ライフサイエンスコーパス: cholesterol sulfate

1 The third composition consisted of no cholesterol sulfate.

2 nd fatty acids (15%) with varying amounts of cholesterol sulfate (0-15%) and cholesterol (15-30%).

3 third component of the migratory pheromone), cholesterol sulfate (6), and squalamine (8)] and model c

4 Cholesterol sulfate, a liver X receptor (LXR) antagonist

5 change in spatial distribution of the sterol cholesterol sulfate, a membrane stabilizing lipid.

6 Here, we show that cholesterol sulfate, a molecule present in relatively hi

7 Cholesterol sulfate, a SC membrane lipid, is synthesized

8 ion in cholesterol, we assessed here whether cholesterol sulfate accumulation and/or cholesterol defi

9 Together, these results suggest that cholesterol sulfate accumulation rather than cholesterol

10 d further suggesting a cellular location for cholesterol sulfate action.

11 ide, and steroid sulfatase, which desulfates cholesterol sulfate, also increased with air exposure.

12 In agreement, topical skin application of cholesterol sulfate, an activator of PKCeta, significant

13 olesterol sulfate and 25% cholesterol or 15% cholesterol sulfate and 15% cholesterol.

14 e lipid interaction than LUVs with either 5% cholesterol sulfate and 25% cholesterol or 15% cholester

15 We found that the LUVs with no cholesterol sulfate and 30% cholesterol exhibited a more

16 rongly inhibit it (coprostanol, androstenol, cholesterol sulfate, and 4-cholestenone).

17 er, with only one of two leaflets containing cholesterol sulfate, and stabilization of the external l

18 Cholesterol sulfate appears to combine the advantages of

19 studies of Adx binding to substrate-free and cholesterol-sulfate-bound CYP46A1 revealed changes in th

20 amide proton exchange in substrate-free and cholesterol-sulfate-bound P450.

21 Doping DMPC/DHPC bicelles with cholesterol sulfate broadens the temperature range over

22 yosis display not only a 10-fold increase in cholesterol sulfate, but also a 50% reduction in cholest

23 Mincle activation by cholesterol sulfate causes the secretion of a range of p

24 precursors, plant sterols, some oxysterols, cholesterol sulfate, cholesterol acetate, and 5-alpha-ch

25 es with this being the area with the highest cholesterol sulfate content suggesting that the physiolo

26 is study was to examine changes in levels of cholesterol sulfate (CS) and activity of its biosyntheti

27 Here we show that cholesterol sulfate (CS), a naturally occurring analog o

28 ol% sterol when cholesterol is replaced with cholesterol sulfate (CS).

29 ulfoconjugation of cholesterol to synthesize cholesterol sulfate (CS).

30 sequence of SSase deficiency, its substrate, cholesterol sulfate (CSO4), accumulates in the epidermis

31 These studies suggest that induction of the cholesterol sulfate cycle enzymes during SC ontogenesis

32 Cholesterol and cholesterol sulfate have been suggested to be RORalpha l

33 The inclusion of 5-15% cholesterol sulfate helps to prevent the collapse of fus

34 Additionally, 88% of cholesterol sulfate in NHEK was membrane-associated furt

35 The presence of cholesterol sulfate in prostate tissues might serve as a

36 Additionally, cholesterol sulfate is a constituent of human platelets,

37 Cholesterol sulfate is a highly amphipathic molecule tha

38 Cholesterol sulfate is a multifunctional sterol metaboli

39 nt suggesting that the physiologic action of cholesterol sulfate is likely carried out in this region

40 Recent evidence suggests that cholesterol sulfate may be an important second messenger

41 Profiles obtained from lamellae with cholesterol sulfate partially substituted for cholestero

42 Yet, topical cholesterol sulfate produced both a barrier abnormality

43 that was reflected in enzymatic activity and cholesterol sulfate production.

44 e for the synthesis of glucosylceramides and cholesterol sulfate, respectively, were accelerated furt

45 nflammatory mediators, and s.c. injection of cholesterol sulfate results in a Mincle-mediated inducti

46 Cholesterol sulfate, the most important sterol sulfate i

47 Finally, addition of exogenous cholesterol sulfate to explants in vitro did not acceler

48 Despite the apparent importance of cholesterol sulfate-to-cholesterol processing for normal

49 esults demonstrate both a potential role for cholesterol sulfate-to-cholesterol processing in normal

50 Cholesterol sulfate was identified as a differentiating