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1 and lay poorly viable eggs with a defective chorion.
2 which caused the malformation of the amnion/chorion.
3 s in blood vessels of floating villi and the chorion.
4 containing Nile red are able to traverse the chorion.
5 in chorionic girdle relative to noninvasive chorion.
6 SNC adhered to the surface and inside of the chorion.
7 D), critical for allantoic elongation to the chorion.
8 unusually high thiol levels in the zebrafish chorion.
9 shapen allantois that fails to fuse with the chorion.
10 ion, is not expressed by cells in the mutant chorion.
11 pic allantois, which failed to fuse with the chorion, a phenotype that leads to subsequent cardiovasc
13 ing lung airway and vascular development and chorion-allantois fusion during placental development.
16 e DNA copy number along the third chromosome chorion amplicon was quantified during multiple developm
17 e the mcm proteins, DmCdc45 localises to the chorion amplification foci in the follicle cells of the
21 hed motor axons, lack of movement within the chorion and abnormal swimming in response to tactile sti
24 ly self-renewing TBPC lines derived from the chorion and alters expression and subcellular localizati
25 have isolated and grown cultures of primary chorion and amnion cells from human cesarean-section pla
26 y (E) 7.5, Irx4 transcripts are found in the chorion and at low levels in a discrete anterior domain
27 companied by lack of allantois fusion to the chorion and increased degeneration and necrosis in neura
29 eles of the chiffon gene cause thin, fragile chorions and female sterility, and were found to elimina
30 the expansion of the ectoplacental cone and chorion, and endoreduplication in trophoblast giant cell
31 f the visceral yolk sac, the ectoderm of the chorion, and subsequently the labyrinthine trophoblast o
33 D45(low)) of fetal origin was present in the chorion at all gestational ages, associated with stromal
37 were capable of transcytosing through intact chorion cell monolayers without disruption of intracellu
42 lymorphonuclear leukocytes in the amnion and chorion define histological chorioamnionitis (HCA), a co
44 hematopoietic potential of the allantois and chorion does not require their union, indicating that it
46 s encode the major protein components of the chorion (eggshell) and are arranged in two clusters in t
49 o meet the demand for the rapid synthesis of chorion (eggshell) proteins, Drosophila ovarian follicle
50 localization, we observe that ACE3, a 440-bp chorion element that contains information sufficient to
54 fy the zinc finger transcriptional regulator chorion factor 2 (CF2) as a factor functioning alongside
55 is regulated by the transcription activator Chorion factor 2 in flies and in tissue-culture cells.
57 in immunodeficient mice, were present in the chorion from 15-24 weeks gestation, but were absent at t
58 ial follicle cells, which begin synchronized chorion gene amplification after three rounds of endocyc
59 tations in Drosophila E2F and DP that affect chorion gene amplification and ORC2 localization in the
60 ion of DNA replication within the context of chorion gene amplification and transcriptional regulatio
61 cells deficient for Chiffon have a defect in chorion gene amplification but still undergo endocycling
62 e-sterile allele of k43 specifically reduces chorion gene amplification in ovarian follicle cells.
65 ns on chromosomes, and in follicle cells the chorion gene amplification loci are well-studied example
67 EP) and recq4(23), which specifically reduce chorion gene amplification of follicle cells by 4-5 fold
68 f2 mutant females display a 50% reduction in chorion gene amplification, and lay poorly viable eggs w
72 amplification defects over a 14-kb domain in chorion gene cluster suggests that RecQ4 may have a spec
73 nt for amplification of the third chromosome chorion gene cluster, ACE3 and Ori-beta, are directly bo
77 the construct did not detectably protect the chorion gene DNA replication origin from position effect
79 loss of Ttk69 in all follicle cells disrupts chorion gene expression and lack of function in dorsal a
80 dest such model systems is the regulation of chorion gene expression during ovarian follicle maturati
83 s large foci that localize to the endogenous chorion gene loci and to active transgenic constructs at
84 ion of replication to amplify the Drosophila chorion gene loci in the follicle cells of egg chambers.
85 ification of the Drosophila third chromosome chorion gene locus requires multiple chromosomal element
86 mmarize almost 40 years' worth of studies on chorion gene regulation while-by comparing Bombyx mori a
87 y associations and faster evolution of early chorion genes and transcriptionally active pseudogenes.
90 e developed a technique to detect amplifying chorion genes in individual follicle cells using BrdU in
92 expression throughout choriogenesis of most chorion genes originally categorized as "middle", and ev
94 ts interrupted by a 164 kb region with 5 non-chorion genes, orthologs of which were on chorion bearin
95 ls to increase the copy number of Drosophila chorion genes, which encode structural components of the
96 cle cells amplify genomic regions containing chorion genes, which facilitate secretion of eggshell pr
100 ults show that Duox-generated H2O2 fuels egg chorion hardening and that this process plays an essenti
101 ion peroxidase (CPO) plays a crucial role in chorion hardening by catalyzing chorion protein cross-li
103 In non-Drosophilid insects, the cuticle, chorion, immune response, silk gland, storage proteins,
104 t viral proteins are present in TBPCs of the chorion in cases of symptomatic congenital infection.
105 suggest that HCMV replicates in TBPCs in the chorion in vivo, interfering with the earliest steps in
107 d that the structural integrity of the outer chorion is dependent upon the presence of a vitelline me
112 minor alleles had 3-fold greater activity in chorion-like trophoblast cells (BeWo, JEG-3 and HTR-8/SV
116 he relative affinities to DNA from the third chorion locus and to random fragments in vitro, and chem
119 To determine the precise structure of the chorion locus, we performed extensive EST analysis, cons
123 placental villous cytotrophoblast cells and chorion membrane extravillous cytotrophoblast cells cont
126 rian membrane perforation repair with amnion-chorion membranes and results obtained from nine cases u
128 e data suggest that an MNL infiltrate in the chorion of the membrane roll marks PTD pathways that are
129 ore than 10 MNLs per high-power field in the chorion of the membrane roll, referred to as MNL-CMR, wa
130 within a cycle, and that specific factors at chorion origins allow them to escape this negative rerep
136 he follicle cells and is required for normal chorion production and dorsal follicle-cell migration.
137 regulate eggshell synthesis suggest that the chorion production and follicle-cell migration defects a
138 cial role in chorion hardening by catalyzing chorion protein cross-linking through dityrosine formati
141 e membrane appears to act as a reservoir for chorion proteins since s36 was found predominantly in th
143 otype-by-environment interactions, including chorion proteins, proteins regulating meiotic recombinat
144 accumulation profiles, s36 and s18, putative chorion proteins, were similarly distributed throughout
146 ant mice, the allantois fails to fuse to the chorion, resulting in a lack of placenta and failure to
147 variant transcripts appear in human amnion, chorion, skeletal muscle, small intestine, and in cell c
149 embryonic mesoderm derivatives including the chorion, the allantois, the amnion and a subset of endot
150 tribution of the fetal membranes, amnion and chorion, to human embryonic and fetal hematopoiesis.
153 expression patterns in 72 samples of amnion, chorion, umbilical cord, and sections of villus parenchy
154 ome localized within distinct regions of the chorion, while others are taken up by the oocyte or beco
155 ile extraembryonic structures, including the chorion, yolk sac blood islands, and allantois appear to
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