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1 versed the decreased contractile response to cholecystokinin octapeptide.
2 enteral feedings, ursodeoxycholic acid, and cholecystokinin-octapeptide.
3 o receive an intravenous injection of either cholecystokinin octapeptide (200 mug/kg in 0.3 mL saline
4 in vivo pancreatic secretion in response to cholecystokinin octapeptide and reduced pancreatic diges
5 ous ligands such as gastrin/carboxy-terminal cholecystokinin octapeptide and selective cholecystokini
7 evis brain shares high affinity for sulfated cholecystokinin octapeptide but has > or = 1000-fold low
8 rats were tested for the ability of systemic cholecystokinin octapeptide (CCK) (0 or 10 microg/kg) to
10 t acini of transgenic mice 5 min after 10 pM cholecystokinin octapeptide (CCK) stimulation was enhanc
11 peridol (HAL-0.12 mg/kg, i.p.), and sulfated cholecystokinin octapeptide (CCK-0.05 and 0.1 mg/kg, i.p
14 traction induced by a continuous intravenous cholecystokinin octapeptide (CCK-8) infusion was determi
18 to intra-arterial injection of low doses of cholecystokinin octapeptide (CCK-8; 10-60 pmol); group B
19 tracerebroventricular (i.c.v.) injections of cholecystokinin-octapeptide (CCK-8) and somatostatin (SS
20 otoxin decreased the contractile response to cholecystokinin octapeptide for up to 96 hours after end
21 hibited by exogenous application of sulfated cholecystokinin octapeptide in a reversible and concentr
22 otoxin abolished the contractile response to cholecystokinin octapeptide in gallbladder strips 4 hour
23 rat model of cardiopulmonary resuscitation, cholecystokinin octapeptide induced mild hypothermia, at
24 with cholesterol-free liposomes for 4 hours, cholecystokinin octapeptide-induced contraction, membran
25 d in a tissue bath to determine responses to cholecystokinin octapeptide or electrical field stimulat
26 maintaining wild-type affinity for sulfated cholecystokinin octapeptide, receptors with increasing a
27 of survival were significantly better in the cholecystokinin octapeptide-treated animals when compare
30 nal discharges in response to 30 and 60 pmol cholecystokinin octapeptide were significantly lower in
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