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1 ge, 1.46-7.90 mm2] in group 3; P = .001) and choroidal (11.83 [5.55] mm2 [range, 4.55-22.14 mm2] in g
2 ptic nerve invasion, or combined non-massive choroidal and prelaminar/laminar optic nerve invasion.
3 ilateral optic disc edema, globe flattening, choroidal and retinal folds, hyperopic refractive error
4                                  To evaluate choroidal and suprachoroidal changes following suprachor
5                        Models of retinal and choroidal angiogenesis, including oxygen-induced retinop
6 ed fatty acids (LC-PUFAs) reduce retinal and choroidal angiogenesis.
7 etermined by dividing vascular area by total choroidal area.
8 older affected members showed severe central choroidal atrophy.
9 o appear in highly myopic eyes with advanced choroidal atrophy.
10 iography revealed gradual restoration of the choroidal blood flow and unmasking of the big choroidal
11 migraine patients without aura and pulsative choroidal blood flow may not be affected during the chro
12              In this model, glucose from the choroidal blood passes through the retinal pigment epith
13                           Abnormal growth of choroidal blood vessels, or choroidal neovascularization
14 entries, 3217 patients with ciliary body and choroidal (CBC) melanoma and 160 with iris melanoma were
15 revalence of drusen-like deposits (DLDs) and choroidal changes in patients with systemic lupus erythe
16        The aim of this study was to evaluate choroidal changes occurring in asymptomatic, acute and c
17 ficant effect in alive rabbit eyes; however, choroidal circulation seems to be a significant effect t
18 sive method of 3D imaging of the retinal and choroidal circulations.
19 nd CH with craniofacial dysmorphic features, choroidal coloboma and endoderm-derived organ malformati
20 (RPE)/choroid and positively correlated with choroidal degeneration in the early stages of retinopath
21 (n = 1; 2.3%), wound leak (n = 1; 2.3%), and choroidal detachment (n = 2; 4.5%).
22  endophthalmitis in 6.3% (3 of 48 eyes), and choroidal detachment or hemorrhage in 8.3% (4 of 48 eyes
23 (all but 2 spontaneously resolving) included choroidal effusion (1), vitreous hemorrhage (3), Desceme
24 P increase (n = 9), pupillary block (n = 1), choroidal effusion (n = 2), CME (n = 4), and redislocati
25 branch retinal artery occlusion and multiple choroidal emboli.
26  family members are expressed in retinal and choroidal endothelial cells (ECs).
27                                        Human choroidal endothelial cells expressed lower TRPV4 mRNA/p
28 xamined by 2 experts to document retinal and choroidal findings.
29 cation of the regional structural context of choroidal flow interest between different imaging modali
30             For each patient, these areas of choroidal flow void on OCTA persisted during the follow-
31                           All eyes had inner choroidal flow void on OCTA that topographically corresp
32 apillary wrinkles (PPW), retinal folds (RF), choroidal folds (CF), and creases using transaxial and e
33 uts experience ophthalmic changes, including choroidal folds, optic disc edema, cotton-wool spots, gl
34 tudy were to further define the functions of choroidal gammadelta T cells and to explore the underlyi
35                    Our data demonstrate that choroidal gammadelta T cells are activated by RPE injury
36  We have previously reported the presence of choroidal gammadelta T cells in a model of chronic degen
37                                              Choroidal gammadelta T cells in protection against retin
38 ns revealed altered outer retinal layers and choroidal hypertransmission.
39 trols with emmetropia in both eyes underwent choroidal imaging using spectral-domain optical coherenc
40 ome contributed to both the acute unilateral choroidal infarction and to the chronic development of b
41                  This revealed an antecedent choroidal infarction that explained the visual field sco
42 erm follow-up of a young female patient with choroidal infarction, primary open angle glaucoma and Fl
43 8 eyes (51%), with characteristic multifocal choroidal infiltrates in 29 eyes (39%).
44 en on multimodal imaging include histiocytic choroidal infiltration causing choroidal lesions, compli
45 e presence of a thin choroid, a perivascular choroidal inflammatory infiltrate, and atrophic changes
46    For the detection of co-occurring massive choroidal invasion and postlaminar optic nerve invasion
47  body/muscle infiltration, massive (>/=3 mm) choroidal invasion, retrolaminar optic nerve invasion, o
48 e investigated the mechanisms underlying the choroidal involution and its long-term impact on retinal
49           We have recently demonstrated that choroidal involution occurs early in retinopathy.
50 retinal vasculitis (374 of 874 [42.8%]), and choroidal involvement (419 of 651 [64.4%]).
51 patients with panuveitis having vitreous and choroidal involvement had a higher risk of treatment fai
52 5% CI, 1.18-11.62; P = .02), and presence of choroidal involvement had an adjusted HR of 2.88 (95% CI
53 were obtained at the outer retinal layer and choroidal layer levels.
54 ment with sorafenib showed regression of the choroidal lesions and resolution of the SRD on multimoda
55                            Patients with few choroidal lesions had a favorable outcome, whereas all p
56 cent on ICG angiography, and correlated with choroidal lesions on SD OCT.
57                                The extent of choroidal lesions varied from few in 2 patients to wides
58                                              Choroidal lesions were identified and analyzed in four o
59                                   Multifocal choroidal lesions were observed bilaterally in all patie
60 e histiocytic choroidal infiltration causing choroidal lesions, complicated by recurrent serous retin
61 is, optic disc swelling, and white-yellowish choroidal lesions.
62 at the outer retinal layer level than at the choroidal level in group 3 (47.70% [31.30%] [range, 13.6
63 laris starting at a size much smaller than a choroidal lobule.
64                           Retinal microglia, choroidal macrophages, and recruited monocytes, collecti
65 atients treated with proton beam therapy for choroidal melanoma (CM), using optical coherence tomogra
66 d effective tumor control for juxtapapillary choroidal melanoma and may be associated with reduced ra
67 3DUS in 45 consecutive patients with primary choroidal melanoma to determine the percentage agreement
68    All patients with primary ciliary body or choroidal melanoma treated with iodine 125 or ruthenium
69 s of 57 patients with a clinically diagnosed choroidal melanoma underwent complete 25-gauge posterior
70 f radiation to the fovea, many patients with choroidal melanoma with foveal involvement maintain usef
71     Forty-seven patients with juxtapapillary choroidal melanoma.
72 dose plaque brachytherapy for juxtapapillary choroidal melanoma.
73  optimal therapeutic dose for juxtapapillary choroidal melanoma.
74 nsecutive cohort study of patients with T3-4 choroidal melanomas according to the 7th edition of the
75 cy of proton beam irradiation (PBI) of large choroidal melanomas has not been reported.
76        Three hundred fifty-one patients with choroidal melanomas located 1 disc diameter (DD) or less
77 s of 57 patients with treatment-naive medium choroidal melanomas without extraocular extension from J
78 owth following globe-preserving treatment of choroidal melanomas, and may be a valuable prognostic in
79 of 203 of the patients with small and medium choroidal melanomas, the effect of a reduced dose of rad
80 who underwent PBI for the treatment of large choroidal melanomas.
81 ive therapeutic option for the management of choroidal metastasis in selected cases.
82 technique that can visualize the retinal and choroidal microvasculature without the injection of exog
83 es were matched at baseline for VA, age, and choroidal neovascular membrane (CNV) size.
84 ) looking at risk factors for development of choroidal neovascular membrane (CNVM) and visual loss.
85                       For analyses by visit: choroidal neovascular membrane activity graded by the tr
86 because this cohort of patients had advanced choroidal neovascular membrane upon enrollment (recurren
87 To describe the characteristics of pediatric choroidal neovascular membranes (CNVs) associated with r
88 in photodynamic therapy for the treatment of choroidal neovascular membranes, has also been shown to
89 ow to monitor for the early detection of the choroidal neovascular stage before substantial vision lo
90 treal SH-11037 (1 muM) significantly reduced choroidal neovascularisation (CNV) lesion volume in the
91 ct Cynomolgus against laser-induced grade IV choroidal neovascularisation (CNV).
92                        In a mouse model with choroidal neovascularisation akin to age-related macular
93 ment epithelium (RPE) deposits, RPE atrophy, choroidal neovascularisation and photoreceptor cell deat
94 ociations between all rare pLoF variants and choroidal neovascularization (CNV) (OR, 1.34; 95% CI, 1.
95 lemonitoring for early detection of incident choroidal neovascularization (CNV) among patients with a
96                                              Choroidal neovascularization (CNV) and capillary dilatio
97 cholesterolemia, worse visual acuity, larger choroidal neovascularization (CNV) area, retinal angioma
98 wever, these mice develop significantly less choroidal neovascularization (CNV) compared to wild-type
99 ace changes over time and their influence on choroidal neovascularization (CNV) development.
100 raphy angiography (OCTA) in the detection of choroidal neovascularization (CNV) in age-related macula
101 acteristics and natural history of quiescent choroidal neovascularization (CNV) in geographic atrophy
102 was an 85.5% mean reduction from baseline in choroidal neovascularization (CNV) size.
103                           In contrast to the choroidal neovascularization (CNV) subtype, the genetic
104  was more prominent in patients with classic choroidal neovascularization (CNV) than those with occul
105                                              Choroidal neovascularization (CNV) was manually segmente
106                The molecular pathogenesis of choroidal neovascularization (CNV), an angiogenic proces
107 data from retinas treated with laser-induced choroidal neovascularization (CNV), bright white-light e
108 normal growth of choroidal blood vessels, or choroidal neovascularization (CNV), is a hallmark of the
109 -induced retinopathy (OIR) and laser-induced choroidal neovascularization (CNV), mimicking hypoxia-me
110 ous proliferation (RAP) lesions, and classic choroidal neovascularization (CNV).
111 ent white dot syndrome (MEWDS) who developed choroidal neovascularization (CNV).
112 of irreversible blindness and manifests with choroidal neovascularization (CNV).
113 cular degeneration (AMD) is characterized by choroidal neovascularization (CNV).
114 te AMD subtypes; geographic atrophy (GA) and choroidal neovascularization (CNV).
115 outcomes in eyes with treatment-naive myopic choroidal neovascularization (mCNV) in the United States
116 l fibrosis after regression of laser-induced choroidal neovascularization and a decrease in mesenchym
117 These retinal lesions can be associated with choroidal neovascularization and central serous choriore
118 6.0 diopters with the presence of subretinal/choroidal neovascularization as indicated by Internation
119 al neovascularization in 3 eyes and inactive choroidal neovascularization in 1 eye.
120 f a hyperflow signal corresponding to active choroidal neovascularization in 3 eyes and inactive chor
121 pressed initiation and growth of spontaneous choroidal neovascularization in a mouse model, and the c
122 aditional multimodal imaging, helps diagnose choroidal neovascularization in patients with Malattia L
123 efined as HM with the presence of subretinal/choroidal neovascularization indicated by the ICD-9-CM d
124            Pigmented mice with laser-induced choroidal neovascularization lesions (n = 7 eyes) were a
125 ffect on both area and number of spontaneous choroidal neovascularization lesions.
126 r of patients with treatment-naive subfoveal choroidal neovascularization receiving intravitreal rani
127  participants, aged >/=50 years, with active choroidal neovascularization secondary to AMD.
128                               The mother had choroidal neovascularization that was treated with bevac
129                       Current treatments for choroidal neovascularization, a major cause of blindness
130 inal pigment epithelium (RPE) abnormalities, choroidal neovascularization, acquired vitelliform lesio
131 ng oxygen-induced retinopathy, laser-induced choroidal neovascularization, and transgenic mouse model
132 models with deficient or spontaneous retinal/choroidal neovascularization, as well as models with ind
133 se vision than their mother, despite lacking choroidal neovascularization, because of the extent of p
134 ctional role for TLR2 in the pathogenesis of choroidal neovascularization, likely by promoting inflam
135 escein-negative intraretinal cystic changes, choroidal neovascularization, serous retinal elevations
136 n 2 (12%), a dome-shaped macula in 1 (6%), a choroidal neovascularization-related subretinal scar in
137 tion of nitric oxide (NO) in macrophages and choroidal neovascularization.
138 2 as a novel therapeutic target for reducing choroidal neovascularization.
139 nd photoreceptors and can be associated with choroidal neovascularization.
140 fundus changes and potentially of subsequent choroidal neovascularization.
141 n strongly implicated in the pathogenesis of choroidal neovascularization.
142 s, both normal and with macular degeneration/choroidal neovascularization.
143 features from pathologic RPE detachments and choroidal neovascularizations (CNVs).
144 ield imaging increased the identification of choroidal nevi by 27% (406 eyes [5.3%] by NMFP vs 545 ey
145 n: Three patients with a recent diagnosis of choroidal nevi underwent a novel adaptive optical assess
146 ld change the current clinical evaluation of choroidal nevi.
147 es in the photoreceptor mosaic overlying the choroidal nevi.
148 or abnormalities in the retina overlying the choroidal nevi.
149 treated with primary proton beam therapy for choroidal or ciliary body melanoma between June 1998 and
150     Severity of acute ischemic damage to the choroidal, outer retinal and optic nerve head.
151    Age and axial length were associated with choroidal parameters in healthy subjects.
152                                        Novel choroidal parameters such as choroidal reflectivity may
153 , was computed using the formula: normalized choroidal reflectivity = (choroidal reflectivity-vitreou
154     There was a negative correlation between choroidal reflectivity and choroidal thickness (r = -0.7
155                                              Choroidal reflectivity and choroidal thickness changes a
156                                              Choroidal reflectivity and choroidal thickness.
157                                       Higher choroidal reflectivity and lower choroidal thickness wer
158      To characterize choroidal thickness and choroidal reflectivity in the eyes of patients with bird
159           Novel choroidal parameters such as choroidal reflectivity may warrant further study in the
160                    An adjusted or normalized choroidal reflectivity, with the RPE as the bright refer
161 ormula: normalized choroidal reflectivity = (choroidal reflectivity-vitreous reflectivity)/RPE reflec
162 P < 0.01) all were significant predictors of choroidal reflectivity.
163 ak, endophthalmitis, intravitreal injection, choroidal retinal vitreal biopsy, and human immunodefici
164 3) and VCT (0.750-0.869), even in eyes where choroidal-scleral junction visibility was <75%.
165         EDI-OCT images were obtained and the choroidal-scleral junction was analyzed through semiauto
166 ak and Arden ratio (P = .06) and presence of choroidal spots on infracyanine green angiograms (80.0%
167 2) mum below the RPE reference plane and the choroidal stalks at 80.4 (24.4) mum below RPE reference
168 e within the dome of the RPE detachment, the choroidal stalks were all in the choroid layer.
169 y lesions in 3 patients leading to localized choroidal thickening with elevation of the overlying ret
170 e LD-BM was influenced significantly more by choroidal thickness (1.14 mum/mum; 95% confidence interv
171                       To identify changes in choroidal thickness (CT) and all retinal layers of diabe
172    The aim of this study was to evaluate the choroidal thickness (CT) changes in obese women, using o
173              To provide a normal database of choroidal thickness (CT) in nine Early Treatment Diabete
174  SS OCT was used to obtain automatic macular choroidal thickness (CT) maps, according to the Early Tr
175 central macula affect the reproducibility of choroidal thickness (CT) measurements on enhanced depth
176 ral retinal thickness (CRT [excluding SND]); choroidal thickness (CT); nasal and temporal retinal thi
177                                       Foveal choroidal thickness (FCT), subretinal hyperreflective ma
178                            A higher baseline choroidal thickness (for every 100-mum increase in choro
179 tiplying the average CVD by macular area and choroidal thickness (obtained with SS-OCT automated soft
180 omatically segment and measure peripapillary choroidal thickness (PCT) from circle scans centered on
181          We found that thinner peripapillary choroidal thickness (PPCT) was independently associated
182 rrelation between choroidal reflectivity and choroidal thickness (r = -0.793; P < 0.001).
183                                    Subfoveal choroidal thickness (SCT) was measured using the SDOCT.
184 measures of rod-mediated dark adaptation and choroidal thickness (time to rod intercept versus choroi
185 idal thickness (time to rod intercept versus choroidal thickness 0.072 (CI95 -0.23 to 0.38) min/100 m
186                                              Choroidal thickness 2.5 degrees above the fovea was 348
187 scores and age, RIT, AMD severity, subfoveal choroidal thickness [SFCT], phakic status, and best-corr
188 ly significant except for the association of choroidal thickness and "peripheral vision." The stronge
189     A negative correlation between subfoveal choroidal thickness and age was detected in all regions
190 ver, because the genetic correlation between choroidal thickness and AMD severity was not significant
191               Phenotypic correlation between choroidal thickness and CARMS category was moderate (Spe
192                              To characterize choroidal thickness and choroidal reflectivity in the ey
193            Repeatability and heritability of choroidal thickness and its phenotypic and genetic corre
194           In subjects with intermediate AMD, choroidal thickness and vessel volume are reduced in the
195 correlate interobserver findings and compare choroidal thickness and visual acuity at each time point
196 ctional measurements of DA testing (RIT) and choroidal thickness are associated with patient-reported
197 hy, central retinal thickness, and subfoveal choroidal thickness are likely to be valuable in monitor
198        For data analysis, we considered mean choroidal thickness as the arithmetic mean value of the
199                                          The choroidal thickness at the foveal center was measured.
200   There was no statistical difference in the choroidal thickness between the different time points (b
201                   Choroidal reflectivity and choroidal thickness changes are evident in active and in
202                                              Choroidal thickness changes were similar despite differe
203 a reference plane to reduce the influence of choroidal thickness changes.
204            Active BSCR patients showed lower choroidal thickness compared with controls (P < 0.01).
205                                              Choroidal thickness decreased 6 months after surgery (P
206                                    Subfoveal choroidal thickness decreased significantly in both typi
207                                    Subfoveal choroidal thickness decreased with age, whereas central
208 al thickness when trying to estimate overall choroidal thickness from any central measurement.
209 choroidal injection of CLS-TA does not alter choroidal thickness in eyes with macular edema due to RV
210                              The increase in choroidal thickness is a feature of the LHON acute stage
211                                      Macular choroidal thickness measured to the outer choroidal vess
212 e interobserver correlation was obtained for choroidal thickness measurements (r = 0.97, P < 0.0001)
213                                              Choroidal thickness measurements are more reproducible w
214                                              Choroidal thickness therefore may capture variation not
215 oveal fluid and a reduction of the subfoveal choroidal thickness to 271 mum after a 3 months follow-u
216 e test for genetic variation associated with choroidal thickness to determine the overlap in genetic
217 macular subretinal fluid with an increase of choroidal thickness up to 341 mum.
218                               Mean subfoveal choroidal thickness was 156 mum (147, 42-362 mum) for GA
219                                 Reduction in choroidal thickness was associated with elevated C-react
220                                    Subfoveal choroidal thickness was greater in patients with SLE (P
221                                              Choroidal thickness was highly repeatable within individ
222                                              Choroidal thickness was measured at each time point (bas
223                           Baseline subfoveal choroidal thickness was not significantly different betw
224      Higher choroidal reflectivity and lower choroidal thickness were documented in inactive BSCR pat
225 ayer thickness, in addition to peripapillary choroidal thickness were measured.
226      There is a consistent overestimation of choroidal thickness when trying to estimate overall chor
227     DRI SS OCT provides a topographic map of choroidal thickness with an ETDRS layout.
228            Baseline predictors (particularly choroidal thickness) for functional response (best-corre
229 dal thickness (for every 100-mum increase in choroidal thickness) was found to be a positive predicto
230             On multiple regression analysis, choroidal thickness, age, and disease duration (all P <
231 ar coherence tomography was used to quantify choroidal thickness, and fundus photography was used to
232 jects, to evaluate macular and peripapillary choroidal thickness, and retinal nerve fiber layer (RNFL
233             CR also worsened with increasing choroidal thickness, but was not affected by retinal thi
234 ce of thickest point from the foveal center, choroidal thickness, choroidal volume, choroidal vascula
235  wide range of individual rates of change of choroidal thickness, from -20.00 to 17.09 mum/year (mean
236 on the LLQ, with both reduced DA and reduced choroidal thickness, in a population of older adults wit
237 levations mimicking retinal folds, increased choroidal thickness, lack of rapid visual recovery, and
238                                              Choroidal thickness, refraction and ocular axial length
239 urements of RNFL thickness and peripapillary choroidal thickness, respectively.
240 ickness, VCT), outer choroid stroma (stromal choroidal thickness, SCT), or inner scleral border (tota
241 ckness, SCT), or inner scleral border (total choroidal thickness, TCT) showed no significant changes
242 o the outer choroidal vessel lumen (vascular choroidal thickness, VCT), outer choroid stroma (stromal
243 d PEDs associated with polyps and a variable choroidal thickness, while CSC patients had a thick chor
244 nalyses, RIT had a stronger association than choroidal thickness.
245 longed DA testing (longer RIT) and decreased choroidal thickness.
246 ly in eyes with subretinal fluid and greater choroidal thickness.
247 daptation can be attributed to variations in choroidal thickness.
248 ti-VEGF injections, PDT therapy, or baseline choroidal thickness.
249  with enhanced depth imaging mode to measure choroidal thickness.
250  focal dark adaptation (DA) testing and with choroidal thickness.
251 eme lighting," and "mobility" also including choroidal thickness.
252                   Choroidal reflectivity and choroidal thickness.
253 I SS OCT, and line measurements of subfoveal choroidal thicknesses (SFCT) were also performed.
254 es (100%), retinal thinning in 8 eyes (89%), choroidal thinning in 7 eyes (78%), and colobomatouslike
255                      Our study suggests that choroidal thinning in eyes with high myopia is associate
256        To evaluate the rate of peripapillary choroidal thinning in glaucoma patients and healthy cont
257                                  Significant choroidal thinning occurred in most patients (25/28 [89%
258                    The rate of peripapillary choroidal thinning was not significantly different betwe
259  thinning of the retinal pigment epithelium, choroidal thinning, undifferentiated nuclear layers of t
260 ocalizing with severe RPE depigmentation and choroidal thinning.
261                                              Choroidal tuberculosis is present in 5-20% of patients w
262  (95% CI: 0.59, 0.80; P = .0004) for massive choroidal tumor invasion (n = 219).
263                   All relapsing patients had choroidal tumors and 15 presented with visual symptoms.
264 s with high myopia, after adjusting for age, choroidal vascular and stromal areas were significantly
265 nter, choroidal thickness, choroidal volume, choroidal vascular and stromal areas within the macular
266 l imaging findings in AMN, with attention to choroidal vascular changes.
267                                   To compare choroidal vascular density (CVD) and volume (CVV) in dia
268                                              Choroidal vascular density (CVD) was calculated as a per
269                                              Choroidal vascular density and volume are significantly
270                                   To compare choroidal vascular features of eyes with and without sub
271   These findings suggest that areas of inner choroidal vascular flow void on OCTA are seen in patient
272    Compared to control eyes with emmetropia, choroidal vascularity was greater in eyes with high myop
273 ular (6 mm) and foveal (1.5 mm) regions, and choroidal vascularity, which was determined by dividing
274                     SS OCT en face images of choroidal vasculature were also captured and converted t
275                 En face SS-OCT images of the choroidal vasculature were binarized.
276 ess in vivo visualization of the retinal and choroidal vasculature.
277 re observed in 7 cases, including polypoidal choroidal vasculopathy (PCV) and macular fibrosis or atr
278 and volume predict retreatment in polypoidal choroidal vasculopathy (PCV).
279 erstanding of the pathogenesis of polypoidal choroidal vasculopathy (PCV).
280                     Historically, polypoidal choroidal vasculopathy complexes are less responsive to
281 l of intravitreal aflibercept for polypoidal choroidal vasculopathy in 21 eyes was conducted.
282                         Eyes with polypoidal choroidal vasculopathy previously treated with ranibizum
283 ions with regression of polyps in polypoidal choroidal vasculopathy.
284 ting aflibercept in management of polypoidal choroidal vasculopathy.
285  further exploration of the contributions of choroidal vessel disease to diabetic eye disease pathoge
286                      The presence of a large choroidal vessel elevating the RPE and the absence of ab
287 ar choroidal thickness measured to the outer choroidal vessel lumen (vascular choroidal thickness, VC
288 pro-angiogenic proteins that promote ex vivo choroidal vessel sprouting.
289                                         Mean choroidal vessel volume was also significantly reduced i
290                                              Choroidal vessel volume was calculated by multiplying th
291 ted from the presence of an underlying large choroidal vessel.
292  demonstrated hyperreflectivity around large choroidal vessels and at the level of the choriocapillar
293 , depigmented fundus, unmasking of the large choroidal vessels and optic atrophy; fluorescein angiogr
294 was calculated as a percent area occupied by choroidal vessels in a 6-mm-diameter submacular circular
295 , calculated as the percent area occupied by choroidal vessels in the central macular region (6-mm-di
296 In all eyes, RPE humps corresponded to large choroidal vessels lifting the RPE.
297       The dense autonomic innervation of the choroidal vessels predisposes them particularly to vasos
298 horoidal blood flow and unmasking of the big choroidal vessels.
299 ells in subretinal space, and a reduction of choroidal vessels.
300 from the foveal center, choroidal thickness, choroidal volume, choroidal vascular and stromal areas w

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