ライフサイエンスコーパス: chordae

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1 ately before and after dividing second-order chordae.

2 e reported mechanical weakness of myxomatous chordae.

3 apsed or flail segment, and 88% for ruptured chordae.

4 for off-pump transapical implantation of neo-chordae.

5 V repair with implantation of artificial neo-chordae.

6 The GAG classes elevated in the myxomatous chordae are associated with matrix microstructure and el

7 xtensible than normal valves, and myxomatous chordae are more mechanically compromised than leaflets.

8 of the anterior leaflet second-order "strut" chordae are unknown.

9 vular repair by severing second-order mitral chordae can improve annuloplasty by reducing papillary m

10 Cutting a minimum number of basal chordae can improve coaptation and reduce ischemic MR.

11 ic results of cutting thickened secondary MV chordae combined with a shallow septal muscular resectio

12 ction, demonstrating the importance of these chordae for LV structure and function.

13 Leaflets and chordae from myxomatous valves (n = 41 ULP, 31 BLP) and

14 Chordae from ULP had 62% more GAGs than those from BLP,

15 Myxomatous leaflets and chordae had 3% to 9% more water content and 30% to 150%

16 Cutting anterior mitral leaflet second-order chordae has been proposed for repair in ischemic mitral

17 erimposed fibrous tissue on both leaflet and chordae is likely the result of subsequent abnormal cont

18 Chordal rupture (i.e., missing chordae) occurred in all 37 patients, but finding indivi

19 Cutting second-order chordae resulted in LV systolic dysfunction and neither

20 Cutting the 2 central basal chordae reversed this without prolapse.

21 Thickened chordae showed endothelial and subendothelial alpha-smoo

22 e volume overload by percutaneously severing chordae tendinae of the mitral apparatus with a bioptome

23 ntric mitral regurgitation usually caused by chordae tendinae rupture or papillary muscle dysfunction

24 ncluded in this TACT (Transapical Artificial Chordae Tendinae) trial.

25 annulus (MA), papillary muscle (PM), and the chordae tendineac, chordal-sparing MVR is popular.

26 les, the fibrous tissue of cardiac valve and chordae tendineae and the course of coronary arteries.

27 he atrioventricular heart valve leaflets and chordae tendineae are composed of diverse cell lineages

28 hordae tendineae; and 2) the absence of many chordae tendineae on the ventricular surfaces of the lea

29 nitor cell diversification into leaflets and chordae tendineae that share inductive interactions and

30 produced by previous operation severing the chordae tendineae were examined.

31 genes scleraxis and tenascin, present in the chordae tendineae.

32 tic bleeding, and rupture of tricuspid valve chordae tendineae.

33 urfaces of the leaflets and surrounding many chordae tendineae; and 2) the absence of many chordae te

34 Biochemical changes were more pronounced in chordae than in leaflets.

35 f malformations of the papillary muscles and chordae, that can be detected by transthoracic and trans

36 Preservation of chordae to at least 1 mitral valve leaflet decreased ear

37 -pump transapical implantation of artificial chordae to correct MR is technically safe and feasible;

38 let and reattachment of the anterior leaflet chordae to either the anterior annulus (n = 7) or poster

39 ng a limited number of critically positioned chordae to the leaflet base that most restrict closure b

40 The strut chordae were encircled with exteriorized wire snares.

41 In 4 patients neo-chordae were not placed for technical and/or patient-spe

42 Both the leaflet and chordae were separated from the superimposed fibrous tis

43 sequent abnormal contact of the leaflets and chordae with one another.

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