1  HBDt structure has its highest affinity for chondroitin 6 sulfate.

2 ionic substrates such as keratan sulfate and chondroitin-6-sulfate.

3 arily from elevated levels of hyaluronan and chondroitin-6-sulfate.

4  substrate, this HL has weak activity toward chondroitin 6-sulfate and dermatan sulfate and can compl

5  lyase (HL), which degrades hyaluronan (HA), chondroitin 6-sulfate, and dermatan sulfate of the extra

6 strated that the DSP GAG attachments contain chondroitin 6-sulfate, but not keratan sulfate, heparan

7 0-fold for cathepsin K but only 25-fold with chondroitin-6-sulfate (C-6S).

8 wise degradation of keratan sulfate (KS) and chondroitin-6-sulfate (C6S).

9  sulfated GAGs (chondroitin-4-sulfate [C4S], chondroitin-6-sulfate [C6S], and dermatan sulfate [DS])

10                             In contrast, the chondroitin 6-sulfate CSC and heparin do not exert these

11 etyl-galactosamines in dermatan-4-sulfate or chondroitin-6-sulfate prevents the HC transfer.

12 o the deduced sequence of IMPG1, a purported chondroitin 6-sulfate proteoglycan core protein, which b

13 nd does not react to a specific antibody for chondroitin 6-sulfate proteoglycan.

14  (ECM) molecules, including chondroitin-4 or chondroitin-6 sulfate proteoglycans (CSPGs) and tenascin

15                  The mutant also synthesizes chondroitin 6-sulfate rather than only chondroitin 4-sul

16      PDGF also stimulated an increase in the chondroitin-6-sulfate to chondroitin-4-sulfate ratio of

17  IC(50) of 2,000 to 5,000 microg/mL, whereas chondroitin 6-sulfate was essentially inactive.

18                                          The chondroitin-6-sulfate was the major glycosaminoglycan sp