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1 ic vesicle, and a medium size vesicle in the chromaffin cell.
2 wo sequential priming steps in mouse adrenal chromaffin cells.
3 e nerve and form postsynaptic neuroendocrine chromaffin cells.
4 iated fusion pore expansion in mouse adrenal chromaffin cells.
5 ippocampus, dorsal root ganglia, and adrenal chromaffin cells.
6 ventional active zones and in neuroendocrine chromaffin cells.
7 augmented T-type Ca2+ current in IH-treated chromaffin cells.
8 correlation, confirming similar findings in chromaffin cells.
9 stimulated catecholamine release in cultured chromaffin cells.
10 spatial distribution of calcium channels in chromaffin cells.
11 d released by exocytosis in PC12 and primary chromaffin cells.
12 adapted from a model for fast exocytosis in chromaffin cells.
13 typical transmitter storage and release from chromaffin cells.
14 nd neuronal cells, including sympathoadrenal chromaffin cells.
15 on with a D1-like receptor on bovine adrenal chromaffin cells.
16 omosyn-syntaxin 1A complexes in live adrenal chromaffin cells.
17 type/reporter plasmids were transfected into chromaffin cells.
18 receptors on exocytosis from bovine adrenal chromaffin cells.
19 se frequency of catecholamine in dissociated chromaffin cells.
20 ant (>90%) mode of secretion in calf adrenal chromaffin cells.
21 calcium-triggered catecholamine release from chromaffin cells.
22 Ca) was produced by P2Y receptors in adrenal chromaffin cells.
23 motions related to the secretory response in chromaffin cells.
24 le priming and secretory amplitude in living chromaffin cells.
25 e in the Ca2+-cooperativity of exocytosis in chromaffin cells.
26 ficant reduction in DCG formation in adrenal chromaffin cells.
27 human embryonic kidney HEK293-S3 and adrenal chromaffin cells.
28 is a rare, but clinically important tumor of chromaffin cells.
29 18-1/syntaxin1A interaction in HEK293-S3 and chromaffin cells.
30 ive anion equilibrium potential, depolarizes chromaffin cells.
31 coupling after this peptidergic stimulus to chromaffin cells.
32 1-L61) were transiently expressed in adrenal chromaffin cells.
33 secretory response triggered by the toxin in chromaffin cells.
34 athetic neurons appears to represent adrenal chromaffin cells.
35 K"- or "Maxi K"-encoding Slo gene in adrenal chromaffin cells.
36 synapses as well as secretory cells, such as chromaffin cells.
37 d," was confirmed by immunohistochemistry at chromaffin cells.
38 in the exocytotic membrane fusion process in chromaffin cells.
39 ranscriptional activation of the VIP gene in chromaffin cells.
40 ease and extrusion from vesicles at mast and chromaffin cells.
41 ar calcium release, and elevation of cAMP in chromaffin cells.
42 ht to mediate BK channel inactivation in rat chromaffin cells.
43 ion is severely inhibited in Ophn1 knock-out chromaffin cells.
44 SPNs), respectively, from adrenal medullary chromaffin cells.
45 ind Rab3a-GTP and to stimulate exocytosis in chromaffin cells.
46 ar the plasma membrane (PM) of living bovine chromaffin cells.
47 ed action potentials (sAP) in bovine adrenal chromaffin cells.
48 properties of epinephrine-secreting adrenal chromaffin cells.
49 nist-induced hGH release from bovine adrenal chromaffin cells.
50 n the exocytotic membrane fusion reaction in chromaffin cells.
51 ease from the regulated secretory pathway in chromaffin cells.
52 ilar time course in AtT-20, CHO, and adrenal chromaffin cells.
53 ecretion observed in digitonin-permeabilized chromaffin cells.
54 was observed in native channels from bovine chromaffin cells.
55 hn1 knock-out mice and OPHN1-silenced bovine chromaffin cells.
56 ipid microdomains at the exocytotic sites in chromaffin cells.
57 he nAChR subtypes expressed by human adrenal chromaffin cells.
58 and is also essential for vesicle docking in chromaffin cells.
59 med during catecholamine exocytosis in mouse chromaffin cells.
60 nk exocytosis to compensatory endocytosis in chromaffin cells.
61 nergic stimulated catecholamine release from chromaffin cells.
62 duced bulk endocytosis also occurs in bovine chromaffin cells.
63 tivator (tPA) (over many seconds) in adrenal chromaffin cells.
64 large, dense core vesicles (LDCVs) in mouse chromaffin cells.
65 uptake at the synaptic terminals and adrenal chromaffin cells.
66 mice, we find that NPY is synthesized by all chromaffin cells.
67 es in a millisecond time resolution in mouse chromaffin cells.
68 successfully from multiple individual living chromaffin cells.
69 AP-25 x synaptotagmin-1 interaction in mouse chromaffin cells.
70 iNW-FET) to detect the K(+)-efflux from live chromaffin cells.
71 holamines released from small populations of chromaffin cells.
72 echolamines, exclusively, from fetal adrenal chromaffin cells.
74 e changes in the surface membrane of adrenal chromaffin cells after stimulation of exocytosis with a
75 e via desensitization/down-regulation of the chromaffin cell alpha(2)-adrenergic receptors that norma
76 n of neural crest-derived catecholaminergic (chromaffin) cells already associated with blood vessels
80 in the purified large dense-core vesicles of chromaffin cells and associated with synaptotagmin-1.
81 an directly affect the secretory capacity of chromaffin cells and contribute, in part, to elevated ca
84 and vesicular catecholamine transporters of chromaffin cells and facilitates localization of the pri
85 investigated this question in mouse adrenal chromaffin cells and found that SNAP-25 inhibits Ca(2+)
86 nergic receptors are found on bovine adrenal chromaffin cells and have been implicated in the facilit
87 (APWs) using both native channels in adrenal chromaffin cells and heterologously expressed channels i
88 irst study of EP receptor signaling in mouse chromaffin cells and identifies a molecular mechanism fo
90 tory capacity measured amperometrically from chromaffin cells and in the expression of tyrosine hydro
91 nd [Ca2+]i responses in neonatal rat adrenal chromaffin cells and involves reactive oxygen species (R
94 repinephrine released from adrenal medullary chromaffin cells and norepinephrine released locally fro
95 mega-shaped structure in live neuroendocrine chromaffin cells and pancreatic beta-cells, visualized u
97 ibitor of catecholamine release from adrenal chromaffin cells and postganglionic sympathetic axons.
98 protein released from secretory granules of chromaffin cells and sympathetic nerves, triggers endoth
100 hrough voltage-gated Ca2+ channels in bovine chromaffin cells and the domain of this receptor variant
101 n the development of sympathetic neurons and chromaffin cells and the mechanisms involved in Lin28B-i
102 proximity to the surface of adherent bovine chromaffin cells and to amperometrically record single e
104 at controls epinephrine release from adrenal chromaffin cells and, consequently, hepatic glucose prod
105 that is found in neurons, platelets, adrenal chromaffin cells, and a few other neurosecretory cells.
106 epinephrine is released from adrenomedullary chromaffin cells, and compensatory glucogenesis ensues.
107 ed transcriptional mechanisms in transfected chromaffin cells, and concluded with observations on blo
108 n of individual secretory granules in living chromaffin cells, and related their mobilities to postfu
109 , is preferentially phosphorylated in intact chromaffin cells, and the levels of annexin 7 phosphoryl
113 onclude that sympathetic neurons and adrenal chromaffin cells are more vulnerable to diabetes than pa
114 findings demonstrate that the fetal adrenal chromaffin cells are the source for acute hypoxaemia-ind
117 d catestatin is secreted from neuroendocrine chromaffin cells as an autocrine regulator of nicotine-s
118 hromocytoma cells and bovine adrenomedullary chromaffin cells as detected by Northern blotting, Weste
119 acitance measurements to probe exocytosis in chromaffin cells at low concentrations of intracellular
121 ion did not evoke action potential firing in chromaffin cells but did cause a persistent subthreshold
122 brane area through exocytosis does not swell chromaffin cells but may decrease membrane tension.
123 nt from mature secretory vesicles in adrenal chromaffin cells, but localizes to a compartment near th
124 ls (a rat insulinoma cell line) and cultured chromaffin cells, but not in AtT-20 cells (derived from
125 ediating SNARE-dependent exocytosis in mouse chromaffin cells, but the role of a closely related calc
126 or monitoring single vesicle exocytosis from chromaffin cells by constant potential amperometry as we
127 alin in secretory vesicles of neuroendocrine chromaffin cells by immunofluorescent confocal and immun
128 sponse element motif, an effect confirmed in chromaffin cells by site-directed mutagenesis on the tra
132 e was measured from bovine adrenal medullary chromaffin cell (CC) cultures maintained over a period o
135 o shrink the Omega-profile in neuroendocrine chromaffin cells containing approximately 300 nm vesicle
136 are increased by Sp and that elevate 5-HT in chromaffin cell cultures, suggesting direct metabolic si
137 describes the primary culture of individual chromaffin cells derived by enzymatic digestion from the
140 l sympathetic neuroblast hyperplasia, blocks chromaffin cell differentiation, and ultimately triggers
141 evoked secretion simultaneously from several chromaffin cells directly cultured on the device surface
142 owever, unlike hypothalamic nerve terminals, chromaffin cells do not display syntilla activation by d
143 synapses lacking Munc13s, the corresponding chromaffin cells do not exhibit a vesicle docking defect
145 techolamine biosynthetic capacity of adrenal chromaffin cells during periods of sustained catecholami
147 Application of etomidate directly to the chromaffin cells elicited robust catecholamine secretion
149 e) Ca2+ and voltage-dependent K+ channels in chromaffin cells exhibit an inactivation that probably a
150 ding potentials to remove inactivation, many chromaffin cells exhibit N-type calcium channel currents
152 ously reported that N-type current in bovine chromaffin cells exhibits very little voltage-dependent
153 evious studies have shown that naive adrenal chromaffin cells express a nominal Ca(v)3.2-dependent co
155 d secretion from Munc18-1-null mouse adrenal chromaffin cells expressing Munc18-1 mutants designed to
156 residues, single exocytotic events in bovine chromaffin cells expressing R198Q, R198E, K201Q, or K201
158 arge dense core vesicle (LDCV) exocytosis in chromaffin cells follows a well characterized process co
161 A30P or wild-type (WT) alpha-syn, as well as chromaffin cells from control and alpha-syn null mice, t
164 e have examined the kinetics of secretion in chromaffin cells from mice lacking phosphatidylinositol
166 nctional properties and cell excitability in chromaffin cells from normal and hypophysectomized (pitu
167 SCR-1 calcium-insensitive mutant or by using chromaffin cells from PLSCR-1(-)/(-) mice prevents outwa
170 s highly expressed in bovine adrenomedullary chromaffin cells, human pheochromocytoma tissue, PC12 ph
172 ime-lapse imaging of Lifeact-GFP-transfected chromaffin cells in combination with fluorescent 70 kDa
176 es and other hormones, released from adrenal chromaffin cells in response to Ca(2+) influx through vo
177 to the extent of catecholamine release from chromaffin cells in response to stimulation by carbachol
181 0 were colocalized to the Golgi apparatus of chromaffin cells in vivo and shared localization with CH
182 We hypothesize that Slo splicing in adrenal chromaffin cells in vivo is differentially regulated by
183 ociated with distinctive membrane changes of chromaffin cells including increased electron density, a
184 evealed decreased LDCV size in noradrenergic chromaffin cells, increased adrenal norepinephrine and e
185 he enhancement of secretion in permeabilized chromaffin cells indicates that N-terminal Rim1 does not
186 ated exocytic function in Mecp2(-/y) adrenal chromaffin cells, indicating that the Mecp2 null mutatio
187 pression of Ca(v)3.2 channels in MPC 9/3L-AH chromaffin cells induced low-threshold secretion that co
189 under physiological electrical stimulation, chromaffin cells internalise membrane via two distinct p
191 ominant calcium signal regulating release in chromaffin cells is generated by the cooperative action
192 Transient stimulation of secretion in calf chromaffin cells is invariably followed by rapid endocyt
194 heteromeric nAChR expressed by human adrenal chromaffin cells is the alpha3beta4* subtype (asterisk i
199 equently, the absence of Snapin in embryonic chromaffin cells leads to a significant reduction of cal
200 These findings suggest one way in which chromaffin cells may regulate cargo release is via diffe
203 e docking, neither synchronized secretion in chromaffin cells nor Ca(2+)-triggered SUV-GUV fusion was
204 exhibiting differential mobility shifting to chromaffin cell nuclear proteins during EMSA, binding of
205 own to accumulate mainly in association with chromaffin cells, occasional nerve endings and macrophag
206 also expressed in neonatal adrenal medullary chromaffin cells of rats and mice whose hypoxia-evoked c
211 c neurons are severely depleted in CIPA, but chromaffin cells of the adrenal medulla are spared.
216 ak amplitude of nicotine-induced currents in chromaffin cells or in human embryonic kidney cells ecto
217 cement of exocytosis by PMA in either bovine chromaffin cells or the INS-1 insulin-secreting cell lin
221 suggest that PROG inhibits CA secretion from chromaffin cells predominantly by rapidly inhibiting nAC
225 est that a spontaneous syntilla, at least in chromaffin cells, releases Ca2+ into a cytosolic microdo
226 wise, inactivation of the V0 a1-I subunit in chromaffin cells resulted in a decreased frequency and p
227 comparable with those of bPAC1hop in bovine chromaffin cells resulted in acquisition by PC12-G cells
228 hat overexpression of neuronal AP-3 in mouse chromaffin cells results in a striking decrease in the n
229 rometric measurements of exocytosis in mouse chromaffin cells revealed that syb2 TMD mutations altere
232 CG10 by synthetic siRNAs virtually abolished chromaffin cell secretion of a transfected CHGA-EAP chim
234 extraordinary accumulation of solutes inside chromaffin cell secretory vesicles, although this has ye
235 r with time, that vesicles in bovine adrenal chromaffin cells segregate into distinct populations, ba
237 l firing rates, set by the sympathetic tone, chromaffin cells selectively release catecholamines at a
240 e mice, expressing Cre recombinase under the chromaffin cell-specific phenylethanolamine N-methyltran
243 gnals was severalfold faster than in adrenal chromaffin cells, suggesting profound differences in the
248 medulla gland and of cultured human adrenal chromaffin cells that demonstrated prominent expression
250 hromocytoma is a rare but important tumor of chromaffin cells that is frequently considered in the ev
251 Src family kinases (SFKs) are abundant in chromaffin cells that reside in the adrenal medulla and
252 ase, present in the local environment of the chromaffin cell, that selectively cleaves CgA to generat
255 , high-affinity nAChRs expressed in cultured chromaffin cells, they do not appear to be involved in f
256 1 was to greatly increase the sensitivity of chromaffin cells to channel formation by alpha-latrotoxi
259 tentials delivered at 0.5 Hz) causes adrenal chromaffin cells to selectively release catecholamines t
260 hetic tone, basal synaptic excitation drives chromaffin cells to selectively secrete modest levels of
261 CPE and PC activity in extracts of cultured chromaffin cells; total protein levels were unaltered fo
263 ies have demonstrated that adrenal medullary chromaffin cells transplanted into the spinal subarachno
264 ection of catecholamine release from adrenal chromaffin cells trapped in a microfluidic network.
265 ificity for the diagnosis of adrenomedullary chromaffin cell tumors can be jeopardized by physiologic
267 We studied insertion of granules in bovine chromaffin cells using capacitance as a measure of plasm
268 stics of individual secretory events in calf chromaffin cells using catecholamine amperometry combine
269 ne release in individual quantal events from chromaffin cells using cell-attached patch amperometry.
270 s of exocytosis from populations of mast and chromaffin cells using chemoreceptive neuron MOS (CnuMOS
272 Experiments on diI-stained bovine adrenal chromaffin cells using polarized TIRFM demonstrate rapid
273 strate that CIH increases the RRP in adrenal chromaffin cells via ROS-mediated activation of PKC and
274 Secretion of catestatin intermediates from chromaffin cells was accompanied by the cosecretion of c
276 histamine in H(1) receptor/TRPC4-expressing chromaffin cells was comparable with that triggered by a
277 ing of individual exocytotic fusion pores in chromaffin cells was imaged electrochemically with high
278 eover, regulated secretion of endopin 2 from chromaffin cells was induced by nicotine and KCl depolar
279 lamines, measured by amperometry in cultured chromaffin cells, was found to be increased either by pr
283 ophysiological measurements in mouse adrenal chromaffin cells, we show that PI(4,5)P2 uncaging potent
287 egulates stimulus-secretion coupling, bovine chromaffin cells were infected with Semliki Forest virus
291 vity was also evident in a subset of adrenal chromaffin cells where labeling appeared to be predomina
292 red how alpha-syn overexpression in PC12 and chromaffin cells, which exhibit low endogenous alpha-syn
293 s to hypoxia by the carotid body and adrenal chromaffin cells, which regulate cardio-respiratory func
295 y monitoring CME of single vesicles in mouse chromaffin cells with cell-attached capacitance measurem
297 s and whole-cell recordings from rat adrenal chromaffin cells with parallel experiments on inactivati
299 in each inhibited catecholamine release from chromaffin cells, with superior potency for the shorter
300 foundly impairs priming of granules in mouse chromaffin cells without altering catecholamine release
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