ライフサイエンスコーパス: chromatin condensation

1 echanical properties of the nucleus and also chromatin condensation.

2 ould then be disassembled to allow metaphase chromatin condensation.

3 ted with DNA replication, transcription, and chromatin condensation.

4 es; DNA methylation likely occurred prior to chromatin condensation.

5 tion of these chromosomes without detectable chromatin condensation.

6 pological state, chromosome segregation, and chromatin condensation.

7 accelerates its proteolytic degradation and chromatin condensation.

8 cleus and the inhibition of acinus-dependent chromatin condensation.

9 ists against apoptotic cleavage and prevents chromatin condensation.

10 Cytochrome c was also found to induce chromatin condensation.

11 ment of a sub-G(0)/G(1) region, and distinct chromatin condensation.

12 phosphatidylserine exposure, DNA damage and chromatin condensation.

13 ncident with the first detectable changes in chromatin condensation.

14 f the gene to DNA demethylation but not with chromatin condensation.

15 electively killing cancer cells by premature chromatin condensation.

16 anscriptional silencing, in part by inducing chromatin condensation.

17 g of P2 and, consequently, the completion of chromatin condensation.

18 domain results in histone deacetylation and chromatin condensation.

19 removal, tau is hyperphosphorylated prior to chromatin condensation.

20 transcription complexes and locally blocking chromatin condensation.

21 oisson's ratio, apparent elastic modulus and chromatin condensation.

22 ing somatic cells during S phase to maintain chromatin condensation.

23 and caspases completely abolishes apoptotic chromatin condensation.

24 is not essential for histone displacement or chromatin condensation.

25 hondrial transmembrane potential and nuclear chromatin condensation.

26 ssive effects of histone hypoacetylation and chromatin condensation.

27 d by deacetylation of histone H4 lysines and chromatin condensation.

28 biotin nick end labeling, DNA laddering, and chromatin condensation.

29 ation, extensive surface bleb formation, and chromatin condensation.

30 nocytes and thymocytes are also resistant to chromatin condensation.

31 of short, active transcription units hinders chromatin condensation.

32 it was surprising that transfection induced chromatin condensation.

33 ot the result of the temperature shift or of chromatin condensation.

34 disrupt nucleosomal dynamics due to aberrant chromatin condensation.

35 ntified a potential direct role for CED-4 in chromatin condensation.

36 endent manner that has long been linked with chromatin condensation.

37 e associations within an array, facilitating chromatin condensation.

38 me filament, with a role similar to that for chromatin condensation.

39 ntiation, meiotic cell divisions and extreme chromatin condensation.

40 sine residues in histone tails, resulting in chromatin condensation.

41 histone acetylation and inducing peripheral chromatin condensation.

42 NA Polymerase II, perhaps by promoting local chromatin condensation.

43 ase in response to DL, blocking load-induced chromatin condensation.

44 miR-191 or knockdown of Riok3 or Mxi1 blocks chromatin condensation.

45 esistant form of cyclin A2 partially rescued chromatin condensation.

46 ry step in cell replication and is linked to chromatin condensation.

47 re and block the binding of the regulator of chromatin condensation 1 (RCC1) acidic patch-binding pro

48 Moreover, the regulator of chromatin condensation 1-like domain of PAM (amino acids

49 Furthermore, the regulator of chromatin condensation 1-like domain of PAM is sufficien

50 nine nucleotide exchange factor regulator of chromatin condensation 1.

51 t HP1(Hsalpha)-nucleosome interactions cause chromatin condensation, a process that regulates many ch

52 ol cells in that they show reduced degree of chromatin condensation, absent nuclear fragmentation, in

53 ine methylation, histone hypoacetylation and chromatin condensation act together in a discrete region

54 is capable of inducing DNA fragmentation and chromatin condensation after cleavage by caspase-3.

55 tic phosphoproteins and inhibiting premature chromatin condensation after DNA damage or hydroxyurea.

56 hypoacetylation of associated histones, and chromatin condensation, all characteristics of a transcr

57 ctivated state showing a decrease in nuclear chromatin condensation, altered histone deacetylase expr

58 ifferentiate, they are capable of undergoing chromatin condensation, although many abnormal cells are

59 he N-termini perform essential roles in both chromatin condensation and also bind specific chromatin-

60 ations, centriole separation, and prophase I chromatin condensation and also cause anaphase I bridges

61 nstrated that overexpression of Plk3 induces chromatin condensation and apoptosis.

62 is in a cell-free system revealed a block in chromatin condensation and apoptotic body formation when

63 slocates into the nucleus, where it promotes chromatin condensation and apoptotic cell death.

64 rough the determination of apoptosis-related chromatin condensation and biochemically through the mea

65 istics of PCD of mammalian cells: NO induced chromatin condensation and caspase-like activity in Arab

66 totic entry, presumably as a prerequisite to chromatin condensation and cell division.

67 anges characteristic of apoptosis, including chromatin condensation and cell shrinkage.

68 tin F confers protection against TPT-induced chromatin condensation and cleavage of PARP, cell detach

69 suggests that chondrocytes likely experience chromatin condensation and decondensation during a daily

70 UBF trimethylation leads to nucleolar chromatin condensation and decreased rDNA transcriptiona

71 plicity of infection (MOI) of 25:1 developed chromatin condensation and DNA fragmentation at 4 h and

72 r77 induction, which is involved in inducing chromatin condensation and DNA fragmentation.

73 adative events of apoptosis, such as nuclear chromatin condensation and DNA fragmentation.

74 the induction of apoptotic cell morphology, chromatin condensation and DNA fragmentation.

75 of MST1, which functions to induce apoptotic chromatin condensation and DNA fragmentation.

76 modifications could affect gene regulation, chromatin condensation and DNA packaging.

77 nd extend the argument that the processes of chromatin condensation and DNA replication are linked.

78 ll death (PCD) at late stages typically show chromatin condensation and endonucleolytic cleavage prio

79 ythroblasts resulted in severe inhibition of chromatin condensation and enucleation but otherwise had

80 lation of miR-191 is essential for erythroid chromatin condensation and enucleation by allowing up-re

81 Hypermethylation in Th2 cells results in chromatin condensation and exclusion of CREB proteins fr

82 This result was confirmed using assays for chromatin condensation and for DNA fragmentation.

83 However, the normal progression of chromatin condensation and fragmentation accompanying pr

84 nuclei was coupled with the documentation of chromatin condensation and fragmentation by confocal mic

85 s from synaptosomes exposed to Abeta induced chromatin condensation and fragmentation in isolated nuc

86 ADP-ribose) polymerase was cleaved, and (iv) chromatin condensation and fragmentation of cellular DNA

87 LZ increased Caspase -3, -8 and -9 activity, chromatin condensation and fragmentation, data indicativ

88 wed characteristic apoptotic changes such as chromatin condensation and fragmentation, membrane blebb

89 age, membrane blebbing, vesicle release, and chromatin condensation and fragmentation.

90 Histone H1 phosphorylation affects chromatin condensation and function, but little is known

91 thin the gene body of SLFN11, inducing local chromatin condensation and gene silencing.

92 tant epigenetic mark that is known to induce chromatin condensation and gene silencing.

93 f the condensin complex that plays a role in chromatin condensation and has also been associated with

94 pase proteolysis of RAD21 precedes apoptotic chromatin condensation and has important functional cons

95 a substantial ( approximately 7 h) delay in chromatin condensation and histone H3 phosphorylation, a

96 eplacement results in an abnormal pattern of chromatin condensation and in reduced fertility.

97 tone-modifying enzymes that together promote chromatin condensation and inactivation.

98 -expression of ced-9 prevented CED-4-induced chromatin condensation and lethality, and caused the rel

99 pression in S. pombe resulted in rapid focal chromatin condensation and lethality.

100 vealed widening of intercellular spaces with chromatin condensation and margination in the upper stra

101 cytoplasmic membrane blebbing (zeiosis), and chromatin condensation and margination, and decrease in

102 the mechanism by which DFF40/CAD influences chromatin condensation and nuclear collapse during apopt

103 The distinct features of chromatin condensation and nuclear DNA fragmentation ind

104 sphorylation of nuclear targets critical for chromatin condensation and nuclear envelope breakdown.

105 activation of caspases and characterized by chromatin condensation and nuclear fragmentation (type I

106 aspase-3-like proteases preceded TAM-induced chromatin condensation and nuclear fragmentation, the ty

107 d to the cytoplasm early in apoptosis before chromatin condensation and nuclear fragmentation.

108 % versus 8.7%, respectively), as detected by chromatin condensation and nuclear fragmentation.

109 was confirmed by: (a) morphology, revealing chromatin condensation and nuclear fragmentation; (b) fl

110 ding cytoplasmic shrinkage and condensation, chromatin condensation and nuclear membrane blebbing.

111 DNA methylation has been implicated in chromatin condensation and nuclear organization, especia

112 ethylation, and show a strong correlation of chromatin condensation and nuclear stiffness with the ex

113 reorganization within the nucleus, including chromatin condensation and nucleoprotein immobilization.

114 2 activity is necessary for death-associated chromatin condensation and other manifestations of apopt

115 Following chromatin condensation and perinuclear centrosome aggreg

116 uding apoptotic morphology with mitosis-like chromatin condensation and prelytic genome digestion.

117 es to the nuclei of hepatic cells to control chromatin condensation and proliferation.

118 at has been implicated in inducing apoptotic chromatin condensation and regulating mRNA processing.

119 gical changes suggesting that the process of chromatin condensation and rod cell structural integrity

120 death was apoptotic, as indicated by marked chromatin condensation and the requirement for caspase-3

121 monstrate the presence or absence of nuclear chromatin condensation and thus confirm that ISEL-positi

122 tivator-dependent histone acetylation had on chromatin condensation and to ascertain whether acetylat

123 of core histone N-terminal tails influences chromatin condensation and transcription.

124 of histones by histone deacetylases promotes chromatin condensation and transcriptional repression.

125 A/B and histone deacetylases to induce local chromatin condensation and transcriptional repression.

126 7), which serves as an epigenetic signal for chromatin condensation and transcriptional repression.

127 ranscription factors, resulting in localized chromatin condensation and transcriptional silencing.

128 other chromatin-associated proteins to cause chromatin condensation and transcriptional silencing.

129 ted DNA, resulting in histone deacetylation, chromatin condensation and transcriptional silencing.

130 rget HDAC activity to chromatin resulting in chromatin condensation and transcriptional silencing.

131 Electron microscopic examination showed chromatin condensation and vacuolization in a fraction o

132 d caspase-3 activation and apoptotic nuclear chromatin condensation and was inhibited by the proteaso

133 totic markers such as nuclear fragmentation, chromatin condensation, and accumulation of reactive oxy

134 cking down of acinus substantially decreases chromatin condensation, and depletion of Akt provokes th

135 assessed by plasma membrane bleb formation, chromatin condensation, and DNA fragmentation.

136 fnt, all of which are required for germ cell chromatin condensation, and drastically altered pattern

137 It also lacks caspase-3 activation, chromatin condensation, and endonuclease-mediated DNA de

138 uclear morphology consistent with apoptosis, chromatin condensation, and formation of apoptotic bodie

139 iated with triggering histone deacetylation, chromatin condensation, and gene silencing.

140 Cell morphology, chromatin condensation, and genomic DNA fragmentation me

141 cleosomal interactions during salt-dependent chromatin condensation, and here we used it to examine i

142 ternalization, increased caspase-3 activity, chromatin condensation, and nuclear fragmentation.

143 rks of PCD such as accumulation of H(2)O(2), chromatin condensation, and oligonucleosomal fragmentati

144 nuclear opening formation, histone release, chromatin condensation, and terminal erythroid different

145 a high frequency as shown by TUNEL staining, chromatin condensation, and the appearance of morphologi

146 e key implications for both the mechanism of chromatin condensation, and the regulation of genomic fu

147 lts in rapid deacetylation of histone H4 K8, chromatin condensation, and transcription silencing.

148 morphology in which nuclear changes such as chromatin condensation are absent or reduced; in additio

149 ylation of histone H4 lysines and coincident chromatin condensation are critically involved in the si

150 TGHQ-induced histone H3 phosphorylation and chromatin condensation are inhibited by PD098059, which

151 Apoptotic DNA fragmentation and chromatin condensation are mediated by the caspase-activ

152 th the defect in clonal proliferation and in chromatin condensation are rescued by mutations known to

153 es have implicated histone deacetylation and chromatin condensation as critical mechanisms of transcr

154 duced apoptosis in C6 cells morphologically (chromatin condensation as detected by light microscopy)

155 KLP38B is required for normal chromatin condensation as embryos from KLP38B mutant mot

156 ence for CD94-induced apoptosis includes: 1) chromatin condensation as measured by increased fluoresc

157 with drug resistance, showed characteristic chromatin condensation as well as distinct poly(ADP-ribo

158 of histone H3 is accompanied by increases in chromatin condensation, as observed with the use of 4,6-

159 tive EGL cells includes a failure to exhibit chromatin condensation (assessed with TOPRO), phosphatid

160 ytes in cultures that contained 2.4G2 showed chromatin condensation, binding of Annexin-V, and fas in

161 nucleus, histone displacement, initiation of chromatin condensation, binding of protamines to DNA, or

162 vented DNA fragmentation and reduced nuclear chromatin condensation but also preserved mitochondrial

163 ce cells treated with toxic ONs did not show chromatin condensation, but did exhibit high-extracellul

164 with higher levels of actin organization and chromatin condensation, but only after 24 h in culture.

165 uces apoptotic morphological changes such as chromatin condensation, but the mechanism is not clear.

166 spermatids as they undergo restructuring and chromatin condensation, but unlike SSTK, is not retained

167 an index of apoptosis, relative to measuring chromatin condensation by acridine orange analysis.

168 other hand, knocking down cyclin B1 delayed chromatin condensation by only about one hour.

169 In vitro, H2A.W enhances chromatin condensation by promoting fiber-to-fiber inter

170 modification at lysine 9 directly regulates chromatin condensation by recruiting MENT to chromatin i

171 equired cell-autonomously for proper meiotic chromatin condensation, cell cycle progression and sperm

172 n both cell lines, and there was evidence of chromatin condensation, cell shrinkage and apoptotic bod

173 ion in bcl-2 protein expression, followed by chromatin condensation, chromosomal DNA fragmentation, a

174 ical changes on electron microscopy: nuclear chromatin condensation, compaction of cytoplasmic organe

175 r-chromatid cohesion (Cohesin: Smc1 & Smc3), chromatin condensation (Condensin: Smc2 & Smc4), and DNA

176 n of histone H1 is thought to have a role in chromatin condensation/decondensation, and we asked whet

177 dCap-D3 mutants also exhibit chromatin condensation defects, and mutant alleles of dC

178 t Jhdm2a-deficient mice exhibit post-meiotic chromatin condensation defects, and that JHDM2A directly

179 g presence of intracellular EMCV virions and chromatin condensation; detection of virus-induced chrom

180 aracteristics of apoptosis assessed included chromatin condensation, DNA cleavage, and phosphatidylse

181 in synthesis because cycloheximide inhibited chromatin condensation, DNA fragmentation and activation

182 bility was due to apoptosis characterized by chromatin condensation, DNA fragmentation by agarose gel

183 is, resulting in decreased colony formation, chromatin condensation, DNA fragmentation, and caspase a

184 These cells exhibited chromatin condensation, DNA fragmentation, and laddering

185 Further, 11 and 18 caused chromatin condensation, DNA fragmentation, and loss of m

186 or phenotypes following peptide binding were chromatin condensation, DNA fragmentation, annexin V bin

187 roduces key elements of apoptosis, including chromatin condensation, DNA fragmentation, caspase activ

188 ifferent cell lines as evidenced by enhanced chromatin condensation, DNA fragmentation, or an enhance

189 cal features similar to apoptosis, including chromatin condensation, DNA laddering, cell shrinkage, a

190 se to DNA damage and regulate KAP1-dependent chromatin condensation, DNA repair, and gene expression.

191 Thus, Akt inhibits chromatin condensation during apoptosis by phosphorylati

192 icient to trigger both DNA fragmentation and chromatin condensation during apoptosis.

193 The mechanisms of chromatin condensation during erythropoiesis are unclear

194 e from the opening is a critical step toward chromatin condensation during erythropoiesis in mice.

195 to be important for histone displacement and chromatin condensation during mammalian spermatogenesis.

196 rbf1 mutant larvae have extensive defects in chromatin condensation during mitosis.

197 rly genes upon mitogenic stimulation, and to chromatin condensation during mitotic/meiotic events.

198 For all tested loci, chromatin condensation during photomorphogenesis does no

199 m female passage of human chromosome 21, the chromatin condensation during spermatogenesis and the ex

200 he p53 gain of function could interfere with chromatin condensation either by blocking mitotic shutdo

201 ty is repressed by overlapping mechanisms of chromatin condensation, epigenetic transcriptional silen

202 r classical features of apoptosis, including chromatin condensation, externalization of phosphatidyls

203 promoter, causing histone deacetylation and chromatin condensation, further reducing ER transcript l

204 Apoptotic chromatin condensation has been causally linked to the p

205 cell lines involving CpG island methylation, chromatin condensation, histone deacetylation and MeCP2

206 embrane blebbing, nuclear fragmentation, and chromatin condensation (Hoechst staining), terminal deox

207 perhaps helping to explain how NIMA promotes chromatin condensation in A. nidulans and when expressed

208 d), cytochrome c redistribution, and nuclear chromatin condensation in approximately 12% of the cells

209 aired Chk1 activation, and induced premature chromatin condensation in cells, indicating a defect in

210 , a constitutively nuclear cyclin B1 rescued chromatin condensation in cyclin A2 knockdown cells.

211 oles of various candidate mitotic cyclins in chromatin condensation in HeLa cells.

212 Chromatin condensation in interphase cells is characteri

213 uggesting that LMNB1 is required to maintain chromatin condensation in interphase nuclei.

214 PAN/DFF40 [4,7,8] (CAD) can induce apoptotic chromatin condensation in isolated HeLa cell nuclei in t

215 CE and DCP-1 in vitro, and which can trigger chromatin condensation in isolated nuclei.

216 in step 11 to 13 spermatids and progressive chromatin condensation in later spermatids, but condensa

217 fluorescent protein, mMaroon1, to visualize chromatin condensation in mitosis.

218 hat mediates regulated DNA fragmentation and chromatin condensation in response to apoptotic signals.

219 These data indicate that chromatin condensation in response to DL is regulated th

220 1), and Prm2, all of which are essential for chromatin condensation in spermatids.

221 are insulated from developmentally regulated chromatin condensation in terminally differentiated cell

222 nsation, endoplasmic reticulum dilation, and chromatin condensation in the COLO 16 cells.

223 ammed cell death (PCD) or apoptosis, such as chromatin condensation in the nucleus and blebbing of th

224 ing by de novo DNA methylation and increased chromatin condensation in the SV40 promoter was the majo

225 ignaling in KRAS-mutant NSCLC cells promotes chromatin condensation in vitro and in vivo, thereby res

226 histones have long been known to facilitate chromatin condensation in vitro.

227 l for the induction of DNA fragmentation and chromatin condensation in vivo, but is not required for

228 cid) induced rapid ATP release, resulting in chromatin condensation independent of loading.

229 )-fluoromethylketone; this failed to inhibit chromatin condensation induced by DNase II.

230 regulation of Claspin augments the premature chromatin condensation induced by hydroxyurea, inhibits

231 h and 8 h, respectively, whereas changes in chromatin condensation induced by MOIs of 10:1 and 1:1 r

232 53 protected granule cells from AraC-induced chromatin condensation, internucleosomal cleavage, and a

233 Chromatin condensation is a ubiquitous feature of metazo

234 Abnormal sperm chromatin condensation is common in sterile men, and our

235 Pachytene chromatin condensation is disrupted, and although some m

236 tene when chromosome homologues pair and sex chromatin condensation is initiated.

237 lin A2 knockdowns, arguing that the delay in chromatin condensation is not secondary to a delay in S-

238 nus, a nuclear factor required for apoptotic chromatin condensation, is a direct target of Akt.

239 uires the amino-terminal "RCC1 (regulator of chromatin condensation)-like" GEF domain.

240 ssociated with dramatic nuclear alterations: chromatin condensation, loss of nuclear lamins, and brea

241 el which, along with the rapid nature of the chromatin condensation (<20 s), reveals the basic physic

242 e, hallmark properties of apoptosis, such as chromatin condensation, may be regulated by posttranslat

243 s, short term DL elicits a rapid increase in chromatin condensation, mediated by acto-myosin based ce

244 s was followed by apoptosis, as reflected by chromatin condensation, membrane blebbing, cell detachme

245 During this active phase there was chromatin condensation, mitochondria were swollen but re

246 ence-based cellular assay for MT morphology, chromatin condensation, mitotic arrest, and cellular tox

247 ment of apoptosis was evaluated by examining chromatin condensation, nuclear DNA fragmentation, and c

248 whereas DIDS induced an apoptotic phenotype (chromatin condensation, nuclear fragmentation and cleava

249 ochrome C and apoptosis inducing factor, (4) chromatin condensation, nuclear lamin A and DNA cleavage

250 Later, nuclear chromatin condensation occurred, a characteristic of apo

251 As chromatin condensation occurs even when CAD activity is

252 High expression of CKT2 correlated with chromatin condensation of spermatids in murine testes.

253 ctedly caused hypermethylation and increased chromatin condensation of the transgene in both the G10

254 ytes released by nuclear envelope breakdown, chromatin condensation, or cytokinesis and does not resu

255 , including nuclear crenellation, absence of chromatin condensation, organelle swelling, and accumula

256 olality on nuclear morphology (p < 0.01) and chromatin condensation (p < 0.001) also differed between

257 Although DEVD inhibited the chromatin condensation, PARP cleavage, release of apopto

258 they showed altered cytosine methylation and chromatin condensation patterns, and a correlation betwe

259 Premature chromatin condensation (PCC) is a hallmark of mammalian

260 Chromatin condensation plays an important role in the re

261 and 6 days after ovariectomy showed nuclear chromatin condensation principally in plasma cells.

262 cium signaling as a central mediator of this chromatin condensation process.

263 over an unexpected link between pRB/RBF1 and chromatin condensation, providing a mechanism by which t

264 mechanistic functions of linker histones in chromatin condensation, purified histone H5 has been ass

265 ght while some transcripts for Regulators of Chromatin Condensation (RCC1) declined in the dark.

266 region by lac operator repeats led to local chromatin condensation, recruitment of nuclear receptor

267 ent kinase inhibitor induced cell shrinkage, chromatin condensation, reorganization of actin microfil

268 on inhibits perinatal nucleation of regional chromatin condensation resulting in continued elevated t

269 Evaluation of DNA fragmentation and chromatin condensation revealed that CART 55-102 reduced

270 e chromatin, and the latter revealed various chromatin condensation states in live cells.

271 The resulting change in chromatin condensation stiffened the MSC nucleus, making

272 However, there was a reduction in chromatin condensation, suggesting that changes in the n

273 Mammalian erythropoiesis involves chromatin condensation that is initiated in the early st

274 of loading events led to a greater degree of chromatin condensation that persisted for longer periods

275 euronal death was characterized by extensive chromatin condensation that was reminiscent of, but not

276 by BRG-1 involved nucleosome recruitment and chromatin condensation, thereby abolishing Sp1 binding.

277 Here we show that MST1 induces apoptotic chromatin condensation through its phosphorylation of hi

278 plication of DL elicited a rapid increase in chromatin condensation through purinergic signaling medi

279 tosolic vacuolization and multifocal nuclear chromatin condensation, thus distinguishing this form of

280 modifications of nucleosomal histones alter chromatin condensation to regulate transcriptional activ

281 termini are required to achieve higher order chromatin condensation, two of which apparently involve

282 lear ultrastructure remained normal, without chromatin condensation, until the nuclear envelope fragm

283 il domain performs multiple functions during chromatin condensation via distinct molecular interactio

284 sistent with this possibility, we found that chromatin condensation was tightly associated temporally

285 nce no evidence of tubulin reorganization or chromatin condensation was visible.

286 ex interactions of the H3 tail domain during chromatin condensation, we have developed a new site-dir

287 Endoplasmic reticulum dilation and chromatin condensation were also observed in the rho(0)

288 later when full-blown DNA fragmentation and chromatin condensation were first observed (6 h).

289 anisms underlying the acetylation effects on chromatin condensation were investigated by analyzing th

290 during chromatin assembly and salt-dependent chromatin condensation were investigated using defined c

291 rtain whether acetylation-induced changes in chromatin condensation were related to changes in RNA po

292 DFF40 also triggered chromatin condensation when incubated with nuclei.

293 2 expression) had little effect on premature chromatin condensation, when combined with ATR-kd expres

294 measured by propidium iodide (PI) uptake and chromatin condensation, whereas they remained relatively

295 propose that lola is necessary for complete chromatin condensation which occurs during programmed ce

296 Our findings suggest that this unique chromatin condensation, which can affect interpretations

297 es in chromatin modifications that result in chromatin condensation, which is a likely contributing f

298 yperosmotic conditions (>400 mOsm/kg) induce chromatin condensation, while hypoosmotic conditions (10

299 ration of 30 kb DNA fragments within 3 h and chromatin condensation within 6 h, when nuclei were incu

300 caspase-3 activation, DNA fragmentation, and chromatin condensation within 72 hours of infection.