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1 hese pre-beta-HDL not involving apoE, HL, or cholesteryl ester transfer protein.
2 sma lecithin-cholesteryl acyltransferase and cholesteryl ester transfer protein activities were lower
4 se was unaffected by psyllium intake whereas cholesteryl ester transfer protein activity was 18% lowe
5 y result directly from an increase in plasma cholesteryl ester transfer protein activity whose effect
6 ty, HDL ability to esterify cholesterol, and cholesteryl ester transfer protein activity), HDL antiox
7 ding expression of the emerging drug targets cholesteryl ester transfer protein and apolipoprotein (a
8 protein E and change in LDL cholesterol, and cholesteryl ester transfer protein and change in HDL cho
9 l candidate genes, such as apolipoprotein E, cholesteryl ester transfer protein and hepatic lipase; (
10 tigate the mechanisms of action, we measured cholesteryl ester transfer protein and indexes of plasma
12 Since previous studies have shown that human cholesteryl ester transfer protein and lecithin:choleste
14 erol acyltransferase), transfer cholesterol (cholesteryl ester transfer protein) and hydrolyze lipids
15 It also is the major plasma inhibitor of cholesteryl ester transfer protein, and appears to inter
16 e, which occurs in plasma, is facilitated by cholesteryl ester transfer protein, and generates a TG-e
17 of coexpression of scavenger receptor BI or cholesteryl ester transfer protein, both of which promot
19 o converted to o-quinone 28, which inhibited cholesteryl ester transfer protein (CETP) activity and L
22 he role of cholesteryl ester transfer (CET), cholesteryl ester transfer protein (CETP) activity, and
24 ough the relationship between the actions of cholesteryl ester transfer protein (CETP) and atheroscle
25 oprotein AI-CIII-AIV, on chromosome 11q; for cholesteryl ester transfer protein (CETP) and lecithin:c
27 of lipid-binding proteins that also contains cholesteryl ester transfer protein (CETP) and phospholip
28 al to the host defence against bacteria, and cholesteryl ester transfer protein (CETP) and phospholip
30 sma phospholipid transfer protein (PLTP) and cholesteryl ester transfer protein (CETP) are homologous
31 nd environmental factors influence LDL size, cholesteryl ester transfer protein (CETP) being one of t
34 ockout mice, and human apolipoprotein (apo)B/cholesteryl ester transfer protein (CETP) double transge
41 is study was to identify associations of the cholesteryl ester transfer protein (CETP) gene with coro
42 soleucine to valine (I405V) variation in the cholesteryl ester transfer protein (CETP) gene, which is
44 We have hypothesized that the absence of cholesteryl ester transfer protein (CETP) in LCAT-Tg mic
45 xplore two HDL-C raising target modulations, Cholesteryl Ester Transfer Protein (CETP) inhibition and
46 ofuroquinoline derivatives exhibiting potent cholesteryl ester transfer protein (CETP) inhibition at
47 ne is consistent with a protective effect of cholesteryl ester transfer protein (CETP) inhibition on
49 te the efficacy and safety of torcetrapib, a cholesteryl ester transfer protein (CETP) inhibitor, in
56 cuses on the studies with niacin and the new cholesteryl ester transfer protein (CETP) inhibitors tor
57 mine the recent advances in our knowledge of cholesteryl ester transfer protein (CETP) inhibitors, he
76 terol acyltransferase (LCAT) and transfer by cholesteryl ester transfer protein (CETP) were measured
77 ls can be increased by >50% by inhibition of cholesteryl ester transfer protein (CETP), a molecule th
85 frequency for the TaqI B1/B2 polymorphism in cholesteryl ester transfer protein, consistent with the
87 oproteinemia seems associated with a related cholesteryl ester transfer protein genotype difference.
89 has been known for over a decade but, unlike cholesteryl ester transfer protein, has been investigate
92 s; adipose tissue-specific overexpression of cholesteryl ester transfer protein in mice reduces the p
93 carry cholesterol accepted from LDL+HDL via cholesteryl ester transfer protein in vivo, may contribu
94 Initial studies addressing the effect of cholesteryl ester transfer protein inhibition on cardiov
96 and low-density lipoprotein cholesterol, the cholesteryl ester transfer protein inhibitor torcetrapib
98 s after ACS to treatment with dalcetrapib (a cholesteryl ester transfer protein inhibitor) or placebo
99 te the safety and efficacy of anacetrapib, a cholesteryl ester transfer protein inhibitor, in patient
100 , 0.94-1.09) vs 0.90 (95% CI, 0.89-0.91) for cholesteryl ester transfer protein inhibitors (P = .002)
101 Emerging HDL-raising therapies (such as cholesteryl ester transfer protein inhibitors and 1,2-di
105 apeutic agents such as fibrates, niacin, and cholesteryl ester transfer protein inhibitors that are k
106 t should be possible to develop more optimal cholesteryl ester transfer protein inhibitors that do no
107 less, drugs that raise HDL-C concentrations, cholesteryl ester transfer protein inhibitors, are in la
110 specific and, since the crystal structure of cholesteryl ester transfer protein is now known, it shou
111 lipoprotein-cholesterol, i.e. inhibition of cholesteryl ester transfer protein, is markedly effectiv
112 at overexpression of LCAT in the presence of cholesteryl ester transfer protein leads to both hyperal
113 apo B, apo C-III, apo E, lipoprotein lipase, cholesteryl ester transfer protein, lecithin:cholesterol
114 and significant evidence for linkage of the cholesteryl ester transfer protein locus (chromosome 16)
115 of postprandial TRLs in plasma increased the cholesteryl ester transfer protein-mediated transfer of
118 hat do not form a nonproductive complex with cholesteryl ester transfer protein on the high-density l
119 zed in the presence of PLA2 by the action of cholesteryl ester transfer protein or by guanidine hydro
120 d had shifted to normal HDL (in mice lacking cholesteryl ester transfer protein) or to apoB containin
121 -cholesterol fractional esterification rate, cholesteryl ester transfer protein, phospholipid transfe
122 holesterol concentration and adipocyte size; cholesteryl ester transfer protein TaqIB polymorphism is
123 Asp), apolipoprotein E (Apo E2, E3, and E4), cholesteryl ester transfer protein (TaqIB), and leptin r
124 , apolipoprotein E (Apo E2, E3, and E4), and cholesteryl ester transfer protein (TaqIB)] and equol pr
127 ipoprotein (LDL) receptor-deficient, or apoB/cholesteryl ester transfer protein transgenic background
128 tein) or to apoB containing lipoproteins (in cholesteryl ester transfer protein transgenic mice).
129 t not in apoA-II0, LDL receptor 0, apoE0, or cholesteryl ester transfer protein transgenic mice.
131 a lipoproteins in an animal model expressing cholesteryl ester transfer protein was evaluated by gene
132 for hepatic lipase, endothelial lipase, and cholesteryl ester transfer protein were analyzed, patien
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