ライフサイエンスコーパス: chondroblast

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1 lin is expressed by immature osteoblasts and chondroblasts.

2 into osteoblasts, adipocytes, myoblasts, or chondroblasts.

3 n to myoblasts, adipocytes, osteoblasts, and chondroblasts.

4 These chondroblasts and myoblasts lie close together within ea

5 ms of derived connective tissue cells (e.g., chondroblasts and osteoblasts).

6 profile of MPCs induced to differentiate to chondroblasts and osteoblasts, significant differences i

7 evelopment of a highly proliferating pool of chondroblasts between the epiphyses and metaphyses of fu

8 duction of c-Fos in vitro in osteoblasts and chondroblasts caused an increase in Fgfr1 RNA and FGFR1

9 everal molecular markers required for normal chondroblast differentiation (&agr; 1(II) collagen, Fgfr

10 tion factors that act redundantly to promote chondroblast differentiation in all cartilages of the mo

11 ption factors that redundantly promote early chondroblast differentiation.

12 ha in limb development and the regulation of chondroblast differentiation.

13 hypertrophic chondrocyte markers, epiphyseal chondroblasts ectopically activate hypertrophic chondroc

14 Sox6 are thus redundant, potent enhancers of chondroblast functions, thereby essential for endochondr

15 apoptosis, and decreased differentiation of chondroblasts in Meckel's cartilage.

16 While chondroblasts in the center of cartilages ultimately act

17 these cells with the ability to give rise to chondroblasts in vivo.

18 Ps) induce differentiation of osteoblast and chondroblast lineage cells from uncommitted mesenchymal

19 any other genes occurs within 3 days, before chondroblast phenotypic genes are detectable.

20 In these double mutants, chondroblasts poorly differentiate.

21 on of growth plate columnar zones by keeping chondroblasts proliferating and by delaying chondrocyte

22 fferentiation was at the late-blastema/early-chondroblast stage.

23 of other cell types such as preadipocytes or chondroblasts, the effect of hypoxia on myogenesis is in

24 In contrast, adhesion of auricular chondroblasts to fibrillin-1 was only partially inhibite

25 While differentiation of chondroblasts to pre-hypertrophic chondrocytes (IHH expr

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