ライフサイエンスコーパス: chromatin remodeling

1 mmune cells through modulation of epigenetic chromatin remodeling.

2 involved in RNA processing, DNA repair, and chromatin remodeling.

3 s involved in transcriptional regulation and chromatin remodeling.

4 low adaptation to new conditions by delaying chromatin remodeling.

5 , in regulating pathologic transcription and chromatin remodeling.

6 ranscription, recombination, DNA repair, and chromatin remodeling.

7 ects on DNA binding affinity and large-scale chromatin remodeling.

8 omal DNA on one side of the dyad axis during chromatin remodeling.

9 gnaling pathways, epigenetic modulation, and chromatin remodeling.

10 ociated proteins, including many involved in chromatin remodeling.

11 ct with CSB and greatly enhance CSB-mediated chromatin remodeling.

12 contribute to transcriptional regulation and chromatin remodeling.

13 nvolved in antifibrotic activity in skin and chromatin remodeling.

14 through promoting glycolysis and subsequent chromatin remodeling.

15 ion of neurodevelopmental diseases linked to chromatin-remodeling.

16 cells, Cbx3/HP1gamma insufficiency leads to chromatin remodeling accompanied by enhanced Prf1, Gzmb

17 These factors are primarily involved in chromatin remodeling, actin assembly, mRNA 3'-end proces

18 pairs in length and enhances the ATPase and chromatin remodeling activities of CHD2.

19 ggested mechanisms by which NKX2-1 acts with chromatin remodeling activities to regulate gene express

20 These results reveal the chromatin remodeling activity of shelterin and demonstra

21 rough recruitment of Brg1-associated SWI-SNF chromatin remodeling activity.

22 many sites in nucleosomes without the aid of chromatin-remodeling agents and without irreversibly dis

23 is improved by small molecules that promote chromatin remodeling and activate the TGFbeta, Shh and W

24 pothesize that NUPR1 plays an active role in chromatin remodeling and carcinogenesis, together with P

25 t featuring dysregulation of RNA processing, chromatin remodeling and cell-cycle genes enriched for p

26 tional factors participate in the process of chromatin remodeling and facilitate the binding of SMAD3

27 e fbp1 promoter, this transcription promotes chromatin remodeling and fbp1 mRNA expression.

28 We also found that impaired chromatin remodeling and fbp1(+) mRNA transcription in p

29 nucleosomal response, and acts as a brake on chromatin remodeling and gene regulation.

30 ers SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling and global methylation patterns tha

31 n NUPR1 is a multifunctional IDP involved in chromatin remodeling and in the development and progress

32 Hence, glucose fluctuations contribute to chromatin remodeling and may explain persistent vascular

33 as induced in MCF7 cells by agents affecting chromatin remodeling and methylation.

34 ion and localization are tightly linked with chromatin remodeling and might be crucial for transition

35 nized group of Mendelian disorders involving chromatin remodeling and modification.

36 ome rearrangements through sequence-targeted chromatin remodeling and provide insight into targeted a

37 transcription elongation factors to mediate chromatin remodeling and regulate tumorigenic gene expre

38 eraction with CHD9 and BRM, thereby blocking chromatin remodeling and robust GR binding at GR-binding

39 regulated circuit that integrates microRNA, chromatin remodeling and the reprogramming transcription

40 regulatory elements of these genes result in chromatin remodeling and transcription activation.

41 ed by adenosine triphosphate (ATP)-dependent chromatin remodeling and transcription.

42 netic readers, which perform a vital role in chromatin remodeling and transcriptional regulation.

43 Notably, Blimp-1 recruited chromatin-remodeling and histone-modifying complexes to

44 Here we assess histone modification, chromatin remodeling, and DNA methylation processes that

45 enetic factors such as histone modification, chromatin remodeling, and DNA methylation.

46 dysfunction, mitochondrial oxidative stress, chromatin remodeling, and genomic instability.

47 DNA methylation-mediated gene activation and chromatin remodeling, and provides a general framework t

48 racellular processes such as the cell cycle, chromatin remodeling, and transcriptional regulation in

49 NAP1L1 is a nuclear chaperone involved in chromatin remodeling, and we demonstrated the NAP1L1-dep

50 Chromatin remodeling appears to be the basis for the dyn

51 ell lineages, loss of proliferative control, chromatin remodeling as well as extensive morphological

52 Together, these data strongly implicate the chromatin remodeling associated with SWI/SNF complex mem

53 ium and kinase signaling cooperate to induce chromatin remodeling at approximately 2100 chromatin reg

54 shing a connection between transcription and chromatin remodeling at CpG islands by histone demethyla

55 that are regulated by SOX10 and MITF and for chromatin remodeling at distal and proximal regulatory s

56 reatment with PMA or ionomycin alone induces chromatin remodeling at far fewer regions ( approximatel

57 9 and BRM were required for GR occupancy and chromatin remodeling at GR-binding regions associated wi

58 eer" factor, suggested a model whereby FoxO1 chromatin remodeling at regulatory targets facilitates b

59 x2 and Brg1 occupancy occurs coincident with chromatin remodeling at some of these loci.

60 we demonstrate that fibroblasts derived from chromatin remodeling ATPase LSH (HELLS)-null mouse embry

61 The Arabidopsis SWI/SNF chromatin-remodeling ATPase BRAHMA (BRM) modulates respo

62 a physical and genetic interactions with the chromatin-remodeling ATPase BRAHMA.

63 Chromatin remodeling ATPases and their associated comple

64 MONOPTEROS block recruitment of the SWI/SNF chromatin remodeling ATPases in addition to recruiting a

65 in understanding the roles and activities of chromatin remodeling ATPases in plants.

66 l ChIP assays demonstrated co-occupancy with chromatin remodeling BRG1/Brahma-associated factors.

67 While chromatin remodeling by CSB and NAP1-like proteins is cr

68 echanism by which NAP1-like proteins enhance chromatin remodeling by CSB remains unknown.

69 chromatin structure, which is susceptible to chromatin remodeling by ISWI.

70 d transcriptional activator binding required chromatin remodeling by NuRD-associated Mi2beta/CHD4, bu

71 s the deacetylation process, which decreases chromatin remodeling by RSC.

72 nd gene networks, our study demonstrates how chromatin remodeling can dictate gene expression distrib

73 ctly impede Cas9 binding and cleavage, while chromatin remodeling can restore Cas9 access.

74 Target genes implicated in transcription, chromatin remodeling, cellular processes, and hormone me

75 ns were predominantly in genes that regulate chromatin remodeling, chromosome alignment, and stabilit

76 l groups, which include activated signaling, chromatin remodeling, cohesin complex, methylation, NPM1

77 Here we show that the Ino80 chromatin remodeling complex (Ino80C) directly prevents

78 Here we show that Mot1, Ino80 chromatin remodeling complex (Ino80C), and NC2 co-locali

79 Subunits of the SWI/SNF chromatin remodeling complex are frequently mutated in h

80 Here, we identify the yeast ISW1 chromatin remodeling complex as an unanticipated mRNP nu

81 indicate that the growth-regulating SWI/SNF chromatin remodeling complex associated with ANGUSTIFOLI

82 with components of B-WICH, an ATP-dependent chromatin remodeling complex associated with rDNA transc

83 It recruited the SWI/SNF chromatin remodeling complex ATPase BRG1 to promote nucl

84 associated factors-containing complex (PBAF) chromatin remodeling complex component BRG1-associated f

85 ith the chromatin remodeler BRG1 and the BAF chromatin remodeling complex in brown adipocytes.

86 e variant H2A.Z into nucleosomes by the SWR1 chromatin remodeling complex is a critical step in eukar

87 The INO80 chromatin remodeling complex is an evolutionarily conser

88 Here we show that the INO80 chromatin remodeling complex is required for oncogenic t

89 c studies in fruit flies have implicated the chromatin remodeling complex nucleosome remodeling facto

90 cleosome remodeling and deacetylation (NuRD) chromatin remodeling complex opposes this transcriptiona

91 FR-regulated microRNA pathway in the SWI/SNF chromatin remodeling complex suggests that EGFR-mediated

92 y sensor AMPKs, mitochondrial biogenesis and chromatin remodeling complex SWI/SNF activation, which m

93 ew set of genes encoding subunits of the BAF chromatin remodeling complex that exhibited Ras-mediated

94 which EBF2 cooperates with a tissue-specific chromatin remodeling complex to activate brown fat ident

95 acts with and recruits a tissue-specific BAF chromatin remodeling complex to brown fat gene enhancers

96 l enhancers and recruiting the SWI/SNF (BAF) chromatin remodeling complex to establish accessible chr

97 proposing that distinct associations of the chromatin remodeling complex with specific GRFs tightly

98 levant transcription factors and the SWI/SNF chromatin remodeling complex, and surprisingly, associat

99 Haploinsufficiency of ARID1B, a component of chromatin remodeling complex, causes intellectual disabi

100 ARID1A, a subunit of the SWI/SNF chromatin remodeling complex, is frequently mutated in c

101 Here, we show that INO80, a chromatin remodeling complex, is required for SE-mediate

102 tial ATPase subunit of the mammalian SWI/SNF chromatin remodeling complex, uses the energy from ATP h

103 Smarca4 -encoded BRG1 subunit of the SWI/SNF chromatin remodeling complex, we employed in vitro model

104 ow that the BRG1/BRM-associated factor (BAF) chromatin remodeling complex, which is mutated in over 2

105 asymmetric nucleosomes are bound by the RSC chromatin remodeling complex, which is required for main

106 the nucleus and is a component of the INO80 chromatin remodeling complex, which is responsible for t

107 DPF3 (BAF45c) is a member of the BAF chromatin remodeling complex.

108 n remodelers, to form a variant of the ISW1a chromatin remodeling complex.

109 on with and likely regulation of the SWI/SNF chromatin remodeling complex.

110 velopmental regulation of the AN3-associated chromatin remodeling complex.

111 through a direct interaction with the INO80 chromatin remodeling complex.

112 the switch-sucrose non-fermentable (SWI-SNF) chromatin remodeling complex.

113 transcription network by targeting the INO80 chromatin remodeling complex.

114 ding BRG1, the ATPase subunit of the SWI/SNF chromatin remodeling complex; and appropriate genomic co

115 ate that H3K4me1 augments association of the chromatin-remodeling complex BAF to enhancers in vivo an

116 7ac, and H4K5ac as well as by recruitment of chromatin-remodeling complex containing BRG-1.

117 demonstrated that LEDGF/p75 complexes with a chromatin-remodeling complex facilitates chromatin trans

118 ng defective/sucrose nonfermenting (SWI/SNF) chromatin-remodeling complex in regulating the behaviora

119 1, a subunit of the evolutionarily conserved chromatin-remodeling complex SWI/SNF.

120 mportant lineage-specific role for a SWI/SNF chromatin-remodeling complex that collaborates with core

121 HJURP selectively recruits the condensin II chromatin-remodeling complex to facilitate CENP-A deposi

122 of BRG1-associated factors (BAF), an SWI/SNF chromatin-remodeling complex with known repressive funct

123 re known to alter the composition of the BAF chromatin-remodeling complex, causing ejection and degra

124 BAF47), a core subunit of the SWI/SNF (BAF) chromatin-remodeling complex, is inactivated in nearly a

125 RCA4 is the catalytic subunit of the SWI/SNF chromatin-remodeling complex, which alters the interacti

126 eterminant is Swi1, a subunit of the SWI/SNF chromatin-remodeling complex.

127 subunit of the switch/sucrose nonfermentable chromatin-remodeling complex.

128 modeling factor (NURF), a member of the ISWI chromatin-remodeling complex.

129 that is a putative member of the BAF swi/snf chromatin-remodeling complex.

130 ATP-dependent chromatin remodeling complexes alter chromatin structure

131 These findings demonstrate interplay between chromatin remodeling complexes and histone modifications

132 ATP-dependent chromatin remodeling complexes are essential for transcr

133 d that genes that encode subunits of SWI/SNF chromatin remodeling complexes are frequently mutated ac

134 Accordingly, ATP-dependent chromatin remodeling complexes are important regulators

135 hat functions in part through recruitment of chromatin remodeling complexes containing methyl-CpG bin

136 Among them, the ATP-dependent chromatin remodeling complexes control the chromatin arc

137 However, whether chromatin remodeling complexes have the ability to regul

138 catalytic subunits of distinct ATP-dependent chromatin remodeling complexes implicated in transcripti

139 important roles for BAZ1B and its associated chromatin remodeling complexes in NAc in the regulation

140 nes, regulatory interactions of histones and chromatin remodeling complexes in response to dynamic an

141 gulatory switch for other PTMs, and connects chromatin remodeling complexes into gene transcription a

142 ed to switch/sucrose nonfermenting (SWI/SNF) chromatin remodeling complexes is presented.

143 However, how chromatin remodeling complexes regulate DNA damage check

144 et is associated with its failure to recruit chromatin remodeling complexes to the Ifng gene promoter

145 0s) that tether SWITCH/SUCROSE NONFERMENTING chromatin remodeling complexes to transcription factors

146 gh interactions with RNA-binding proteins in chromatin remodeling complexes, and modulate dynamic and

147 incorporated into promoters by SWR-C-related chromatin remodeling complexes, but whether it is also a

148 We show that BAZ1B, a component of chromatin remodeling complexes, in the nucleus accumbens

149 , an accessory subunit of the ISWI family of chromatin remodeling complexes, is upregulated in the nu

150 a growing list of nuclear proteins including chromatin remodeling complexes, transcription factors an

151 epressors that link transcription factors to chromatin remodeling complexes.

152 ne modifications, microRNA interactions, and chromatin remodeling complexes.

153 SMARCA2 are important components of SWI/SNF chromatin remodeling complexes.

154 on of the surrounding chromatin structure by chromatin remodeling complexes.

155 rum of human disorders caused by ablation of chromatin remodeling complexes.

156 sine signaling kinases and components of the chromatin remodeling complexes.

157 ranscription factors that recruit activating chromatin remodeling complexes.

158 onfigurations such as histone chaperones and chromatin remodeling complexes.

159 Chromatin-remodeling complexes and post-translational hi

160 Genes encoding subunits of SWI/SNF (BAF) chromatin-remodeling complexes are collectively mutated

161 BRD9 and BRD7 are part of the human SWI/SNF chromatin-remodeling complexes BAF and PBAF.

162 The Swi/Snf (PBAF and BAF) chromatin-remodeling complexes contribute to DNA damage-

163 ATP-dependent chromatin-remodeling complexes utilize the energy from A

164 nd current state of the dysregulation of the chromatin remodeling components as the driving mechanism

165 evidence have suggested that alterations in chromatin remodeling components contribute to cancer ini

166 ways, the AP-1 transcription factor network, chromatin remodeling, cytokine signaling pathways, cell

167 ed by MyoD, and a strong correlation between chromatin-remodeling deficiencies and incomplete gene ex

168 this spectrum overlaps with that of several chromatin-remodeling developmental disorders.

169 Large-scale chromatin remodeling during mitosis is catalyzed by a he

170 Chromatin remodeling during the GG-NER process is theref

171 Here the authors show that the chromatin remodeling enzyme ATRX is a regulator of TIS a

172 Here, we demonstrate that the chromatin remodeling enzyme ATRX is required for therapy

173 nd Brg1, the ATPase of the mammalian SWI/SNF chromatin remodeling enzyme required for PPARgamma2 acti

174 The chromatin-remodeling enzyme CHD4 maintains vascular inte

175 Human ALC1 is an oncogene-encoded chromatin-remodeling enzyme required for DNA repair that

176 ly, our data identify that the ATP-dependent chromatin-remodeling enzyme Snf2 plays a fundamental rol

177 n Helicase DNA-binding protein 4 (CHD4) is a chromatin-remodeling enzyme that has been reported to re

178 Chromatin remodeling enzymes act to dynamically regulate

179 rvation that the INO80C and SWR1C/SWR1/SWR-C chromatin remodeling enzymes catalyze replacement of nuc

180 which supports the activity of ATP-dependent chromatin remodeling enzymes during hormone-dependent tr

181 Adenosine 5'-triphosphate (ATP)-dependent chromatin remodeling enzymes play essential biological r

182 We further find that chromatin remodeling enzymes stimulate Cas9 activity on

183 m, exploiting gene-specific requirements for chromatin remodeling enzymes to selectively influence DN

184 se-DNA-binding (CHD) family of ATP-dependent chromatin remodeling enzymes, comprising CHD6, CHD7, CHD

185 nes, regulators of cell differentiation, and chromatin remodeling enzymes.

186 gene-specific requirements for ATP-dependent chromatin remodeling enzymes.

187 d population has widespread misexpression of chromatin-remodeling enzymes and myeloid differentiation

188 riptional regulation is modulated in part by chromatin-remodeling enzymes that control gene accessibi

189 a catalytic subunit of the mammalian SWI/SNF chromatin-remodeling enzymes, is required for both myobl

190 (switching/sucrose nonfermenting)-dependent chromatin remodeling establishes coordinated gene expres

191 SBF binding at URS2-L sites depends on prior chromatin remodeling events at the upstream URS1 region.

192 ith core histones during repair of DSBs, and chromatin remodeling events such as histone H2A phosphor

193 histone posttranslational modifications and chromatin-remodeling events in the etiology of MDD, it h

194 nges in the histone modification profile and chromatin-remodeling events leading to Sp7 gene expressi

195 Here, we show that the chromatin remodeling factor Brg1 is required for enhance

196 e zipper transcription factor 1 (BACH1), the chromatin remodeling factor chromodomain helicase DNA-bi

197 Chromatin remodeling factor LSH is critical for normal d

198 Chromatin remodeling factor metastatic tumor protein 1 (

199 egulator(s) and relationship with the master chromatin remodeling factor MTA1, continues to be poorly

200 ediated miR-7 suppresses the coupling of the chromatin remodeling factor SMARCD1 with p53, resulting

201 ndings illustrate how cancer cells utilize a chromatin remodeling factor to engage a core survival pa

202 Mutations in genes encoding chromatin-remodeling factor Brg1/SmarcA4 and its associa

203 Moreover, the chromatin-remodeling factor Hmga2 in the skin plays a cr

204 -specific chaperone, DAXX, together with the chromatin-remodeling factor, ATRX, regulates H3.3 deposi

205 nd tyrosine kinases and their interplay with chromatin remodeling factors in cancer cells.

206 helicase DNA-binding family of ATP-dependent chromatin remodeling factors play essential roles during

207 ally, NAP1 histone chaperones, ATP-dependent chromatin remodeling factors, and some histone-modifying

208 matin fibers, and how they are influenced by chromatin remodeling factors.

209 Transcription factors and chromatin-remodeling factors have been implicated in reg

210 ration events, which inappropriately recruit chromatin-remodeling factors to elicit the aberrant tran

211 The disrupted genes primarily clustered in chromatin remodeling functional nodes and in the PI3K pa

212 The chromatin remodeling gene CHD8 represents a central node

213 The chromatin remodeling gene, AT-rich interactive domain 1A

214 De novo truncating mutations in ARID1B, a chromatin-remodeling gene, cause Coffin-Siris syndrome,

215 ARID1A, an SWI/SNF chromatin-remodeling gene, is commonly mutated in cancer

216 indings demonstrate a role for Tet genes and chromatin remodeling genes in the formation of cerebella

217 el recurrent CNVs including deletions at two chromatin-remodeling genes RERE and NPM2 This method wil

218 Gene mutations in the chromatin remodeling groups were relatively more frequen

219 l differentiation and myelination depends on chromatin remodeling, histone acetylation, and methylati

220 ws that Reln gene expression is regulated by chromatin remodeling, identifies CHD7 as a previously un

221 anscription itself is an active component of chromatin remodeling in 1-cell embryos.

222 the specific molecular mechanisms governing chromatin remodeling in CLL are unknown.

223 stabilizes chromatin as evidenced by faster chromatin remodeling in its absence.

224 Chromatin remodeling in late genes is characterized by a

225 es renal inflammation by several mechanisms: chromatin remodeling in promoter regions of specific gen

226 ells showed impaired nucleosome eviction and chromatin remodeling in response to anthracycline treatm

227 moresistance and highlight the importance of chromatin remodeling in response to cytotoxic chemothera

228 reveal an essential role of INO80-dependent chromatin remodeling in SE function and suggest a novel

229 on the mechanisms that control higher-order chromatin remodeling in the context of epidermal keratin

230 ter, suggesting that FHL2 may be involved in chromatin remodeling in the control of TGF-beta1 gene tr

231 First, Tup11/12 inhibit chromatin remodeling in the fbp1(+) promoter region wher

232 lated to glutamatergic neurotransmission and chromatin remodeling in the human striatum.

233 etic landscapes suggests a critical role for chromatin remodeling in these processes.

234 The facilitated chromatin remodeling in turn correlated with more effici

235 process of lung carcinogenesis is linked to chromatin remodeling, inflammation, and tumor microenvir

236 pathways (protein synthesis and degradation, chromatin remodeling, intracellular signaling), which ar

237 Because chromatin remodeling is a necessary step in transcriptio

238 nd microRNAs by cytosine DNA methylation and chromatin remodeling is critical for the initiation and

239 Chromatin remodeling is essential for establishing diver

240 Our results suggest that REST-mediated chromatin remodeling is required in neural progenitors f

241 programs, such as histone modifications and chromatin remodeling, is not well understood.

242 Adenosine triphosphate-dependent chromatin remodeling machines play a central role in gen

243 to discuss the current state of knowledge of chromatin remodeling mechanisms and highlight exciting o

244 protein-DNA complexes independent of classic chromatin remodeling mechanisms.

245 of some vertebrates, possibly as a result of chromatin-remodeling mechanisms that enforce terminal di

246 While chromatin remodeling mediated by post-translational modi

247 se transcription, while the Swi/Snf and SAGA chromatin remodeling/modification factors are dispensabl

248 e Sig-1Rs bind NE protein emerin and recruit chromatin-remodeling molecules, including lamin A/C, bar

249 Chromatin remodeling motors play essential roles in all

250 d within 48 h after MS1 induction; out of 14 chromatin-remodeling mutants studied, expression of clus

251 cording to endogenous yeast DNA sequence and chromatin-remodeling network, as judged by a yeast-like

252 stinct H1 subtypes may mediate the extensive chromatin remodeling occurring during epigenetic reprogr

253 Conversely, dramatic chromatin remodeling occurs in naive CD4(+) T cells duri

254 Chromatin remodeling of early genes during the inflammat

255 -specific enhancer that was found to mediate chromatin remodeling of the EDC in an AP-1 dependent man

256 idate risk genes with predicted functions in chromatin remodeling or neurodevelopment, including ACTL

257 Using a different chromatin remodeling paradigm that is JIL-1 dependent, w

258 can serve as a key regulatory mechanism for chromatin remodeling possibly leading to anxiolysis.

259 ied include genes involved in transcription, chromatin remodeling, post-translational modifications,

260 In a unique global chromatin remodeling process during mammalian spermiogen

261 Chromatin remodeling processes mediated by the SWI/SNF c

262 Consequently, diRNA-mediated chromatin remodeling promoted DSB repair by enhancing th

263 we examined mouse embryos deficient for the chromatin remodeling protein ATRX to determine the cellu

264 We show that the chromatin remodeling protein HMGA1 functions as a downst

265 ntially expressed epigenetic regulators, the chromatin remodeling protein HMGA2 is significantly upre

266 s growth inhibitory in vivo In addition, the chromatin remodeling protein INO80C was identified as a

267 Mutations of the chromatin remodeling protein Lsh/HELLS can cause the hum

268 nriched expression was CHD2, which encodes a chromatin remodeling protein mutated to cause human epil

269 Loss of the chromatin-remodeling protein ATRX associates with ALT in

270 distinct subsets of genes regulated by these chromatin remodeling proteins after chronic exposure to

271 ATP-dependent chromatin remodeling proteins are being implicated incre

272 Chromatin remodeling proteins are frequently dysregulate

273 stablish an in vivo function of CHD Type III chromatin remodeling proteins in this process, and revea

274 Proper cooperation of C/EBP and chromatin remodeling proteins is essential for the termi

275 n of complexes of C/EBP family proteins with chromatin remodeling proteins.

276 identified CHD6 from a screen among several chromatin-remodeling proteins as a putative epigenetic m

277 d Dmp1 promoters and prevented KAT6A-induced chromatin remodeling, repressing gene transcription.

278 that stimulation of OX40 triggered a robust chromatin remodeling response and produced a "closed" ch

279 Chromatin remodeling seems to regulate the patterns of p

280 CHD8 and discovered distinct differences in chromatin remodeling specificities and activities among

281 Here, we will review the chromatin remodeling steps that occur immediately after

282 (MBD3), to show how the core deacetylase and chromatin-remodeling sub-modules associate in vivo.

283 Loss-of-function mutations in SWI/SNF chromatin-remodeling subunit genes are observed in many

284 ated haploinsufficiency of ARID1B, a SWI/SNF chromatin-remodeling subunit, in short stature, autism s

285 res BRG1 (SMARCA4), the ATPase engine of the chromatin remodeling SWI/SNF complex (also called BAF).

286 ew mechanisms underlying the function of the chromatin remodeling SWI/SNF complex in controlling gene

287 The multi-subunit chromatin-remodeling SWI/SNF complex determines gene exp

288 on in a functional network that converged on chromatin remodeling, synapse transmission and cell cycl

289 te that interplay of p300-HDAC2-Sin3A in the chromatin remodeling system is involved in HIF-1alpha de

290 uncovers the different steps of CSB-mediated chromatin remodeling that can be regulated by NAP1L1.

291 This event initiated extensive chromatin remodeling that reprogrammed immune response g

292 Chromatin remodeling through histone acetyltransferase (

293 xpected direct role for diRNAs in regulating chromatin remodeling to facilitate DSB repair, revealing

294 programs in mouse germ cells are reshaped by chromatin remodeling to orchestrate the onset of develop

295 ys in vitro, and the native region undergoes chromatin remodeling, transcribes a bidirectional enhanc

296 assumed to be a side effect of the extensive chromatin remodeling underlying the epigenetic reprogram

297 n that can be relieved through ATP-dependent chromatin remodeling via complexes such as the switch-su

298 umoylation activity, impairing KAP1-mediated chromatin remodeling, which is important for efficiently

299 ATP-dependent chromatin remodeling, which repositions and restructures

300 lve rapid gene expression changes and global chromatin remodeling, yet the underlying regulatory path