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1 me in the production of acetylcholine (i.e., choline acetyltransferase).
2 uronal markers neurofilament 200 (NF200) and choline acetyltransferase.
3 d simultaneously for tyrosine hydroxylase or choline acetyltransferase.
4 of the neurotransmitter-synthesizing enzyme choline acetyltransferase.
5 ns were OFQ-IR, all OFQ-IR neurons expressed choline acetyltransferase.
6 mesopontine tegmental area colocalizing with choline acetyltransferase.
7 se structures in some neurons that stain for choline acetyltransferase.
8 75 and trkA, and with the cholinergic marker choline acetyltransferase.
9 cells expressing FLI were immunopositive for choline acetyltransferase.
10 s only 13% of m2-stained neurons colocalized choline acetyltransferase.
11 munoreactivity for nitric oxide synthase and choline acetyltransferase.
12 enteric plexus of the ileum expressed IR for choline acetyltransferase (56.9%), substance P (55.0%),
13 nological and histological stains, including choline acetyltransferase, acetylcholinesterase, tyrosin
14 n examined two cholinergic synaptic markers, choline acetyltransferase activity (ChAT) and [3H]hemich
17 de recognized VIP-binding sites and enhanced choline acetyltransferase activity as well as cognitive
19 ds of mice lacking one or both genes contain choline acetyltransferase activity comparable to that of
21 duced a significant reduction in hippocampal choline acetyltransferase activity in all tested groups
23 opsied brain showed a pronounced decrease in choline acetyltransferase activity in the frontal and te
24 triatum of AChE-/- mice showed no changes in choline acetyltransferase activity or levels of the vesi
29 central nervous system cholinergic indices--choline acetyltransferase and acetylcholinesterase activ
31 and a significant decrease in expression of choline acetyltransferase and calretinin, demonstrating
32 Presently, such maps are unavailable for choline acetyltransferase and carnitine acetyltransferas
33 ating normal prefrontal immunoreactivity for choline acetyltransferase and dopamine beta-hydroxylase.
36 hese bundles contained acetylcholinesterase, choline acetyltransferase and nerve growth factor recept
37 cholinergic neurons as manifested by loss of choline acetyltransferase and p75NGFR immunoreactivity.
39 ls were induced to become neurons expressing choline acetyltransferase and substance P and formed neu
40 motoneurons and interneurons that expressed choline acetyltransferase and substance P, and in inhibi
41 ssion of the cholinergic gene locus encoding choline acetyltransferase and the vesicular acetylcholin
42 stochemical experiments, using antibodies to choline acetyltransferase and vesicular acetylcholine tr
43 ted cholinergic gene locus, we now show that choline acetyltransferase and vesicular acetylcholine tr
44 terneurons and projection neurons containing choline acetyltransferase and/or NADPH diaphorase were s
45 uorescence staining using antibodies against choline-acetyltransferase and neurofilament was performe
46 cytochemistry for dopamine beta-hydroxylase, choline acetyltransferase, and serotonin in the primate
47 noreactivity was coexpressed with calbindin, choline acetyltransferase, and substance P in the myente
48 contained relatively dense staining for TH, choline acetyltransferase, and substance P, whereas vaso
49 3, alpha5, alpha7, and beta2 nAChR subunits, choline acetyltransferase, and the asymmetric and globul
50 ncluding calbindin, calretinin, parvalbumin, choline acetyltransferase, and tyrosine hydroxylase.
52 unoreactive for cholecystokinin precursor or choline acetyltransferase, because both types of amacrin
53 dGs were usually negative for glycine, ChAT (choline acetyltransferase), bNOS (brain-type nitric oxid
54 in induced dendritic degeneration, decreased choline acetyltransferase but increased nitric oxide syn
55 jecting neurons were immunoreactive (IR) for choline acetyltransferase but not nitric oxide synthase
57 detection of NOS with tyrosine hydroxylase, choline acetyltransferase, calbindin, calretinin, and se
58 acrine cell markers gamma-aminobutyric acid, choline acetyltransferase, calretinin, or tyrosine hydro
59 and expression of alpha-smooth muscle actin, choline acetyltransferase, CD45, glial fibrillary acidic
61 alretinin-IR, 5-HT-accumulating, and ON-type choline acetyltransferase (ChAT) ACs in wild-type and Ch
62 sease (AD), loss of cortical and hippocampal choline acetyltransferase (ChAT) activity has been corre
63 ne-p75-SAP produced a dose-dependent loss of choline acetyltransferase (ChAT) activity in the hippoca
64 of 192Sap produced 50 and 80% depletions of choline acetyltransferase (ChAT) activity in the neocort
65 nths of age we observed a 40-50% decrease in choline acetyltransferase (ChAT) activity in two of thre
67 tration of NGF on P4 or later induced septal choline acetyltransferase (ChAT) activity to the same re
68 physin-immunoreactive presynaptic terminals, choline acetyltransferase (ChAT) activity, and ChAT-posi
74 f 5 days, alterations in the localisation of choline acetyltransferase (ChAT) and calretinin immunore
75 Light microscopic analyses revealed that choline acetyltransferase (ChAT) and GABA immunoreactive
78 etinogram (ERG), the retinal localization of choline acetyltransferase (ChAT) and neuronal nitric oxi
79 TrkA tyrosine phosphorylation in the septum, choline acetyltransferase (ChAT) and p75 neurotrophin re
80 splanted cells developed immunoreactivity to choline acetyltransferase (ChAT) and sent axons into the
81 procedures using antibodies directed against choline acetyltransferase (ChAT) and the dopamine D5 rec
82 , p75-positive (p75+) cells were enriched in choline acetyltransferase (ChAT) and the high-affinity c
83 ated by immunohistochemical visualization of choline acetyltransferase (ChAT) and the low-affinity ne
86 fects of one week of estrogen replacement on choline acetyltransferase (ChAT) and trkA mRNA expressio
88 Third, we correlated levels of freezing with choline acetyltransferase (ChAT) and vesicular acetylcho
89 NF stimulates mRNA and protein expression of choline acetyltransferase (ChAT) and vesicular acetylcho
90 acing of nigral efferents with pre-embedding choline acetyltransferase (ChAT) and/or glutamate (Glu)
92 sion of the critical neurotransmitter enzyme choline acetyltransferase (ChAT) by in vitro motor neuro
94 ncurrent staining for the cholinergic marker choline acetyltransferase (ChAT) demonstrated that virtu
96 , abnormally ubiquitinated proteins, reduced choline acetyltransferase (ChAT) enzyme expression, frag
99 ping septum that increases ACh synthesis and choline acetyltransferase (Chat) gene expression both in
100 both induce GAL gene expression and inhibit choline acetyltransferase (ChAT) gene expression in this
101 cent microspheres was combined with nNOS and choline acetyltransferase (ChAT) immunocytochemistry, an
102 xidase (WGA-HRP) combined with glutamate and choline acetyltransferase (ChAT) immunohistochemical and
103 ex by using retrograde tracing combined with choline acetyltransferase (ChAT) immunohistochemistry in
104 olinergic amacrine cells were identified via choline acetyltransferase (ChAT) immunohistochemistry.
105 s Q140 males at 1 and 4 months of age, using choline acetyltransferase (ChAT) immunolabeling to ident
106 sed stereological estimates of the number of choline acetyltransferase (ChAT) immunopositive neurons
110 ew intrinsic CARTp-IR neurons also exhibited choline acetyltransferase (ChAT) immunoreactivity, indic
111 Intrinsic NOS-IR cardiac neurons exhibited choline acetyltransferase (ChAT) immunoreactivity, indic
112 50% of muOR-immunoreactive neurons contained choline acetyltransferase (ChAT) immunoreactivity, where
114 (VLDB) revealed enhanced optical density for choline acetyltransferase (ChAT) in both young and aged
115 tochemical staining for this peptide and for choline acetyltransferase (ChAT) in macaque monkeys, in
116 ed axons and interneurons immunoreactive for choline acetyltransferase (ChAT) in regions of the execu
117 Immunohistochemistry 14 d after MI revealed choline acetyltransferase (ChAT) in stellate sympathetic
118 edium spiny neurons (MSNs) and D2-expressing choline acetyltransferase (ChAT) interneurons express Sl
119 liest ray-finned fishes, the distribution of choline acetyltransferase (ChAT) is described in Polypte
123 the labeled neurons in IF5 were found to be choline acetyltransferase (ChAT) negative, indicating th
124 tions were stained immunohistochemically for choline acetyltransferase (ChAT) or p140trk (trk) or his
125 brains were sectioned and immunolabeled for choline acetyltransferase (ChAT) or p75-neurotrophin rec
127 in-2 (ChR2) protein under the control of the choline acetyltransferase (ChAT) promoter (ChAT-ChR2-EYF
129 enzyme glutamic acid decarboxylase (GAD) and choline acetyltransferase (ChAT) revealed that all CG ne
133 ibody raised against the biosynthetic enzyme choline acetyltransferase (ChAT) was applied to individu
135 ), neuronal nitric oxide synthase (nNOS), or choline acetyltransferase (ChAT) were performed in whole
136 dentified a previously unknown population of choline acetyltransferase (ChAT)(+) neurons residing in
137 %-60% decrease of both parvalbumin (PV)+ and choline acetyltransferase (ChAT)+ cholinergic interneuro
138 ties, cortical projections and expression of choline acetyltransferase (ChAT), a synthetic enzyme for
139 f the cholinergic system in the hippocampus: choline acetyltransferase (ChAT), acetylcholinesterase (
140 end stage/140 days were analyzed for p75NTR, choline acetyltransferase (ChAT), activating transcripti
141 ightly fixed rabbit retinas with antisera to choline acetyltransferase (ChAT), AMPA receptor subunits
142 , aspartate, gamma-aminobutyric acid (GABA), choline acetyltransferase (ChAT), and a N-methyl-D-aspar
146 ctors including acetylcholinesterase (AChE), choline acetyltransferase (ChAT), choline transporter 1
147 nergic because of their immunoreactivity for choline acetyltransferase (ChAT), contradictory findings
148 to identify GABA and Fos protein, as well as choline acetyltransferase (ChAT), in histological sectio
149 of a known estrogen-responsive gene product, choline acetyltransferase (ChAT), in the hippocampus and
150 plex synergistically recruits another enzyme choline acetyltransferase (ChAT), leading to enhanced se
151 ) by establishing and analyzing mice lacking choline acetyltransferase (ChAT), the biosynthetic enzym
152 been reported to potentiate the activity of choline acetyltransferase (ChAT), the enzyme that produc
155 onditional mutant in which the gene encoding choline acetyltransferase (ChAT), the sole synthetic enz
157 By 10 d, saporin eliminated staining for choline acetyltransferase (ChAT), the synthetic enzyme f
158 unoreactivity was also compared with that of choline acetyltransferase (ChAT), tyrosine hydroxylase (
159 on of several specialized enzymes, including choline acetyltransferase (ChAT), vesicular acetylcholin
160 vasoactive intestinal polypeptide (VIP), and choline acetyltransferase (ChAT), was used to characteri
161 l mouse model of the ACh-synthesizing enzyme choline acetyltransferase (Chat), we report that local e
162 howed that CD4(+) T lymphocytes that express choline acetyltransferase (ChAT), which catalyzes the sy
163 istribution of nitric oxide synthase (NOS)-, choline acetyltransferase (ChAT)-, and vasoactive intest
164 ial septum containing hippocampal-projecting choline acetyltransferase (ChAT)-, neuronal nitric oxide
165 s estrogen replacement prevented the loss of choline acetyltransferase (ChAT)-containing cells in the
166 ssion of the tdTomato fluorescent protein in choline acetyltransferase (ChAT)-expressing cells were u
168 The number, size, and staining intensity of choline acetyltransferase (ChAT)-immunopositive cells in
169 d adjacent VTA contain moderate to intensely choline acetyltransferase (ChAT)-immunopositive neurons.
173 y vulnerable to glaucomatous damage, whereas choline acetyltransferase (ChAT)-positive and glycinergi
174 n individuals under age 65 years, 72% of the choline acetyltransferase (ChAT)-positive BFCN contained
176 as measured by increased c-Fos expression in choline acetyltransferase (ChAT)-positive interneurons.
177 oyed 43% of Nissl-stained neurons and 70% of choline acetyltransferase (ChAT)-positive neurons 5 days
178 led significant decrease in the soma size of choline acetyltransferase (ChAT)-positive neurons in the
190 Multiple tyrosine hydroxylase (Th)::Cre and choline acetyltransferase (Chat)::Cre lines were produce
191 By the expression of fluorescent proteins in choline acetyltransferase (ChAT)::Cre(+) transgenic rats
193 onjugates and a polyclonal antiserum against choline acetyltransferase (ChAT; Chemicon Ab 143), that
195 composed of two distinct genetic functions: choline acetyltransferase (ChAT; EC 2.3.1.6), the enzyme
196 rkA receptor) but not others (e.g., cortical choline acetyltransferase [ChAT] activity, the number of
197 m control ganglia and explanted ganglia were choline acetyltransferase(ChAT)-immunoreactive, indicati
198 ring of cholinergic interneurons (expressing choline acetyltransferase; ChAT) and GABAergic interneur
199 l image analysis of localising antibodies to choline acetyltransferase, co-localised with protein gen
200 linergic signaling, including a reduction in choline acetyltransferase enzymatic activity and the num
202 ns expressing parvalbumin or somatostatin or choline acetyltransferase exhibited varying degrees of e
203 cholinergic forebrain neurons, we activated choline acetyltransferase expressing neurons using chann
205 -day differences in acetylcholinesterase and choline acetyltransferase expression and general neurona
206 te that the normal developmental increase of choline acetyltransferase expression becomes critically
207 ated and nonphosphorylated neurofilament and choline acetyltransferase expression were comparable bet
208 movement and death, and reduce transport of choline acetyltransferase from cell bodies to the synaps
209 ysis of temperature-sensitive mutants in the choline acetyltransferase gene revealed that the sprouti
210 cular acetylcholine transporter gene and the choline acetyltransferase gene, was investigated in PC12
211 expression of tyrosine hydroxylases 1 and 2, choline acetyltransferase, histidine decarboxylase, or o
212 Second, AMNs were counted in double-stained choline acetyltransferase immunocytochemical and NADPH d
213 h we did not visualize colocalization of m2+/choline acetyltransferase-immunonegative neurons and the
214 bel immunohistochemistry to demonstrate that choline acetyltransferase-immunopositive fibers in the S
216 nducted quantitative comparative analyses of choline acetyltransferase-immunoreactive axons in cortic
217 correlated significantly with the number of choline acetyltransferase-immunoreactive cells in the NB
218 ealed a few dystrophic neurites and neurons, choline acetyltransferase-immunoreactive fibers were not
224 We found that upregulated translation of choline acetyltransferase in the CPEB2 KO dorsal motor n
225 eover, neurons double-labeled with c-Fos and choline acetyltransferase in the NAmb were identified in
226 lum (p < 0.005, one-way ANOVA) and for ChAT (choline acetyltransferase) in the male vagal lobe and th
227 alpha 7 nicotinic acetylcholine receptor and choline acetyltransferase, in A beta 42-burdened, activa
228 or the axonal transport of a soluble enzyme, choline acetyltransferase, in a specific subset of neuro
230 aced-starburst amacrine cells (identified by choline acetyltransferase-IR) but in less than 50% of th
231 revealed that m2 was detected in only 14% of choline acetyltransferase-labeled neurons, whereas only
234 s of ACh associated with increased levels of choline acetyltransferase mRNA and decreased cholinester
236 th antibodies against the CB(1) receptor and choline acetyltransferase, neurofilament proteins, calbi
237 sin (eNpHR), or channelrhodopsin-2 (ChR2) in Choline acetyltransferase neurons (ChAT(+)) or Arch in L
239 din DK28, calretinin, nitric oxide synthase, choline-acetyltransferase, neuropeptide Y, serotonin, ne
240 oviruses targeted to atrial ganglia enhanced choline acetyltransferase-nNOS co-localization (P < 0.05
241 etween the autistic and comparison groups in choline acetyltransferase or acetylcholinesterase activi
242 ity of choline had any effect on hippocampal choline acetyltransferase or acetylcholinesterase activi
243 orated with vehicle, or with dsRNA targeting choline acetyltransferase or axonin-1, did not exhibit t
244 affold by mapping neurons immunopositive for choline acetyltransferase or retrogradely labeled from c
245 ed pulp macrophages, but they do not express choline acetyltransferase or vesicular acetylcholine tra
246 de (nitric oxide synthase) or acetylcholine (choline acetyltransferase) or in situ hybridization for
247 into the median raphe (MR) and parvalbumin, choline acetyltransferase, or calretinin as well as doub
249 e-expressing, but not acetylcholinesterase-, choline acetyltransferase-, or tryptophan hydroxylase-ex
250 controls: median percentage surface area of choline acetyltransferase over PGP was 17.5% (range 10.0
251 on of eGFP in intrinsic cholinergic neurons (choline acetyltransferase positive) and intrinsic adrene
252 number of cells was 197 +/- 23, and 92% were choline acetyltransferase positive, implying a cholinerg
253 All chambers of the heart were innervated by choline acetyltransferase-positive axons, implying choli
255 eurotrophin receptor (p75(NTR))-positive and choline acetyltransferase-positive cells in this region
257 ly higher number (10-15% increase) of septal choline acetyltransferase-positive cells than the vehicl
258 of this nAChR subtype reduces the number of choline acetyltransferase-positive cells, macrophages, a
259 revented Abeta-induced apoptosis and loss of choline acetyltransferase-positive cholinergic neurons.
261 der basal conditions, pERK is upregulated in choline acetyltransferase-positive interneurons in DKO m
264 hs the numbers of pedunculopontine tegmentum choline acetyltransferase-positive neurons were reduced
265 sferase enzymatic activity and the number of choline acetyltransferase-positive neurons, as well as,
266 In GAD67-GFP knock-in mice, BF cholinergic (choline acetyltransferase-positive) neurons were intermi
267 ic mice expressing cre-recombinase under the choline acetyltransferase promoter were used to selectiv
269 S1/CHGFP mice and reversed the reductions in choline acetyltransferase protein levels in the hippocam
270 cetylcholine receptor, acetylcholinesterase, choline acetyltransferase, rapsyn, MuSK and Na(v)1.4.
271 ns and fewer than 2% of neuropeptide Y (NPY)/choline acetyltransferase secretomotor neurons, and P2Y2
272 combined with peroxidase immunostaining for choline acetyltransferase showed that 60% of all synapti
273 burst amacrine cells with antibodies against choline acetyltransferase showed that the CD15-positive
274 GFP-positive efferents that co-expressed choline acetyltransferase specifically terminated in the
275 f the m2-labeled neurons, whereas only 6% of choline acetyltransferase-stained perikarya were dual la
277 d cells were reacted with antibodies against choline acetyltransferase, substance P and the neurokini
278 t five different vesicular proteins (UNC-17, choline acetyltransferase, Synaptotagmin, Synaptobrevin,
280 These included the localisation of Thy-1 and choline acetyltransferase, the a- and b-wave amplitudes
281 lethanolamine N-methyltransferase (PNMT) and choline acetyltransferase, the synthesizing enzymes for
282 in the density of fibers immunoreactive for choline acetyltransferase, this same treatment markedly
283 protein alignment data, was used to redesign choline acetyltransferase to accommodate L-carnitine as
284 In this study, we used an immunolabeling for choline acetyltransferase to demonstrate, for the first
285 The cells were also double-labelled for choline acetyltransferase to determine if they were cont
286 perior salivatory nucleus, or for PRV-Ba and choline acetyltransferase to establish the neurochemical
287 mRNA combined with immunohistochemistry for choline acetyltransferase to reveal cholinergic neurons.
290 hoprotein, calretinin, acetylcholinesterase, choline acetyltransferase, tyrosine hydroxylase, calbind
291 tylated tubulin, human neuronal protein C/D, choline acetyltransferase, tyrosine hydroxylase, neurona
292 , and human neuronal protein C/D, as well as choline acetyltransferase, tyrosine hydroxylase, nitric
293 TRPC6-IR fibers were not immunoreactive for choline acetyltransferase, tyrosine hydroxylase, or subs
296 ine was synthesized in yolk sacs, but 70 kDa choline acetyltransferase was undetectable by immunoblot
298 id decarboxylase, glycine transporter-2, and choline acetyltransferase were used as markers for GABAe
299 he concurrent visualization of tauopathy and choline acetyltransferase were used to determine if the
300 e C, parvalbumin, calbindin, calretinin, and choline acetyltransferase were used to stain different r
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