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1 s, both normal and with macular degeneration/choroidal neovascularization.
2 arization in vivo in a laser injury model of choroidal neovascularization.
3 f an atrophic scar at the fovea or following choroidal neovascularization.
4  255 treatment-naive patients with subfoveal choroidal neovascularization.
5 nd a larger increase in total lesion area of choroidal neovascularization.
6 s investigated in the laser-induced model of choroidal neovascularization.
7  of the photoreceptor outer segments (OS) to choroidal neovascularization.
8 y contributor to increased susceptibility to choroidal neovascularization.
9 and thus reduce vascular leakage and retinal-choroidal neovascularization.
10 fluorescence and the volume of laser-induced choroidal neovascularization.
11 each eye of WT and COX-2 null mice to induce choroidal neovascularization.
12  of reactive oxygen species, contributing to choroidal neovascularization.
13 ent in vitro and in a model of laser-induced choroidal neovascularization.
14 ischemic retinopathy (OIR) and laser-induced choroidal neovascularization.
15 acterized by drusen, geographic atrophy, and choroidal neovascularization.
16 VEGF-A secretion and angiogenic phenotype in choroidal neovascularization.
17 insight into the pathogenesis of corneal and choroidal neovascularization.
18 oxygen-induced retinopathy and laser-induced choroidal neovascularization.
19  the efficacy and safety of PDT for treating choroidal neovascularization.
20 d macular degeneration and other diseases of choroidal neovascularization.
21 r deposits, Bruch's membrane thickening, and choroidal neovascularization.
22 d in the most effective means of identifying choroidal neovascularization.
23 tion and the risks of retinal detachment and choroidal neovascularization.
24 MVECs suggest it may play a critical role in choroidal neovascularization.
25 F-induced subretinal neovascularization, and choroidal neovascularization.
26  degeneration is to avoid the development of choroidal neovascularization.
27 d strong up-regulation of alpha(5)beta(1) in choroidal neovascularization.
28 erlying vasculature, geographic atrophy, and choroidal neovascularization.
29 ssion of Ang2 also resulted in regression of choroidal neovascularization.
30 ies exploring new therapeutic approaches for choroidal neovascularization.
31 tion of nitric oxide (NO) in macrophages and choroidal neovascularization.
32 2 as a novel therapeutic target for reducing choroidal neovascularization.
33 nd photoreceptors and can be associated with choroidal neovascularization.
34 fundus changes and potentially of subsequent choroidal neovascularization.
35 cterized by fibrous membrane formation after choroidal neovascularization.
36 D OCT in 44% of the cases with no associated choroidal neovascularization.
37 ive cystoid macular edema or exacerbation of choroidal neovascularization.
38 e a strong risk factor in the development of choroidal neovascularization.
39 n strongly implicated in the pathogenesis of choroidal neovascularization.
40 sumed Ocular Histoplasmosis Syndrome-related choroidal neovascularization.
41 fundus changes and potentially of subsequent choroidal neovascularization.
42 larger increase in mean total lesion area of choroidal neovascularization (3.00 mm(2)) than eyes with
43 xtreme cases, apoE4 mice also develop marked choroidal neovascularization, a hallmark of exudative AM
44                       Current treatments for choroidal neovascularization, a major cause of blindness
45 inal pigment epithelium (RPE) abnormalities, choroidal neovascularization, acquired vitelliform lesio
46 eposition of drusen-like material or aborted choroidal neovascularization adjacent to the natural bre
47 g of the macular choroid (affected or not by choroidal neovascularization), along with the significan
48                Likewise, mouse laser-induced choroidal neovascularization, an injury that involves LP
49 l fibrosis after regression of laser-induced choroidal neovascularization and a decrease in mesenchym
50 These retinal lesions can be associated with choroidal neovascularization and central serous choriore
51  suppress hemangiogenesis in mouse models of choroidal neovascularization and dermal wound healing in
52 eripapillary atrophy were also common, while choroidal neovascularization and Fuchs spot were rare.
53 ration (AMD) often involve persons with both choroidal neovascularization and geographic atrophy (GA)
54                   Some patients also develop choroidal neovascularization and glaucoma.
55 ivo screening strategy with rodent models of choroidal neovascularization and iterative compound desi
56 oidal choroidal vasculopathy is a variant of choroidal neovascularization and neovascular age related
57                                  We document choroidal neovascularization and other AMD-like ocular p
58 a role in the development and maintenance of choroidal neovascularization and provides a target for t
59 F/rGel also caused significant regression of choroidal neovascularization and regression of retinal n
60 dels of neovascular pathology [laser-induced choroidal neovascularization and the very low density li
61 s of ocular neovascularization: laser injury choroidal neovascularization and VEGF-induced corneal ne
62 y criteria for CATT specified that eyes have choroidal neovascularization and visual acuity between 2
63 phenotypes for the disease: neovascular AMD (choroidal neovascularization) and geographic atrophy.
64 ot, myopic chorioretinal atrophy, and myopic choroidal neovascularization) and optic disc (optic nerv
65   They may represent an early sign of active choroidal neovascularization, and should prompt to early
66 hy developed in areas previously occupied by choroidal neovascularization, and the other 5 eyes had a
67             Four stage 3 eyes had associated choroidal neovascularization, and these eyes had 20/60 p
68 lial growth factor antagonists also suppress choroidal neovascularization, and transgenic expression
69 ng oxygen-induced retinopathy, laser-induced choroidal neovascularization, and transgenic mouse model
70 ere older age, VA of 20/40 or better, larger choroidal neovascularization area, presence of geographi
71 6.0 diopters with the presence of subretinal/choroidal neovascularization as indicated by Internation
72 models with deficient or spontaneous retinal/choroidal neovascularization, as well as models with ind
73 in ischemic retina and significantly reduced choroidal neovascularization at Bruch's membrane rupture
74 se vision than their mother, despite lacking choroidal neovascularization, because of the extent of p
75 ith UPR inhibitors or siRNAs reduced retinal/choroidal neovascularization by a further 25% to 40%, an
76 s may contribute to macular degeneration and choroidal neovascularization by causing ER stress leadin
77               Digoxin suppresses retinal and choroidal neovascularization by reducing HIF-1alpha leve
78 ells (HSCs) have been shown to contribute to choroidal neovascularization by signaling through the SD
79         In age-related macular degeneration, choroidal neovascularization can be observed without the
80 rly onset macular degenerative disease, with choroidal neovascularization causing a loss of vision in
81  risk of scarring were predominantly classic choroidal neovascularization (CNV) (aHR, 3.1; CI, 2.4-3.
82 ociations between all rare pLoF variants and choroidal neovascularization (CNV) (OR, 1.34; 95% CI, 1.
83 the mechanism of cell death in laser-induced choroidal neovascularization (CNV) after photodynamic th
84 peptide fragments on vessel sprouting and on choroidal neovascularization (CNV) after subconjunctival
85 if they had low vision in the fellow eye and choroidal neovascularization (CNV) along with (1) no res
86 lemonitoring for early detection of incident choroidal neovascularization (CNV) among patients with a
87                                              Choroidal neovascularization (CNV) and capillary dilatio
88       Inflammation and immune cells regulate choroidal neovascularization (CNV) and could become ther
89  gamma-secretase could inhibit laser-induced choroidal neovascularization (CNV) and if this was assoc
90  in two in vivo murine models, laser-induced choroidal neovascularization (CNV) and melanoma growth.
91                     Wet AMD includes typical choroidal neovascularization (CNV) and polypoidal choroi
92                           Eyes with advanced choroidal neovascularization (CNV) and poor visibility o
93 s investigated the effects of doxycycline on choroidal neovascularization (CNV) and regulation of MMP
94 cting and quantifying experimentally induced choroidal neovascularization (CNV) and vascular changes
95 .96 to 0.97 for quantitative measurements of choroidal neovascularization (CNV) area and total area o
96                                        Total choroidal neovascularization (CNV) area increased by 1.1
97 cholesterolemia, worse visual acuity, larger choroidal neovascularization (CNV) area, retinal angioma
98 on methods that are used to detect new-onset choroidal neovascularization (CNV) as a result of age-re
99 s to develop and characterize a rat model of choroidal neovascularization (CNV) as occurs in age-rela
100 es of previously untreated, active subfoveal choroidal neovascularization (CNV) associated with AMD w
101 l (ie, ocular) complement factor H (CFH) and choroidal neovascularization (CNV) associated with wet a
102                        The relative risk for choroidal neovascularization (CNV) based on drusen area,
103                                  In the eye, choroidal neovascularization (CNV) causes blindness in p
104 wever, these mice develop significantly less choroidal neovascularization (CNV) compared to wild-type
105 ace changes over time and their influence on choroidal neovascularization (CNV) development.
106 of fluorescein angiograms from patients with choroidal neovascularization (CNV) due to age-related ma
107 also called FLT1), in patients with blinding choroidal neovascularization (CNV) from age-related macu
108  to identify patients with exudative AMD and choroidal neovascularization (CNV) in 1 or both eyes res
109  suppress retinal vessel leakage and inhibit choroidal neovascularization (CNV) in a laser-induced CN
110                                              Choroidal neovascularization (CNV) in age-related macula
111 ibitor of pathological angiogenesis, such as choroidal neovascularization (CNV) in age-related macula
112 raphy angiography (OCTA) in the detection of choroidal neovascularization (CNV) in age-related macula
113 study, we describe a new method for inducing choroidal neovascularization (CNV) in C57BL/6 mice, an a
114 acteristics and natural history of quiescent choroidal neovascularization (CNV) in geographic atrophy
115 macular edema (DME), retinal vein occlusion, choroidal neovascularization (CNV) in high myopia, and o
116 ) has been implicated in the pathogenesis of choroidal neovascularization (CNV) in neovascular AMD, w
117 e (CAM) assay and laser-induced experimental choroidal neovascularization (CNV) in rats.
118 nhibitor, 10 weeks post-injection suppresses choroidal neovascularization (CNV) in rats.
119 ry outcome measure was time to occurrence of choroidal neovascularization (CNV) in the study eye.
120 cell adhesion and proliferation in vitro and choroidal neovascularization (CNV) in vivo was examined.
121                           Adjacent to active choroidal neovascularization (CNV) in wet AMD, CC dropou
122                          Here we report that choroidal neovascularization (CNV) incited by injury was
123                                              Choroidal neovascularization (CNV) is a defining feature
124                                              Choroidal neovascularization (CNV) is a major cause of v
125                                              Choroidal neovascularization (CNV) is a severe complicat
126                                Laser-induced choroidal neovascularization (CNV) is a widely used mode
127                                              Choroidal neovascularization (CNV) is the end point of s
128                                              Choroidal neovascularization (CNV) is the leading cause
129 luded lower BCVA, younger age, smaller total choroidal neovascularization (CNV) leakage area, smaller
130 acterize techniques for measuring changes in choroidal neovascularization (CNV) lesion size and fluor
131 change in angiographic total lesion size and choroidal neovascularization (CNV) lesion size.
132                          Eyes with subfoveal choroidal neovascularization (CNV) lesions had a lower g
133 onstrated by increased size of laser-induced choroidal neovascularization (CNV) lesions.
134        AMG 386 was tested in a laser-induced choroidal neovascularization (CNV) model in monkeys usin
135                         In the laser-induced choroidal neovascularization (CNV) model, the CNV area w
136 treatment/intervention modality of the laser choroidal neovascularization (CNV) model, the efficacy o
137 tion of laser rupture of Bruch's membrane, a choroidal neovascularization (CNV) model.
138 -induced retinopathy (OIR) and laser-induced choroidal neovascularization (CNV) models.
139 he corneal micropocket and the laser-induced choroidal neovascularization (CNV) mouse models.
140                                              Choroidal neovascularization (CNV) of the macular area o
141  were subdivided into 3 groups: eyes without choroidal neovascularization (CNV) or chorioretinal atro
142 s with AMD with severe atrophy due to either choroidal neovascularization (CNV) or geographic atrophy
143 degeneration (AMD) to the advanced stages of choroidal neovascularization (CNV) or geographic atrophy
144 lantation of 7KCh-exposed microglia promoted choroidal neovascularization (CNV) relative to control m
145 elial progenitor cells (EPCs) are present in choroidal neovascularization (CNV) secondary to age-rela
146 ty required angiographic and OCT evidence of choroidal neovascularization (CNV) secondary to age-rela
147                      Patients with subfoveal choroidal neovascularization (CNV) secondary to AMD were
148 ty of Ranibizumab in Patients with Subfoveal Choroidal Neovascularization (CNV) Secondary to AMD, Pro
149       A total of 151 patients with subfoveal choroidal neovascularization (CNV) secondary to neovascu
150 DRS letters, angiographic total lesion size, choroidal neovascularization (CNV) size, and optical coh
151 was an 85.5% mean reduction from baseline in choroidal neovascularization (CNV) size.
152                           In contrast to the choroidal neovascularization (CNV) subtype, the genetic
153 ecrosis factor (TNF)-alpha has been found in choroidal neovascularization (CNV) surgically removed fr
154  was more prominent in patients with classic choroidal neovascularization (CNV) than those with occul
155 -induced retinopathy (OIR) and laser-induced choroidal neovascularization (CNV) to assess the contrib
156                                              Choroidal neovascularization (CNV) was manually segmente
157 corneal neovascularization and laser-induced choroidal neovascularization (CNV) were examined in TIMP
158 tinopathy (OIR) model and the laser model of choroidal neovascularization (CNV) were used to test the
159 t epithelium derived factor (PEDF) may cause choroidal neovascularization (CNV), a key event in wet A
160   To investigate the genetic contribution to choroidal neovascularization (CNV), a leading cause of b
161                The molecular pathogenesis of choroidal neovascularization (CNV), an angiogenic proces
162 play an important role in the development of choroidal neovascularization (CNV), an integral componen
163                    Older age, larger area of choroidal neovascularization (CNV), and elevation of ret
164 data from retinas treated with laser-induced choroidal neovascularization (CNV), bright white-light e
165                     In 2 eyes with secondary choroidal neovascularization (CNV), distinct en face SS-
166 normal growth of choroidal blood vessels, or choroidal neovascularization (CNV), is a hallmark of the
167 t epithelium derived factor (PEDF) may cause choroidal neovascularization (CNV), key event of wet AMD
168 -induced retinopathy (OIR) and laser-induced choroidal neovascularization (CNV), mimicking hypoxia-me
169                                              Choroidal neovascularization (CNV), the advanced stage o
170 arly always precede and increase the risk of choroidal neovascularization (CNV), the late vision-thre
171 ibodies within retinal lesions in a model of choroidal neovascularization (CNV).
172 s ligand (FasL) function in a mouse model of choroidal neovascularization (CNV).
173 ing hyperreflective material consistent with choroidal neovascularization (CNV).
174 ch's membrane (BrM) into the retina, forming choroidal neovascularization (CNV).
175 te AMD subtypes; geographic atrophy (GA) and choroidal neovascularization (CNV).
176 that occur in vivo during the development of choroidal neovascularization (CNV).
177 he wet form of the disease, characterized by choroidal neovascularization (CNV).
178 AMD-related vision loss, is characterized by choroidal neovascularization (CNV).
179 plex using the murine model of laser-induced choroidal neovascularization (CNV).
180 ive B6-gld mice were laser treated to induce choroidal neovascularization (CNV).
181 cular degeneration (AMD) is characterized by choroidal neovascularization (CNV).
182 imens from mice treated with laser to induce choroidal neovascularization (CNV).
183 tance of PLCgamma1 and c-Cbl in experimental choroidal neovascularization (CNV).
184  to sites of complement activation, in mouse choroidal neovascularization (CNV).
185 MD, including an increased susceptibility to choroidal neovascularization (CNV).
186 ay have a permissive effect on the growth of choroidal neovascularization (CNV).
187 e retina, an abnormality also referred to as choroidal neovascularization (CNV).
188 ted the role of nAChR in the pathogenesis of choroidal neovascularization (CNV).
189 ay plays a central role in the laser-induced choroidal neovascularization (CNV).
190 ic therapy (PDT) in a laser-induced model of choroidal neovascularization (CNV).
191 ys of complement activation in laser-induced choroidal neovascularization (CNV).
192 5 fellow eyes of 55 patients with unilateral choroidal neovascularization (CNV).
193  CGC-11144 and CGC-11150 in a mouse model of choroidal neovascularization (CNV).
194 moses also occur in various animal models of choroidal neovascularization (CNV).
195 ous proliferation (RAP) lesions, and classic choroidal neovascularization (CNV).
196 ent white dot syndrome (MEWDS) who developed choroidal neovascularization (CNV).
197 of irreversible blindness and manifests with choroidal neovascularization (CNV).
198 cular degeneration (AMD) is characterized by choroidal neovascularization (CNV).
199 permeability, while there was no evidence of choroidal neovascularization (CNV).
200  were also more susceptible to laser-induced choroidal neovascularization (CNV).
201  drusen; retinal pigment epithelial changes; choroidal neovascularization (CNV); atrophy; and hypoaut
202 nfidence interval [CI], 53-442), followed by choroidal neovascularization (CNV; OR, 90; 95% CI, 26-31
203 features from pathologic RPE detachments and choroidal neovascularizations (CNVs).
204 had reduced dihydroethidium fluorescence and choroidal neovascularization compared with wild-type con
205                                    Bilateral choroidal neovascularization developed in only 1 patient
206                     The treatment options of choroidal neovascularization due to age-related macular
207 ed from 1134 of 1146 patients with subfoveal choroidal neovascularization due to AMD with minimally c
208 Cs suggests a possible role for potentiating choroidal neovascularization during the pathology of neo
209 cious in mouse models of geographic atrophy, choroidal neovascularization, graft-versus-host disease,
210 ous in vivo systems, including laser-induced choroidal neovascularization, growth factor-induced corn
211                         Stage 3 eyes without choroidal neovascularization had a mean presenting visua
212 betic macular edema, retinal vein occlusion, choroidal neovascularization) have shown promising resul
213              Areas of geographic atrophy and choroidal neovascularization imaged by FA were depicted
214 al neovascularization in 3 eyes and inactive choroidal neovascularization in 1 eye.
215 silar insufficiency in 2.8% of eyes; non-AMD choroidal neovascularization in 2.3% of eyes; retinitis
216 f a hyperflow signal corresponding to active choroidal neovascularization in 3 eyes and inactive chor
217 pressed initiation and growth of spontaneous choroidal neovascularization in a mouse model, and the c
218 al scars, which are thought to progress into choroidal neovascularization in a small proportion of pa
219 , and is likely to play an important role in choroidal neovascularization in age-related macular dege
220 fied as a risk factor for the development of choroidal neovascularization in age-related macular dege
221 y for the Treatment of Predominantly Classic Choroidal Neovascularization in AMD (ANCHOR) trials show
222 s, retinal vascular leakage, and retinal and choroidal neovascularization in animal models of ROP and
223 EGF signaling exists and may be important in choroidal neovascularization in human age-related macula
224 s immunofluorescent staining for rGel within choroidal neovascularization in mice and regression of t
225 travitreal administration of oligo G reduces choroidal neovascularization in mice.
226 ote various pathologic conditions, including choroidal neovascularization in models of neovascular ag
227 s showed excellent short-term suppression of choroidal neovascularization in patients with exudative
228 aditional multimodal imaging, helps diagnose choroidal neovascularization in patients with Malattia L
229 rsy that surrounds the optimal treatment for choroidal neovascularization in patients with presumed o
230 ies demonstrated that aflibercept suppresses choroidal neovascularization in several animal models.
231        We used a laser-injury wound model of choroidal neovascularization in the eye to determine the
232 yes and retinal hemorrhages with an apparent choroidal neovascularization in the left eye, which was
233 n, the authors used a model of laser-induced choroidal neovascularization in the mouse eye and invest
234  reduce pro-angiogenic isoforms, and prevent choroidal neovascularization in vivo.
235 efined as HM with the presence of subretinal/choroidal neovascularization indicated by the ICD-9-CM d
236                                   Subsequent choroidal neovascularization is limited to the area dema
237 ted macular degeneration eyes with subfoveal choroidal neovascularization is uniform across a wide ra
238 inistration of IL-4 attenuates laser-induced choroidal neovascularization (L-CNV) due to specific IL-
239 -induced retinopathy (OIR) and laser-induced choroidal neovascularization (LCNV).
240                               The pattern of choroidal neovascularization lesion size enlargement in
241                      The main determinant of choroidal neovascularization lesion size enlargement is
242            Pigmented mice with laser-induced choroidal neovascularization lesions (n = 7 eyes) were a
243 e first realistic means to address subfoveal choroidal neovascularization lesions from age-related ma
244 ed BRB leakage and the size of laser-induced choroidal neovascularization lesions.
245 ffect on both area and number of spontaneous choroidal neovascularization lesions.
246 ctional role for TLR2 in the pathogenesis of choroidal neovascularization, likely by promoting inflam
247                                              Choroidal neovascularization location as well as retinal
248 outcomes in eyes with treatment-naive myopic choroidal neovascularization (mCNV) in the United States
249     Furthermore, analysis in a laser-induced choroidal neovascularization model showed that depletion
250 nd showed good efficacy in the laser induced choroidal neovascularization model.
251 ssel growth were also determined in a murine choroidal neovascularization model.
252 pathological angiogenesis in a laser-induced choroidal neovascularization mouse model.
253 or ATF6 in the oxygen-induced retinopathy or choroidal neovascularization mouse models caused an appr
254 inal hemorrhage (n = 3, 0.9%), active myopic choroidal neovascularization (n = 3, 0.9%), and no case
255                                  Retinal and choroidal neovascularization (NV) and vascular leakage c
256                                 In contrast, choroidal neovascularization occurred equally well in bi
257                                 Furthermore, choroidal neovascularization occurred in 15% of the mice
258                     Stage 3 resembles occult choroidal neovascularization, occurs primarily in older
259 d associations of ARMS2 and CFH with type of choroidal neovascularization on fluorescein angiography
260 ioretinopathy, and did not have a history of choroidal neovascularization or photodynamic therapy.
261 -related macular degeneration independent of choroidal neovascularization or retinal pigment epitheli
262 95% CI, 1.35-6.08; P = .006), larger area of choroidal neovascularization (OR for a >4-disc area vs <
263 ors with early-stage AMD (P = 0.028), active choroidal neovascularization (P = 0.020), and disciform
264 l loss may occur during a preoccult phase of choroidal neovascularization, prior to the development o
265 inal vasculitis, multifocal choroiditis, and choroidal neovascularization, providing for the first ti
266 r of patients with treatment-naive subfoveal choroidal neovascularization receiving intravitreal rani
267 n 2 (12%), a dome-shaped macula in 1 (6%), a choroidal neovascularization-related subretinal scar in
268 retinal angiomatous proliferation, and mixed choroidal neovascularization, respectively.
269 ow, the most common form of late-stage AMD - choroidal neovascularization - responds to treatment wit
270 lar Endothelial Growth Factor in Age-Related Choroidal Neovascularization) results were not released
271  classic or occult (with no classic lesions) choroidal neovascularization secondary to age-related ma
272 en-label study in 15 patients with subfoveal choroidal neovascularization secondary to AMD at a clini
273 l Neovascularization with or without Classic Choroidal Neovascularization Secondary to AMD Study), wh
274  participants, aged >/=50 years, with active choroidal neovascularization secondary to AMD.
275                                          For choroidal neovascularization secondary to inflammatory d
276 escein-negative intraretinal cystic changes, choroidal neovascularization, serous retinal elevations
277  photoreceptor degeneration, and exaggerated choroidal neovascularization similar to AMD.
278 mL vs 69.3 pg/mL) and did not correlate with choroidal neovascularization size and lesion type.
279                            In the B50 group, choroidal neovascularization size was smaller (0.73 vs 1
280 ular Degeneration: Detection of Onset of new Choroidal neovascularization Study (AMD DOC Study) evalu
281 f visual acuity, on average, at detection of choroidal neovascularization than do individuals using s
282  pegaptanib sodium and PDT on lesion size in choroidal neovascularization than with each monotherapy.
283                               The mother had choroidal neovascularization that was treated with bevac
284 giogenic isoforms of VEGF-A as inhibitors of choroidal neovascularization, the authors used a model o
285 ntagonist, caused significant suppression of choroidal neovascularization; the area of neovasculariza
286  classic or occult (with no classic lesions) choroidal neovascularization to receive 24 monthly intra
287 ependence on VEGF-A in models of corneal and choroidal neovascularization using a combination of reag
288                                              Choroidal neovascularization was diagnosed in 8 of 27 ey
289 tioned CD200R(-/-) BMMPhi, and laser-induced choroidal neovascularization was enhanced in CD200R-defi
290                                              Choroidal neovascularization was generated by laser inju
291                                              Choroidal neovascularization was generated in mice using
292                                              Choroidal neovascularization was induced by 532-nm diode
293 ralizing protein can result in regression of choroidal neovascularization, which is sometimes associa
294 itreal morpholino injection suppressed laser choroidal neovascularization while increasing sKDR.
295 or the average person with classic subfoveal choroidal neovascularization, while laser photocoagulati
296                 Parstatin potently inhibited choroidal neovascularization with an IC(50) of approxima
297  detachment associated with occult subfoveal choroidal neovascularization with intravitreal ranibizum
298 ty of Ranibizumab in Subjects with Subfoveal Choroidal Neovascularization with or without Classic Cho
299 or the treatment of occult/minimally classic choroidal neovascularization with ranibizumab is greater
300 8(-/-)Abca4(-/-) mice for 4 months prevented choroidal neovascularization without changing retinal VE

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