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1 ing complex termed ToRC (Toutatis-containing chromatin remodeling complex).
2 Cys) in INO80D, a subunit of the human INO80 chromatin remodeling complex.
3 litate the removal of nucleosomes by the RSC chromatin remodeling complex.
4 nits of the yeast SWI/SNF and vertebrate BAF chromatin remodeling complex.
5 e genesis of cancers driven by mutation of a chromatin remodeling complex.
6 ic loss of SMARCB1, a subunit of the SWI/SNF chromatin remodeling complex.
7 nucleosome remodeling and deacetylase (NuRD) chromatin remodeling complex.
8 ted protein Arp8 is a component of the INO80 chromatin remodeling complex.
9 18(Hamlet) subunit (Dmp18) of the SWR1/SRCAP chromatin remodeling complex.
10 and recruiting the Sin3-histone deacetylase chromatin remodeling complex.
11 nd ARP9, each encoding components of the RSC chromatin remodeling complex.
12 the switch-sucrose non-fermentable (SWI-SNF) chromatin remodeling complex.
13 nown to interact with hSNF2H to form an ISWI chromatin remodeling complex.
14 mo-1 protein, which is a subunit of the PBAF chromatin remodeling complex.
15 he epigenome as a core member of the SWI/SNF chromatin remodeling complex.
16 which encodes the BAF180 subunit of the PBAF chromatin remodeling complex.
17 transcription network by targeting the INO80 chromatin remodeling complex.
18 DPF3 (BAF45c) is a member of the BAF chromatin remodeling complex.
19 n remodelers, to form a variant of the ISW1a chromatin remodeling complex.
20 on with and likely regulation of the SWI/SNF chromatin remodeling complex.
21 velopmental regulation of the AN3-associated chromatin remodeling complex.
22 through a direct interaction with the INO80 chromatin remodeling complex.
23 of the Polycomb repressive complex 2 (PRC2) chromatin-remodeling complex.
24 o regulation at multiple levels in the INO80 chromatin-remodeling complex.
25 t on the structure and function of the INO80 chromatin-remodeling complex.
26 nent of the evolutionarily conserved SWI/SNF chromatin-remodeling complex.
27 integrity of a repressor form of the SWI/SNF chromatin-remodeling complex.
28 role in recruitment of an activating SWI/SNF chromatin-remodeling complex.
29 iptional repressor and component of the NuRD chromatin-remodeling complex.
30 modulate protein-protein interactions at the chromatin-remodeling complex.
31 bunit, MCRS1, is shared with the human INO80 chromatin-remodeling complex.
32 eterminant is Swi1, a subunit of the SWI/SNF chromatin-remodeling complex.
33 subunit of the switch/sucrose nonfermentable chromatin-remodeling complex.
34 modeling factor (NURF), a member of the ISWI chromatin-remodeling complex.
35 that is a putative member of the BAF swi/snf chromatin-remodeling complex.
36 turnover that are typically bound by the RSC chromatin-remodeling complex.
37 ranscription factors that recruit activating chromatin remodeling complexes.
38 histone posttranslational modifications, and chromatin remodeling complexes.
39 ogenic regulatory factors, microRNAs and BAF chromatin remodeling complexes.
40 atin propagation by transcription-associated chromatin remodeling complexes.
41 rum of human disorders caused by ablation of chromatin remodeling complexes.
42 ependent on Nap1l1-coupled SWI/SNF and INO80 chromatin remodeling complexes.
43 sine signaling kinases and components of the chromatin remodeling complexes.
44 which is a subunit of PBAF-specific Swi/Snf chromatin remodeling complexes.
45 onfigurations such as histone chaperones and chromatin remodeling complexes.
46 ion depends upon the antagonistic actions of chromatin remodeling complexes.
47 moved, however, by a class of ATP dependent chromatin remodeling complexes.
48 , RBBP1) as part of SIN3.histone deacetylase chromatin remodeling complexes.
49 c subset of transcriptional coactivators and chromatin remodeling complexes.
50 epressors that link transcription factors to chromatin remodeling complexes.
51 ne modifications, microRNA interactions, and chromatin remodeling complexes.
52 SMARCA2 are important components of SWI/SNF chromatin remodeling complexes.
53 on of the surrounding chromatin structure by chromatin remodeling complexes.
54 both directly and by targeting or activating chromatin-remodeling complexes.
55 al posttranslational modifications (PTMs) on chromatin-remodeling complexes.
56 catalytic subunits of the SWI/SNF family of chromatin-remodeling complexes.
62 ucted mammalian genetic models for the INO80 chromatin remodeling complex and investigated the impact
63 le of Mediator in recruitment of the Swi/Snf chromatin remodeling complex and its role, along with co
64 PRMT7 interacts with the BRG1-based hSWI/SNF chromatin remodeling complex and specifically methylates
65 f a "trimeric minimal RSC" (RSCt) of the RSC chromatin remodeling complex and the effect of nucleotid
66 8 genes encode proteins involved in multiple chromatin remodeling complexes and are likely to play im
68 sh a direct connection between ATP-dependent chromatin remodeling complexes and checkpoint regulation
69 and facilitates transcription by recruiting chromatin remodeling complexes and general transcription
70 These findings demonstrate interplay between chromatin remodeling complexes and histone modifications
71 functioning in a critical interface between chromatin remodeling complexes and the genome to direct
72 ar processes and are essential components of chromatin remodeling complexes and transcription factor
73 which functions as a subunit of the SWI/SNF chromatin-remodeling complex and a tumor suppressor in f
74 on is well established, but the link between chromatin-remodeling complexes and DNA repair remains un
75 eral Tat-activated kinases and by recruiting chromatin-remodeling complexes and histone-modifying enz
77 tion in INO80D, a subunit of the human INO80 chromatin remodeling complex, and accelerated arterial a
78 the SWItch/Sucrose NonFermentable (SWI/SNF) chromatin remodeling complex, and analyzed its role in m
79 levant transcription factors and the SWI/SNF chromatin remodeling complex, and surprisingly, associat
80 of the nucleosome remodeling and deacetylase chromatin remodeling complex, and we demonstrate that CH
81 gh interactions with RNA-binding proteins in chromatin remodeling complexes, and modulate dynamic and
82 mutations in CHD4/Mi2b, a member of the NuRD-chromatin-remodeling complex, and TAF1, an element of th
83 cally, mitotic RCS1 associates with the NuRD chromatin-remodeling complex, and this RCS1 complex is l
85 ding BRG1, the ATPase subunit of the SWI/SNF chromatin remodeling complex; and appropriate genomic co
87 genes encoding subunits of the SWI/SNF (BAF) chromatin remodeling complex are mutated in 20% of all h
90 rg/Brahma-associated factors)] ATP-dependent chromatin remodeling complexes are essential for formati
92 d that genes that encode subunits of SWI/SNF chromatin remodeling complexes are frequently mutated ac
93 nucleosome remodeling and deacetylase (NuRD) chromatin remodeling complexes are important regulators
95 in genes encoding subunits of SWI/SNF (BAF) chromatin remodeling complexes are particularly prevalen
96 at switching/sucrose nonfermenting (SWI/SNF) chromatin remodeling complexes are recruited to the Igh
97 one acetyltransferases and the BRM and Brg-1 chromatin remodeling complexes are recruited to viral IE
100 Genes encoding subunits of SWI/SNF (BAF) chromatin-remodeling complexes are collectively mutated
101 ties are regulated by additional subunits of chromatin-remodeling complexes are less well understood.
102 tion of Schwann cells and implicate the NuRD chromatin remodeling complex as a requisite factor in ti
105 t ATPase of the mammalian SWI/SNF (mSWI/SNF) chromatin remodeling complex, as being essential for the
106 fied BRG1, the ATPase subunit of the SWI/SNF chromatin-remodeling complex, as an E2F6 interacting pro
107 indicate that the growth-regulating SWI/SNF chromatin remodeling complex associated with ANGUSTIFOLI
108 with components of B-WICH, an ATP-dependent chromatin remodeling complex associated with rDNA transc
110 uitment of the SWItch/Sucrose NonFermentable chromatin remodeling complex ATPase enzyme SMARCA4 (also
111 ate that H3K4me1 augments association of the chromatin-remodeling complex BAF to enhancers in vivo an
113 re is increasing evidence that ATP-dependent chromatin remodeling complexes based on the alternative
114 brahma (Brm), a subunit of the ATP-dependent chromatin-remodeling complex BRG/brahma-associated facto
115 ts recruitment of a component of the SWI/SNF chromatin remodeling complex, Brg1, and RNA polymerase I
116 incorporated into promoters by SWR-C-related chromatin remodeling complexes, but whether it is also a
117 SWI/SNF-like Brg/Brm-associated factor (BAF) chromatin remodeling complexes by creating distinct poly
118 enesis by demonstrating that disruption of a chromatin remodeling complex can largely, if not complet
119 nase activity and suggest that ATP-dependent chromatin remodeling complexes can regulate nonchromatin
120 Haploinsufficiency of ARID1B, a component of chromatin remodeling complex, causes intellectual disabi
121 1-GCN5L acetylase] and TFTC (GCN5 and TRRAP) chromatin remodeling complexes, causes the neurodegenera
122 re known to alter the composition of the BAF chromatin-remodeling complex, causing ejection and degra
123 r it resulted in increased expression of the chromatin remodeling complex CBP/p300, as well as decrea
125 indings define a direct relationship between chromatin-remodeling complexes, chromatin structure, and
126 ll, Kubik et al. (2015) describe how the RSC chromatin remodeling complex collaborates with two DNA s
127 UNC13D intron 1 recruitment of STAT4 and the chromatin remodeling complex component BRG1, diminishing
128 associated factors-containing complex (PBAF) chromatin remodeling complex component BRG1-associated f
129 ither of the two subunits of a transcription/chromatin remodeling complex consisting of DAXX (death-d
131 pe II NTE strain prevents the recruitment of chromatin remodeling complexes containing Brahma-related
132 (BAF250A) promotes the formation of SWI/SNF chromatin remodeling complexes containing BRG1 or BRM.
133 hat functions in part through recruitment of chromatin remodeling complexes containing methyl-CpG bin
135 60 complex (TIP60.com) is a tumor suppressor chromatin-remodeling complex containing RVB proteins.
141 During development, trithorax group (trxG) chromatin remodeling complexes counteract repression by
142 f these genes requires their modification by chromatin remodeling complexes (CRCs), which are recruit
143 witch (SWI)/Sucrose Nonfermenting (SNF)-type chromatin-remodeling complexes (CRCs) are involved in re
145 00, CBP, PCAF, and GCN5 or the BRM and Brg-1 chromatin remodeling complexes did not diminish IE gene
148 we explore the role of the mammalian SWI/SNF chromatin-remodeling complex during spermatogenesis usin
151 demonstrated that LEDGF/p75 complexes with a chromatin-remodeling complex facilitates chromatin trans
154 leukemia cells require the mammalian SWI/SNF chromatin remodeling complex for their survival and aber
155 plant SWITCH/SUCROSE NONFERMENTING (SWI/SNF) chromatin remodeling complexes formed around the ATPases
156 unifying concepts on how these two opposing chromatin remodeling complexes function selectively at t
159 F, an evolutionarily conserved ATP-dependent chromatin-remodeling complex, has an important role in t
162 ormone receptors mediated the association of chromatin remodeling complexes, histone modification, an
164 itching defective/sucrose nonfermenting-type chromatin remodeling complex, (iii) transcription coacti
165 catalytic subunits of distinct ATP-dependent chromatin remodeling complexes implicated in transcripti
168 icated the role of the SWI/SNF ATP-dependent chromatin remodeling complex in nuclear excision repair
169 Recent findings have implicated the NuRD chromatin remodeling complex in the sophisticated choreo
170 t roles of the SNR1/SNF5 subunit and the Brm chromatin remodeling complex in transcription regulation
171 n appears to be regulated by Brg1-containing chromatin remodeling complexes in a partially SRF-depend
172 important roles for BAZ1B and its associated chromatin remodeling complexes in NAc in the regulation
173 nes, regulatory interactions of histones and chromatin remodeling complexes in response to dynamic an
175 tudies highlight an unsuspected role for BAF chromatin remodeling complexes in the maintenance of HSC
176 ng defective/sucrose nonfermenting (SWI/SNF) chromatin-remodeling complex in regulating the behaviora
179 c and mechanistic basis for the BRG1 and BRM chromatin-remodeling complexes in regulating gene expres
180 ing factors CTCF, RAD21, SMC3, and YY1/INO80 chromatin-remodeling complexes in repressor depletion an
181 ed factor (BAF) complexes (mammalian SWI/SNF chromatin remodeling complexes) in several human intelle
182 a polybromo protein in ATP-dependent SWI/SNF chromatin remodeling complexes, in coronary development.
184 re of chromatin), an abundant, ATP-dependent chromatin-remodeling complex, in the cellular response t
186 the SWItch/Sucrose NonFermentable (SWI/SNF) chromatin remodeling complex, including all three putati
187 the nucleus and bind multiple components of chromatin-remodeling complexes, including Polycomb group
190 up protein (HMGB) that is a component of the chromatin-remodeling complex INO80, which is involved in
194 gulatory switch for other PTMs, and connects chromatin remodeling complexes into gene transcription a
195 e variant H2A.Z into nucleosomes by the SWR1 chromatin remodeling complex is a critical step in eukar
201 macrophages, the mammalian Swi/Snf-like BAF chromatin remodeling complex is recruited to many TLR4 t
203 se observations suggest that prior action of chromatin remodeling complexes is necessary for the acti
208 al helicase of the mammalian SWI/SNF-related chromatin remodeling complex, is required for Schwann ce
210 , an accessory subunit of the ISWI family of chromatin remodeling complexes, is upregulated in the nu
211 BAF47), a core subunit of the SWI/SNF (BAF) chromatin-remodeling complex, is inactivated in nearly a
212 t of the Brg1/Brahma-associated factor (BAF) chromatin-remodeling complex, is required in NCCs to dir
213 hat BRG1, which is the core subunit of eight chromatin-remodeling complexes, is essential not only fo
215 he composition of a SIN3.histone deacetylase chromatin remodeling complex, leads to altered gene expr
217 s suggest that the proteasome and the hINO80 chromatin-remodeling complex may cooperate to regulate t
218 e further propose that guiding ATP-dependent chromatin-remodeling complexes may be a more general fun
219 chromatin regulators, including the neuronal chromatin remodeling complex (nBAF) component SS18L1 (al
220 activation of other individual ATP-dependent chromatin remodeling complexes negligibly affects report
221 c studies in fruit flies have implicated the chromatin remodeling complex nucleosome remodeling facto
222 TA-1, its cofactor FOG-1, and the associated chromatin remodeling complex NuRD in the developmental s
225 ssembly and remodeling factor) ATP-dependent chromatin-remodeling complex, occurring in the nucleus a
226 cleosome remodeling and deacetylation (NuRD) chromatin remodeling complex opposes this transcriptiona
228 nteractions between individual ATP-dependent chromatin remodeling complexes play critical role in bot
231 iochemical and structural studies of the RSC chromatin-remodeling complex prompt a proposal for the r
235 ied that the Osa-containing SWI/SNF (Brahma) chromatin-remodeling complex regulates Drosophila midgut
238 t that the combined action of activators and chromatin remodeling complexes remove the -1 nucleosome
240 H3K14ac have a higher affinity for purified chromatin remodeling complex RSC (Remodels the Structure
245 TP binding proteins that are part of various chromatin-remodeling complexes, such as Ino80, SNF2-rela
246 FR-regulated microRNA pathway in the SWI/SNF chromatin remodeling complex suggests that EGFR-mediated
247 y sensor AMPKs, mitochondrial biogenesis and chromatin remodeling complex SWI/SNF activation, which m
249 e yeast PHO8 gene was found to depend on the chromatin-remodeling complex SWI/SNF, whereas activation
254 ew set of genes encoding subunits of the BAF chromatin remodeling complex that exhibited Ras-mediated
255 the polycomb repressive complex 2 (PRC2), a chromatin remodeling complex that mediates silencing of
256 nting protein 2 homologue (hSNF2H) to form a chromatin remodeling complex that participates in severa
257 The SWI/SNF complex is an ATP-dependent chromatin remodeling complex that plays pivotal roles in
258 several nuclear proteins including SIN3:HDAC chromatin remodeling complexes that are involved in repr
259 ctivity of the SWI/SNF-related ATP-dependent chromatin remodeling complexes that control the accessib
260 mportant lineage-specific role for a SWI/SNF chromatin-remodeling complex that collaborates with core
261 the Foxp transcription factors and the NuRD chromatin-remodeling complex that modulates transcriptio
263 d from yeast to man and many are subunits of chromatin-remodeling complexes that facilitate transcrip
264 he remodels the structure of chromatin (RSC) chromatin remodeling complex, the nuclear motor Kip1, th
266 which EBF2 cooperates with a tissue-specific chromatin remodeling complex to activate brown fat ident
267 acts with and recruits a tissue-specific BAF chromatin remodeling complex to brown fat gene enhancers
268 o signaling pathways that direct the SWI/SNF chromatin remodeling complex to cardiomyogenic loci in m
269 l enhancers and recruiting the SWI/SNF (BAF) chromatin remodeling complex to establish accessible chr
270 Nucleosome Remodeling and Deacetylase (NuRD) chromatin remodeling complex to regulate gene transcript
271 We show that recruitment of the Swi/Snf chromatin remodeling complex to the induced CHA1 promote
272 dentified, but the contributions of specific chromatin remodeling complexes to peripheral nerve myeli
273 f bromodomain proteins, directs multisubunit chromatin remodeling complexes to specific acetyl-nucleo
274 ppear to act as scaffolds to further recruit chromatin remodeling complexes to specific target genes.
275 et is associated with its failure to recruit chromatin remodeling complexes to the Ifng gene promoter
276 d by recruiting both histone deacetylase and chromatin remodeling complexes to their promoters by the
277 0s) that tether SWITCH/SUCROSE NONFERMENTING chromatin remodeling complexes to transcription factors
278 We show that recruitment of the SWI/SNF chromatin-remodeling complex to both RSSs increases coup
279 HJURP selectively recruits the condensin II chromatin-remodeling complex to facilitate CENP-A deposi
280 t locus, we show that Oct1 recruits the NuRD chromatin-remodeling complex to promote a repressed stat
281 recruiting and interacting with the SWI/SNF chromatin-remodeling complex to specifically mediate Neu
282 a growing list of nuclear proteins including chromatin remodeling complexes, transcription factors an
283 , a component of the WICH complex (WSTF-ISWI chromatin-remodeling complex), under basal conditions in
285 tial ATPase subunit of the mammalian SWI/SNF chromatin remodeling complex, uses the energy from ATP h
287 y elements in the histone promoters, the RSC chromatin-remodeling complex, various histone chaperones
289 Smarca4 -encoded BRG1 subunit of the SWI/SNF chromatin remodeling complex, we employed in vitro model
290 ow that the BRG1/BRM-associated factor (BAF) chromatin remodeling complex, which is mutated in over 2
291 asymmetric nucleosomes are bound by the RSC chromatin remodeling complex, which is required for main
292 the nucleus and is a component of the INO80 chromatin remodeling complex, which is responsible for t
293 es and is an essential component of the SWR1 chromatin remodeling complex, which regulates transcript
294 RCA4 is the catalytic subunit of the SWI/SNF chromatin-remodeling complex, which alters the interacti
295 lly, EcR genetically interacts with the NURF chromatin-remodeling complex, which we previously showed
296 ecialized and novel cardiac-enriched SWI/SNF chromatin-remodeling complexes, which are required for h
297 dentified and purified a human ATP-dependent chromatin remodeling complex with similarity to the Sacc
298 proposing that distinct associations of the chromatin remodeling complex with specific GRFs tightly
299 is a unique and defining subunit of the BAF chromatin remodeling complex with the potential to facil
300 of BRG1-associated factors (BAF), an SWI/SNF chromatin-remodeling complex with known repressive funct
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