1 ssion of retinal responses via activation by cholinergic agents.

2 contributes to the antinociceptive action of cholinergic agents.

3 ng the antinociceptive action of intrathecal cholinergic agents.

4 dent or voltage-independent calcium channels.Cholinergic agents (ACh, nicotine, muscarine, bethanecho

5 aneous retinal waves of action potentials by cholinergic agents altered the subsequent eye-specific l

6 nergic agent, we verified that delivery of a cholinergic agent by means of topical application can le

7 served that nicotine (2 mg/kg) and the novel cholinergic agent CAP55 (12 mg/kg) inhibit endothelial c

8                 Nitric oxide (NO) donors and cholinergic agents decrease intraocular pressure, in par

9 s have suggested the potential usefulness of cholinergic agents for ADHD.

10 in the antinociceptive action of intrathecal cholinergic agents in normal rats and in a rat model of

11 lgesic mechanisms and the site of actions of cholinergic agents in the spinal cord are not fully unde

12     However, the mechanisms of the action of cholinergic agents in the spinal cord are not fully unde

13                  There is also evidence that cholinergic agents may have disease-modifying effects.

14 several types of cells, we hypothesized that cholinergic agents might have similar effects on keratin

15 rect inhibitory effects of acetylcholine and cholinergic agents on tumor necrosis factor (TNF)-induce

16                                     For each cholinergic agent, we verified that delivery of a cholin

17 oM nicotine) and impermeant (5 mM carbachol) cholinergic agents were used to define specific [3H]epib