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1 It is also a core protein of cholesterol gallstones.
2 s frequently associated with the presence of cholesterol gallstones.
3 increased biliary cholesterol secretion and cholesterol gallstones.
4 ecifically required for biofilm formation on cholesterol gallstones.
5 ary lipid metabolism and the pathogenesis of cholesterol gallstones.
6 or uninfected mice fed a chow diet developed cholesterol gallstones.
7 a critical role in 17beta-estradiol-induced cholesterol gallstones.
8 ile revealed cholesterol crystals as well as cholesterol gallstones.
9 trategy for treating hypercholesterolemia or cholesterol gallstones.
10 wild-type mice developed significantly more cholesterol gallstones (27%-80% prevalence) than Rag mic
11 known until recently whether the endemic of cholesterol gallstones among certain southwestern Americ
12 pies that may alleviate biofilm formation on cholesterol gallstones and the chronic carrier state.
15 odentium and fed a lithogenic diet developed cholesterol gallstones at 80% prevalence by 8 weeks comp
17 Apob48 or Apob100 reduces susceptibility to cholesterol gallstones by decreasing intestinal absorpti
18 romising strategy for preventing or treating cholesterol gallstones by inhibiting intestinal choleste
19 rome, as hyperglycemia, atherosclerosis, and cholesterol gallstones can all be caused by insulin resi
25 tible wild-type mice that recapitulate human cholesterol gallstone disease, treatment with a syntheti
26 tible wild-type mice that recapitulate human cholesterol gallstone disease, treatment with a syntheti
27 Because hyperhomocysteinemia can occur in cholesterol gallstone disease, we hypothesized that this
31 mbrane composition, such as that observed in cholesterol gallstone disease.Due to the challenges in d
33 (LIRKO mice) are markedly predisposed toward cholesterol gallstone formation due to at least two dist
35 play a major role in the pathophysiology of cholesterol gallstone formation in mice and perhaps huma
39 athway has implications for atherosclerosis, cholesterol gallstone formation, and some lipid storage
40 s intestinal cholesterol absorption, hepatic cholesterol gallstone formation, and the accumulation of
41 lesterol hypersecretion as the mechanism for cholesterol gallstone formation, thereby drawing a link
53 monella can form biofilms on the surfaces of cholesterol gallstones in the gallbladders of mice and h
54 in has a central role in the pathogenesis of cholesterol gallstones, in part because of its ability t
55 have been implicated in the pathogenesis of cholesterol gallstones-including those developing in acr
59 the effects of Helicobacter spp infection in cholesterol gallstone pathogenesis in the highly suscept
60 ammation are likely to be crucial factors in cholesterol gallstone pathogenesis, rather then merely t
61 layed statistically significant increases in cholesterol gallstone prevalence compared with uninfecte
62 elicobacter cinaedi , and H rodentium gave a cholesterol gallstone prevalence of 40%, 30%, and 20%, r
63 sorption, biliary cholesterol secretion, and cholesterol gallstone prevalence were compared in knocko
64 modify current views of the pathogenesis of cholesterol gallstones, promote further research on the
65 o and that bile-induced biofilm formation on cholesterol gallstones promotes gallbladder colonization
70 1(+/+)) mice fed a lithogenic diet developed cholesterol gallstones that supported biofilm formation
71 of the immune system in the pathogenesis of cholesterol gallstones was not considered a valid topic
72 ecular mechanisms of how estrogen influences cholesterol gallstones, we studied gonadectomized AKR/J
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