nicotiana benthamiana) induced chlorosis.

5 rce's disease of grape and citrus variegated chlorosis.

6 oved fertility and also reversed interveinal chlorosis.

7 grapevines that leads to leaf scorching and chlorosis.

8 , leading to accumulation of sugars and leaf chlorosis.

9 ssion of AvrB in rpm1 plants results in leaf chlorosis.

10 Fe deficiency symptoms, such as interveinal chlorosis.

11 ase symptoms, with plants showing no visible chlorosis.

12 racterized by necrotic lesions surrounded by chlorosis.

13 nts with reduced CpNifS expression exhibited chlorosis, a disorganized chloroplast structure, and stu

14 r the tic40 and hsp93-V mutations, exhibited chlorosis, aberrant chloroplast biogenesis, and ineffici

15 plants were found to have significantly less chlorosis after treatment with the superoxide-generating

16 Other phenotypes, however, such as chlorosis along the leaf veins, are likely caused by thi

17 ns 16% pRBR binding activity, only developed chlorosis along the veins, and viral DNA, AL1 protein an

18 t for the tolerance to Fe deficiency-induced chlorosis, also on soil substrate.

19 tically low Fe concentrations and, hence, Fe chlorosis, although the transcriptional Fe deficiency re

20 of silencing from germination rapidly caused chlorosis and a strong developmental phenotype that led

21 hereas ethylene insensitivity led to reduced chlorosis and ABA deficiency to reduced anthocyanin accu

22 ough leaves and cotyledons continued to show chlorosis and altered chloroplasts.

23 we found that GPA feeding induced premature chlorosis and cell death, and increased the expression o

24 in alkaline soil, fro7 seedlings show severe chlorosis and die without setting seed unless watered wi

25 ight promoted the development of interveinal chlorosis and growth inhibition in the transgenic plants

26 Furthermore, development of chlorosis and growth inhibition was dependent on growth

27 a was supplemented with ribose, which led to chlorosis and growth inhibition.

28 iron deficiencies, measured as reduced leaf chlorosis and improved maintenance of the photosynthetic

29 seedlings showed severe growth defects, leaf chlorosis and leaf shrinkage.

30 ection of Brachypodium with PMV+SPMV induced chlorosis and necrosis of leaves, reduced seed set, caus

31 measurements of leaf arching, increased leaf chlorosis and necrosis, and altered UV-B regulation of s

32 while S. albescens suffered reduced growth, chlorosis and necrosis, impaired photosynthesis, and hig

33 sical defense marker, and symptoms including chlorosis and necrosis.

34 multiplication, and delays the onset of leaf chlorosis and necrosis.

35 showed a distinct phenotype characterized by chlorosis and reduced plant size, as well as hypersensit

36 Both chlorosis and ribose accumulation were abolished upon th

37 cluding Arabidopsis to actinonin resulted in chlorosis and severe reductions in plant growth and deve

38 We suggest that the chlorosis and stunting in P6-transgenic and CaMV-infecte

39 for mutants that suppressed the phenotype of chlorosis and stunting.

40 anisms underlying the onset of Fe-deficiency chlorosis and the maintenance of photosynthetic function

41 thesize any nicotianamine, shows strong leaf chlorosis, and is sterile.

42 ere obvious, including impaired growth, leaf chlorosis, and necrosis and curling of leaf margins.

43 ymptoms such as developmental abnormalities, chlorosis, and necrosis.

44 coronatine, a major determinant of the leaf chlorosis associated with DC3000 pathogenesis.

45 umber of mutants exhibiting photorespiratory chlorosis at ambient CO(2), including several with defec

46 ed for optimal activity in tomato, including chlorosis, changes in chloroplast structure, cell wall t

47 at 22 degrees C but showed chilling-induced chlorosis, confirming that the gene is essential for low

48 ees C exhibits a pattern of chilling-induced chlorosis consistent with a disruption of chloroplast de

49 Two Xylella diseases, citrus variegated chlorosis (CVC) and Pierce's disease (PD) of grapevines,

50 d with the modified viral vectors manifested chlorosis due to silencing of either ChlI or PDS in appr

51 ron uptake, resulting in impaired growth and chlorosis during iron limitation.

52 nd produced phenotypes of starvation-induced chlorosis during short-day growth conditions and extende

53 ormal phenotype characterized by interveinal chlorosis, growth inhibition and weakening of stems and

54 to promote further growth (HopM1 and HopE1), chlorosis (HopG1), lesion formation (HopAM1-1), and near

55 ic studies of abiotic stress iron deficiency chlorosis (IDC) of soybean is reported.

56 rmination rates, slow growth rates, moderate chlorosis, impaired fertility and reduced long term seed

57 screen for mutants that lack AvrB-dependent chlorosis in an rpm1 background, we isolated TAO1 (targe

58 Significantly, starch accumulation precedes chlorosis in cells that will become a yellow sector.

59 lence of Pst DC3000 and for the induction of chlorosis in host plants.

60 in wild-type plants but strongly exacerbated chlorosis in irt1 plants, indicating that manganese anta

61 abnormalities, ranging from a characteristic chlorosis in leaves to a necrosis and large inhibition o

62 on of ascorbate occurred before the onset of chlorosis in Mn-stressed plants, especially in cv ZPV-29

63 tors (14 of 63 tested) induced cell death or chlorosis in N. benthamiana.

64 molecular mechanisms that may contribute to chlorosis in plants when exposed to metals.

65 ing type III effectors; however, it promotes chlorosis in the model plant Nicotiana benthamiana in a

66 S in transgenic plants also resulted in leaf chlorosis, increased light sensitivity, and dwarfism due

67 y metals mimics iron (Fe) deficiency-induced chlorosis, indicating a disturbance in Fe homeostasis.

68 expression lines are slightly chlorotic, and chlorosis is rescued by exogenous iron.

69 l response that occurs prior to the onset of chlorosis, namely the disconnection of the LHCI antenna

70 t not limited to severe dwarfing appearance, chlorosis, nearly complete reduction of internodes and a

71 esponse and low sulfur levels activating the chlorosis or bleaching response.

72 en to yellow-green, a process referred to as chlorosis or bleaching.

73 ivity (approximately 90% or more), developed chlorosis or necrosis on some of their lower leaves.

74 enotype at 22 degrees C, it has a pronounced chlorosis phenotype at 8 degrees C that is correlated wi

75 The symptoms included leaf chlorosis, restriction of root elongation, and eventual

76 Chlorosis resulting from application of F. oxysporum cul

77 d to a severe reduction in growth and strong chlorosis symptoms.

78 ed delay of transition to flowering and mild chlorosis symptoms.

79 iscA and sufA mutant strains exhibited less chlorosis than the wild type.

80 nd the double mutant (k1 k3) displayed rapid chlorosis upon high light stress.

81 l regions of the bipartite genome of Lettuce chlorosis virus (LCV), a member in the genus Crinivirus

82 Host chlorosis was associated with virulence, whereas necroti

83 This induced chlorosis was dependent on ENHANCED DISEASE RESISTANCE1,

84 IF response was retained in NahG leaves and chlorosis was more pronounced than in the wild-type.

85 Anthocyanin accumulation, stunting, and chlorosis were common symptoms.

86 eir ability for reduction of iron deficiency chlorosis were explored.

87 Reduced development and chlorosis were observed for plants exposed to highly neg

88 necrosis (the D192K mutant), or an atypical chlorosis with necrotic flecking (the L194A mutant).

89 20 results in extensive necrosis and limited chlorosis within 5-6 days post-inoculation (d.p.i.), whi