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1 al activity of Sox9, the master regulator of chondrogenesis.
2 Canonical Wnt signaling strongly inhibits chondrogenesis.
3 d transforming growth factor beta to support chondrogenesis.
4 collagen content, and mineralization during chondrogenesis.
5 owth plate chondrocytes where it facilitates chondrogenesis.
6 ssion of several miRNAs was regulated during chondrogenesis.
7 ommit to an osteogenic identity and suppress chondrogenesis.
8 ypertrophic chondrocytes and is required for chondrogenesis.
9 maintenance and the suppression of in vitro chondrogenesis.
10 Edn1 signaling and NC differentiation during chondrogenesis.
11 -associated proteins, similar to its role in chondrogenesis.
12 nd that Alcama interacts with Nadl1.1 during chondrogenesis.
13 n to activate downstream target genes during chondrogenesis.
14 ocytes and mesenchymal stem cells undergoing chondrogenesis.
15 addition to its previously described role in chondrogenesis.
16 thritis arises from abnormal and accelerated chondrogenesis.
17 ned the role of receptor Smads 1, 5 and 8 in chondrogenesis.
18 cts miR-199a(*)-mediated repression of early chondrogenesis.
19 e RhoA in such cultures can markedly enhance chondrogenesis.
20 lycan desulfation is a critical regulator of chondrogenesis.
21 ix (ECM) components normally associated with chondrogenesis.
22 that NF-kappaB-p65 facilitates growth plate chondrogenesis.
23 wn-regulation of Sox9, a master regulator of chondrogenesis.
24 e now identify a possible role for GATA-6 in chondrogenesis.
25 ally regulated exon 2 splicing switch during chondrogenesis.
26 o affected as a result of the delay in early chondrogenesis.
27 tissue, creating the correct environment for chondrogenesis.
28 le in the TRANSFAC database, as important to chondrogenesis.
29 criptional regulatory networks that regulate chondrogenesis.
30 Runx2 fulfills antagonistic functions during chondrogenesis.
31 lop normally until the onset of craniofacial chondrogenesis.
32 1) is a previously unrecognized regulator of chondrogenesis.
33 efect under a periosteal flap into goats for chondrogenesis.
34 gnaling pathways are essential regulators of chondrogenesis.
35 nts for BMP signaling in multiple aspects of chondrogenesis.
36 fering RNA inhibits Col2a1 expression during chondrogenesis.
37 genesis and in the activation of Sox9 during chondrogenesis.
38 g is essential for multiple aspects of early chondrogenesis.
39 (COL2A1) is developmentally regulated during chondrogenesis.
40 and in concert with Sox proteins to regulate chondrogenesis.
41 PGC-1alpha) as a coactivator for Sox9 during chondrogenesis.
42 ctivates Sox9-dependent transcription during chondrogenesis.
43 model for the initial events of mesenchymal chondrogenesis.
44 and one from the oral ectoderm that promotes chondrogenesis.
45 ing limb, suggesting that it plays a role in chondrogenesis.
46 ndroprogenitors and has an essential role in chondrogenesis.
47 wth factor in the regulation of growth plate chondrogenesis.
48 fect in cartilage formation during postnatal chondrogenesis.
49 ctor that is required in successive steps of chondrogenesis.
50 velopment, and its deletion in mice disrupts chondrogenesis.
51 act as an important SOX9 co-activator during chondrogenesis.
52 endent transcriptional activation in primary chondrogenesis.
53 ranscriptional complex at the early stage of chondrogenesis.
54 DMP1 (dentin matrix protein 1) in postnatal chondrogenesis.
55 ents such as angiogenesis, osteogenesis, and chondrogenesis.
56 sion complexes during the early steps of MPC chondrogenesis.
57 -kappaB, which may play an important role in chondrogenesis.
58 irect transcriptional target of Runx2 during chondrogenesis.
59 ction of osteogenesis and the suppression of chondrogenesis.
60 ed longitudinal bone growth and growth plate chondrogenesis.
61 transcriptional repressor to promote somitic chondrogenesis.
62 inal bone growth by stimulating growth plate chondrogenesis.
63 nhibitor (SST0001), which strongly inhibited chondrogenesis.
64 he chondrocyte differentiation programme and chondrogenesis.
65 genesis, patterning of the oral skeleton and chondrogenesis.
66 density micromass' using TGF-beta3 to induce chondrogenesis.
67 suppresses osteogenesis, whereas it promotes chondrogenesis.
68 eurin/NFAT signaling pathway during tracheal chondrogenesis.
69 mimic mesenchymal condensation leading into chondrogenesis.
70 rning and at later post-patterning stages of chondrogenesis.
71 v3.2 is responsible for Ca(2+) influx during chondrogenesis.
72 smic reticulum stress transducer crucial for chondrogenesis.
73 ch mimics the function of nSMase2, inhibited chondrogenesis.
74 2+) channel Cav3.2 is essential for tracheal chondrogenesis.
75 ng a secreted factor during TGF-beta-induced chondrogenesis.
76 naling proteins including BMPs, and restrict chondrogenesis.
77 nally shifted phalangeal joints and impaired chondrogenesis.
78 etas (TGFbeta), regulate multiple aspects of chondrogenesis.
79 contribution of these N-cadherin peptides to chondrogenesis.
80 signaling required for joint patterning and chondrogenesis.
81 te decisions from osteogenesis to adipo- and chondrogenesis.
82 ractions during mesenchymal condensation and chondrogenesis.
83 more widely used chemical induced method for chondrogenesis.
84 and Sox9 may determine the earliest steps of chondrogenesis.
85 of chondrocyte hypertrophy and growth plate chondrogenesis, although the specific molecular mechanis
87 eta1 is a growth factor that is critical for chondrogenesis and binds to both biglycan and fibromodul
88 with exogenous heparanase greatly stimulated chondrogenesis and bone morphogenetic protein signaling
89 nction and that its absence leads to ectopic chondrogenesis and cartilage formation in conjunction wi
91 tides and evaluated their role in regulating chondrogenesis and cartilage matrix deposition by encaps
94 that Twist1 contributes to the repression of chondrogenesis and chondrocyte gene expression resulting
99 romodulin as novel key players in regulating chondrogenesis and ECM turnover during temoporomandibula
101 cle provides an overview of the processes of chondrogenesis and endochondral ossification and their c
104 ormone-related peptide, negatively regulates chondrogenesis and endochondral ossification via associa
107 t within an arthritic joint may also inhibit chondrogenesis and induce degradation of native and engi
111 plate chondrocytes facilitates growth plate chondrogenesis and longitudinal bone growth by inducing
112 Growth hormone (GH) stimulates growth plate chondrogenesis and longitudinal bone growth with its sti
113 determine whether CaR regulates growth plate chondrogenesis and longitudinal bone growth, we chose an
116 important in the regulation of angiogenesis, chondrogenesis and osteogenesis during limb development,
117 cascades in response to TGF-beta to control chondrogenesis and osteogenesis during mandibular develo
119 in human chondrocytes and potently inhibited chondrogenesis and osteogenesis using in vitro models.
123 involvement of actin binding proteins during chondrogenesis and provide a molecular basis to a human
125 P-4 by genetically engineered MDSCs enhanced chondrogenesis and significantly improved articular cart
127 trast, treatment with TGFbeta also supported chondrogenesis and stimulated Sox9 expression, but faile
129 findings identify JAWS as a key regulator of chondrogenesis and synovial joint positioning required f
130 PR1B are functionally redundant during early chondrogenesis and that BMP signaling is required for ch
131 ore suggest that GATA-6 also plays a role in chondrogenesis and that Gpr49 is a potential direct targ
132 he SRY-related SOX9 gene is involved in both chondrogenesis and the early steps of mammalian sex dete
133 novel genes that regulate human growth plate chondrogenesis and thereby contribute to the normal vari
135 of mTOR is necessary for cell proliferation, chondrogenesis, and cartilage growth during bone develop
137 transcription factor germane to osteogenesis/chondrogenesis, and increased migratory ability in KFs.
138 l proliferation and matrix remodeling during chondrogenesis, and is a key regulator coupling extracel
139 kewise, nMSCs presented compromised in vitro chondrogenesis, and Meckel's cartilage was underdevelope
141 expression of SOX9, the master regulator of chondrogenesis, and reducing SOX9 dosage allowed chondro
142 is a transcriptional activator required for chondrogenesis, and SOX5 and SOX6 are closely related DN
143 or), an important regulator of angiogenesis, chondrogenesis, and wound healing, is overexpressed in a
144 The abnormal progression of hypertrophic chondrogenesis appeared to be associated with the sustai
145 tiviral HMGB2 transduction of MSC suppressed chondrogenesis as reflected by an inhibition of Col2a1 a
147 CM1 seems to be critical for PTHrP action in chondrogenesis, as blockage of ECM1 nearly abolishes PTH
148 miR-199a(*) significantly inhibited early chondrogenesis, as revealed by the reduced expression of
149 mportant roles in both sex determination and chondrogenesis, as well as genes responsible for the pro
151 e to orchestrate spatiotemporal programs for chondrogenesis autonomously, and to implement cartilage
152 nstrate that FoxA factors are induced during chondrogenesis, bind to conserved binding sites in the c
153 ndicate that heparanase is able to stimulate chondrogenesis, bone morphogenetic protein signaling, ce
154 g the many factors involved in regulation of chondrogenesis, bone morphogenetic proteins (BMPs) and m
155 OX9 cooperative dimerization is required for chondrogenesis but not for sex determination and may exp
156 C3H10T1/2 cells can prevent the induction of chondrogenesis, but cannot reverse the chondrogenic phen
157 nt dimer when it activates genes involved in chondrogenesis, but functions as a monomer to activate g
158 scription factor Sox9 is necessary for early chondrogenesis, but its subsequent roles in the cartilag
159 redundant L-Sox5 and Sox6 proteins to effect chondrogenesis, but the mode of action of the trio remai
160 indicate that IGF-I stimulates growth plate chondrogenesis by activating NF-kappaB-p65 in chondrocyt
161 by increased chondrocyte gene expression and chondrogenesis by facilitating bone morphogenetic protei
162 ore, previous investigations of induction of chondrogenesis by human ESCs required embryoid body form
164 hat paraxis may regulate early events during chondrogenesis by positively directing transcription of
165 g factor beta subunit (CBFbeta), and induces chondrogenesis by regulating the CBFbeta-RUNX1 transcrip
166 n OA mainly by having a beneficial effect on chondrogenesis by the donor and host cells as well as by
167 principal processes underlying growth plate chondrogenesis, chondrocyte proliferation and hypertroph
168 esis RNA), by RNA interference disrupted MSC chondrogenesis, concomitant with reduced cartilage-speci
169 pendent and BMP-responsive manner to promote chondrogenesis, consistent with the ectopic endochondral
170 , decreased proliferation at early stages of chondrogenesis, delayed skeletal vascularization and del
172 limb bud patterning but defective or delayed chondrogenesis due to a lack of Sox9 and Col2a1 expressi
173 tic protein-2 (BMP-2) regulates growth plate chondrogenesis during development and postnatal bone gro
175 owed expression of a marker gene in areas of chondrogenesis during mouse development and in adult cho
176 regulated expression of Sox9, a regulator of chondrogenesis, during initiation of PF suture closure,
177 e the cellular function of key regulators of chondrogenesis found mutated in chondrodysplasia syndrom
179 mbers of the BMP family that are crucial for chondrogenesis, GDF5 and BMP4, regulate the pattern of B
181 r (GDF)-2) potently induces osteogenesis and chondrogenesis, has been implicated in the differentiati
184 gs suggest that BMP-6 is a potent inducer of chondrogenesis in ADAS cells, in contrast to mesenchymal
185 sectioning was used to investigate secondary chondrogenesis in disarticulated craniofacial elements o
186 hibited bone morphogenetic protein-2-induced chondrogenesis in high density micromass cultures of C3H
188 results reveal that the genetic pathway for chondrogenesis in lampreys and gnathostomes is conserved
190 d Kartogenin (KGN) - that greatly stimulates chondrogenesis in marrow-derived mesenchymal stem cells
191 he skeleton and that Tgfbr2 can act to limit chondrogenesis in mesenchymal cells like the interzone.
193 Wnt/beta-catenin signaling by LiCl enhances chondrogenesis in pericyte pellet cultures in the presen
194 itor of BMP receptors drastically attenuated chondrogenesis in recombinant human BMP 2-treated mutant
195 nexpected role of lipids as orchestrators of chondrogenesis in response to oxygen tension which is, a
196 the competence of somitic cells to initiate chondrogenesis in response to subsequent BMP signals by
197 ta-catenin, whereas Topol et al. report that chondrogenesis in the distal mouse limb bud depends on i
198 I collagen were noted when pericytes undergo chondrogenesis in the hydrogel in the absence of inducti
199 se data indicate that Osr1 normally prevents chondrogenesis in the mammalian tongue through repressio
200 cal mechanical environment promote secondary chondrogenesis in the mandibular adductor enthesis of du
201 rogenic genes, including Col2a1, followed by chondrogenesis in the MSC and developing chick limb.
203 steoprogenitor differentiation and perturbed chondrogenesis in the proximal region of the mandible.
204 ression of the myogenic factor Pax3 prevents chondrogenesis in these cells, while chondrogenic factor
206 and FGF8 have strong synergistic effects on chondrogenesis in vitro and are sufficient to promote ou
207 our results indicate that neogenin promotes chondrogenesis in vitro and in vivo, revealing an unexpe
210 and are sufficient to promote outgrowth and chondrogenesis in vivo, suggesting a very specific role
212 istology and expression of genes that affect chondrogenesis, including members of the FGF and BMP pat
213 ors have been identified that play a role in chondrogenesis, including the positive transacting facto
214 ion, which recapitulated the early stages of chondrogenesis, including transient vascularization.
215 iRNA targeting ANGPTL4 prior to induction of chondrogenesis increased expression of type II collagen
216 of precartilage condensations and subsequent chondrogenesis, indicating that downregulation of HA is
219 otides, nicotinamide, and IL-1beta inhibited chondrogenesis-induced down-regulation of cartilage-spec
220 nterior mesenchyme to undergo SOX9-dependent chondrogenesis, instead persisting as an interdigital-li
221 metabolic programming of the MSCs directing chondrogenesis into articular- or epiphyseal cartilage-l
222 rogramming of MSCs by oxygen tension directs chondrogenesis into either permanent or transient hyalin
225 and that repression of Runx2 at the onset of chondrogenesis is a prerequisite for the activation of a
227 n embryonic limb development have shown that chondrogenesis is initiated by cellular condensation, du
228 he transcriptional network that drives early chondrogenesis is intact, and that cell polarity within
230 ed in the developing tongue mesenchyme where chondrogenesis is subsequently activated to form the ect
231 Thus, one mechanism whereby Sox9 regulates chondrogenesis is to promote efficient beta-catenin phos
232 Sox9, a key transcriptional regulator of chondrogenesis, is required for TGF-beta-mediated regula
233 ining gene-9 (Sox9), the master regulator of chondrogenesis, is widely expressed in the nascent tongu
234 morphogenic protein (BMP) signaling promotes chondrogenesis, it is not clear whether BMP-induced chon
235 work and act in concert in the regulation of chondrogenesis.-Kong, L., Zhao, Y.-P., Tian, Q.-Y., Feng
236 blocks Wnt/beta-catenin signaling, inhibited chondrogenesis, leading to reduced Sox-9 and type II col
237 lular nanofiber scaffolds supported enhanced chondrogenesis marked by proteoglycan production minimal
238 hat laminins and nidogen-2 drive CPCs toward chondrogenesis may help in the elucidation of new treatm
240 ht of the inhibitory effects on growth plate chondrogenesis mediated by other FGFs, we hypothesized t
243 migration beneath the brain, requirements in chondrogenesis occur later, as cells form separate trabe
244 r thereafter in the course of BMP2-triggered chondrogenesis of a micromass culture of pluripotent C3H
246 e metabolism and is critical for accelerated chondrogenesis of ank/ank mesenchymal precursors and P(i
247 silencing suppressed Creb3l2 expression and chondrogenesis of ATDC5 cells, whereas infection of Alg2
248 e methods are similar to those published for chondrogenesis of bone marrow-derived mesenchymal stem c
249 mportant to better understand signal-induced chondrogenesis of chondrogenic progenitors in physiologi
252 TGF-beta1 treatment initiates and maintains chondrogenesis of MPCs through the differential chondro-
253 ptides onto HA hydrogels promotes both early chondrogenesis of MSCs and cartilage-specific matrix pro
254 at this 3D hydrogel environment supports the chondrogenesis of MSCs, and here we demonstrate through
255 s antagonist, soluble Flt-1 (sFlt-1), on the chondrogenesis of skeletal muscle-derived stem cells (MD
258 al, nonredundant role for CasR in regulating chondrogenesis or osteogenesis, but further studies are
259 ACVR1 as the underlying cause of the ectopic chondrogenesis, osteogenesis and joint fusions seen in F
261 condensation and has key roles in regulating chondrogenesis, osteogenesis, and bone and mineral homeo
262 s (e.g., type X collagen) by MSCs undergoing chondrogenesis raises concern for a tissue engineering a
263 ntetheinase, which inhibits synthesis of the chondrogenesis regulator glutathione, since we observed
264 02723505, which we termed ROCR (regulator of chondrogenesis RNA), by RNA interference disrupted MSC c
265 udal (occipital) chondrocranium, followed by chondrogenesis rostrally to form the nasal capsule, and
268 reduced expression of early marker genes for chondrogenesis such as cartilage oligomeric matrix prote
269 ation requirements for embryonic mesenchymal chondrogenesis, suggesting common chondro-regulatory mec
270 f Sox9 to both inhibit myogenesis and induce chondrogenesis, suggesting that Nkx3.2 is required for S
271 the 3'UTR of Sox9, the central initiator of chondrogenesis, suggesting that Twist1 might directly re
272 d that adopting an osteogenesis-inducing and chondrogenesis-suppressing cell fate in the ventral mese
274 ytes, a mechanism resembling in vivo somitic chondrogenesis that is not recapitulated with TGFbeta.
275 rk provides new insights into the control of chondrogenesis that may ultimately lead to a stem cell-b
276 in BMP-2-mediated initiation of mesenchymal chondrogenesis that results in activation of Sox9 at lea
278 The cells displayed a propensity to undergo chondrogenesis that was enhanced by treatment with exoge
279 ates one of the major pathways that promotes chondrogenesis, the transforming growth factor beta rece
280 RNAs was measured in the ATDC5 cell model of chondrogenesis using microarray and was verified using q
281 producible model system of human ESC-induced chondrogenesis, using a novel direct plating method in w
282 ndent on Vanin-1; however, Vanin-1 regulates chondrogenesis via glutathione metabolism and is critica
283 asia, identifying a mechanism that regulates chondrogenesis via modulation of SOX9 ubiquitination.
284 ese results demonstrate that Phlpp1 controls chondrogenesis via multiple mechanisms and that Phlpp1 i
289 pand our understanding of the role of VHL in chondrogenesis, we conditionally deleted VHL in mesenchy
290 ha in mesenchymal condensations and in early chondrogenesis, we conditionally inactivated Hif-1alpha
291 herin, and Wnt signaling in condensation and chondrogenesis, we have examined here their involvement,
292 gain insight into the role of ANGPTL4 during chondrogenesis, we used recombinant ANGPTL4 as well as a
293 air, the effects of selective EP agonists on chondrogenesis were examined in E11.5 long-term limb bud
295 av3.2 overexpression in ATDC5 cells enhances chondrogenesis, which could be blunted by both blocking
296 evaluate the effects of STC on growth plate chondrogenesis, which is the primary determinant of long
297 of the most important of these is secondary chondrogenesis, which occurs following ossification with
298 essential role for barx1 at early stages of chondrogenesis within the developing zebrafish viscerocr
300 promoting effect on IGF-1 expression and on chondrogenesis, yet it is not known whether other signal
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