ライフサイエンスコーパス: choriomeningitis

1 hat cause human diseases such as lymphocytic choriomeningitis, Bolivian hemorrhagic fever, and Lassa

2 murine gamma-herpesvirus 68 and lymphocytic choriomeningitis clone 13 and reversed T cell exhaustion

3 complexes formed during chronic lymphocytic choriomeningitis infection can interfere with Fcgamma-re

4 ion in humans and during chronic lymphocytic choriomeningitis infection in mice.

5 onstrated in vivo during chronic lymphocytic choriomeningitis infection.

6 , we focused on two RNA viruses: lymphocytic choriomeningitis (LCMV) and influenza (Flu).

7 tion, including the well-studied lymphocytic choriomeningitis (LCMV) and Pichinde (PICV) viruses, sev

8 s antigen tetramers and to LCMV (lymphocytic choriomeningitis)-reactive CD8+ T cells.

9 we have developed a recombinant lymphocytic choriomeningitis virus (LCMV) (rLCMVDeltaGP/GFP) where w

10 l known pathogenic arenaviruses, lymphocytic choriomeningitis virus (LCMV) and Lassa, Junin, Machupo,

11 C from the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) and LASV, which correlated

12 ith two natural mouse pathogens, lymphocytic choriomeningitis virus (LCMV) and murine cytomegalovirus

13 Prf1(--)) mice are infected with lymphocytic choriomeningitis virus (LCMV) and secondary (noninherite

14 e generated after infection with lymphocytic choriomeningitis virus (LCMV) and that these CD4 T cells

15 re we show that the arenaviruses lymphocytic choriomeningitis virus (LCMV) and the clinically used Ju

16 NPs) of the Old World arenavirus lymphocytic choriomeningitis virus (LCMV) and the New World arenavir

17 hmania major and 2 wk later with lymphocytic choriomeningitis virus (LCMV) and then monitored the cou

18 Mice were infected with lymphocytic choriomeningitis virus (LCMV) and treated with Mn(III)te

19 ections, such as infections with lymphocytic choriomeningitis virus (LCMV) and vaccinia virus, where

20 nrelated and pathogenic viruses, lymphocytic choriomeningitis virus (LCMV) and vaccinia virus.

21 In this study, using lymphocytic choriomeningitis virus (LCMV) and Zika virus (ZIKV) in w

22 endrocyte glycoprotein (MOG) and lymphocytic choriomeningitis virus (LCMV) antigens, respectively.

23 Using lymphocytic choriomeningitis virus (LCMV) as a model of a persistent

24 ion with a persistent variant of lymphocytic choriomeningitis virus (LCMV) but have no impact on vira

25 We show that lymphocytic choriomeningitis virus (LCMV) can also inhibit macrophag

26 Lymphocytic choriomeningitis virus (LCMV) can cause acute fatal dise

27 Lymphocytic choriomeningitis virus (LCMV) can establish acute or chr

28 f life-long chronic infection by lymphocytic choriomeningitis virus (LCMV) Cl 13 but does not affect

29 t mice chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13) are highly

30 is C virus or those of mice with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13), result in

31 ) when chronically infected with lymphocytic choriomeningitis virus (LCMV) clone 13 (CL13).

32 infection with HIV in humans or lymphocytic choriomeningitis virus (LCMV) clone 13 in mice.

33 ly induced on APCs at day 2 post-lymphocytic choriomeningitis virus (LCMV) clone 13 infection, but is

34 T cells from mice infected with lymphocytic choriomeningitis virus (LCMV) clone 13 into recipient mi

35 During chronic infection with lymphocytic choriomeningitis virus (LCMV) clone 13, miR-31-deficent

36 with that of a persistent virus, lymphocytic choriomeningitis virus (LCMV) clone 13, on early innate

37 urs after mice are infected with lymphocytic choriomeningitis virus (LCMV) clone 13, which is used as

38 Lymphocytic choriomeningitis virus (LCMV) clone 13, which normally e

39 ic event are more susceptible to lymphocytic choriomeningitis virus (LCMV) clone-13 infection exhibit

40 se against a massively exhausted lymphocytic choriomeningitis virus (LCMV) epitope.

41 mice persistently infected with lymphocytic choriomeningitis virus (LCMV) exhibit a severe defect in

42 we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global perturbatio

43 eficient animals failed to clear lymphocytic choriomeningitis virus (LCMV) from hematopoietic organs

44 unized with H(2)O(2)-inactivated lymphocytic choriomeningitis virus (LCMV) generated cytolytic, multi

45 d mice with Ad5 vectors encoding lymphocytic choriomeningitis virus (LCMV) glycoprotein (GP) and exam

46 denovirus vectors expressing the lymphocytic choriomeningitis virus (LCMV) glycoprotein (GP), followe

47 d35, and Ad48 vectors expressing lymphocytic choriomeningitis virus (LCMV) glycoprotein (GP).

48 and CD8(+) T cells responding to lymphocytic choriomeningitis virus (LCMV) identified multiple genes

49 acute primary infection with the lymphocytic choriomeningitis virus (LCMV) in mice is significantly f

50 itis B and C virus in humans and lymphocytic choriomeningitis virus (LCMV) in mice.

51 dotyped with the glycoprotein of lymphocytic choriomeningitis virus (LCMV) in vitro.

52 nges the outcome to a subsequent lymphocytic choriomeningitis virus (LCMV) infection and can result i

53 rus-specific CTLs during chronic lymphocytic choriomeningitis virus (LCMV) infection and suppressed C

54 fied during chronic versus acute lymphocytic choriomeningitis virus (LCMV) infection and suppresses T

55 f IAPs in T-cell immunity during lymphocytic choriomeningitis virus (LCMV) infection by pharmacologic

56 hough cellular immunity to acute lymphocytic choriomeningitis virus (LCMV) infection has been well ch

57 Recent studies in chronic lymphocytic choriomeningitis virus (LCMV) infection have defined a P

58 ction over the course of chronic lymphocytic choriomeningitis virus (LCMV) infection in an IL-10 repo

59 CD4(+) and CD8(+) T cells during lymphocytic choriomeningitis virus (LCMV) infection in an IL-21-depe

60 In contrast to MCMV, lymphocytic choriomeningitis virus (LCMV) infection in mice seems to

61 e to establishment of persistent lymphocytic choriomeningitis virus (LCMV) infection in mice through

62 on of CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection in mice.

63 ade during an acute or a chronic lymphocytic choriomeningitis virus (LCMV) infection in mice.

64 Lymphocytic choriomeningitis virus (LCMV) infection induces robust t

65 Using the lymphocytic choriomeningitis virus (LCMV) infection model, we show t

66 Using acute and chronic lymphocytic choriomeningitis virus (LCMV) infection models, we deter

67 n kinetics and viral loads after lymphocytic choriomeningitis virus (LCMV) infection of Ctx(-) mice.

68 we used a mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection to address this

69 cific CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection to examine the T

70 rom aseptic meningitis following lymphocytic choriomeningitis virus (LCMV) infection to hemorrhagic f

71 CD8(+) T-cell response to acute lymphocytic choriomeningitis virus (LCMV) infection was predominantl

72 during the first week of chronic lymphocytic choriomeningitis virus (LCMV) infection, before severe d

73 are essential for clearance of a lymphocytic choriomeningitis virus (LCMV) infection, but the virus c

74 t al. report that in response to lymphocytic choriomeningitis virus (LCMV) infection, fully different

75 During persistent lymphocytic choriomeningitis virus (LCMV) infection, IFNalpha and IF

76 In a mouse model of persistent lymphocytic choriomeningitis virus (LCMV) infection, it was shown th

77 ormation following immunization, lymphocytic choriomeningitis virus (LCMV) infection, or herpes simpl

78 pathology during early systemic lymphocytic choriomeningitis virus (LCMV) infection, suggesting a ho

79 bit reduced IFN-I responses upon lymphocytic choriomeningitis virus (LCMV) infection, which affects t

80 tion in vitro and in vivo during lymphocytic choriomeningitis virus (LCMV) infection.

81 HLH-like disease severity after lymphocytic choriomeningitis virus (LCMV) infection.

82 exhaustion in mice with chronic lymphocytic choriomeningitis virus (LCMV) infection.

83 cation in the context of chronic lymphocytic choriomeningitis virus (LCMV) infection.

84 uction at late stages of chronic lymphocytic choriomeningitis virus (LCMV) infection.

85 de in the mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection.

86 the manifestations of HLH after lymphocytic choriomeningitis virus (LCMV) infection.

87 lls are virus specific following lymphocytic choriomeningitis virus (LCMV) infection.

88 arly stages of acute and chronic lymphocytic choriomeningitis virus (LCMV) infection.

89 istributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected human patho

90 istributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected human patho

91 istributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected human patho

92 istributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is an important neglected

93 th the clone 13 (CL13) strain of lymphocytic choriomeningitis virus (LCMV) is extensively used as a m

94 The prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is widely used as a model

95 hronic Clone 13 (C13) strains of lymphocytic choriomeningitis virus (LCMV) led to two distinct phases

96 Ins2-GP(Tg) mice injected with lymphocytic choriomeningitis virus (LCMV) lost islet sympathetic ner

97 tion with the clone 13 strain of lymphocytic choriomeningitis virus (LCMV) or influenza virus.

98 In mice infected with lymphocytic choriomeningitis virus (LCMV) or Listeria monocytogenes

99 at infection of mice with either lymphocytic choriomeningitis virus (LCMV) or pneumonia virus of mice

100 tion with the systemic pathogens lymphocytic choriomeningitis virus (LCMV) or vaccinia virus (VACV).

101 using RIP-LCMV mice expressing a lymphocytic choriomeningitis virus (LCMV) protein in the beta-cells.

102 low-dose infection of mice with lymphocytic choriomeningitis virus (LCMV) results in massive expansi

103 ation after acute infection with lymphocytic choriomeningitis virus (LCMV) strain Armstrong.

104 tigen variant-expressing chronic lymphocytic choriomeningitis virus (LCMV) strains, we uncovered that

105 mice persistently infected with lymphocytic choriomeningitis virus (LCMV) suppressed multiple Fcgamm

106 The lymphocytic choriomeningitis virus (LCMV) system constitutes one of

107 stently infected from birth with lymphocytic choriomeningitis virus (LCMV) to demonstrate that therap

108 e used the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to develop a general molec

109 nant engineering of trisegmented lymphocytic choriomeningitis virus (LCMV) to express two genes of in

110 prototypic Old World arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere with RIG-I/MA

111 ity of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere with the indu

112 ) consisting of 2 nonreplicating lymphocytic choriomeningitis virus (LCMV) vectors expressing the hum

113 ced T(regs) generated upon acute Lymphocytic Choriomeningitis Virus (LCMV) WE and Vaccinia Virus (VV)

114 Lymphocytic choriomeningitis virus (LCMV) WE variant 2.2 (v2.2) gene

115 ses to chimeric vaccines against lymphocytic choriomeningitis virus (LCMV) were assessed in mice that

116 NP) of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) with the least frequently

117 nic infections such as HIV, HPV, lymphocytic choriomeningitis virus (LCMV), and schistosomiasis to ev

118 NP of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV), as well as those of the h

119 y molecule expression induced by lymphocytic choriomeningitis virus (LCMV), CD28/B7-mediated costimul

120 prototype member in the family, lymphocytic choriomeningitis virus (LCMV), disables the host innate

121 rf1(-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV), disease is driven by over

122 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a model for dissecting

123 stributed prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a neglected human path

124 ting mice with L. guyanensis and lymphocytic choriomeningitis virus (LCMV), or the sand fly-transmitt

125 We infected mice with chronic lymphocytic choriomeningitis virus (LCMV), performed RNA sequencing

126 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), provides investigators wi

127 ding Ebola virus, Marburg virus, lymphocytic choriomeningitis virus (LCMV), rabies virus, and Lassa v

128 Lymphocytic choriomeningitis virus (LCMV), the prototype arenavirus,

129 in memory phase challenged with lymphocytic choriomeningitis virus (LCMV), which we show in this stu

130 T(M) from lymphocytic choriomeningitis virus (LCMV)-immune and H60-vaccinated

131 (AP)-fed controls, LP feeding in lymphocytic choriomeningitis virus (LCMV)-immune mice resulted in a

132 In this environment, lymphocytic choriomeningitis virus (LCMV)-infected iFRCs activated n

133 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8(+) T cells ca

134 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8+ T cells in n

135 sion of CCL21 in murine spleens, lymphocytic choriomeningitis virus (LCMV)-specific T cell responses

136 e against infection with chronic lymphocytic choriomeningitis virus (LCMV).

137 ent of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

138 ector T cells postinfection with lymphocytic choriomeningitis virus (LCMV).

139 vivo during acute infection with lymphocytic choriomeningitis virus (LCMV).

140 at replicate the negative-strand lymphocytic choriomeningitis virus (LCMV).

141 ged with the Armstrong strain of lymphocytic choriomeningitis virus (LCMV).

142 against viral infection, such as lymphocytic choriomeningitis virus (LCMV).

143 fected with the mouse arenavirus lymphocytic choriomeningitis virus (LCMV).

144 e and infected the chimeras with lymphocytic choriomeningitis virus (LCMV).

145 tosis (HLH) after infection with lymphocytic choriomeningitis virus (LCMV).

146 ASP-deficient (WAS KO) mice with lymphocytic choriomeningitis virus (LCMV).

147 ponse to many viruses, including lymphocytic choriomeningitis virus (LCMV).

148 f1 (-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV).

149 n mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

150 on for the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

151 II molecules were infected with lymphocytic choriomeningitis virus (LCMV).

152 ed after systemic infection with lymphocytic choriomeningitis virus (LCMV).

153 R signals after clearance of the lymphocytic choriomeningitis virus (LCMV).

154 ing acute infection of mice with lymphocytic choriomeningitis virus (LCMV).

155 but not chronic, infection with lymphocytic choriomeningitis virus (LCMV).

156 n-deficient mice is triggered by lymphocytic choriomeningitis virus (LCMV).

157 n mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

158 s following acute infection with lymphocytic choriomeningitis virus (LCMV).

159 cular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV).

160 st three distinct ssRNA viruses: lymphocytic choriomeningitis virus (LCMV); influenza A virus (IAV);

161 th a variant glycoprotein of the lymphocytic choriomeningitis virus (LCMV-GP), creating a replicating

162 urine cytomegalovirus (MCMV) and lymphocytic choriomeningitis virus (LCMV; Cl13), in their natural ro

163 18-transgenic (Il18tg) mice with lymphocytic choriomeningitis virus (LCMV; strain Armstrong).

164 ollowed by replication-defective lymphocytic choriomeningitis virus (rLCMV) boost elicited robust CD4

165 We constructed recombinant lymphocytic choriomeningitis virus (rLCMV) featuring either addition

166 nts of the prototypic arenavirus lymphocytic choriomeningitis virus (rLCMVs), whose S-IGRs were repla

167 on CD8(+) T cells during chronic lymphocytic choriomeningitis virus and hepatitis C virus infections.

168 s also required for infection of lymphocytic choriomeningitis virus and human parainfluenza virus typ

169 expand and to protect from acute lymphocytic choriomeningitis virus and Leishmania major parasite inf

170 cribe its use in the contexts of lymphocytic choriomeningitis virus and Mycobacterium tuberculosis in

171 ell responses to infections with lymphocytic choriomeningitis virus and the intracellular bacteria Li

172 marenaviruses, the widely spread lymphocytic choriomeningitis virus and the recently described Wenzho

173 us and EBV in humans and between lymphocytic choriomeningitis virus and vaccinia virus in mice.

174 f a naive C57BL/6 recipient with lymphocytic choriomeningitis virus and vaccinia virus, followed by c

175 owed enhanced viral clearance in lymphocytic choriomeningitis virus and vesicular stomatitis virus in

176 administered at the beginning of lymphocytic choriomeningitis virus Armstrong infection anti-CD137 in

177 ent antiviral immunity using the lymphocytic choriomeningitis virus Armstrong strain acute infection

178 Using lymphocytic choriomeningitis virus Armstrong to probe the response t

179 ed by acute viral infection with lymphocytic choriomeningitis virus Armstrong.

180 with the natural murine pathogen lymphocytic choriomeningitis virus become more resistant to apoptosi

181 T cells during acute and chronic lymphocytic choriomeningitis virus challenges, but did not affect th

182 Pathogenic HIV or lymphocytic choriomeningitis virus chronic infections display a pers

183 that the Old World arenaviruses lymphocytic choriomeningitis virus clone 13 (LCMV Cl13) and Lassa vi

184 ist chronic viral infection with lymphocytic choriomeningitis virus clone 13 (LCMV cl13).

185 ltering Slamf6(+) numbers during lymphocytic choriomeningitis virus clone 13 infection.

186 viral control during persistent lymphocytic choriomeningitis virus clone 13 infection.

187 d prevented chronic infection in lymphocytic choriomeningitis virus clone 13- and reduced viral load

188 The G protein from lymphocytic choriomeningitis virus endowed VSV with the ability to s

189 ns expressing well-characterized lymphocytic choriomeningitis virus epitopes, we found that survival

190 adenovirus vector expressing the lymphocytic choriomeningitis virus glycoprotein (LCMVgp) (Ad5gp) inc

191 e during chronic infections with lymphocytic choriomeningitis virus in mice and hepatitis C virus in

192 slow-spreading acute) isolate of lymphocytic choriomeningitis virus induced large-scale microbiome sh

193 Infection with lymphocytic choriomeningitis virus induces monopoiesis in wild-type

194 T cell attrition in response to lymphocytic choriomeningitis virus infection and during CoB during t

195 ne responses to acute and chronic lymphocyte choriomeningitis virus infection are regulated by type 1

196 normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generated an increa

197 with this finding, we show that lymphocytic choriomeningitis virus infection can directly modulate t

198 responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lambdaR1-deficie

199 ls are comparable in controlling lymphocytic choriomeningitis virus infection in mice and suppress gr

200 TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, and determined

201 cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, and PD-1(hi) c

202 In a model of chronic lymphocytic choriomeningitis virus infection in mice, we show that e

203 levels of memory following acute lymphocytic choriomeningitis virus infection in mice.

204 c effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.

205 pecific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.

206 the control of acute and chronic lymphocytic choriomeningitis virus infection in the joint and spleen

207 (+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we found that th

208 a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin-deficient m

209 Here we demonstrate that chronic lymphocytic choriomeningitis virus infection rapidly triggers severe

210 sic manner early following acute lymphocytic choriomeningitis virus infection to suppress the magnitu

211 ntiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.

212 ent and serum IFN-I responses to lymphocytic choriomeningitis virus infection were augmented in newly

213 show that during chronic murine lymphocytic choriomeningitis virus infection, activation of AKT and

214 ced viral clearance in models of lymphocytic choriomeningitis virus infection, and also protection fr

215 ring the early stages of chronic lymphocytic choriomeningitis virus infection, and that this early T

216 in immunization; however, during lymphocytic choriomeningitis virus infection, B cells induce T(FH) d

217 In chronic lymphocytic choriomeningitis virus infection, blockade of type I IFN

218 Upon lymphocytic choriomeningitis virus infection, Cmah(-/-) mice make mo

219 R cells predominated response to lymphocytic choriomeningitis virus infection, comprising up to 60% o

220 gulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting functional ca

221 Following acute lymphocytic choriomeningitis virus infection, memory CD8 T cells in

222 Here we show that, following lymphocytic choriomeningitis virus infection, resident meningeal mac

223 the experimental mouse model of lymphocytic choriomeningitis virus infection, we demonstrate that th

224 Following lymphocytic choriomeningitis virus infection, we found most killer c

225 (+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show that memory ce

226 CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type I IFN levels

227 ls to mount a robust response to lymphocytic choriomeningitis virus infection, with both quantitative

228 reased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-deficient memo

229 ion to those in acute or chronic lymphocytic choriomeningitis virus infection.

230 ecific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.

231 nd in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.

232 e or different times after acute lymphocytic choriomeningitis virus infection.

233 ctor CD8 T cells following acute lymphocytic choriomeningitis virus infection.

234 function in viral clearance upon lymphocytic choriomeningitis virus infection.

235 iles and chromatin states during lymphocytic choriomeningitis virus infection.

236 l generation and responses after lymphocytic choriomeningitis virus infection.

237 by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.

238 T cell attrition resulting from lymphocytic choriomeningitis virus infection.

239 nd defective immune responses to lymphocytic choriomeningitis virus infection.

240 loping HLH immunopathology after lymphocytic choriomeningitis virus infection.

241 RM subsets was overcome by acute lymphocytic choriomeningitis virus infection; nevertheless, memory v

242 y also show that chronic HIV and lymphocytic choriomeningitis virus infections have a very different

243 d were unable to resolve chronic lymphocytic choriomeningitis virus infections.

244 responding to acute and chronic lymphocytic choriomeningitis virus infections.

245 tion during primary responses to lymphocytic choriomeningitis virus lowered the magnitude of CD8 Ag-s

246 ve upon this, we used the murine lymphocytic choriomeningitis virus model and adenoviral vectors to c

247 ivation was also verified in the lymphocytic choriomeningitis virus model, in which IFN-beta promoter

248 by two other NS RNA viruses, the lymphocytic choriomeningitis virus of the Arenaviridae family and hu

249 the impact of an infection with lymphocytic choriomeningitis virus on prenatal allospecific toleranc

250 protracted viral infection with lymphocytic choriomeningitis virus or during autoimmune diabetes dev

251 cell cycle after infection with lymphocytic choriomeningitis virus or vesicular stomatitis virus.

252 ot acute, infection of mice with lymphocytic choriomeningitis virus results in a marked expansion of

253 ction of Spi6 knockout mice with lymphocytic choriomeningitis virus revealed impaired survival of CD8

254 is cell turnover, we transferred lymphocytic choriomeningitis virus specific memory CD8 T cells into

255 ponses in mice given variants of lymphocytic choriomeningitis virus that cause acute or persisting in

256 in mice lacking B cells, whereas lymphocytic choriomeningitis virus titers were dramatically increase

257 in (Prf1)(KO) mice infected with lymphocytic choriomeningitis virus to genetically eliminate either I

258 gainst representative Old World (lymphocytic choriomeningitis virus) and New World (Junin virus) aren

259 detected in cells infected with lymphocytic choriomeningitis virus, an ambisense RNA virus, and minu

260 for vesicular stomatitis virus, lymphocytic choriomeningitis virus, and dengue virus but not for the

261 llaris, Cryptococcus neoformans, lymphocytic choriomeningitis virus, and West Nile virus.

262 found that in mice infected with lymphocytic choriomeningitis virus, colocalization of virus-specific

263 After infection of mice with lymphocytic choriomeningitis virus, IL-2Ralpha-deficient effector CD

264 murine CMV (MCMV), but not with lymphocytic choriomeningitis virus, induce CD25 on NK cells, along w

265 iciently promote CD8 immunity to lymphocytic choriomeningitis virus, nor did cav-1(-/-) OT-1(+) CD8(+

266 After infection with lymphocytic choriomeningitis virus, pearl mice developed all key fea

267 Using murine infection with lymphocytic choriomeningitis virus, we demonstrate that, in contrast

268 phase of an acute infection with lymphocytic choriomeningitis virus, we found that virus-specific CD8

269 fluenza hemagglutinin or GP from lymphocytic choriomeningitis virus, which enter through late endosom

270 The clone 13 (Cl13) variant of lymphocytic choriomeningitis virus--a prototype of Old World arenavi

271 ring splenocytes from individual lymphocytic choriomeningitis virus-immune donors into paired recipie

272 es development in the CD8-driven lymphocytic choriomeningitis virus-induced model of type 1 diabetes.

273 dual role in the development of lymphocytic choriomeningitis virus-induced, T cell-mediated hepatiti

274 e highly dynamic model system of lymphocytic choriomeningitis virus-mediated hepatitis and bone marro

275 ther germinal center B cells nor lymphocytic choriomeningitis virus-specific Ab levels were influence

276 sed Ig switch and low avidity of lymphocytic choriomeningitis virus-specific Abs despite intact IL-6R

277 fic human CD8 T cells or chronic lymphocytic choriomeningitis virus-specific CD8 T cells from mice; 3

278 al T-cell functions, "exhausted" lymphocytic choriomeningitis virus-specific cells losing the capacit

279 nsion of both primary and memory lymphocytic choriomeningitis virus-specific CTL, which could be corr

280 cells and the systemic levels of lymphocytic choriomeningitis virus-specific IgG2 Abs were dramatical

281 rom peripheral T cells using the lymphocytic choriomeningitis virus-specific P14 TCR.

282 ection, Cmah(-/-) mice make more lymphocytic choriomeningitis virus-specific T cells than WT mice, an

283 gnaling blockade on the residual lymphocytic choriomeningitis virus-triggered CTL response detected i

284 on in response to infection with lymphocytic choriomeningitis virus.

285 fate during acute infection with lymphocytic choriomeningitis virus.

286 derived from the glycoprotein of lymphocytic choriomeningitis virus.

287 ing acute infection of mice with lymphocytic choriomeningitis virus.

288 response to acute infection with lymphocytic choriomeningitis virus.

289 tion during acute infection with lymphocytic choriomeningitis virus.

290 highly disseminating variant of lymphocytic choriomeningitis virus.

291 alitis from organ donor-acquired lymphocytic choriomeningitis virus.

292 iral immunity after infection by lymphocytic choriomeningitis virus.

293 ty to mount a recall response to lymphocytic choriomeningitis virus.

294 mice persistently infected with lymphocytic choriomeningitis virus.

295 N1; measles; dengue; rabies; and lymphocytic choriomeningitis virus.

296 ell immunity by using the murine lymphocytic choriomeningitis virus.

297 s, including West Nile virus and lymphocytic choriomeningitis virus.

298 tions after acute infection with lymphocytic choriomeningitis virus.

299 nses during acute infection with lymphocytic choriomeningitis virus.

300 y ablated in T cells, with acute lymphocytic choriomeningitis virus.